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PLoS ONE, ISSN 1932-6203, 04/2009, Volume 4, Issue 4, pp. e5385 - e5385
Journal Article
European Journal of Pediatrics, ISSN 0340-6199, 8/2015, Volume 174, Issue 8, pp. 1085 - 1092
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2002, Volume 99, Issue 10, pp. 7021 - 7026
Insufficient oxygen and nutrient supply often restrain solid tumor growth, and the hypoxia-inducible factors (HIF) 1α and HIF-2α are key transcription... 
Tumor cell line | Chromaffin cells | Biological Sciences | Oxygen | Phenotypes | Neurons | Cell lines | HeLa cells | Hypoxia | Cellular differentiation | Tumors | IN-VITRO | HOMEOSTASIS | FACTOR-1-ALPHA | INDUCIBLE FACTOR 1-ALPHA | MULTIDISCIPLINARY SCIENCES | SYMPATHETIC NERVOUS-SYSTEM | N-MYC | TUMOR-SUPPRESSOR PROTEIN | DIFFERENTIATION | HIF-1-ALPHA | TRANSCRIPTION FACTOR | Vascular Endothelial Growth Factor A | Vascular Endothelial Growth Factors | Humans | Transplantation, Heterologous | Neuropeptide Y - genetics | Helix-Loop-Helix Motifs | Neuroblastoma | Sympathetic Nervous System - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit | Oxygen - metabolism | Cell Hypoxia | Insulin-Like Growth Factor II - genetics | Lymphokines - genetics | Trans-Activators - genetics | Neoplasms, Experimental | Female | Tumor Cells, Cultured | Basic Helix-Loop-Helix Transcription Factors | Gene Expression | Neural Crest - cytology | Down-Regulation | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Transcription Factors - metabolism | Tyrosine 3-Monooxygenase - genetics | Phenotype | Animals | Mice, Nude | Endothelial Growth Factors - genetics | Paraganglia, Chromaffin - cytology | Biomarkers | Trans-Activators - metabolism | Mice | HeLa Cells | Growth | Genetic aspects | Research | Gene expression | hypoxia-inducible factor 1a | hypoxia-inducible factor 2a | Pediatrics | Tumor Cells | Support | DNA-Binding Proteins : genetics | Medical and Health Sciences | Trans-Activators : genetics | Biological Markers | Medicin och hälsovetenskap | Pediatrik | Lymphokines : genetics | Trans-Activators : metabolism | Neural Crest : cytology | Transplantation | Klinisk medicin | Nude | Neoplasms | Cultured | Chromaffin : cytology | Transcription Factors : genetics | Human | Transcription Factors : metabolism | Experimental | Paraganglia | Tyrosine 3-Monooxygenase : genetics | Endothelial Growth Factors : genetics | Clinical Medicine | Insulin-Like Growth Factor II : genetics | Animal | Oxygen : metabolism | Sympathetic Nervous System : metabolism | Cancer and Oncology | Non-U.S. Gov't | Heterologous | Neuropeptide Y : genetics | Hela Cells | Cancer och onkologi
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 12/2015, Volume 289, Issue 3, pp. 457 - 465
Epigenetic modifications of DNA and histones alter cellular phenotypes without changing genetic codes. Alterations of epigenetic marks can be induced by... 
PCBs | ER-α | JMJD2B | H3K9me3 | Epigenetics and vascular inflammation | p65 | POLYCHLORINATED-BIPHENYLS PCBS | PERSISTENT ORGANIC POLLUTANTS | FACTOR C-REL | HISTONE LYSINE METHYLATION | REACTIVE PROTEIN EXPRESSION | ESTROGEN-RECEPTOR-ALPHA | GENOME-WIDE ANALYSIS | GENE-EXPRESSION | CARDIOVASCULAR-DISEASE | PHARMACOLOGY & PHARMACY | TOXICOLOGY | TRANSCRIPTION-FACTOR | ER-alpha | Inflammation - chemically induced | Environmental Pollutants - adverse effects | Humans | Tumor Necrosis Factor-alpha - genetics | Endothelium, Vascular - drug effects | Vascular Cell Adhesion Molecule-1 - genetics | Epigenesis, Genetic - drug effects | eIF-2 Kinase - genetics | Cell Adhesion - genetics | Interleukin-6 - genetics | Cells, Cultured | Up-Regulation - genetics | C-Reactive Protein - genetics | Signal Transduction - genetics | Cell Adhesion - drug effects | Up-Regulation - drug effects | Histones - genetics | Polychlorinated Biphenyls - adverse effects | Methylation - drug effects | NF-kappa B - genetics | Signal Transduction - drug effects | Epigenesis, Genetic - genetics | Intercellular Adhesion Molecule-1 - genetics | Inflammation - genetics | Endothelial Cells - drug effects | Index Medicus | ESTROGENS | IMMUNITY | TRANSLOCATION | METHYLATION | RNA | LYSINE | CARDIOVASCULAR DISEASES | POLYCHLORINATED BIPHENYLS | 60 APPLIED LIFE SCIENCES | HISTONES | GLOBULINS | NECROSIS | POLYMERASE CHAIN REACTION | LYMPHOKINES | ENDOTHELIUM | INFLAMMATION | PHOSPHATES | LEUKEMIA | ANDROGENS | RECEPTORS | OXIDOREDUCTASES | epigenetics and vascular inflammation
Journal Article
by Peden, John F and Hopewell, Jemma C and Saleheen, Danish and Chambers, John C and Hager, Jorg and Soranzo, Nicole and Collins, Rory and Danesh, John and Elliott, Paul and Farrall, Martin and Stirrups, Kathy and Zhang, Weihua and Hamsten, Anders and Parish, Sarah and Lathrop, Mark and Watkins, Hugh and Clarke, Robert and Deloukas, Panos and Kooner, Jaspal S and Goel, Anuj and Ongen, Halit and Strawbridge, Rona J and Heath, Simon and Mälarstig, Anders and Helgadottir, Anna and Öhrvik, John and Murtaza, Muhammed and Potter, Simon and Hunt, Sarah E and Delepine, Marc and Jalilzadeh, Shapour and Axelsson, Tomas and Syvanen, Ann-Christine and Gwilliam, Rhian and Bumpstead, Suzannah and Gray, Emma and Edkins, Sarah and Folkersen, Lasse and Kyriakou, Theodosios and Franco-Cereceda, Anders and Gabrielsen, Anders and Seedorf, Udo and Eriksson, Per and Offer, Alison and Bowman, Louise and Sleight, Peter and Armitage, Jane and Peto, Richard and Abecasis, Goncalo and Ahmed, Nabeel and Caulfield, Mark and Donnelly, Peter and Froguel, Philippe and Kooner, Angad S and McCarthy, Mark I and Samani, Nilesh J and Scott, James and Sehmi, Joban and Silveira, Angela and Hellénius, Mai-Lus and van't Hooft, Ferdinand M and Olsson, Gunnar and Rust, Stephan and Assman, Gerd and Barlera, Simona and Tognoni, Gianni and Franzosi, Maria Grazia and Linksted, Pamela and Green, Fiona R and Rasheed, Asif and Zaidi, Moazzam and Shah, Nabi and Samuel, Maria and Mallick, Nadeem H and Azhar, Muhammad and Zaman, Khan S and Samad, Abdus and Ishaq, Mohammad and Gardezi, Ali R and Fazal-ur-Rehman, Memon and Frossard, Philippe M and Spector, Tim and Peltonen, Leena and Nieminen, Markku S and Sinisalo, Juha and Salomaa, Veikko and Ripatti, Samuli and Bennett, Derrick and Leander, Karin and Gigante, Bruna and de Faire, Ulf and Pietri, Silvia and Gori, Francesca and Marchioli, Roberto and Sivapalaratnam, Suthesh and Kastelein, John J. P and Trip, Mieke D and Theodoraki, Eirini V and Dedoussis, George V and Engert, Jamie C and ... and Coronary Artery Dis C4D Genetics C and MuTHER Consortium and Coronary Artery Disease (C4D) Genetics Consortium and The Coronary Artery Disease (C4D) Genetics Consortium and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Molekylär medicin and Uppsala universitet and Institutionen för medicinska vetenskaper
Nature genetics, ISSN 1061-4036, 2011, Volume 43, Issue 4, pp. 339 - 344
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2016, Volume 478, Issue 2, pp. 845 - 851
Platelet-derived growth factor D (PDGF-D) signaling plays significant roles during the development and progression of human malignancies via interacting with... 
TSCC | PDGF-D/PDGFRβ | p38/AKT/ERK/EMT signal pathway | HEAD | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICROENVIRONMENTAL REGULATION | EMT | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | OVEREXPRESSION | BIOPHYSICS | DISSEMINATION | PDGF-D/PDGFR beta | NECK | BIOLOGICAL INSIGHTS | RNA, Small Interfering - genetics | Phosphorylation | Cadherins - metabolism | Vimentin - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | Epithelial-Mesenchymal Transition - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Antigens, CD - genetics | Platelet-Derived Growth Factor - genetics | Proto-Oncogene Proteins c-akt - genetics | Antigens, CD - metabolism | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | Lymphokines - genetics | Snail Family Transcription Factors - genetics | Vimentin - genetics | Lymphokines - antagonists & inhibitors | Lymphokines - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Cadherins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Tongue Neoplasms - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Snail Family Transcription Factors - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Neoplasm Invasiveness | Tongue Neoplasms - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Tongue Neoplasms - pathology | Disease Progression | Platelet-Derived Growth Factor - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Line, Tumor | Platelet-Derived Growth Factor - antagonists & inhibitors | Cell Movement | RNA, Small Interfering - metabolism
Journal Article
Genes and Development, ISSN 0890-9369, 2000, Volume 14, Issue 4, pp. 391 - 396
In glioblastoma-derived cell lines, PTEN does not significantly alter apoptotic sensitivity or cause complete inhibition of DNA synthesis. However, in these... 
PTEN | Angiogenesis | Gene expression | Glioblastoma | SURVIVAL PATHWAY | angiogenesis | KINASE | AKT | DEVELOPMENTAL BIOLOGY | INDUCTION | CELL-SURVIVAL | HYPOXIA | BRAIN-TUMORS | glioblastoma | GENETICS & HEREDITY | GERMLINE MUTATIONS | TUMOR-SUPPRESSOR | gene expression | ENDOTHELIAL GROWTH-FACTOR | Prostaglandin-Endoperoxide Synthases - biosynthesis | Vascular Endothelial Growth Factor A | Insulin-Like Growth Factor I - pharmacology | Vascular Endothelial Growth Factors | Humans | Neoplasm Proteins - physiology | Protein-Serine-Threonine Kinases | Brain Neoplasms - pathology | Cyclooxygenase 1 | Phosphofructokinase-1 - genetics | Phosphoric Monoester Hydrolases - deficiency | Recombinant Fusion Proteins - physiology | Phosphofructokinase-1 - biosynthesis | Genetic Complementation Test | Hypoxia-Inducible Factor 1, alpha Subunit | Lymphokines - genetics | Transfection | Glioblastoma - genetics | PTEN Phosphohydrolase | Gene Deletion | Lymphokines - biosynthesis | Tumor Cells, Cultured | Culture Media, Serum-Free - pharmacology | Neoplasm Proteins - genetics | Gene Expression Regulation, Neoplastic - genetics | Prostaglandin-Endoperoxide Synthases - genetics | DNA-Binding Proteins - physiology | Phosphoric Monoester Hydrolases - genetics | Hypoxia-Inducible Factor 1 | Isoenzymes - genetics | Neoplasm Proteins - biosynthesis | Brain Neoplasms - genetics | Phosphotransferases (Alcohol Group Acceptor) - genetics | Endothelial Growth Factors - biosynthesis | Phosphoric Monoester Hydrolases - physiology | Disease Progression | Membrane Proteins | Proto-Oncogene Proteins c-akt | Endothelial Growth Factors - genetics | Glioblastoma - pathology | Proto-Oncogene Proteins - physiology | Nuclear Proteins - physiology | Transcription Factors | Cell Hypoxia - genetics | Isoenzymes - biosynthesis | Tumor Suppressor Proteins | Apoptosis | Phosphotransferases (Alcohol Group Acceptor) - biosynthesis | Tumor suppressor genes | Genetic aspects | Research | Neovascularization | Cytochemistry | Glioblastoma multiforme | Carcinogenesis | Research Communication
Journal Article
Cancer Science, ISSN 1347-9032, 09/2015, Volume 106, Issue 9, pp. 1143 - 1152
Journal Article
Nature Genetics, ISSN 1061-4036, 01/2001, Volume 28, Issue 2, pp. 131 - 138
Hypoxia stimulates angiogenesis through the binding of hypoxia-inducible factors to the hypoxia-response element in the vascular endothelial growth factor... 
NERVOUS-SYSTEM | ANGIOGENESIS | VEGF | GENETICS & HEREDITY | FACTOR MESSENGER-RNA | SPINAL-CORD | FACTOR EXPRESSION | ISCHEMIA | AMYOTROPHIC-LATERAL-SCLEROSIS | MODEL | INDUCIBLE FACTOR-1 | Receptors, Vascular Endothelial Growth Factor | Sequence Deletion | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Humans | Nerve Degeneration - physiopathology | Electrophysiology | Nerve Degeneration - genetics | Axons - physiology | Motor Neurons - pathology | Neuropilin-1 | Receptors, Growth Factor - genetics | Lymphokines - genetics | Lymphokines - metabolism | Response Elements - genetics | Binding Sites | Promoter Regions, Genetic | Motor Neurons - physiology | Amyotrophic Lateral Sclerosis - genetics | Muscular Atrophy - pathology | Muscular Atrophy - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Nerve Tissue Proteins - genetics | Peripheral Nerves - pathology | Nerve Degeneration - pathology | Mice, Knockout | Nerve Tissue Proteins - metabolism | Amyotrophic Lateral Sclerosis - pathology | Animals | Muscle Contraction | Receptor Protein-Tyrosine Kinases - genetics | Endothelial Growth Factors - genetics | Spinal Cord - physiology | Muscle Fibers, Skeletal - pathology | Mice | Cell Hypoxia - genetics | Receptors, Growth Factor - metabolism | Gene mutations | Physiological aspects | Hypoxia | Genetic aspects | Research | Neovascularization | Health aspects | Vascular endothelial growth factor
Journal Article
Genes and Development, ISSN 0890-9369, 08/2003, Volume 17, Issue 15, pp. 1835 - 1840
Several platelet-derived growth factor (PDGF) and vascular endothelial growth factor (VEGF) family members display C-terminal protein motifs that confer... 
Pericyte | Cell retention | Retina | Mesangial cell | PDGF | Heparan sulphate proteoglycan | DIABETIC-RETINOPATHY | VEGF ISOFORMS | pericyte | ANGIOGENESIS | A-CHAIN | DEVELOPMENTAL BIOLOGY | mesangial cell | CELL BIOLOGY | cell retention | retina | IN-VIVO | GENETICS & HEREDITY | MICE | heparan sulphate proteoglycan | HEPARAN-SULFATE PROTEOGLYCANS | GROWTH-FACTOR | BINDING | SMOOTH-MUSCLE CELLS | Kidney - physiology | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Microcirculation - metabolism | Retina - metabolism | Vascular Endothelial Growth Factors | Glomerulosclerosis, Focal Segmental - genetics | Molecular Sequence Data | Intercellular Signaling Peptides and Proteins - metabolism | Lymphokines - metabolism | Protein Structure, Tertiary | Amino Acid Sequence | Proto-Oncogene Proteins c-sis - genetics | Proto-Oncogene Proteins c-sis - metabolism | Retinal Degeneration - genetics | Pericytes - metabolism | Proteinuria - genetics | Amino Acid Motifs | Retina - physiology | Phenotype | Platelet-Derived Growth Factor - metabolism | Animals | Endothelium, Vascular - metabolism | Alleles | Mice | Models, Genetic | Mutation | Microscopy, Fluorescence | Cell Movement | Proteins | Blood platelets | Secretion | Analysis | Genetic research | Genetic aspects | Growth factors | Endothelium | Research Communications | Glomerulosclerosis; Focal Segmental/genetics | Intercellular Signaling Peptides and Proteins/metabolism | Platelet-Derived Growth Factor/metabolism | Proto-Oncogene Proteins c-sis/genetics/metabolism | Proteinuria/genetics | Models; Genetic | Microcirculation/metabolism | Lymphokines/metabolism | Microscopy; Fluorescence | Pericytes/metabolism | Kidney/physiology | Protein Structure; Tertiary | Retinal Degeneration/genetics | Endothelial Growth Factors/metabolism | Endothelium; Vascular/metabolism | Retina/metabolism/physiology
Journal Article