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Journal Article
Science Translational Medicine, ISSN 1946-6234, 06/2015, Volume 7, Issue 292, pp. 292ra98 - 292ra98
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 11/2012, Volume 164, Issue 1, pp. 49 - 57
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 10/2013, Volume 272, Issue 1, pp. 127 - 136
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53906
Background: MicroRNAs are short regulatory RNAs that negatively modulate protein expression at a post-transcriptional and/or translational level and are deeply... 
BREAST-CANCER | LUNG-CANCER | SMALL RNAS | INDUCED APOPTOSIS | GASTRIC-CANCER | CYCLIN D1 | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER CELLS | TUMOR-SUPPRESSOR PTEN | MESENCHYMAL TRANSITION | EXPRESSION | MicroRNAs - antagonists & inhibitors | Apoptosis - drug effects | Humans | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Bone Neoplasms - pathology | Proto-Oncogene Proteins c-akt - genetics | Bone Neoplasms - metabolism | Luciferases | Female | Antineoplastic Agents - pharmacology | Bone Neoplasms - genetics | Gene Expression Regulation, Neoplastic - drug effects | 3' Untranslated Regions | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Genes, Reporter | Osteosarcoma - metabolism | PTEN Phosphohydrolase - genetics | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Cisplatin - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | MicroRNAs - genetics | Osteosarcoma - genetics | Cell Cycle - drug effects | Osteosarcoma - pathology | Drug Resistance, Neoplasm - drug effects | MicroRNA | Osteosarcoma | Analysis | Genes | Luciferase | Genetic research | Genetic transcription | Cisplatin | Apoptosis | Immunohistochemistry | Post-transcription | Biotechnology | Pathogenesis | Oncology | AKT protein | Retailing | Metastasis | Kinases | Phosphatase | Tissues | Assaying | Osteoblasts | Bone surgery | Cancer therapies | Proteins | Liver cancer | Cell growth | Bioaccumulation | Cell cycle | Biocompatibility | Bioinformatics | Cell survival | MiRNA | Breast cancer | Gene expression | Real time | Ribonucleic acid--RNA | Survival | 1-Phosphatidylinositol 3-kinase | Bone cancer | Chemotherapy | Inhibitors | 3' Untranslated regions | MicroRNAs | Orthopedics | Cell lines | Prostate | Viability | PTEN protein | RNA | Ribonucleic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, pp. e0159998 - e0159998
Recent research found that Tiron was an effective antioxidant that could act as the intracellular reactive oxygen species (ROS) scavenger or alleviate the... 
IRRADIATION | OXIDATIVE STRESS | HUMAN SKIN | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | MECHANISMS | SODIUM 4,5-DIHYDROXYBENZENE-1,3-DISULFONATE | INDUCTION | C-JUN | NF-KAPPA-B | PROTEIN-KINASES | Promoter Regions, Genetic - radiation effects | Binding Sites - radiation effects | Humans | Transcriptional Activation - drug effects | Skin Aging - radiation effects | 1,2-Dihydroxybenzene-3,5-Disulfonic Acid Disodium Salt - pharmacology | Promoter Regions, Genetic - drug effects | Transcription Factor AP-1 - metabolism | Dermis - drug effects | Cytoprotection - drug effects | Signal Transduction - radiation effects | Dermis - cytology | Matrix Metalloproteinase 1 - genetics | Fibroblasts - metabolism | MAP Kinase Signaling System - radiation effects | Dermis - metabolism | Skin Aging - drug effects | Transcriptional Activation - radiation effects | Cells, Cultured | Dermis - radiation effects | Matrix Metalloproteinase 3 - metabolism | Antioxidants - pharmacology | Ultraviolet Rays - adverse effects | MAP Kinase Signaling System - drug effects | Fibroblasts - radiation effects | Signal Transduction - drug effects | Fibroblasts - drug effects | Transcription Factor AP-1 - radiation effects | Cytoprotection - genetics | Matrix Metalloproteinase 1 - metabolism | Cytoprotection - radiation effects | Matrix Metalloproteinase 3 - genetics | Genetic aspects | Skin | Genetic transcription | RNA | Analysis | Enzyme-linked immunosorbent assay | Oxidative stress | Reactive oxygen species | Transcription factors | Senescence | Oncology | Activation | Matrix metalloproteinase | Kinases | Proteins | Antioxidants | Signal transduction | Consent | Transcription activation | Fibroblasts | Aging | Biocompatibility | Metalloproteinase | Interstitial collagenase | Stromelysin 1 | Wound healing | U.V. radiation | Activator protein 1 | MAP kinase | Gene expression | Medicine | Signaling | Collagen | In vivo methods and tests | Binding sites | Apoptosis | Index Medicus
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 03/2003, Volume 95, Issue 6, pp. 437 - 448
Background: Interferon alpha (IFN-alpha) has antiangiogenic activity, although the underlying mechanism of action is unclear. Because human neuroendocrine (NE)... 
CELLS | INHIBITION | THERAPY | ONCOLOGY | SP1 | CLINICAL-APPLICATIONS | PROTEIN-KINASE | DOWN-REGULATION | EXPRESSION | CANCER | BINDING | Immunohistochemistry | Interferon-alpha - pharmacology | Transcription, Genetic - drug effects | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Luciferases - metabolism | Vascular Endothelial Growth Factors | Receptors, Vascular Endothelial Growth Factor - drug effects | Transplantation, Heterologous | Pregnancy Proteins - metabolism | Promoter Regions, Genetic - drug effects | RNA, Messenger - metabolism | Biomarkers, Tumor | Intercellular Signaling Peptides and Proteins - metabolism | Lymphokines - genetics | Receptors, Vascular Endothelial Growth Factor - metabolism | Time Factors | Lymphokines - metabolism | Antineoplastic Agents - pharmacology | Electrophoretic Mobility Shift Assay | Gene Expression Regulation, Neoplastic - drug effects | Receptors, Vascular Endothelial Growth Factor - genetics | Tumor Cells, Cultured | Lymphokines - drug effects | Enzyme-Linked Immunosorbent Assay | Neuroendocrine Tumors - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Angiogenesis Inhibitors - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Endothelial Growth Factors - genetics | Neuroendocrine Tumors - drug therapy | Genes, Reporter - drug effects | Mice | Neovascularization, Pathologic - metabolism | Neuroendocrine Tumors - blood supply | Receptors, Immunologic - metabolism | Measurement | Physiological aspects | Vascular endothelial growth factor | Interferon alpha | Interferon-alpha/pharmacology | Endothelial Growth Factors/genetics/metabolism | Tumor Cells; Cultured | Tumor Markers; Biological | Neuroendocrine Tumors/blood supply/drug therapy/metabolism | Antineoplastic Agents/pharmacology | Pregnancy Proteins/metabolism | Luciferases/metabolism | Promoter Regions (Genetics)/drug effects | Research Support; Non-U.S. Gov't | Genes; Reporter/drug effects | Mice; Nude | Gene Expression Regulation; Neoplastic/drug effects | RNA; Messenger/metabolism | Receptors; Vascular Endothelial Growth Factor/drug effects/genetics/metabolism | Receptors; Immunologic/metabolism | Intercellular Signaling Peptides and Proteins/genetics/metabolism | Neovascularization; Pathologic/drug therapy/metabolism | Angiogenesis Inhibitors/pharmacology | Lymphokines/drug effects/genetics/metabolism | Transcription; Genetic/drug effects | Transplantation; Heterologous
Journal Article
Plant Cell, ISSN 1040-4651, 3/2015, Volume 27, Issue 3, pp. 772 - 786
Journal Article
Cancer Research, ISSN 0008-5472, 06/2010, Volume 70, Issue 12, pp. 5184 - 5193
MicroRNAs (miRNA) have rapidly emerged as modulators of gene expression in cancer in which they may have great diagnostic and therapeutic import.... 
MIGRATION | INVASIVE GROWTH | INHIBITION | METASTASIS | THERAPY | ONCOLOGY | DOWN-REGULATION | HUMAN HEPATOCELLULAR-CARCINOMA | IDENTIFICATION | MICRORNA EXPRESSION | CANCER | Proto-Oncogene Proteins c-met - metabolism | Luciferases - metabolism | Antibiotics, Antineoplastic - pharmacology | Apoptosis - drug effects | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Male | Intracellular Signaling Peptides and Proteins - metabolism | RNA, Messenger - metabolism | Carcinoma, Hepatocellular - drug therapy | Carcinoma, Hepatocellular - genetics | Aged, 80 and over | Female | Liver Neoplasms - pathology | Intracellular Signaling Peptides and Proteins - genetics | Liver Cirrhosis - genetics | Protein-Serine-Threonine Kinases - metabolism | Liver Cirrhosis - drug therapy | Liver Neoplasms - genetics | RNA, Messenger - genetics | Liver Neoplasms - drug therapy | Protein-Serine-Threonine Kinases - genetics | Cell Adhesion - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Proto-Oncogene Proteins c-met - genetics | Cell Movement - drug effects | Carcinoma, Hepatocellular - pathology | Liver Cirrhosis - pathology | Aged | Cell Proliferation - drug effects | TOR Serine-Threonine Kinases | MicroRNAs - physiology | Cell Cycle - drug effects | Doxorubicin - pharmacology
Journal Article
Cancer Cell, ISSN 1535-6108, 2008, Volume 13, Issue 3, pp. 249 - 260
The Hedgehog (Hh) pathway plays critical roles in normal development and in tumorigenesis. We generated Gli-luciferase transgenic mice to evaluate the Smo... 
CELLBIO | DEVBIO | CELL LUNG-CANCER | TARGETED THERAPY | VERATRUM CALIFORNICUM | STEM-CELLS | IN-VITRO | ONCOLOGY | SIGNALING PATHWAY | SONIC HEDGEHOG | PROSTATE-CANCER | INDIAN HEDGEHOG | SMALL-MOLECULE INHIBITOR | CELL BIOLOGY | Growth Plate - pathology | Bone and Bones - pathology | Luciferases - metabolism | Receptors, G-Protein-Coupled - metabolism | Hedgehog Proteins - metabolism | Microscopy, Video | Antineoplastic Agents - administration & dosage | Recombinant Fusion Proteins - metabolism | Bone Remodeling - drug effects | Antineoplastic Agents - toxicity | Bone and Bones - drug effects | Luciferases - genetics | Dose-Response Relationship, Drug | Chondrocytes - drug effects | Time Factors | Kruppel-Like Transcription Factors - metabolism | Bone and Bones - metabolism | Smoothened Receptor | Growth Plate - drug effects | Chondrocytes - metabolism | Animals, Newborn | Chondrocytes - pathology | Cerebellar Neoplasms - drug therapy | Calcification, Physiologic - drug effects | Administration, Oral | Osteogenesis - drug effects | Cells, Cultured | Mice, Transgenic | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Receptors, G-Protein-Coupled - antagonists & inhibitors | Cell Proliferation - drug effects | Mice | Zinc Finger Protein GLI1 | Bone and Bones - embryology | Medulloblastoma - drug therapy | Kruppel-Like Transcription Factors - genetics | Microscopy, Fluorescence | Aging - metabolism | Luciferase | Genetic engineering | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e61792
MicroRNAs (miRNAs) have been implicated to play a central role in the development of drug resistance in a variety of malignancies. However, many studies were... 
CELLS | ACTIVATION | DRUG-RESISTANCE | MAPK PATHWAYS | MULTIDISCIPLINARY SCIENCES | CHEMOTHERAPY | Cell Count | Humans | MicroRNAs - metabolism | NF-kappa B - metabolism | Stomach Neoplasms - pathology | Luminescent Measurements | Fluorouracil - therapeutic use | Herpes Simplex Virus Protein Vmw65 - metabolism | Female | p38 Mitogen-Activated Protein Kinases - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Transgenes | Genes, Reporter | Luciferases, Firefly - metabolism | Stomach Neoplasms - genetics | Reproducibility of Results | Up-Regulation - genetics | Signal Transduction - genetics | Stomach Neoplasms - drug therapy | Symporters - metabolism | Up-Regulation - drug effects | Diagnostic Imaging | Drug Resistance, Neoplasm - genetics | Animals | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Fluorouracil - pharmacology | Mice | MicroRNAs - genetics | Drug resistance in microorganisms | Care and treatment | MicroRNA | Genes | Luciferase | Imaging systems | Genetic aspects | Stomach cancer | Cancer | Nuclear medicine | Biotechnology | Bioluminescence | 5-Fluorouracil | Genomics | Luminescence | Chemoresistance | Retailing | Drug resistance | Kinases | Neurogenesis | Cancer therapies | Sodium iodide | Proteins | Biomedical materials | Reporter gene | Imaging | Xenografts | Cell cycle | Sodium iodide symporter | Life sciences | Cardiology | Gastric cancer | Radioactivity | Pathogens | NF-κB protein | Iodides | Multidrug resistance | MiRNA | Etoposide | Gene expression | Gene fusion | Myogenesis | Chemotherapy | Hospitals | Ribonucleic acids | Sodium | MicroRNAs | Laboratory animals
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2010, Volume 285, Issue 27, pp. 20940 - 20951