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1. Full Text Statin-Associated Side Effects
by Thompson, Paul D., MD and Panza, Gregory, MS and Zaleski, Amanda, MS and Taylor, Beth, PhD
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2016, Volume 67, Issue 20, pp. 2395 - 2410
Abstract Hydroxy-methyl-glutaryl-coenzyme A (HMG-CoA) reductase inhibitors or statins are well tolerated, but associated with various statin-associated... 
Cardiovascular | Internal Medicine | rhabdomyolysis | skeletal muscle | interstitial lung disease | myopathy | C-REACTIVE PROTEIN | RANDOMIZED CONTROLLED-TRIALS | CARDIAC & CARDIOVASCULAR SYSTEMS | FORCE 2014 UPDATE | PRIMARY-PREVENTION TRIAL | INDUCED MUSCLE TOXICITY | INTERSTITIAL LUNG-DISEASE | CENTRAL-NERVOUS-SYSTEM | OF-THE-LITERATURE | CORONARY-HEART-DISEASE | TYPE-2 DIABETES-MELLITUS | Liver Function Tests | Sleep Wake Disorders - chemically induced | Hydroxymethylglutaryl-CoA Reductase Inhibitors - immunology | Myalgia - chemically induced | Humans | Hyperlipidemias - drug therapy | Risk Factors | Lung Diseases, Interstitial - chemically induced | Tendon Injuries - chemically induced | Necrosis - chemically induced | Brain - drug effects | Rupture - chemically induced | Hydroxymethylglutaryl-CoA Reductase Inhibitors - adverse effects | Stroke - chemically induced | Cognition Disorders - etiology | Depression - chemically induced | Rhabdomyolysis - chemically induced | Antibodies - blood | Diabetes Mellitus - chemically induced | Acute Kidney Injury - chemically induced | Muscular Diseases - chemically induced | Testosterone - blood | Autoimmune Diseases - chemically induced | Hyperlipidemias - complications | Viral antibodies | Complications and side effects | Enzymes | Thiols | Antibodies | Diabetes | Cardiology | Statins | Cardiovascular agents | Creatine kinase | Muscles | Creatine | Respiratory tract diseases | Studies | Musculoskeletal system | Genotype & phenotype | Mortality | Clinical trials | Lipids | Clinical medicine
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2. Full Text TRPV4 inhibition counteracts edema and inflammation and improves pulmonary function and oxygen saturation in chemically induced acute lung injury
by Balakrishna, Shrilatha and Song, Weifeng and Achanta, Satyanarayana and Doran, Stephen F and Liu, Boyi and Kaelberer, Melanie M and Yu, Zhihong and Sui, Aiwei and Cheung, Mui and Leishman, Emma and Eidam, Hilary S and Ye, Guosen and Willette, Robert N and Thorneloe, Kevin S and Bradshaw, Heather B and Matalon, Sadis and Jordt, Sven-Eric
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 07/2014, Volume 307, Issue 2, pp. L158 - L172
The treatment of acute lung injury caused by exposure to reactive chemicals remains challenging because of the lack of mechanism-based therapeutic approaches.... 
Acute lung injury | Chlorine | TRPV4 | HYPERREACTIVITY | ACTIVATION | PHYSIOLOGY | BARRIER | MECHANISMS | IDENTIFICATION | acute lung injury | FAILURE | chlorine | RESPIRATORY-DISTRESS-SYNDROME | RESPONSES | RNA-SEQ | RESPIRATORY SYSTEM | CHANNELS | Anti-Inflammatory Agents - pharmacology | Humans | TRPV Cation Channels - agonists | Rats | Hydrochloric Acid - toxicity | Male | Chlorine - toxicity | TRPV Cation Channels - deficiency | Animals | Pneumonia - drug therapy | Acute Lung Injury - chemically induced | TRPV Cation Channels - antagonists & inhibitors | HEK293 Cells | Acute Lung Injury - drug therapy | Mice | Bronchoalveolar Lavage Fluid - chemistry | Physiological aspects | Care and treatment | Inflammation | Properties | Lung diseases | Membrane proteins
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3. Full Text Rutin decreases lipopolysaccharide-induced acute lung injury via inhibition of oxidative stress and the MAPK–NF-κB pathway
by Yeh, Chung-Hsin and Yang, Jiann-Jou and Yang, Ming-Ling and Li, Yi-Ching and Kuan, Yu-Hsiang
Free Radical Biology and Medicine, ISSN 0891-5849, 04/2014, Volume 69, pp. 249 - 257
Acute lung injury (ALI) is a serious disease with unacceptably high mortality and morbidity rates. Up to now, no effective therapeutic strategy for ALI has... 
Acute lung injury | NF-κB | Free radicals | CAT | Lipopolysaccharide | SOD | HO-1 | Rutin | MAPK | GPx | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RATS | NEUTROPHILS | MECHANISMS | THERAPEUTIC APPLICATIONS | QUERCETIN | IN-VITRO | NF-kappa B | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | MICE | EXPRESSION | Mitogen-Activated Protein Kinase Kinases - genetics | Heme Oxygenase-1 - biosynthesis | Lipopolysaccharides - toxicity | Rutin - toxicity | Superoxide Dismutase - biosynthesis | Humans | Acute Lung Injury - genetics | Glutathione Peroxidase - biosynthesis | Gene Expression Regulation - drug effects | Animals | Acute Lung Injury - chemically induced | NF-kappa B - genetics | Signal Transduction - drug effects | Rutin - administration & dosage | Acute Lung Injury - drug therapy | Mice | Oxidative Stress - drug effects | Superoxide Dismutase-1 | Catalase - biosynthesis
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4. Full Text Antifibrotic and Anti-inflammatory Activity of the Tyrosine Kinase Inhibitor Nintedanib in Experimental Models of Lung Fibrosis
by Wollin, L and Maillet, I and Quesniaux, V and Holweg, A and Ryffel, B
JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS, ISSN 0022-3565, 05/2014, Volume 349, Issue 2, pp. 209 - 220
The tyrosine kinase inhibitor nintedanib (BIBF 1120) is in clinical development for the treatment of idiopathic pulmonary fibrosis. To explore its mode of... 
MESSENGER-RNA EXPRESSION | IMATINIB | DISEASE | TISSUE INHIBITOR | BLEOMYCIN | PHARMACOLOGY & PHARMACY | MICE | QUALITY-OF-LIFE | IDIOPATHIC PULMONARY-FIBROSIS | FIBROBLASTS | ENDOTHELIAL GROWTH-FACTOR | Fibrosis - drug therapy | Lung Injury - pathology | Phosphorylation | Protein-Tyrosine Kinases - metabolism | Humans | Pneumonia - pathology | Bleomycin | Pneumonia - chemically induced | Silicon Dioxide | Anti-Inflammatory Agents - therapeutic use | Indoles - pharmacology | Cell Differentiation | Lung - metabolism | Granuloma - chemically induced | Lung Injury - drug therapy | Fibroblasts - metabolism | Myofibroblasts - pathology | Lung - pathology | Fibrosis - chemically induced | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Fibroblasts - pathology | Myofibroblasts - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Granuloma - pathology | Animals | Pneumonia - drug therapy | Fibroblasts - drug effects | Lung - drug effects | Granuloma - drug therapy | Indoles - therapeutic use | Cell Proliferation - drug effects | Fibrosis - pathology | Lung Injury - chemically induced | Protein-Tyrosine Kinases - antagonists & inhibitors
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5. Full Text The monocrotaline model of pulmonary hypertension in perspective
by Gomez-Arroyo, Jose G and Farkas, Laszlo and Alhussaini, Aysar A and Farkas, Daniela and Kraskauskas, Donatas and Voelkel, Norbert F and Bogaard, Harm J
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 02/2012, Volume 302, Issue 4, pp. L363 - L369
Gomez-Arroyo JG, Farkas L, Alhussaini AA, Farkas D, Kraskauskas D, Voelkel NF, Bogaard HJ. The monocrotaline model of pulmonary hypertension in perspective. Am... 
Myocarditis | Acute lung injury | Hepatic venocclusive disease | Monocrotaline | Pulmonary hypertension | CROTALARIA SPECTABILIS | PHYSIOLOGY | pulmonary hypertension | myocarditis | PYRROLIZIDINE ALKALOIDS | monocrotaline | HEART-FAILURE | acute lung injury | ARTERIAL ENDOTHELIAL-CELLS | RESPIRATORY-DISTRESS-SYNDROME | TREATED RATS | CYTOCHROME-P450 3A | HEPATIC VENOOCCLUSIVE DISEASE | RESPIRATORY SYSTEM | hepatic venocclusive disease | RIGHT-VENTRICULAR HYPERTROPHY | VASCULAR CHANGES | Acute Lung Injury - physiopathology | Lung - pathology | Myocarditis - chemically induced | Humans | Hypertension, Pulmonary - physiopathology | Chemical and Drug Induced Liver Injury - physiopathology | Hypertension, Pulmonary - chemically induced | Animals | Acute Lung Injury - chemically induced | Myocarditis - pathology | Chemical and Drug Induced Liver Injury - pathology | Lung - blood supply | Hypertension, Pulmonary - pathology | Myocarditis - physiopathology | Disease Models, Animal | Biomechanics | Physiological aspects | Physiology, Pathological | Development and progression | Research
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6. Full Text Human Umbilical Cord Mesenchymal Stem Cells Reduce Fibrosis of Bleomycin-Induced Lung Injury
by Moodley, Yuben and Atienza, Daniel and Manuelpillai, Ursula and Samuel, Chrishan S and Tchongue, Jorge and Ilancheran, Sivakami and Boyd, Richard and Trounson, Alan
American Journal of Pathology, The, ISSN 0002-9440, 2009, Volume 175, Issue 1, pp. 303 - 313
Acute respiratory distress syndrome is characterized by loss of lung tissue as a result of inflammation and fibrosis. Augmenting tissue repair by the use of... 
Pathology | MATRIX METALLOPROTEINASES | GROWTH-FACTOR-BETA | EPITHELIAL-CELLS | INDUCED PULMONARY-FIBROSIS | BONE-MARROW | TISSUE INHIBITORS | TIME-COURSE | PATHOLOGY | EXPRESSION | RELAXIN | RESPIRATORY-DISTRESS-SYNDROME | Immunohistochemistry | Inflammation - chemically induced | Respiratory Distress Syndrome, Adult - pathology | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Pulmonary Fibrosis - therapy | Antibiotics, Antineoplastic - toxicity | Humans | RNA, Messenger - analysis | Macrophage Migration-Inhibitory Factors - metabolism | Lung Injury - therapy | Mesenchymal Stromal Cells | Polymerase Chain Reaction | Bleomycin - toxicity | Disease Models, Animal | Respiratory Distress Syndrome, Adult - therapy | Enzyme-Linked Immunosorbent Assay | Matrix Metalloproteinase 2 - metabolism | Pulmonary Fibrosis - pathology | Mice, SCID | Blotting, Western | Tissue Inhibitor of Metalloproteinases - metabolism | Cord Blood Stem Cell Transplantation | Collagen - metabolism | Inflammation - therapy | Animals | Pulmonary Fibrosis - chemically induced | Mice | Lung Injury - chemically induced | Fetal Blood | Transforming Growth Factor beta - metabolism | Mesenchymal Stem Cell Transplantation | Index Medicus | Abridged Index Medicus | Regular
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7. Full Text Protective effects of polydatin on lipopolysaccharide-induced acute lung injury through TLR4-MyD88-NF-κB pathway
by Jiang, Qi and Yi, Min and Guo, Qianqian and Wang, Huimin and Wang, Ciman and Meng, Shanshan and Liu, Chao and Fu, Yeliu and Ji, Hui and Chen, Tong
International Immunopharmacology, ISSN 1567-5769, 12/2015, Volume 29, Issue 2, pp. 370 - 376
The purpose of this study was to investigate the protective effect of PD against lipopolysaccharide (LPS)-induced acute lung injury (ALI) and explore its... 
TLR4-MyD88-NF-κB pathway | Cytokines | BEAS-2B cells | LPS | ALI | Polydatin | VITRO | RAW 264.7 CELLS | SALIDROSIDE | PLATYCODIN D | IMMUNOLOGY | INTERLEUKIN-8 | TLR4-MyD88-NF-kappa B pathway | EPITHELIAL-CELLS | INFLAMMATION | IN-VIVO | DISEASE | PHARMACOLOGY & PHARMACY | MICE | Cell Line | Stilbenes - therapeutic use | Toll-Like Receptor 4 - drug effects | Male | Lipopolysaccharides | Myeloid Differentiation Factor 88 - drug effects | Animals | Acute Lung Injury - chemically induced | Signal Transduction - drug effects | Acute Lung Injury - prevention & control | Glucosides - therapeutic use | Bronchoalveolar Lavage Fluid - cytology | Leukocyte Count | Mice | Mice, Inbred BALB C | Cytokines - biosynthesis | NF-kappa B - drug effects
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8. Full Text Selective visualization of endogenous hypochlorous acid in zebrafish during lipopolysaccharide-induced acute liver injury using a polymer micelles-based ratiometric fluorescent probe
by Zhang, Peisheng and Wang, Hong and Hong, Yongxiang and Yu, Maolin and Zeng, Rongjin and Long, Yunfei and Chen, Jian
Biosensors and Bioelectronics, ISSN 0956-5663, 01/2018, Volume 99, pp. 318 - 324
The development of effective method for monitoring of endogenous hypochlorous acid (HClO) in vivo is of great significance for early diagnosis of... 
Ratiometric fluorescence | Polymer micelles | Zebrafish | HClO | ELECTROCHEMISTRY | CHEMISTRY, ANALYTICAL | AQUEOUS-MEDIA | MITOCHONDRIA | REACTIVITY | SENSOR | NANOSCIENCE & NANOTECHNOLOGY | MECHANISMS | HYDROGEN-SULFIDE | SENSITIVE DETECTION | NANOPARTICLES | BIOPHYSICS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | IN-VIVO | LIVING CELLS | Fluorescent Dyes - chemistry | Limit of Detection | Liver - pathology | Lipopolysaccharides - toxicity | Hypochlorous Acid - isolation & purification | Acute Lung Injury - diagnosis | Animals | Acute Lung Injury - chemically induced | Liver - diagnostic imaging | Micelles | Biosensing Techniques | Fluorescence Resonance Energy Transfer | Polymers - chemistry | Hypochlorous Acid - chemistry | Polymer industry | Polymers | Mitogens | Liver | Investigations | Usage | Fluorescence
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9. Full Text Lineage-negative progenitors mobilize to regenerate lung epithelium after major injury
by Vaughan, Andrew E and Brumwell, Alexis N and Xi, Ying and Gotts, Jeffrey E and Brownfield, Doug G and Treutlein, Barbara and Tan, Kevin and Tan, Victor and Liu, Feng Chun and Looney, Mark R and Matthay, Michael A and Rock, Jason R and Chapman, Harold A
Nature, ISSN 0028-0836, 01/2015, Volume 517, Issue 7536, pp. 621 - 625
Broadly, tissue regeneration is achieved in two ways: by proliferation of common differentiated cells and/or by deployment of specialized stem/progenitor... 
ALVEOLAR | MAINTENANCE | STEM-CELLS | IN-VITRO | REPAIR | AIRWAY | MULTIDISCIPLINARY SCIENCES | MOUSE | BIOLOGY | NOTCH | TRANSPLANTATION | Lung Injury - pathology | Cell Proliferation | Epithelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Male | Stem Cells - cytology | Lung - cytology | Phosphoproteins - metabolism | Stem Cells - metabolism | Stem Cell Transplantation | Cysts - metabolism | Lung Injury - virology | Bleomycin | Trans-Activators - genetics | Female | Epithelial Cells - cytology | Re-Epithelialization | Lung - pathology | Signal Transduction | Cell Separation | Orthomyxoviridae Infections - pathology | Epithelial Cells - pathology | Phosphoproteins - genetics | Lung - physiology | Cysts - pathology | Cell Lineage | Animals | Keratin-5 - metabolism | Trans-Activators - metabolism | Mice | Lung Injury - chemically induced | Orthomyxoviridae Infections - virology | Physiological aspects | Genetic aspects | Regeneration (Biology) | Research | Epithelium | Lung diseases
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10. Full Text Allogeneic Human Mesenchymal Stem Cells for Treatment of E. coli Endotoxin-Induced Acute Lung Injury in the Ex vivo Perfused Human Lung
by Jae W. Lee and Xiaohui Fang and Naveen Gupta and Vladimir Serikov and Michael A. Matthay and John A. Clements
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2009, Volume 106, Issue 38, pp. 16357 - 16362
Recent studies have suggested that bone marrow-derived multipotent mesenchymal stem cells (MSCs) may have therapeutic applications in multiple clinical... 
Pulmonary alveoli | Lungs | Epithelial cells | Medical treatment | Endotoxins | Small interfering RNA | Pulmonary edema | Average fixed cost | Mesenchymal stem cells | Lung injury | Acute respiratory distress syndrome | Keratinocyte growth factor | Alveolar fluid clearance | pulmonary edema | keratinocyte growth factor | MULTIDISCIPLINARY SCIENCES | ALPHA | SODIUM-CHANNEL | INTRATRACHEAL INSTILLATION | REPAIR | ENGRAFTMENT | alveolar fluid clearance | KERATINOCYTE | EPITHELIAL FLUID TRANSPORT | EDEMA | DECREASES EXPRESSION | acute respiratory distress syndrome | STROMAL CELLS | Fibroblast Growth Factor 7 - pharmacology | Lung Injury - surgery | Humans | Middle Aged | Culture Media, Conditioned - pharmacology | Male | Transplantation, Homologous | Mesenchymal Stromal Cells - cytology | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - drug effects | Pulmonary Edema - prevention & control | Adult | Female | Pulmonary Edema - pathology | Leukocyte Count | Lung - metabolism | Pulmonary Edema - metabolism | Neutrophils - pathology | Acute Disease | Lung - pathology | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Sodium Channel Blockers - pharmacology | Recombinant Proteins - pharmacology | Escherichia coli - chemistry | Fibroblast Growth Factor 7 - genetics | Mesenchymal Stem Cell Transplantation - methods | Body Fluids - metabolism | Perfusion | Amiloride - pharmacology | In Vitro Techniques | Lung - surgery | Lung Injury - chemically induced | Care and treatment | Escherichia coli infections | Research | Lung diseases | Stem cells | Index Medicus | Biological Sciences
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11. Full Text Molecular hydrogen ameliorates lipopolysaccharide-induced acute lung injury in mice through reducing inflammation and apoptosis
by Xie, Keliang and Yu, Yonghao and Huang, Yi and Zheng, Lina and Li, Jipeng and Chen, Hongguang and Han, Huanzhi and Hou, Lichao and Gong, Gu and Wang, Guolin
Shock, ISSN 1073-2322, 05/2012, Volume 37, Issue 5, pp. 548 - 555
Acute lung injury (ALI) is still a leading cause of morbidity and mortality in critically ill patients. Recently, our and other studies have found that... 
Acute lung injury | Inflammation | Hydrogen gas | Chemokines | Apoptosis | SURGERY | OXIDATIVE STRESS | ACTIVATION | INHALATION | apoptosis | FACTOR-KAPPA-B | chemokines | RAT MODEL | RESPIRATORY-DISTRESS-SYNDROME | hydrogen gas | inflammation | SALINE PROTECTS | GAS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ISCHEMIA/REPERFUSION | EPIDEMIOLOGY | CRITICAL CARE MEDICINE | Inflammation - chemically induced | Lung - pathology | Hydrogen - pharmacology | Lipopolysaccharides - toxicity | Cytokines - metabolism | Apoptosis - drug effects | Male | NF-kappa B - metabolism | Inflammation - metabolism | Animals | Acute Lung Injury - chemically induced | Inflammation - drug therapy | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Lung - metabolism | Mice
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