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NATURE CHEMICAL BIOLOGY, ISSN 1552-4450, 01/2015, Volume 11, Issue 1, pp. 9 - 15
Mitochondria have a well-recognized role in the production of ATP and the intermediates needed for macromolecule biosynthesis, such as nucleotides.... 
CELL LUNG-CANCER | OXIDATIVE-PHOSPHORYLATION | METFORMIN | INHIBITION | GLUCOSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | TUMOR-CELLS | DEGENERATIVE DISEASES | ENERGY-METABOLISM | GLUTAMINE-METABOLISM
Journal Article
Nature, ISSN 0028-0836, 01/2009, Volume 457, Issue 7225, pp. 102 - 106
Metastatic progression depends on genetic alterations intrinsic to cancer cells as well as the inflammatory microenvironment of advanced tumours. To understand... 
LEWIS-LUNG-CARCINOMA | ANGIOGENESIS | CANCER METASTASIS | INFLAMMATION | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | HYALURONAN | PROTEOGLYCAN | TUMOR-GROWTH | INNATE IMMUNITY | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | Carcinoma, Lewis Lung - secretion | Toll-Like Receptor 2 - agonists | Lung Neoplasms - metabolism | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Culture Media, Serum-Free - metabolism | Lipopolysaccharide Receptors - metabolism | Macrophages - secretion | Lung Neoplasms - secondary | Liver Neoplasms - secondary | Interleukin-6 - metabolism | Toll-Like Receptor 6 - metabolism | Mice, Inbred C57BL | Tumor Necrosis Factor-alpha - secretion | Toll-Like Receptor 2 - metabolism | Versicans - pharmacology | Carcinoma, Lewis Lung - metabolism | Interleukin-6 - secretion | Macrophage Activation | Macrophages - metabolism | Animals | Neoplasm Metastasis - pathology | Carcinoma, Lewis Lung - pathology | Mice | Versicans - metabolism | Culture Media, Conditioned - metabolism | Care and treatment | Cell receptors | Cancer cells | Physiological aspects | Genetic aspects | Metastasis | Research | Health aspects | Risk factors | Proteins | Immunization | Adrenal glands | Rodents | Mortality | Gram-positive bacteria | Cancer | Apoptosis | Cell adhesion & migration | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2014, Volume 289, Issue 41, pp. 28660 - 28675
The cadmium-transformed human lung bronchial epithelial BEAS-2B cells exhibit a property of apoptosis resistance as compared with normal non-transformed... 
TARGET | OVEREXPRESSION | BCL-2 | OXIDATIVE STRESS | ACTIVATION | TRANSCRIPTION FACTOR NRF2 | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | AUTOPHAGY | P62 | BINDING | Neoplasm Transplantation | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | NF-E2-Related Factor 2 - antagonists & inhibitors | Superoxide Dismutase - genetics | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Epithelial Cells - drug effects | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | bcl-X Protein - genetics | Apoptosis - genetics | Lung Neoplasms - pathology | Male | Cell Transformation, Neoplastic - chemically induced | Green Fluorescent Proteins - genetics | Bronchi - drug effects | Proto-Oncogene Proteins c-bcl-2 - metabolism | Cell Transformation, Neoplastic - genetics | Bronchi - metabolism | NF-E2-Related Factor 2 - genetics | Autophagy - genetics | Bronchi - pathology | Genes, Reporter | Superoxide Dismutase - metabolism | RNA-Binding Proteins - antagonists & inhibitors | Lung Neoplasms - genetics | Cell Line | Green Fluorescent Proteins - metabolism | Cadmium - toxicity | Signal Transduction | Catalase - genetics | Epithelial Cells - pathology | Cell Transformation, Neoplastic - metabolism | Catalase - metabolism | Animals | Mice, Nude | NF-E2-Related Factor 2 - metabolism | Mice | Cell Transformation, Neoplastic - pathology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Index Medicus | Cadmium | Reactive Oxygen Species (ROS) | Nuclear Factor 2 (Erythroid-derived 2-like Factor) (NFE2L2) (Nrf2) | Autophagy | Carcinogenesis | p62 | Cell Biology | Apoptosis
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 11/2013, Volume 191, Issue 10, pp. 5182 - 5195
The Wnt signaling network, an ancient signaling system governing ontogeny and homeostatic processes, has recently been identified to exert immunoregulatory... 
SIGNAL-TRANSDUCTION | TARGET | STEM-CELLS | ACTIVATION | PATHWAY | GLYCOGEN-SYNTHASE KINASE-3 | GENE-EXPRESSION | C-MYC | IMMUNOLOGY | BETA-CATENIN | INFLAMMATORY RESPONSE | Lung - microbiology | Tuberculosis, Pulmonary - metabolism | Cell Proliferation | Granuloma - metabolism | Toll-Like Receptor 2 - genetics | Tumor Necrosis Factor-alpha - genetics | Wnt Signaling Pathway - immunology | Mycobacterium tuberculosis - immunology | Arginase - biosynthesis | NF-kappa B - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Proto-Oncogene Proteins - biosynthesis | Wnt Proteins - metabolism | RNA, Messenger - biosynthesis | Wnt Proteins - genetics | Bone Marrow Cells - immunology | Lung - metabolism | Macrophages - immunology | Proto-Oncogene Proteins - metabolism | Lung - pathology | Mice, Inbred C57BL | Myeloid Differentiation Factor 88 - genetics | Pertussis Toxin - metabolism | Proto-Oncogene Proteins - genetics | Toll-Like Receptor 2 - metabolism | Proto-Oncogene Proteins c-myc - metabolism | beta Catenin - metabolism | Mice, Knockout | Cell Differentiation - immunology | Macrophages - metabolism | Tuberculosis, Pulmonary - immunology | Animals | Ki-67 Antigen - genetics | Ki-67 Antigen - biosynthesis | Mice | Wnt Proteins - biosynthesis | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | Culture Media, Conditioned - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2014, Volume 289, Issue 30, pp. 20813 - 20823
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2015, Volume 290, Issue 17, pp. 11177 - 11187
Cachexia occurs in patients with advanced cancers. Despite the adverse clinical impact of cancer-induced muscle wasting, pathways causing cachexia are... 
TRANSCRIPTION FACTORS | SKELETAL-MUSCLE | PROTEIN-DEGRADATION | MYOSTATIN GENE | PATHWAY | ATROPHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | KAPPA-B | UP-REGULATION | EXPRESSION | CATABOLIC CONDITIONS | Colonic Neoplasms - genetics | SKP Cullin F-Box Protein Ligases - genetics | Caspase 3 - metabolism | Ubiquitin - metabolism | Muscle, Skeletal - metabolism | Colonic Neoplasms - metabolism | Tripartite Motif Proteins | Proteolysis | Caspase 3 - genetics | Muscle Proteins - metabolism | Myostatin - genetics | CCAAT-Enhancer-Binding Protein-delta - metabolism | Myostatin - metabolism | Cachexia - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Cachexia - genetics | Ubiquitin-Protein Ligases - metabolism | SKP Cullin F-Box Protein Ligases - metabolism | Ubiquitin - genetics | Carcinoma, Lewis Lung - metabolism | Mice, Knockout | Muscle Proteins - genetics | Proteasome Endopeptidase Complex - genetics | Animals | Carcinoma, Lewis Lung - genetics | CCAAT-Enhancer-Binding Protein-delta - genetics | Cachexia - pathology | Colonic Neoplasms - pathology | Carcinoma, Lewis Lung - pathology | Cell Line, Tumor | Mice | Muscle, Skeletal - pathology | Proteasome Endopeptidase Complex - metabolism | Ubiquitin-Protein Ligases - genetics | Index Medicus | Ubiquitin | Signal Transduction | STAT3 | Caspase | Cancer Cachexia | Cancer Biology | Muscle Atrophy | Caspase-3
Journal Article
International Journal of Cancer, ISSN 0020-7136, 04/2012, Volume 130, Issue 7, pp. 1695 - 1705
Lung cancer is one of the most commonly occurring malignancies. It has been reported that mammalian target of rapamycin (mTOR) is phosphorylated in lung cancer... 
lung cancer | signaling | PI3K | fisetin | mTOR | Akt | flavonoid | dual inhibition | ACTIVATED PROTEIN-KINASE | CYCLE ARREST | KAPPA-B | TUBEROUS-SCLEROSIS | ONCOLOGY | PROSTATE-CANCER | GROWTH | TUMOR-SUPPRESSOR | MTOR INHIBITORS | PI3K/AKT/MTOR PATHWAY | EXPRESSION | Protein Kinases - metabolism | Lung Neoplasms - drug therapy | TOR Serine-Threonine Kinases - metabolism | Tumor Suppressor Proteins - antagonists & inhibitors | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Lung Neoplasms - metabolism | Eukaryotic Initiation Factor-4E - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | TOR Serine-Threonine Kinases - antagonists & inhibitors | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Regulatory-Associated Protein of mTOR | Flavonoids - pharmacology | Phosphorylation - drug effects | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Rapamycin-Insensitive Companion of mTOR Protein | Tumor Suppressor Proteins - metabolism | Lung Neoplasms - enzymology | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Ribosomal Protein S6 Kinases, 70-kDa - antagonists & inhibitors | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | MTOR Associated Protein, LST8 Homolog | Carcinoma, Non-Small-Cell Lung - metabolism | Carrier Proteins - antagonists & inhibitors | PTEN Phosphohydrolase - metabolism | Eukaryotic Initiation Factor-4E - antagonists & inhibitors | Carrier Proteins - metabolism | Cell Line, Tumor | Stem Cells - drug effects | Cell Proliferation - drug effects | Carcinoma, Non-Small-Cell Lung - drug therapy | Adaptor Proteins, Signal Transducing - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Kinases | Phosphorylation | Lung cancer | Index Medicus
Journal Article
Journal of Thoracic Oncology, ISSN 1556-0864, 04/2010, Volume 5, Issue 4, pp. 436 - 441
The dichotomization of non-small cell carcinoma (NSCLC) subtype into squamous (SQCC) and adenocarcinoma (ADC) has become important in recent years and is... 
Immunohistochemistry | Adenocarcinoma | Squamous cell carcinoma | Lung | Cytology | Non-small cell carcinoma | EGFR | Cancer | Transbronchial needle aspirate | CARBOPLATIN | MARKER | PHASE-II | COMBINATION | PACLITAXEL | PULMONARY ADENOCARCINOMAS | FINE-NEEDLE-ASPIRATION | ONCOLOGY | RESPIRATORY SYSTEM | TRANSCRIPTION FACTOR-I | EXPRESSION | Receptor, Epidermal Growth Factor - genetics | Thyroid Nuclear Factor 1 | Prognosis | Prospective Studies | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Humans | Lung Neoplasms - metabolism | Carcinoma, Non-Small-Cell Lung - diagnosis | Cytodiagnosis - methods | Immunoenzyme Techniques | Carcinoma, Squamous Cell - diagnosis | Small Cell Lung Carcinoma - metabolism | Adenocarcinoma - metabolism | DNA Mutational Analysis | Sensitivity and Specificity | Mucins - metabolism | Polymerase Chain Reaction | Biomarkers, Tumor - metabolism | Adenocarcinoma - genetics | Retrospective Studies | Lung Neoplasms - genetics | Diagnosis, Differential | Tumor Suppressor Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Genotype | Nuclear Proteins - metabolism | Survival Rate | Small Cell Lung Carcinoma - genetics | Mutation - genetics | Transcription Factors - metabolism | Biopsy, Fine-Needle | Adenocarcinoma - diagnosis | Keratin-5 - metabolism | Small Cell Lung Carcinoma - diagnosis | Trans-Activators - metabolism | Biomarkers, Tumor - genetics | Keratin-6 - metabolism | DNA, Neoplasm - genetics | Lung Neoplasms - diagnosis | Neoplasm Staging | Index Medicus
Journal Article