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Cancer Treatment Reviews, ISSN 0305-7372, 2016, Volume 44, pp. 51 - 60
Highlights • Role of immune checkpoint inhibition in the management of key malignancies. • Description of rates of immune-related adverse events and timing of... 
Hematology, Oncology and Palliative Medicine | Pembrolizumab | Immune-related adverse events | Renal cancer | Ipilimumab | Nivolumab | Lung cancer | Melanoma | Immune-checkpoint inhibitors | OPEN-LABEL | SINGLE-ARM | ANTI-CTLA-4 ANTIBODY | METASTATIC MELANOMA | ONCOLOGY | ADVANCED MELANOMA | LONG-TERM SAFETY | MALIGNANT MESOTHELIOMA | ENTERIC NEUROPATHY | ADVERSE EVENTS | IPILIMUMAB RETREATMENT | Lung Neoplasms - drug therapy | Skin Neoplasms - drug therapy | Humans | Immunosuppressive Agents - therapeutic use | Antibodies, Monoclonal - adverse effects | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Adrenal Cortex Hormones - therapeutic use | Diarrhea - chemically induced | Infliximab - therapeutic use | Drug Eruptions - drug therapy | Antineoplastic Agents - adverse effects | Colitis - chemically induced | Antilymphocyte Serum - therapeutic use | Colitis - drug therapy | Carcinoma, Renal Cell - drug therapy | Chemical and Drug Induced Liver Injury - etiology | Pituitary Diseases - chemically induced | Pituitary Diseases - drug therapy | Antibodies, Monoclonal, Humanized - adverse effects | Drug Eruptions - etiology | Neoplasms - drug therapy | Cyclosporine - therapeutic use | Tacrolimus - therapeutic use | Mycophenolic Acid - analogs & derivatives | Mycophenolic Acid - therapeutic use | Melanoma - drug therapy | Chemical and Drug Induced Liver Injury - drug therapy | Diarrhea - drug therapy | CTLA-4 Antigen - antagonists & inhibitors | Kidney Neoplasms - drug therapy | Thyroiditis - drug therapy | Thyroiditis - chemically induced | Index Medicus
Journal Article
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 05/2011, Volume 6, Issue 5, pp. 966 - 973
Journal Article
JAMA, ISSN 0098-7484, 10/2011, Volume 306, Issue 14, pp. 1574 - 1581
Journal Article
JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS, ISSN 0022-3565, 05/2014, Volume 349, Issue 2, pp. 209 - 220
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e56407
Background: Nuclear factor-kappa B (NF-kappa B) is a central transcriptional factor and a pleiotropic regulator of many genes involved in acute lung injury.... 
INDUCED MITOGENIC FACTOR | IN-VITRO | ACTIVATION | INHIBITION | PI-3K/AKT-NF-KAPPA-B SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | ALVEOLAR FLUID CLEARANCE | EPITHELIAL SODIUM-CHANNEL | PANICULATA | EXPRESSION | RESPIRATORY-DISTRESS-SYNDROME | Bronchoalveolar Lavage Fluid | Protein Binding - genetics | Cell Count | Pneumonia - pathology | Cell Survival - genetics | Acute Lung Injury - genetics | Male | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Protective Agents - therapeutic use | Vascular Endothelial Growth Factor A - genetics | Lipopolysaccharides | Alveolar Epithelial Cells - pathology | Protective Agents - pharmacology | Protein Binding - drug effects | Pulmonary Edema - complications | Pulmonary Edema - pathology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - drug therapy | Acute Lung Injury - metabolism | Alveolar Epithelial Cells - ultrastructure | Pneumonia - genetics | Diterpenes - pharmacology | Pulmonary Edema - genetics | Cell Survival - drug effects | Diterpenes - therapeutic use | Cytokines - metabolism | DNA - metabolism | Down-Regulation - drug effects | Acute Lung Injury - pathology | Pulmonary Edema - drug therapy | Gene Expression Regulation - drug effects | Animals | Pneumonia - drug therapy | Transcription Factor RelA - metabolism | Pneumonia - complications | Mice | Mice, Inbred BALB C | Vascular Cell Adhesion Molecule-1 - metabolism | Peroxidase - metabolism | Vascular cell adhesion molecule 1 | Phosphorylation | Epithelial cells | Kinases | Macrophages | Inactivation | Cell adhesion & migration | Interleukin 6 | Ultrastructure | Rodents | Inhibition | Alveoli | Vascular endothelial growth factor | Drug dosages | Deoxyribonucleic acid--DNA | Medicinal plants | Pathogens | Edema | NF-κB protein | Deactivation | Therapeutic applications | Traditional Chinese medicine | Leukocytes (neutrophilic) | Inflammation | Tumor necrosis factor-α | Gene expression | Medicine | Hospitals | Lungs | Respiratory distress syndrome | Rheumatoid arthritis | Hypoxia | In vivo methods and tests | Laboratory animals | In vitro methods and tests | Binding sites | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2012, Volume 122, Issue 7, pp. 2661 - 2671
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1379 - 1387
Membrane sphingolipids are metabolized to sphingosine-1-phosphate (S1P), a bioactive lipid mediator that regulates many processes in vertebrate development,... 
MEDICINE, RESEARCH & EXPERIMENTAL | SPHINGOSINE 1-PHOSPHATE RECEPTOR | ENDOTHELIAL-CELLS | KINASE 1 | IN-VIVO | TRANSPORTER SPNS2 | VASCULAR LEAK | MICE | PERSISTENT STAT3 ACTIVATION | LYMPHOCYTE EGRESS | FINGOLIMOD FTY720 | Receptors, Lysosphingolipid - antagonists & inhibitors | Humans | Cell Physiological Phenomena - physiology | Lysophospholipids - agonists | Neoplasms - blood | Propylene Glycols - therapeutic use | Sphingosine - agonists | Acute Lung Injury - drug therapy | Hematopoietic Stem Cell Mobilization | Autoimmune Diseases - metabolism | Autoimmune Diseases - drug therapy | Disease Models, Animal | Influenza, Human - metabolism | Lysophospholipids - antagonists & inhibitors | Cardiovascular Diseases - metabolism | Sphingosine - antagonists & inhibitors | Sphingosine - physiology | Fingolimod Hydrochloride | Receptors, Lysosphingolipid - physiology | Sphingolipids - metabolism | Neovascularization, Physiologic - physiology | Sphingosine - analogs & derivatives | Animals | Neurogenesis - physiology | Membrane Lipids - metabolism | Sphingosine - therapeutic use | Mice | Anemia, Sickle Cell - blood | Lysophospholipids - physiology | Multiple Sclerosis - drug therapy | Cellular signal transduction | Genetic aspects | Disease susceptibility | Health aspects | Sphingosine | Plasma | Pathogenesis | Lipids | Kinases | Metabolism | Phosphatase | Endothelium | Inflammatory bowel disease | Vertebrates | Metabolites | Rodents | Atherosclerosis | Ligands | Crystal structure | Review
Journal Article