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Pathology, ISSN 0031-3025, 02/2009, Volume 41, Issue 2, pp. 118 - 126
The aim of this study was to investigate the mechanism of PRL-3 in inducing angiogenesis and lymphangiogenesis to promote distant and lymph node metastasis in... 
ERK1/2 | metastasis | Lung cancer | VEGF | Rho | VEGF-C | PRL-3 | Metastasis | Life Sciences & Biomedicine | Pathology | Science & Technology | Immunohistochemistry | Up-Regulation | Phosphorylation | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | Protein Tyrosine Phosphatases - metabolism | rhoA GTP-Binding Protein - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | rhoA GTP-Binding Protein - genetics | Neoplasm Proteins - metabolism | Vascular Endothelial Growth Factor A - genetics | Extracellular Signal-Regulated MAP Kinases - genetics | Cell Movement - physiology | Protein Tyrosine Phosphatases - genetics | Female | Neoplasm Proteins - genetics | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | rho GTP-Binding Proteins - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Lymphangiogenesis - physiology | rhoC GTP-Binding Protein | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Vascular Endothelial Growth Factor C - genetics | Neoplasm Metastasis - genetics | Neoplasm Metastasis - pathology | Vascular Endothelial Growth Factor C - metabolism | rho GTP-Binding Proteins - metabolism | Fluorescent Antibody Technique | Neovascularization, Pathologic - genetics | Neovascularization, Pathologic - metabolism | Carcinoma, Non-Small-Cell Lung, pathology | Lung Neoplasms, genetics | Cell Movement, physiology | Carcinoma, Non-Small-Cell Lung, genetics | Vascular Endothelial Growth Factor C, genetics | Neoplasm Metastasis, pathology | Neovascularization, Pathologic, genetics | Protein Tyrosine Phosphatases, genetics | Extracellular Signal-Regulated MAP Kinases, metabolism | Lymphangiogenesis, physiology | Vascular Endothelial Growth Factor A, metabolism | Neoplasm Proteins, metabolism | Extracellular Signal-Regulated MAP Kinases, genetics | rho GTP-Binding Proteins, genetics | rhoA GTP-Binding Protein, genetics | rhoA GTP-Binding Protein, metabolism | Protein Tyrosine Phosphatases, metabolism | Vascular Endothelial Growth Factor A, genetics | rho GTP-Binding Proteins, metabolism | Carcinoma, Non-Small-Cell Lung, metabolism | Neoplasm Metastasis, genetics | Neoplasm Proteins, genetics | Lung Neoplasms, metabolism | Lung Neoplasms, pathology | Neovascularization, Pathologic, metabolism | Vascular Endothelial Growth Factor C, metabolism | Index Medicus
Journal Article
European journal of cancer (1990), ISSN 0959-8049, 09/2014, Volume 50, Issue 13, pp. 2336 - 2350
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 379, Issue 1, pp. 143 - 153
Highlights • PDACs with an angiogenic gene signature are enriched in lymphangiogenic genes. • Tumors from the KRC PDAC model harbor LECs and have a... 
Hematology, Oncology and Palliative Medicine | TGF-β | Mouse model | Lymphangiogenesis | Pancreatic cancer | TCGA | Life Sciences & Biomedicine | Oncology | Science & Technology | Carcinoma, Pancreatic Ductal - secondary | Receptor, Epidermal Growth Factor - genetics | Receptor, ErbB-2 - genetics | Apoptosis - drug effects | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Neovascularization, Pathologic | Receptor, ErbB-2 - metabolism | Male | Molecular Targeted Therapy | Cell Movement - genetics | Quinolines - pharmacology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Carcinoma, Pancreatic Ductal - genetics | Pancreatic Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Transforming Growth Factor beta - antagonists & inhibitors | Female | Receptor, ErbB-2 - antagonists & inhibitors | Protein-Serine-Threonine Kinases - metabolism | Pyrazoles - pharmacology | Genetic Predisposition to Disease | Neoplasm Invasiveness | Genes, Retinoblastoma | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - enzymology | Angiogenesis Inhibitors - pharmacology | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Gene Expression Profiling - methods | Mice, Transgenic | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - enzymology | Lymphangiogenesis - drug effects | Carcinoma, Pancreatic Ductal - drug therapy | Cell Movement - drug effects | Phenotype | Animals | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Mutation | Quinazolines - pharmacology | Lymphangiogenesis - genetics | Transforming Growth Factor beta - metabolism | Index Medicus | lymphangiogenesis | pancreatic cancer | mouse model
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 03/2017, Volume 127, Issue 4, pp. 1225 - 1240
Journal Article
International journal of oncology, ISSN 1019-6439, 02/2019, Volume 54, Issue 2, pp. 572 - 584
Journal Article
Development (Cambridge), ISSN 1477-9129, 02/2014, Volume 141, Issue 6, pp. 1228 - 1238
In mammals, the homeodomain transcription factor Prox1 acts as the central regulator of lymphatic cell fate. Its restricted expression in a subset of cardinal... 
Endothelial Cells | Species Specificity | Animals, Genetically Modified | Zebrafish Proteins | Zebrafish | Mice, Knockout | Cell Lineage | Animals | Gene Expression Regulation, Developmental | Homeodomain Proteins | Lymphatic Vessels | Cell Differentiation | Mice | Mutation | SOXF Transcription Factors | Lymphangiogenesis | COUP Transcription Factor II | Tumor Suppressor Proteins | Prox1 | Coup-TFII | Nr2f2 | Sox18 | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | SOXF Transcription Factors - metabolism | Lymphatic Vessels - metabolism | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | COUP Transcription Factor II - deficiency | Lymphatic Vessels - cytology | Endothelial Cells - metabolism | Zebrafish Proteins - metabolism | Lymphangiogenesis - physiology | Zebrafish - growth & development | Homeodomain Proteins - genetics | Zebrafish - genetics | Zebrafish Proteins - physiology | Tumor Suppressor Proteins - physiology | Zebrafish Proteins - deficiency | COUP Transcription Factor II - genetics | Endothelial Cells - cytology | Zebrafish - physiology | COUP Transcription Factor II - metabolism | Zebrafish Proteins - genetics | Homeodomain Proteins - physiology | SOXF Transcription Factors - deficiency | SOXF Transcription Factors - genetics | Lymphangiogenesis - genetics | Index Medicus | in-vivo | transcription factor | growth-factor-c | lymphangiogenesis | coup-tfii | prox1 | redundant roles | sox18 | xenopus-laevis | vascular endothelial-cells
Journal Article
Journal Article