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1981, Contemporary immunology, ISBN 0896030121, xv, 437
Book
Lymphokine reports, ISSN 0197-596X, 1980
Journal
Lymphokine reports, ISSN 0197-596X, 1980
Journal
Lymphokine and cytokine research, ISSN 1056-5477, 1991
Journal
Lymphokine research, ISSN 0277-6766, 1982
Journal
Lymphokines, ISSN 0277-013X, 1981
Journal
1981, ISBN 9780124320031, Volume 3., xviii, 450
Book
Journal of biological regulators and homeostatic agents, ISSN 0393-974X, 1987
Journal
Journal of Molecular Biology, ISSN 0022-2836, 2002, Volume 316, Issue 3, pp. 769 - 787
Vascular endothelial growth factor (VEGF) is a potent endothelial cell-specific mediator of angiogenesis and vasculogenesis. VEGF is involved pathologically in... 
protein structure | NMR | angiogenesis | vascular endothelial growth factor | peptide antagonist | Angiogenesis | Peptide antagonist | Protein structure | Vascular endothelial growth factor | MOLECULAR-DYNAMICS | TRIPLE-RESONANCE EXPERIMENTS | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEIN-STRUCTURE DETERMINATION | RECEPTOR-BINDING DOMAIN | BIOACTIVE CONFORMATION | ANGSTROM RESOLUTION | DE-NOVO DESIGN | IMPROVED SENSITIVITY | INTERPROTON DISTANCE RESTRAINTS | Disulfides - metabolism | Receptors, Vascular Endothelial Growth Factor | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Humans | Molecular Sequence Data | Receptors, Growth Factor - antagonists & inhibitors | Crystallography, X-Ray | Endothelial Growth Factors - chemistry | Proto-Oncogene Proteins - chemistry | Vascular Endothelial Growth Factor Receptor-1 | Lymphokines - genetics | Peptides - chemical synthesis | Peptides - metabolism | Alanine - genetics | Solutions | Lymphokines - antagonists & inhibitors | Protein Structure, Quaternary | Lymphokines - metabolism | Nuclear Magnetic Resonance, Biomolecular | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Binding Sites | Dimerization | Protein Structure, Tertiary | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | Proto-Oncogene Proteins - antagonists & inhibitors | Bacteriophages - chemistry | Peptides - chemistry | Protein Structure, Secondary | Models, Molecular | Alanine - metabolism | Mutation - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Receptors, Growth Factor - chemistry | Peptides - pharmacology | Endothelial Growth Factors - antagonists & inhibitors | Lymphokines - chemistry | Endothelial Growth Factors - genetics | Hydrophobic and Hydrophilic Interactions | Receptor Protein-Tyrosine Kinases - chemistry | Receptors, Growth Factor - metabolism
Journal Article
1982, ISBN 9780124320062, Volume 6., xv, 318
Book
Journal of Clinical Investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 3786 - 3799
The molecular mechanisms of acute lung injury resulting in inflammation and fibrosis are not well established. Here we investigate the roles of the IL-1... 
MONOSODIUM URATE CRYSTALS | MEDICINE, RESEARCH & EXPERIMENTAL | INTERLEUKIN-1 RECEPTOR ANTAGONIST | INDUCED CYTOKINE | INDUCED LUNG INJURY | AIRWAY INFLAMMATION | BLEOMYCIN-INDUCED PNEUMONOPATHY | DEFICIENT MICE | ONSET STILLS-DISEASE | NF-KAPPA-B | CUTTING EDGE | Respiratory Distress Syndrome, Adult - pathology | Interleukin-8 - genetics | Sialoglycoproteins - genetics | Interleukin-1beta - pharmacology | Antibiotics, Antineoplastic - toxicity | Humans | Sialoglycoproteins - metabolism | Pneumonia - pathology | Respiratory Distress Syndrome, Adult - metabolism | Receptors, Interleukin-1 Type I - metabolism | Receptors, Interleukin-1 Type I - antagonists & inhibitors | Respiratory Distress Syndrome, Adult - genetics | Lymphokines - genetics | Pneumonia - chemically induced | Bone Marrow Transplantation | Lymphokines - metabolism | Pulmonary Fibrosis - metabolism | Interleukin 1 Receptor Antagonist Protein - pharmacology | Interleukin-8 - metabolism | Bleomycin - toxicity | Disease Models, Animal | Pneumonia - genetics | Receptors, Interleukin-1 Type I - agonists | Respiratory Distress Syndrome, Adult - chemically induced | Myeloid Differentiation Factor 88 - genetics | Pulmonary Fibrosis - pathology | Signal Transduction - genetics | Recombinant Proteins - pharmacology | Mice, Knockout | Transplantation Chimera - genetics | Transplantation Chimera - metabolism | Animals | Signal Transduction - drug effects | Receptors, Interleukin-1 Type I - genetics | Pulmonary Fibrosis - chemically induced | Mice | Pneumonia - metabolism | Chronic Disease | Myeloid Differentiation Factor 88 - metabolism | Complications and side effects | Bleomycin | Pulmonary fibrosis | Dosage and administration | Research | Health aspects | Risk factors | Apoptosis | Pneumonia | Respiratory Distress Syndrome, Adult | Signal Transduction | Interleukin-1beta | Antibiotics, Antineoplastic | Myeloid Differentiation Factor 88 | Recombinant Proteins | Transplantation Chimera | Interleukin-8 | Lymphokines | Life Sciences | Sialoglycoproteins | Immunology | Pulmonary Fibrosis | Interleukin 1 Receptor Antagonist Protein | Receptors, Interleukin-1 Type I
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4171 - 4185
Mucosal-associated invariant T cells (MAITs) have potent antimicrobial activity and are abundant in humans (5%-10% in blood). Despite strong evolutionary... 
MEDICINE, RESEARCH & EXPERIMENTAL | REPERTOIRE | ACTIVATION | HIV-INFECTION | MAIT CELLS | MR1 ANTIGEN PRESENTATION | HUMANS | RECEPTOR | BACTERIAL-INFECTION | LAMINA PROPRIA | SUBSET | Chemotaxis, Leukocyte | Lymphokines - secretion | Gene Rearrangement, alpha-Chain T-Cell Antigen Receptor | Minor Histocompatibility Antigens | Humans | Mice, Congenic - microbiology | Microbiota | Kruppel-Like Transcription Factors - analysis | Female | Urinary Tract Infections - microbiology | Disease Models, Animal | Natural Killer T-Cells - secretion | Lymphocyte Activation | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Mice, Transgenic | Radiation Chimera | Mice, Congenic - immunology | Receptors, Antigen, T-Cell, alpha-beta - genetics | Promyelocytic Leukemia Zinc Finger Protein | Mice, Knockout | Lymphoid Tissue - cytology | Phenotype | Animals | Receptors, Cytokine - analysis | Germ-Free Life | Nuclear Receptor Subfamily 1, Group F, Member 3 - analysis | Lymphocyte Count | Mice, Congenic - genetics | Immunologic Memory | Mice | Polymorphism, Single Nucleotide | Urinary Tract Infections - immunology | Crosses, Genetic | Natural Killer T-Cells - immunology | Pathogens | Medical research | Genotype & phenotype | Hypotheses | Cytokines | Lymphocytes | Laboratories | Rodents | Bacteria | Infections | Genomes | Receptors, Cytokine | Histocompatibility Antigens Class I | Lymphokines | Life Sciences | Immunology | Receptors, Antigen, T-Cell, alpha-beta | Natural Killer T-Cells | Nuclear Receptor Subfamily 1, Group F, Member 3 | Lymphoid Tissue | Kruppel-Like Transcription Factors | Urinary Tract Infections | Mice, Congenic
Journal Article
1979, ISBN 9780121782504, xvi, 626
Book
Nature Genetics, ISSN 1061-4036, 01/2003, Volume 34, Issue 4, pp. 383 - 394
Amyotrophic lateral sclerosis (ALS) is an incurable degenerative disorder of motoneurons. We recently reported that reduced expression of Vegfa causes ALS-like... 
TRANSFORMATION | PATHOGENESIS | GENETIC ASSOCIATION | MESSENGER-RNA | HEAVY NEUROFILAMENT SUBUNIT | DISEASE | GENETICS & HEREDITY | POLYMORPHISM | HIPPOCAMPAL-NEURONS | SPORADIC ALS | ENDOTHELIAL GROWTH-FACTOR | Haplotypes | Vascular Endothelial Growth Factor A | Vascular Endothelial Growth Factors | Humans | Middle Aged | Spinal Cord Ischemia - drug therapy | Child, Preschool | Nerve Degeneration - genetics | Male | Amyotrophic Lateral Sclerosis - drug therapy | Motor Neurons - pathology | Intercellular Signaling Peptides and Proteins - physiology | Genetic Variation | Sweden | Lymphokines - genetics | Spinal Cord Ischemia - pathology | Female | Amyotrophic Lateral Sclerosis - etiology | Cell Death - drug effects | Endothelial Growth Factors - therapeutic use | Child | Ischemia - pathology | Lymphokines - therapeutic use | Motor Neurons - drug effects | Paralysis - etiology | Endothelial Growth Factors - physiology | Amyotrophic Lateral Sclerosis - genetics | Lymphokines - physiology | Intercellular Signaling Peptides and Proteins - genetics | Mice, Transgenic | Mice, Knockout | Amyotrophic Lateral Sclerosis - pathology | Animals | Endothelial Growth Factors - genetics | Alleles | Aged | Intercellular Signaling Peptides and Proteins - therapeutic use | Mice | Motor neurons | Physiological aspects | Amyotrophic lateral sclerosis | Genetic aspects | Diagnosis | Research | Vascular endothelial growth factor | Risk factors
Journal Article
1995, Molecular biology intelligence unit (Unnumbered), ISBN 9781570592539, 123
Book
Journal of Biological Chemistry, ISSN 0021-9258, 04/2003, Volume 278, Issue 15, pp. 12605 - 12608
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 2014, Volume 59, Issue 5, pp. 1402 - 1409.e4
Background Platelet-derived growth factor C (PDGF-C) has been reported to promote angiogenesis independently of vascular endothelial growth factor (VEGF),... 
Surgery | SURGERY | ALPHA-RECEPTOR | VEGF | ENDOTHELIAL-CELLS | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | GENE-THERAPY | PATHOLOGICAL ANGIOGENESIS | MELLITUS | THERAPEUTIC ANGIOGENESIS | EXPRESSION | PDGF-CC | Antibodies, Neutralizing - administration & dosage | Neuropilin-2 - metabolism | Hindlimb | Platelet-Derived Growth Factor - immunology | Lymphokines - deficiency | Neuropilin-1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Male | Ischemia - genetics | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Platelet-Derived Growth Factor - genetics | Recovery of Function | Diabetic Angiopathies - physiopathology | Lymphokines - genetics | Time Factors | Lymphokines - antagonists & inhibitors | Lymphokines - metabolism | Diabetes Mellitus, Experimental - metabolism | Diabetic Angiopathies - genetics | Diabetes Mellitus, Experimental - physiopathology | Muscle, Skeletal - blood supply | Diabetic Angiopathies - metabolism | Gene Transfer Techniques | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Signal Transduction | Diabetes Mellitus, Type 1 - physiopathology | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - genetics | Ischemia - metabolism | Ischemia - therapy | Platelet-Derived Growth Factor - deficiency | Vascular Endothelial Growth Factor A - immunology | Ischemia - physiopathology | Regional Blood Flow | Mice, Knockout | Diabetic Angiopathies - therapy | Platelet-Derived Growth Factor - metabolism | Animals | Lymphokines - immunology | Mice | Platelet-Derived Growth Factor - antagonists & inhibitors | Neovascularization, Physiologic | Platelet-derived growth factor | Diabetes | Vascular endothelial growth factor | Analysis
Journal Article
Journal Article