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Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 104 - 107
Injury causes a systemic inflammatory response syndrome (SIRS) that is clinically much like sepsis. Microbial pathogen-associated molecular patterns (PAMPs)... 
SHOCK | SIRS | OPERATED CALCIUM INFLUX | HUMAN NEUTROPHILS | PEPTIDES | MODELS | MOBILIZATION | CHEMOTAXIS | MULTIDISCIPLINARY SCIENCES | RECEPTORS | TRAUMA | Liver - pathology | Phosphorylation | Humans | Male | Mitochondria - immunology | Liver - injuries | N-Formylmethionine Leucyl-Phenylalanine - metabolism | Liver - immunology | Systemic Inflammatory Response Syndrome - blood | Muscle, Skeletal - immunology | Sepsis - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Neutrophils - metabolism | Calcium Signaling | Femur - injuries | Sepsis - immunology | CpG Islands - immunology | Neutrophils - enzymology | Wounds and Injuries - pathology | Fractures, Bone - immunology | Wounds and Injuries - blood | Cells, Cultured | Neutrophils - immunology | Wounds and Injuries - complications | Rats | Wounds and Injuries - immunology | Acute Lung Injury - pathology | DNA, Mitochondrial - blood | Rats, Sprague-Dawley | DNA, Mitochondrial - immunology | Immunity, Innate - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Mitochondria - secretion | Acute Lung Injury - immunology | Fractures, Bone - pathology | Systemic Inflammatory Response Syndrome - complications | Muscle, Skeletal - pathology | Toll-Like Receptor 9 - metabolism | Sepsis - microbiology | Mitogen-Activated Protein Kinases - metabolism | N-Formylmethionine Leucyl-Phenylalanine - immunology | Infection | Peptides | Physiological aspects | Mitochondrial DNA | Inflammation | Research | Risk factors | Fractures | Bacterial proteins | Rheumatoid arthritis | Trauma | Deoxyribonucleic acid--DNA
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2009, Volume 119, Issue 10, pp. 2898 - 2913
Journal Article
by Wang, J and Li, FQ and Wei, HM and Lian, ZX and Sun, R and Tian, ZG
JOURNAL OF EXPERIMENTAL MEDICINE, ISSN 0022-1007, 11/2014, Volume 211, Issue 12, pp. 2397 - 2410
Influenza in humans is often accompanied by gastroenteritis-like symptoms such as diarrhea, but the underlying mechanism is not yet understood. We explored the... 
MEDICINE, RESEARCH & EXPERIMENTAL | TRACT | HOMEOSTASIS | MUCOSAL IMMUNOLOGICAL SYSTEM | RECOGNITION | GUT | ESCHERICHIA-COLI INFECTION | IMMUNOLOGY | EXPRESSION | T-CELLS | BOWEL-DISEASE | LYMPHOCYTES | Influenza A Virus, H1N1 Subtype - immunology | Intestines - drug effects | Lung - microbiology | Epithelial Cells - metabolism | Receptors, CCR - metabolism | Humans | Influenza A Virus, H1N1 Subtype - physiology | Interleukin-17 - immunology | Male | Interleukin-15 - immunology | Intestines - immunology | Host-Pathogen Interactions - immunology | Orthomyxoviridae Infections - genetics | CD4-Positive T-Lymphocytes - immunology | Antibodies, Neutralizing - immunology | Flow Cytometry | Th17 Cells - metabolism | Chemokines, CC - immunology | Microbiota - immunology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Interleukin-17 - genetics | Antibodies, Neutralizing - pharmacology | Inflammation - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Interleukin-17 - metabolism | Animals | Intestines - microbiology | Receptors, CCR - immunology | Epithelial Cells - immunology | Interleukin-15 - metabolism | Inflammation - genetics | Th17 Cells - immunology | Orthomyxoviridae Infections - virology | Lung - immunology | Orthomyxoviridae Infections - immunology | Chemokines, CC - metabolism | 305
Journal Article
Journal Article
International Immunopharmacology, ISSN 1567-5769, 05/2014, Volume 20, Issue 1, pp. 24 - 32
Journal Article
Mucosal Immunology, ISSN 1933-0219, 05/2010, Volume 3, Issue 3, pp. 291 - 300
Severe respiratory syncytial virus (RSV)-induced bronchiolitis has been associated with a mixed "Th1" and "Th2" cytokine storm. We hypothesized that... 
SYNCYTIAL VIRUS BRONCHIOLITIS | RECEPTOR-GAMMA | MURINE MACROPHAGES | DISEASE | INNATE IMMUNE-RESPONSE | GENE-EXPRESSION | ALVEOLAR MACROPHAGES | PPAR-GAMMA | INFECTION | IMMUNOLOGY | COTTON RAT | Interferon-beta - immunology | Humans | Respiratory Syncytial Viruses - immunology | Respiratory Syncytial Virus Infections - metabolism | Respiratory Syncytial Virus Infections - immunology | Cell Differentiation - genetics | Signal Transduction - immunology | Lung Injury - virology | Lung Injury - metabolism | Interferon-beta - genetics | Pneumonia - immunology | Respiratory Syncytial Virus Infections - virology | Cytokines - genetics | Macrophages, Alveolar - immunology | Respiratory Syncytial Viruses - metabolism | Cytokines - immunology | Macrophages, Alveolar - virology | Cyclooxygenase 2 Inhibitors - pharmacology | Cytokines - metabolism | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Macrophage Activation - immunology | Macrophages, Peritoneal - immunology | Rats | Receptors, Cell Surface - metabolism | Respiratory Syncytial Virus Infections - genetics | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Pneumonia - virology | Receptors, Cell Surface - immunology | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Gene Expression Regulation - drug effects | Cell Differentiation - immunology | Macrophages, Peritoneal - virology | Sigmodontinae | Animals | Interferon-beta - metabolism | Signal Transduction - drug effects | Cell Differentiation - drug effects | Mice | Mice, Inbred BALB C | Macrophages, Alveolar - metabolism | Macrophage Activation - drug effects | Macrophages, Peritoneal - metabolism | Pneumonia - metabolism | Lung Injury - immunology | Receptors, Cell Surface - genetics
Journal Article
Hepatology, ISSN 0270-9139, 11/2012, Volume 56, Issue 5, pp. 1971 - 1982
Acetaminophen (APAP) is a safe analgesic and antipyretic drug. However, APAP overdose leads to massive hepatocyte death. Cell death during APAP toxicity occurs... 
ACETAMINOPHEN-INDUCED HEPATOTOXICITY | CELL INJURY | HEPATOCYTES | STERILE INFLAMMATION | DEPLETION PROTECTS | INFLAMMATORY RESPONSES | FORMYL PEPTIDE RECEPTOR | AUTOIMMUNE HEPATITIS | MICE | CORTICOSTEROIDS | GASTROENTEROLOGY & HEPATOLOGY | Chemokines - blood | Liver - pathology | Acute-Phase Reaction - immunology | Coculture Techniques | Humans | Middle Aged | Toll-Like Receptor 9 - genetics | Male | Receptors, Formyl Peptide - immunology | Acute-Phase Reaction - metabolism | Acute Lung Injury - blood | Necrosis - immunology | Toll-Like Receptor 9 - immunology | Young Adult | Receptors, Interleukin-8B - blood | Systemic Inflammatory Response Syndrome - blood | Mitochondrial Proteins - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Adult | Female | Interleukin-8 - blood | Child | Chemokines - immunology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Neutrophils - immunology | Liver Failure, Acute - chemically induced | Receptors, Interleukin-8B - immunology | DNA, Mitochondrial - blood | Hep G2 Cells | Mice, Knockout | Animals | Analysis of Variance | Acetaminophen | Liver Failure, Acute - immunology | Receptors, Interleukin-8B - metabolism | Adolescent | Acute Lung Injury - immunology | Chemokines - metabolism | Mice | Mitochondrial Proteins - immunology | Cell Movement | Mitochondrial DNA | Liver | Rodents | Hepatology | Chemokines | Index Medicus
Journal Article
Annual Review of Physiology, ISSN 0066-4278, 2011, Volume 73, Issue 1, pp. 479 - 501
Journal Article