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by Iglesias, Adriana I and Mishra, Aniket and Vitart, Veronique and Bykhovskaya, Yelena and Höhn, René and Springelkamp, Henriët and Cuellar-Partida, Gabriel and Gharahkhani, Puya and Bailey, Jessica N. Cooke and Willoughby, Colin E and Li, Xiaohui and Yazar, Seyhan and Nag, Abhishek and Khawaja, Anthony P and Polašek, Ozren and Siscovick, David and Mitchell, Paul and Tham, Yih Chung and Haines, Jonathan L and Kearns, Lisa S and Hayward, Caroline and Shi, Yuan and Van Leeuwen, Elisabeth M and Taylor, Kent D and Wang, Jie Jin and Rochtchina, Elena and Attia, John and Scott, Rodney and Holliday, Elizabeth G and Baird, Paul N and Xie, Jing and Inouye, Michael and Viswanathan, Ananth and Sim, Xueling and Bonnemaijer, Pieter and Rotter, Jerome I and Martin, Nicholas G and Zeller, Tanja and Mills, Richard A and Staffieri, Sandra E and Jonas, Jost B and Schmidtmann, Irene and Boutin, Thibaud and Kang, Jae H and Lucas, Sionne E. M and Wong, Tien Yin and Beutel, Manfred E and Wilson, James F and Allingham, R. Rand and Brilliant, Murray H and Budenz, Donald L and Christen, William G and Fingert, John and Friedman, David S and Gaasterland, Douglas and Gaasterland, Terry and Hauser, Michael A and Kraft, Peter and Lee, Richard K and Lichter, Paul R and Liu, Yutao and Loomis, Stephanie J and Moroi, Sayoko E and Pericak-Vance, Margaret A and Realini, Anthony and Richards, Julia E and Schuman, Joel S and Scott, William K and Singh, Kuldev and Sit, Arthur J and Vollrath, Douglas and Weinreb, Robert N and Wollstein, Gadi and Zack, Donald J and Zhang, Kang and Donnelly, Peter and Barroso, Ines and Blackwell, Jenefer M and Bramon, Elvira and Brown, Matthew A and Casas, Juan P and Corvin, Aiden and Deloukas, Panos and Duncanson, Audrey and Jankowski, Janusz and Markus, Hugh S and Mathew, Christopher G and Palmer, Colin N. A and Plomin, Robert and Rautanen, Anna and Sawcer, Stephen J and Trembath, Richard C and Wood, Nicholas W and Spencer, Chris C. A and Band, Gavin and Bellenguez, Céline and Freeman, Colin and Hellenthal, Garrett and Giannoulatou, Eleni and Pirinen, Matti and ... and Blue Mountains Eye Study-GWAS Grp and NEIGHBORHOOD Consortium and Wellcome Trust Case Control Consor and Blue Mountains Eye Study—GWAS group and Wellcome Trust Case Control Consortium 2 (WTCCC2)
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1 - 11
Central corneal thickness (CCT) is a highly heritable trait associated with complex eye diseases such as keratoconus and glaucoma. We perform a genome-wide... 
DECORIN | CHROMATIN STATES | POLYMORPHISMS | RISK-FACTOR | MULTIDISCIPLINARY SCIENCES | MOUSE | GENE-EXPRESSION | MUTATIONS | HERITABILITY | LUMICAN | KERATOCONUS | Glaucoma | Cornea | Genes | Myopia | Association analysis | Genomes | Gene expression | Tissues | Connective tissues | Keratoconus | Collagen | Eye diseases | Extracellular matrix | Index Medicus
Journal Article
Nature communications, ISSN 2041-1723, 05/2018, Volume 9, Issue 1, p. 1864
Central corneal thickness (CCT) is a highly heritable trait associated with complex eye diseases such as keratoconus and glaucoma. We perform a genome-wide... 
Lumican - metabolism | Glaucoma, Open-Angle - genetics | Humans | Corneal Diseases - metabolism | Corneal Dystrophies, Hereditary - ethnology | Corneal Diseases - genetics | Ehlers-Danlos Syndrome - genetics | Corneal Dystrophies, Hereditary - pathology | Loeys-Dietz Syndrome - metabolism | Mendelian Randomization Analysis | Fibrillin-1 - metabolism | Cornea - pathology | Keratoconus - pathology | Corneal Diseases - pathology | Decorin - genetics | Gene Expression | Eye Diseases, Hereditary - pathology | Lumican - genetics | ADAMTS Proteins - metabolism | Proteoglycans - metabolism | Ehlers-Danlos Syndrome - ethnology | European Continental Ancestry Group | Myopia - pathology | Cornea - metabolism | Myopia - ethnology | Loeys-Dietz Syndrome - pathology | Corneal Diseases - ethnology | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Cornea - abnormalities | Quantitative Trait Loci | Proteoglycans - genetics | ADAMTS Proteins - genetics | Transforming Growth Factor beta2 - metabolism | Corneal Dystrophies, Hereditary - genetics | Glaucoma, Open-Angle - pathology | Keratoconus - metabolism | Loeys-Dietz Syndrome - ethnology | Glaucoma, Open-Angle - ethnology | Myopia - metabolism | Ehlers-Danlos Syndrome - pathology | Marfan Syndrome - ethnology | Keratoconus - genetics | Glaucoma, Open-Angle - metabolism | Fibrillin-1 - genetics | Eye Diseases, Hereditary - ethnology | Genome-Wide Association Study | Quantitative Trait, Heritable | Corneal Dystrophies, Hereditary - metabolism | Marfan Syndrome - genetics | Eye Diseases, Hereditary - genetics | Myopia - genetics | Asian Continental Ancestry Group | Keratoconus - ethnology | Ehlers-Danlos Syndrome - metabolism | Loeys-Dietz Syndrome - genetics | Polymorphism, Single Nucleotide | Transforming Growth Factor beta2 - genetics | Genome, Human | Decorin - metabolism | Eye Diseases, Hereditary - metabolism | Cornea | Decorin | Corneal Dystrophies, Hereditary | Myopia | Corneal Diseases | Marfan Syndrome | Keratoconus | ADAMTS Proteins | Lumican | Transforming Growth Factor beta2 | Ehlers-Danlos Syndrome | Glaucoma, Open-Angle | Fibrillin-1 | Eye Diseases, Hereditary | Proteoglycans | Wellcome Trust Case Control Consortium 2 (WTCCC2) | Blue Mountains Eye Study—GWAS group | Loeys-Dietz Syndrome | NEIGHBORHOOD Consortium
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, pp. e0184028 - e0184028
Journal Article
FEBS Journal, ISSN 1742-464X, 05/2013, Volume 280, Issue 10, pp. 2382 - 2398
During progression to heart failure (HF), myocardial extracellular matrix (ECM) alterations and tissue inflammation are central. Lumican is an ECM‐localized... 
heart failure | lumican | fibrosis | inflammation | pressure overload | LYSYL OXIDASE | DIFFERENTIAL EXPRESSION | RISK-FACTORS | COLLAGEN CROSS-LINKING | BIOCHEMISTRY & MOLECULAR BIOLOGY | METALLOPROTEINASES | IN-VITRO | INHIBITION | LEUCINE-RICH PROTEOGLYCANS | MOUSE MODEL | EXTRACELLULAR-MATRIX | Cardiomyopathy, Dilated - pathology | Interleukin-1beta - pharmacology | Phosphorylation | Chondroitin Sulfate Proteoglycans - genetics | Rats, Wistar | Humans | Middle Aged | Transforming Growth Factor beta1 - metabolism | Extracellular Matrix - metabolism | Stress, Mechanical | Male | NF-kappa B - metabolism | Smad3 Protein - metabolism | RNA, Messenger - metabolism | Smad3 Protein - genetics | Chondroitin Sulfate Proteoglycans - pharmacology | Adult | Female | Keratan Sulfate - pharmacology | Lumican | Lung - metabolism | Fibroblasts - metabolism | Animals, Newborn | Lung - pathology | Collagen Type I - metabolism | Echocardiography | Signal Transduction | Mice, Inbred C57BL | RNA, Messenger - genetics | Chondroitin Sulfate Proteoglycans - metabolism | Keratan Sulfate - metabolism | Organ Size | Rats | Heart Failure - metabolism | Transforming Growth Factor beta1 - genetics | Heart Failure - pathology | Fibroblasts - pathology | Keratan Sulfate - genetics | Cardiomyopathy, Dilated - metabolism | Animals | Mice | Oxidases | Heart failure | Bone morphogenetic proteins | RNA | Collagen | Proteins | Cytokines | Cellular biology | Pathogenesis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, pp. e27574 - e27574
Chronic pancreatitis is a chronic inflammatory disorder of the pancreas. The etiology is multi-fold, but all lead to progressive scarring and loss of... 
CELLS | SPLENIC VEIN-THROMBOSIS | BIOMARKER DISCOVERY | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | CANCER-TISSUE | EXTRACELLULAR-MATRIX | TANDEM MASS-SPECTROMETRY | PROTEINS | PARAFFIN-EMBEDDED TISSUES | STATISTICAL-MODEL | Immunohistochemistry | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Humans | Glycoproteins - metabolism | Adenocarcinoma - metabolism | Lumican | Proteomics - methods | Pancreatic Stellate Cells - pathology | Extracellular Matrix Proteins - metabolism | Acinar Cells - metabolism | Reproducibility of Results | Signal Transduction | Pancreatic Neoplasms - pathology | Chondroitin Sulfate Proteoglycans - metabolism | Keratan Sulfate - metabolism | Pancreas - pathology | Pancreas - metabolism | Pancreatitis, Chronic - pathology | Collagen - metabolism | Acinar Cells - pathology | Proteins - metabolism | Biological Specimen Banks | Staining and Labeling | Versicans - metabolism | Pancreatitis, Chronic - metabolism | Cluster Analysis | Pancreatic Stellate Cells - metabolism | Proteins | Medical research | Pancreatic cancer | Pancreatitis | Medicine, Experimental | Development and progression | Comparative analysis | Formaldehyde | Adenocarcinoma | Pathogenesis | Paraffin | Tissues | Cell activation | Epidermal growth factor | Pathways | Etiology | Extracellular matrix | Lesions | Pancreas | Versican | Ethanol | Glycoproteins | Metabolism | Disease control | Thrombosis | Biological activity | Medicine | Scars | Collagen | Proteomics | Fibrosis | Prothrombin | Functional groups | Cancer | Index Medicus
Journal Article
European respiratory journal, ISSN 0903-1936, 2012, Volume 40, Issue 6, pp. 1362 - 1373
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 3, pp. 612 - 621.e5
Background & Aims Collagen I deposition contributes to liver fibrosis, yet little is known about other factors that mediate this process. Fibromodulin is a... 
Gastroenterology and Hepatology | Liver Disease | Intercellular Communication | Mouse Model | Fibers | DIFFERENTIAL EXPRESSION | LIPID-PEROXIDATION | LUMICAN | GROWTH-FACTOR-BETA | HEPATIC STELLATE CELLS | ABNORMAL COLLAGEN FIBRILS | HEPG2 CELLS | KUPFFER CELLS | EXTRACELLULAR-MATRIX | GASTROENTEROLOGY & HEPATOLOGY | CONDITIONED MEDIUM | Liver - virology | Up-Regulation | Liver - pathology | Cell Proliferation | Carbon Tetrachloride | Coculture Techniques | Humans | Hepatic Stellate Cells - metabolism | Male | RNA, Messenger - metabolism | Liver - drug effects | Time Factors | Hepatitis C - complications | Hepatic Stellate Cells - drug effects | Endothelial Cells - metabolism | Extracellular Matrix Proteins - genetics | Liver - metabolism | Proteoglycans - metabolism | Rats | Liver Cirrhosis, Biliary - pathology | Mice, Knockout | Chemical and Drug Induced Liver Injury - metabolism | Liver Cirrhosis - pathology | Mice | Oxidative Stress - drug effects | Endothelial Cells - pathology | Liver Cirrhosis, Experimental - metabolism | Proteoglycans - genetics | Cell Movement | Extracellular Matrix Proteins - deficiency | Actins - metabolism | Hepatocytes - pathology | Hepatocytes - metabolism | Case-Control Studies | Proteoglycans - deficiency | Transfection | Liver Cirrhosis - metabolism | Liver Cirrhosis, Experimental - pathology | Hepatic Stellate Cells - virology | Chemical and Drug Induced Liver Injury - pathology | Liver Cirrhosis - genetics | Chemical and Drug Induced Liver Injury - etiology | Extracellular Matrix Proteins - metabolism | Hepatic Stellate Cells - pathology | Fibromodulin | Collagen Type I - metabolism | Mice, Inbred C57BL | Antioxidants - pharmacology | Hep G2 Cells | Thioacetamide | Liver - blood supply | Animals | Liver Cirrhosis - virology | Liver Cirrhosis, Experimental - chemically induced | Liver Cirrhosis, Biliary - metabolism | Immunohistochemistry | Liver diseases | Actin | Analysis | Liver | Collagen | Muscle proteins | Index Medicus | Abridged Index Medicus
Journal Article
Atherosclerosis, ISSN 0021-9150, 2015, Volume 241, Issue 2, pp. 701 - 708
Journal Article
Experimental Eye Research, ISSN 0014-4835, 08/2012, Volume 101, pp. 16 - 26
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 07/2008, Volume 10, Issue 4, pp. R79 - R79
Journal Article