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Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2012, Volume 122, Issue 8, pp. 2911 - 2915
Journal Article
by Fu, X and Meng, Z and Liang, W and Tian, Y and Wang, X and Han, W and Lou, G and Lou, F and Yen, Y and Yu, H and Jove, R and Huang, W
Oncogene, ISSN 1476-5594, 2013, Volume 33, Issue 34, pp. 4296 - 4306
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 08/2012, Volume 122, Issue 8, pp. 2871 - 2883
miR-122, an abundant liver-specific microRNA (miRNA), regulates cholesterol metabolism and promotes hepatitis C virus (HCV) replication. Reduced miR-122... 
MICRORNA BINDING | MEDICINE, RESEARCH & EXPERIMENTAL | HEPATOCELLULAR-CARCINOMA | LIPID-METABOLISM | IN-VIVO | GENE-EXPRESSION | MICE | STEATOHEPATITIS | DIFFERENTIATION | HEPATIC STEATOSIS | CANCER | MicroRNAs - therapeutic use | Liver - pathology | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Fatty Liver - pathology | Humans | Cell Survival - genetics | MicroRNAs - metabolism | Liver Neoplasms - therapy | Monocytes - immunology | Genes, myc | Mice, 129 Strain | RNA, Neoplasm - metabolism | Liver Neoplasms, Experimental - metabolism | Liver - immunology | Base Sequence | Carcinoma, Hepatocellular - genetics | Lipids - blood | Monocytes - pathology | Lipid Metabolism - genetics | Liver Neoplasms, Experimental - genetics | 3' Untranslated Regions | Genes, Tumor Suppressor | Neutrophils - pathology | Fatty Liver - genetics | Gene Expression | Fatty Liver - metabolism | Liver Neoplasms - genetics | Liver - metabolism | Neutrophils - immunology | Liver Neoplasms, Experimental - etiology | Mice, Knockout | Animals | Liver Neoplasms - metabolism | RNA, Neoplasm - genetics | Carcinoma, Hepatocellular - therapy | Mice | MicroRNAs - genetics | Cytokines - biosynthesis | Carcinoma, Hepatocellular - metabolism | Fatty Liver - etiology | Liver diseases | MicroRNA | Anti-inflammatory drugs | Tumor necrosis factor | Physiological aspects | Hepatitis C virus | Cholesterol | Tumors
Journal Article
JNCI : Journal of the National Cancer Institute, ISSN 0027-8874, 06/2012, Volume 104, Issue 12, pp. 906 - 922
Background A disintegrin and metalloproteinase 28 (ADAM28) is implicated in tumor growth and metastasis in human breast and non-small cell lung carcinomas. We... 
VONWILLEBRAND-FACTOR | A1 DOMAIN | ANGIOGENESIS | ONCOLOGY | DISINTEGRIN | FACTOR BINDING PROTEIN-3 | TUMOR-CELLS | PROLIFERATION | INTEGRIN ALPHA(V)BETA | GROWTH-FACTOR | METALLOPROTEINASES | Immunohistochemistry | Neoplasms - metabolism | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Immunoblotting | Lung Neoplasms - pathology | Transplantation, Heterologous | Kidney Neoplasms - metabolism | von Willebrand Factor - genetics | Luminescent Measurements | Breast Neoplasms - metabolism | Gene Knockdown Techniques | Neoplasms, Experimental - pathology | Liver Neoplasms - pathology | von Willebrand Factor - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Carcinoma, Renal Cell - pathology | Carcinoma, Non-Small-Cell Lung - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Gene Expression Regulation, Enzymologic | Carcinoma, Renal Cell - metabolism | Two-Hybrid System Techniques | ADAM Proteins - metabolism | Animals | Breast Neoplasms - pathology | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Kidney Neoplasms - pathology | Mice | Neoplasms, Experimental - metabolism | ADAM Proteins - genetics | Neoplasms - pathology | RNA, Small Interfering - administration & dosage | Apoptosis | Carcinoma, Hepatocellular - metabolism | RNA, Small Interfering - metabolism | Physiological aspects | Breast cancer | Metastasis | Research | Lung cancer, Non-small cell | Von Willebrand factor
Journal Article
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2018, Volume 155, Issue 2, pp. 557 - 571.e14
MAF bZIP transcription factor G (MAFG) is activated by the farnesoid X receptor to repress bile acid synthesis. However, expression of MAFG increases during... 
FXR | Obeticholic Acid | S-Adenosylmethionine | Ursodeoxycholic Acid | CELLS | C-MYC | GLUTATHIONE SYNTHESIS | COLON-CANCER | DEOXYCHOLIC-ACID | MICE | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | METHIONINE | Diethylnitrosamine - toxicity | Up-Regulation | Carcinoma, Hepatocellular - mortality | Liver - pathology | Humans | Male | Cholangiocarcinoma - etiology | Liver Neoplasms - mortality | Gene Knockdown Techniques | Cholestasis - etiology | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Liver Neoplasms, Experimental - genetics | Bile Duct Neoplasms - genetics | Gene Expression Regulation, Neoplastic - drug effects | Liver Neoplasms, Experimental - pathology | MafG Transcription Factor - metabolism | MafG Transcription Factor - genetics | Repressor Proteins - metabolism | Liver Neoplasms - virology | Liver Neoplasms - genetics | Mice, Inbred C57BL | Repressor Proteins - genetics | Carcinoma, Hepatocellular - virology | Receptors, Cytoplasmic and Nuclear - agonists | S-Adenosylmethionine - pharmacology | Liver Neoplasms, Experimental - etiology | Bile Duct Neoplasms - etiology | Xenograft Model Antitumor Assays | Cholestasis - pathology | Cholic Acids - pharmacology | Cholangiocarcinoma - pathology | Animals | Mice, Nude | Bile Duct Neoplasms - mortality | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Cholangiocarcinoma - genetics | Mice | Mice, Inbred BALB C | Bile Duct Neoplasms - pathology | RNA, Small Interfering - metabolism | Liver cancer | Analysis | obeticholic acid | ursodeoxycholic acid | S-adenosylmethionine
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2018, Volume 155, Issue 1, pp. 33 - 37.e6
We studied the effects of gut microbiome depletion by oral antibiotics on tumor growth in subcutaneous and liver metastases models of pancreatic cancer, colon... 
Immune Regulation | Tumor Promotion | Gut Bacteria | Metastases | GASTROENTEROLOGY & HEPATOLOGY | IL-17 | T-Lymphocyte Subsets - immunology | Neoplasm Transplantation | Interleukin-17 - immunology | Soft Tissue Neoplasms - immunology | Dysbiosis - immunology | Melanoma, Experimental - immunology | Neoplasm Metastasis - immunology | Melanoma, Experimental - secondary | Liver Neoplasms - secondary | Carcinoma - secondary | Disease Models, Animal | Skin Neoplasms - pathology | Skin Neoplasms - immunology | Soft Tissue Neoplasms - secondary | Pancreatic Neoplasms - pathology | Liver Neoplasms - immunology | Melanoma - secondary | Mice, Knockout | Tumor Microenvironment - immunology | Animals | Gastrointestinal Microbiome - drug effects | Melanoma - immunology | Neoplasms - immunology | Colonic Neoplasms - pathology | Interferon-gamma - immunology | Pancreatic Neoplasms - immunology | Cell Line, Tumor | T-Lymphocytes - immunology | Anti-Bacterial Agents - pharmacology | Mice | Gastrointestinal Microbiome - immunology | Interleukin-10 - immunology | Medical colleges | Microbiota (Symbiotic organisms) | Growth | Liver | Melanoma | Metastasis | B cells | T cells | Biological response modifiers | Liver cancer | Colon cancer | Interleukins | Immunotherapy | Pancreatic cancer | Bacteria | Interferon gamma | metastases | gut bacteria | immune regulation | tumor promotion
Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 83 - 13
Mitochondrial topoisomerase IB (TOP1MT) is a nuclear-encoded topoisomerase, exclusively localized to mitochondria, which resolves topological stress generated... 
CHAIN | RNA GRANULES | REGULATOR | HEPATOCELLULAR-CARCINOMA | ROLES | PROMOTION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | STEM-CELL | DNA TOPOISOMERASES | CANCER | Carcinoma, Hepatocellular - mortality | Liver - pathology | Protein Biosynthesis | Cell Proliferation | Prognosis | Humans | Gene Expression Regulation, Neoplastic | Liver Neoplasms, Experimental - chemically induced | Male | Gene Expression Profiling | Liver Neoplasms - mortality | Carcinogens - toxicity | DNA, Mitochondrial - isolation & purification | Liver Neoplasms, Experimental - metabolism | Cell Nucleus - metabolism | DNA, Mitochondrial - genetics | Carcinoma, Hepatocellular - genetics | Fibroblasts | Female | Liver Neoplasms - pathology | Liver Neoplasms, Experimental - genetics | Liver Neoplasms, Experimental - pathology | Datasets as Topic | DNA Topoisomerases, Type I - metabolism | Liver Neoplasms - genetics | HCT116 Cells | Liver - metabolism | DNA Topoisomerases, Type I - genetics | Mitochondria - metabolism | Mitochondria - pathology | Mice, Knockout | Carcinogenesis - pathology | Xenograft Model Antitumor Assays | Animals | Energy Metabolism | Mice, Nude | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Survival Analysis | Glycolysis | Liver - cytology | Mice | Carcinoma, Hepatocellular - metabolism | Cell proliferation | Phosphorylation | Animal models | Translation | Transcription | Liver | Hepatocellular carcinoma | Mitochondrial DNA | Drug development | Carcinogenesis | Liver cancer | Mitochondria | Carcinogens | Colon cancer | Oxidative phosphorylation | Colon | Cancer | Tumors | Life Sciences | Human health and pathology
Journal Article
Annals of surgery, ISSN 0003-4932, 04/2014, Volume 259, Issue 4, pp. 750 - 759
OBJECTIVE:To assess the contribution of hypoxia and bone marrow–derived cells to aggressive outgrowth of micrometastases after liver surgery. BACKGROUND:Liver... 
hypoxia | colorectal | inflammation | tirapazamine | Cancer stem cell | liver metastases | hypoxia activated prodrugs | surgery | INITIATING CELLS | ACCELERATED OUTGROWTH | SOLID TUMORS | COLON-CANCER | INDUCIBLE FACTORS | TRANSCATHETER ARTERIAL EMBOLIZATION | HEPATOCELLULAR-CARCINOMA | HEPATIC METASTASES | cancer stem cell | COLORECTAL-CANCER | RADIOFREQUENCY ABLATION | Immunohistochemistry | Neoplasm, Residual - pathology | Liver Neoplasms, Experimental - secondary | Reperfusion Injury - etiology | Neoplasm, Residual - metabolism | Triazines - therapeutic use | Humans | Postoperative Complications - metabolism | Hematopoietic Stem Cells - pathology | Catheter Ablation | Male | Antineoplastic Agents - therapeutic use | Liver Neoplasms, Experimental - therapy | Hypoxia - metabolism | Neoplasm Recurrence, Local - pathology | Liver Neoplasms, Experimental - metabolism | Flow Cytometry | Neoplastic Stem Cells - metabolism | Neoplasm Invasiveness - pathology | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Liver Neoplasms, Experimental - pathology | Real-Time Polymerase Chain Reaction | Reperfusion Injury - metabolism | Postoperative Complications - pathology | Hepatectomy - methods | Neoplasm Recurrence, Local - metabolism | Reperfusion Injury - pathology | Hypoxia - etiology | Mice, SCID | Blotting, Western | Phenotype | Animals | Hypoxia - pathology | Mice, Nude | Cell Line, Tumor | Mice, Inbred NOD | Mice | Mice, Inbred BALB C | Colorectal Neoplasms - pathology | Neoplasm Micrometastasis - pathology
Journal Article
Annals of surgical oncology, ISSN 1534-4681, 2018, Volume 25, Issue 9, pp. 2652 - 2660
Journal Article