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American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 09/2011, Volume 301, Issue 3, pp. C559 - C565
Deliu E, Tica AA, Motoc D, Brailoiu GC, Brailoiu E. Intracellular angiotensin II activates rat myometrium. Am J Physiol Cell Physiol 301: C559-C565, 2011.... 
Calcium imaging | Microinjection | Cytosolic calcium concentration | Endoplasmic reticulum | CYCLIC ADP-RIBOSE | PHYSIOLOGY | ANG-II | LIVER LYSOSOMES | RECEPTOR | microinjection | METABOLIC-CLEARANCE | BLOOD-PRESSURE | UTERINE | CELL BIOLOGY | SIGNAL-TRANSDUCTION | RENAL ENDOSOMES | SMOOTH-MUSCLE-CELLS | calcium imaging | cytosolic calcium concentration | endoplasmic reticulum | Myometrium - drug effects | Type C Phospholipases - metabolism | Brefeldin A - pharmacology | Angiotensin II Type 1 Receptor Blockers - pharmacology | Autophagy - drug effects | Endosomes - metabolism | Angiotensin II Type 2 Receptor Blockers - pharmacology | Macrocyclic Compounds - pharmacology | Saralasin - pharmacology | Endoplasmic Reticulum - drug effects | Estrenes - pharmacology | Myometrium - metabolism | Inositol 1,4,5-Trisphosphate Receptors - antagonists & inhibitors | Ryanodine - pharmacology | Angiotensin II - pharmacology | Pyridines - administration & dosage | Endocytosis - drug effects | Enzyme Inhibitors - pharmacology | Losartan - pharmacology | Saralasin - administration & dosage | Rats | Imidazoles - pharmacology | Piperazines - pharmacology | Rats, Sprague-Dawley | Receptor, Angiotensin, Type 2 - metabolism | Signal Transduction - drug effects | Calcium Signaling - drug effects | Models, Biological | Receptor, Angiotensin, Type 1 - metabolism | Oxazoles - pharmacology | Losartan - administration & dosage | Angiotensin II - administration & dosage | Calcium Signaling - physiology | Endoplasmic Reticulum - metabolism | Imidazoles - administration & dosage | Type C Phospholipases - antagonists & inhibitors | Lysosomes - metabolism | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Pyrrolidinones - pharmacology | Macrolides - pharmacology | NADP - analogs & derivatives | Female | NADP - metabolism | Heparin - pharmacology | Angiotensin II - metabolism | Arsenicals - pharmacology | Cells, Cultured | Sirolimus - pharmacology | Myometrium - cytology | Animals | Egtazic Acid - pharmacology | Uterus - cytology | Signal Transduction - physiology | Pyridines - pharmacology | Carbolines - pharmacology | Myometrium | Cell receptors | Calcium channels | Genetic aspects | Properties | Angiotensin | Receptors and Signal Transduction
Journal Article
Gastroenterology, ISSN 0016-5085, 2000, Volume 118, Issue 6, pp. 1149 - 1156
Circulating levels of angiotensin II (ANGII), a powerful vasoconstrictor factor, are frequently increased in chronic liver diseases. In these conditions,... 
FAT-STORING CELLS | RAT-LIVER | GLOMERULAR MESANGIAL CELLS | DEPENDENT CONTRACTILITY | NITRIC-OXIDE | GROWTH-FACTOR | ITO CELLS | GASTROENTEROLOGY & HEPATOLOGY | SMOOTH-MUSCLE CELLS | PORTAL-HYPERTENSION | ALDOSTERONE SYSTEM | Receptor, Angiotensin, Type 1 | Receptors, Angiotensin - metabolism | Receptor, Angiotensin, Type 2 | Antihypertensive Agents - pharmacology | Iodine Radioisotopes | Liver - enzymology | Humans | Penicillamine - analogs & derivatives | Sodium Nitrite - pharmacology | Liver - drug effects | Thymidine - pharmacology | Nitric Oxide Donors - pharmacology | Radioligand Assay | Thymidine - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Dinoprostone - pharmacology | Angiotensin II - pharmacology | Angiotensin II - metabolism | Cell Size - drug effects | Penicillamine - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Losartan - pharmacology | Rats | Imidazoles - pharmacology | Cell Division - drug effects | Indomethacin - pharmacology | Cyclooxygenase Inhibitors - pharmacology | Animals | Vasoconstrictor Agents - metabolism | Liver - cytology | Pyridines - pharmacology | Mitogen-Activated Protein Kinases - metabolism | Cell research | Liver diseases | Platelet-derived growth factor | Prostaglandins E | Nitric oxide | Angiotensin | Physiological aspects | Bookbinding | DNA synthesis | Peptide hormones | Protein kinases
Journal Article
Life Sciences, ISSN 0024-3205, 10/2016, Volume 163, pp. 1 - 10
Journal Article
The Journal of Physiology, ISSN 0022-3751, 12/2016, Volume 594, Issue 23, pp. 7027 - 7047
Key points Candesartan, an inverse agonist of the type 1 angiotensin II receptor (AT1R), causes a concentration‐dependent inhibition of pressure‐dependent... 
type 1 angiotensin II receptor | cytoskeleton | mechanotransduction | actin stress fibres | vascular smooth muscle cell signalling | myogenic response | protein kinase C | RHO-ASSOCIATED KINASE | INTRAVASCULAR PRESSURE | PHYSIOLOGY | CEREBRAL PARENCHYMAL ARTERIOLES | RESISTANCE ARTERIES | PROTEIN-COUPLED RECEPTORS | MYOSIN PHOSPHORYLATION | STRETCH-ACTIVATION | CYTOSKELETON REORGANIZATION | NEUROSCIENCES | VASCULAR SMOOTH-MUSCLE | LIGHT-CHAIN PHOSPHORYLATION | Arterioles - physiology | Antihypertensive Agents - pharmacology | Receptor, Angiotensin, Type 1 - physiology | Tetrazoles - pharmacology | Captopril - pharmacology | Maleimides - pharmacology | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Muscle Development | Receptor, Angiotensin, Type 1 - genetics | Diglycerides - pharmacology | Indoles - pharmacology | Muscle Fibers, Skeletal - physiology | Vasoconstrictor Agents - pharmacology | Protein Kinase C - physiology | Abdominal Muscles - physiology | Cells, Cultured | Losartan - pharmacology | Imidazoles - pharmacology | Protein Kinase C - antagonists & inhibitors | Arterioles - drug effects | Pressure | Rats, Sprague-Dawley | Vasoconstriction - drug effects | Animals | Benzimidazoles - pharmacology | Pyridines - pharmacology | Actins - physiology | Atomic force microscopy | Actin | Angiotensin | Polymerization | Muscles | G proteins | Muscle proteins | Protein kinases | Arteries | Membrane proteins | Cardiovascular Physiology | Skeletal Muscle | Cardiovascular | Research Paper
Journal Article
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 02/2014, Volume 67, pp. 366 - 376
Chronic lead exposure induces hypertension affecting endothelial function. We investigated whether low-concentration lead exposure alters blood pressure and... 
Hypertension | Rat aorta | Lead acetate | Free radicals | Vasoconstrictor prostanoids | ROS | Local renin–angiotensin system | Local renin-angiotensin system | SYSTEM | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTATHIONE-PEROXIDASE | BLOOD-PRESSURE | GUANYLATE-CYCLASE | INDUCED HYPERTENSION | SMOOTH-MUSCLE | NITRIC-OXIDE METABOLISM | ENDOTHELIUM | CYCLOOXYGENASE PATHWAYS | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR-DISEASE | Rats, Wistar | Prostaglandins - pharmacology | Male | Aorta - metabolism | Lead Poisoning - metabolism | Phenylephrine - pharmacology | Acetophenones - pharmacology | Vascular Resistance - drug effects | Blood Pressure - drug effects | Aorta - physiopathology | Lead Poisoning - physiopathology | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Superoxide Dismutase - pharmacology | Vasodilator Agents - pharmacology | Cyclooxygenase 2 Inhibitors - pharmacology | Aorta - drug effects | Losartan - pharmacology | Rats | Antioxidants - pharmacology | Vasoconstriction - drug effects | Indomethacin - pharmacology | Animals | Renin-Angiotensin System - drug effects | Vasodilation - drug effects | Nitric Oxide - metabolism | Chronic Disease | COX-2 inhibitors | Prostaglandins | International agencies | Angiotensin | Dosage and administration | Superoxide | Blood pressure | Indomethacin
Journal Article
Canadian Journal of Physiology and Pharmacology, ISSN 0008-4212, 08/2012, Volume 90, Issue 8, pp. 1105 - 1116
We have previously shown that A10 vascular smooth muscle cells (VSMC) exposed to angiotensin II (Ang II) exhibited overexpression of Giα proteins. In the... 
Giα proteins | AMPc | receptors | PLC | PTK | récepteurs AT | PKC | protéines Giα | cAMP | CaM kinase | Gia proteins | OXIDATIVE STRESS | PHYSIOLOGY | STIMULATION | TYROSINE PHOSPHORYLATION | SPONTANEOUSLY HYPERTENSIVE-RATS | Gi alpha proteins | INVOLVEMENT | AT receptors | RECEPTOR SUBTYPES | INHIBITION | CALCIUM-CHANNELS | PHARMACOLOGY & PHARMACY | ADENYLATE-CYCLASE | KINASE ACTIVATION | Gallic Acid - pharmacology | Nucleic Acid Synthesis Inhibitors - pharmacology | Calcium - metabolism | GTP-Binding Protein alpha Subunits, Gi-Go - biosynthesis | Angiotensin II Type 1 Receptor Blockers - pharmacology | Type C Phospholipases - antagonists & inhibitors | Angiotensin II Type 2 Receptor Blockers - pharmacology | Drug Interactions | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Egtazic Acid - analogs & derivatives | Gallic Acid - analogs & derivatives | Cell Line | Angiotensin II - pharmacology | Losartan - pharmacology | Gene Expression Regulation - physiology | Rats | Imidazoles - pharmacology | Chelating Agents - pharmacology | Gene Expression Regulation - drug effects | Animals | Egtazic Acid - pharmacology | Genistein - pharmacology | Signal Transduction - physiology | Bucladesine - pharmacology | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Dactinomycin - pharmacology | Angiotensin II - physiology
Journal Article
FASEB Journal, ISSN 0892-6638, 03/2017, Volume 31, Issue 3, pp. 1193 - 1203
VEGF inhibitors, including receptor tyrosine kinase inhibitors, are used as adjunct therapies in a number of cancer treatments. An emerging issue with these... 
Hypertension | Cancer therapy | Regional hemodynamics | RTKI | VEGF | SIGNALING PATHWAY INHIBITORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TOXICITY | CANCER | RENAL-CELL CARCINOMA | BLOOD-PRESSURE | CELL BIOLOGY | cancer therapy | GROWTH-FACTOR RECEPTOR | INDUCED HYPERTENSION | TARGETED THERAPIES | regional hemodynamics | BIOLOGY | SUNITINIB | hypertension | FACTOR VEGF RECEPTORS | Niacinamide - analogs & derivatives | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Regional Blood Flow - drug effects | Piperidines - pharmacology | Propranolol - administration & dosage | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Quinazolines - administration & dosage | Adrenergic beta-Antagonists - administration & dosage | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Piperidines - administration & dosage | Consciousness | Pyrimidines - administration & dosage | Losartan - pharmacology | Rats | Pyrimidines - pharmacology | Sulfonamides - pharmacology | Rats, Sprague-Dawley | Niacinamide - administration & dosage | Protein Kinase Inhibitors - administration & dosage | Animals | Phenylurea Compounds - administration & dosage | Hemodynamics - drug effects | Phenylurea Compounds - pharmacology | Protein Kinase Inhibitors - pharmacology | Losartan - administration & dosage | Niacinamide - pharmacology | Quinazolines - pharmacology | Sulfonamides - administration & dosage | Drugs | Physiological effects | Drug development | Arteries | Rodents | Aorta | Protein-tyrosine kinase receptors | Blood pressure | Propranolol | Vascular endothelial growth factor | Protein-tyrosine kinase | Tyrosine | Jugular vein | Kidneys | Regional analysis | Catheters | Inhibitors | Coronary vessels | Phentolamine | Hemodynamics | Circulatory system | Cancer | Peritoneum | Research
Journal Article
Progress in Neuropsychopharmacology & Biological Psychiatry, ISSN 0278-5846, 06/2013, Volume 43, pp. 79 - 88
While it is now well established that the independent brain renin–angiotensin system (RAS) has some important central functions besides the vascular ones, the... 
Angiotensin II | Oxidative stress | Memory | RECEPTOR ANTAGONISTS | AT RECEPTOR | PSYCHIATRY | ALZHEIMERS-DISEASE | MOTOR-ACTIVITY | CONVERTING ENZYME-INHIBITORS | DEFICIENT MICE | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | CLINICAL NEUROLOGY | VASCULAR DEMENTIA | PHARMACOLOGY & PHARMACY | CEREBRAL-BLOOD-FLOW | ANTIHYPERTENSIVE TREATMENTS | Memory - drug effects | Captopril - pharmacology | Rats, Wistar | Angiotensin Receptor Antagonists - pharmacology | Male | Hippocampus - drug effects | Angiotensin II Type 1 Receptor Blockers - pharmacology | Angiotensin II Type 2 Receptor Blockers - pharmacology | Lipid Peroxidation - drug effects | Behavior, Animal - drug effects | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Glutathione Peroxidase - metabolism | Angiotensin II - pharmacology | Losartan - pharmacology | Rats | Memory, Short-Term - drug effects | Imidazoles - pharmacology | Psychomotor Performance - drug effects | Maze Learning - drug effects | Hippocampus - metabolism | Animals | Avoidance Learning - drug effects | Pyridines - pharmacology | Oxidative Stress - drug effects | Oxidases | Phosphates | Niacinamide | Brain | Captopril | Amyloid beta-protein | Proline | Superoxide | Antioxidants | Purines | Angiotensin | Alzheimer's disease
Journal Article