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Journal Article
Cancer cell, ISSN 1535-6108, 2015, Volume 28, Issue 6, pp. 758 - 772
Angiosarcoma/lymphangiosarcoma is a rare malignancy with poor prognosis. We generated a mouse model with inducible endothelial-cell-specific deletion of Tsc1... 
mouse models | mTORC1 signaling | VEGF | vascular anomalies | angiosarcoma | vascular tumor | lymphangiosarcoma | Vascular tumor | Vascular anomalies | Mouse models | MTORC1 signaling | Lymphangiosarcoma | Angiosarcoma | MAMMALIAN TARGET | RAPAMYCIN MTOR | KAPOSIS-SARCOMA | ONCOLOGY | PROTEIN-COUPLED RECEPTOR | IN-VIVO | INFANTILE HEMANGIOMA | TUBEROUS SCLEROSIS | TSC1 | VASCULAR TUMORS | ANGIOSARCOMA | CELL BIOLOGY | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Multiprotein Complexes - metabolism | RNA Interference | Cell Transformation, Neoplastic - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Lymphangiosarcoma - genetics | Tumor Suppressor Proteins - metabolism | Lymphangiosarcoma - metabolism | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Angiogenesis Inhibitors - pharmacology | Genotype | Tumor Burden | Mice, Knockout | Phenotype | Mice, Nude | Proto-Oncogene Proteins c-myc - genetics | Enzyme Activation | Endothelial Cells - pathology | Endothelial Cells - enzymology | Retinal Neovascularization - pathology | Cell Movement | Hypoxia-Inducible Factor 1, alpha Subunit - analysis | Proto-Oncogene Proteins c-myc - analysis | Neovascularization, Pathologic | Autocrine Communication - drug effects | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | TOR Serine-Threonine Kinases - antagonists & inhibitors | Receptors, Vascular Endothelial Growth Factor - metabolism | Transfection | Tumor Suppressor Proteins - genetics | Retinal Neovascularization - genetics | Neoplasm Invasiveness | Cells, Cultured | Vascular Endothelial Growth Factor A - analysis | Lymphangiosarcoma - pathology | Cell Transformation, Neoplastic - metabolism | Proto-Oncogene Proteins c-myc - metabolism | Animals | Lymphangiosarcoma - drug therapy | Protein Kinase Inhibitors - pharmacology | Cell Transformation, Neoplastic - pathology | Endothelial Cells - drug effects | Vascular endothelial growth factor | Analysis | Endothelium | Medical colleges | Development and progression | Endothelial growth factors | Tumors
Journal Article
Journal Article
Journal Article
Journal of Wound Care, ISSN 0969-0700, 06/2015, Volume 24, Issue Sup6, pp. S5 - S7
Journal Article
Journal Article
Journal of veterinary medical science, ISSN 0916-7250, 2014, Volume 77, Issue 3, pp. 371 - 374
Journal Article
Modern pathology, ISSN 1530-0285, 2000, Volume 13, Issue 9, pp. 978 - 987
Journal Article
Journal Article
Journal Article
Cell Cycle, ISSN 1538-4101, 07/2016, Volume 15, Issue 14, pp. 1801 - 1802
Journal Article
Pan African Medical Journal, ISSN 1937-8688, 2014, Volume 19, Issue 1, p. 2
The Stewart-Treves syndrome was first described in 1948, it's an angiosarcoma developed on a longstanding lymphadenomatous limb, more often after radical... 
Angiosarcoma | Stewart-Treves | Syndrome | Hemangiosarcoma - therapy | Breast Neoplasms - surgery | Hemangiosarcoma - pathology | Lymphangiosarcoma - diagnosis | Humans | Female | Aged | Lymphangiosarcoma - pathology | Lymphangiosarcoma - therapy | Mastectomy - adverse effects | Hemangiosarcoma - diagnosis
Journal Article