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Journal of Biological Chemistry, ISSN 0021-9258, 10/2002, Volume 277, Issue 41, pp. 38205 - 38211
Stimulation of human colon cancer cells with insulin-like growth factor 1 (IGF-1) induces expression of the VEGF gene, encoding vascular endothelial growth... 
FACTOR 1-ALPHA HIF-1-ALPHA | CAP-BINDING PROTEIN | TRANSCRIPTIONAL ACTIVITY | ACTIVATED PROTEIN-KINASE | PROLYL HYDROXYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | TUMOR-SUPPRESSOR PROTEIN | TRANSLATION INITIATION | UP-REGULATION | HIF-ALPHA | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | MAP Kinase Signaling System - physiology | Protein-Tyrosine Kinases - metabolism | Vascular Endothelial Growth Factors | Humans | Ubiquitin - metabolism | Eukaryotic Initiation Factor-4E - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Phosphoproteins - metabolism | MAP Kinase Kinase 2 | Colonic Neoplasms - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit | Hypoxia - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Lymphokines - genetics | Lymphokines - metabolism | Repressor Proteins - metabolism | Enzyme Inhibitors - metabolism | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Adaptor Proteins, Signal Transducing | Animals | Carrier Proteins - metabolism | Endothelial Growth Factors - genetics | Carcinoma - metabolism | Insulin-Like Growth Factor I - metabolism | Index Medicus
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 03/2003, Volume 95, Issue 6, pp. 437 - 448
Background: Interferon alpha (IFN-alpha) has antiangiogenic activity, although the underlying mechanism of action is unclear. Because human neuroendocrine (NE)... 
CELLS | INHIBITION | THERAPY | ONCOLOGY | SP1 | CLINICAL-APPLICATIONS | PROTEIN-KINASE | DOWN-REGULATION | EXPRESSION | CANCER | BINDING | Immunohistochemistry | Interferon-alpha - pharmacology | Transcription, Genetic - drug effects | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Luciferases - metabolism | Vascular Endothelial Growth Factors | Receptors, Vascular Endothelial Growth Factor - drug effects | Transplantation, Heterologous | Pregnancy Proteins - metabolism | Promoter Regions, Genetic - drug effects | RNA, Messenger - metabolism | Biomarkers, Tumor | Intercellular Signaling Peptides and Proteins - metabolism | Lymphokines - genetics | Receptors, Vascular Endothelial Growth Factor - metabolism | Time Factors | Lymphokines - metabolism | Antineoplastic Agents - pharmacology | Electrophoretic Mobility Shift Assay | Gene Expression Regulation, Neoplastic - drug effects | Receptors, Vascular Endothelial Growth Factor - genetics | Tumor Cells, Cultured | Lymphokines - drug effects | Enzyme-Linked Immunosorbent Assay | Neuroendocrine Tumors - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Angiogenesis Inhibitors - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Endothelial Growth Factors - genetics | Neuroendocrine Tumors - drug therapy | Genes, Reporter - drug effects | Mice | Neovascularization, Pathologic - metabolism | Neuroendocrine Tumors - blood supply | Receptors, Immunologic - metabolism | Measurement | Physiological aspects | Vascular endothelial growth factor | Interferon alpha | Blood vessels | Therapy | Genes | Tumors | Index Medicus | Interferon-alpha/pharmacology | Endothelial Growth Factors/genetics/metabolism | Tumor Cells; Cultured | Tumor Markers; Biological | Neuroendocrine Tumors/blood supply/drug therapy/metabolism | Antineoplastic Agents/pharmacology | Pregnancy Proteins/metabolism | Luciferases/metabolism | Promoter Regions (Genetics)/drug effects | Research Support; Non-U.S. Gov't | Genes; Reporter/drug effects | Mice; Nude | Gene Expression Regulation; Neoplastic/drug effects | RNA; Messenger/metabolism | Receptors; Vascular Endothelial Growth Factor/drug effects/genetics/metabolism | Receptors; Immunologic/metabolism | Intercellular Signaling Peptides and Proteins/genetics/metabolism | Neovascularization; Pathologic/drug therapy/metabolism | Angiogenesis Inhibitors/pharmacology | Lymphokines/drug effects/genetics/metabolism | Transcription; Genetic/drug effects | Transplantation; Heterologous
Journal Article
Genes and Development, ISSN 0890-9369, 08/2003, Volume 17, Issue 15, pp. 1835 - 1840
Several platelet-derived growth factor (PDGF) and vascular endothelial growth factor (VEGF) family members display C-terminal protein motifs that confer... 
Pericyte | Cell retention | Retina | Mesangial cell | PDGF | Heparan sulphate proteoglycan | DIABETIC-RETINOPATHY | VEGF ISOFORMS | pericyte | ANGIOGENESIS | A-CHAIN | DEVELOPMENTAL BIOLOGY | mesangial cell | CELL BIOLOGY | cell retention | retina | IN-VIVO | GENETICS & HEREDITY | MICE | heparan sulphate proteoglycan | HEPARAN-SULFATE PROTEOGLYCANS | GROWTH-FACTOR | BINDING | SMOOTH-MUSCLE CELLS | Kidney - physiology | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Microcirculation - metabolism | Retina - metabolism | Vascular Endothelial Growth Factors | Glomerulosclerosis, Focal Segmental - genetics | Molecular Sequence Data | Intercellular Signaling Peptides and Proteins - metabolism | Lymphokines - metabolism | Protein Structure, Tertiary | Amino Acid Sequence | Proto-Oncogene Proteins c-sis - genetics | Proto-Oncogene Proteins c-sis - metabolism | Retinal Degeneration - genetics | Pericytes - metabolism | Proteinuria - genetics | Amino Acid Motifs | Retina - physiology | Phenotype | Platelet-Derived Growth Factor - metabolism | Animals | Endothelium, Vascular - metabolism | Alleles | Mice | Models, Genetic | Mutation | Microscopy, Fluorescence | Cell Movement | Proteins | Blood platelets | Secretion | Analysis | Genetic research | Genetic aspects | Growth factors | Endothelium | Index Medicus | Research Communications | Glomerulosclerosis; Focal Segmental/genetics | Intercellular Signaling Peptides and Proteins/metabolism | Platelet-Derived Growth Factor/metabolism | Proto-Oncogene Proteins c-sis/genetics/metabolism | Proteinuria/genetics | Models; Genetic | Microcirculation/metabolism | Lymphokines/metabolism | Microscopy; Fluorescence | Pericytes/metabolism | Kidney/physiology | Protein Structure; Tertiary | Retinal Degeneration/genetics | Endothelial Growth Factors/metabolism | Endothelium; Vascular/metabolism | Retina/metabolism/physiology
Journal Article
Circulation, ISSN 0009-7322, 07/2003, Volume 108, Issue 2, pp. 198 - 204
Background-Intravitreal neovascular diseases, as in ischemic retinopathies, are a major cause of blindness. Because inflammatory mechanisms influence vitreal... 
Prostaglandins | Ischemia | Diabetes mellitus | Vasculature | vasculature | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | OXYGEN-INDUCED RETINOPATHY | diabetes mellitus | OCULAR NEOVASCULARIZATION | ischemia | RETINAL NEOVASCULARIZATION | INHIBITION | IN-VIVO | prostaglandins | PERIPHERAL VASCULAR DISEASE | PLATELET-ACTIVATING-FACTOR | GROWTH-FACTOR | PROSTAGLANDIN E-2 | Retina - drug effects | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Humans | Middle Aged | Astrocytes - pathology | Male | Astrocytes - enzymology | Receptors, Lipoprotein - metabolism | Diabetic Retinopathy - complications | Receptors, Prostaglandin E, EP4 Subtype | Retinal Vessels - pathology | Lymphokines - metabolism | Ischemia - pathology | Disease Models, Animal | Enzyme Inhibitors - pharmacology | Rats | Retinal Vessels - drug effects | Rats, Sprague-Dawley | Receptors, Prostaglandin E, EP3 Subtype | Cell Division - drug effects | Dinoprostone - metabolism | Membrane Proteins | Receptors, Scavenger | Neovascularization, Pathologic - drug therapy | Receptors, Immunologic | Mice | Thrombospondin 1 - metabolism | Vitreoretinopathy, Proliferative - enzymology | Retina - pathology | Diabetic Retinopathy - drug therapy | Vascular Endothelial Growth Factor A | Vitreoretinopathy, Proliferative - complications | Cyclooxygenase 2 | Ischemia - enzymology | Diabetic Retinopathy - pathology | Neovascularization, Pathologic - pathology | Intercellular Signaling Peptides and Proteins - metabolism | CD36 Antigens - metabolism | Ischemia - complications | Isoenzymes - metabolism | Retina - enzymology | Adult | Female | Astrocytes - drug effects | Vitreoretinopathy, Proliferative - drug therapy | Mice, Inbred C57BL | Vitreoretinopathy, Proliferative - pathology | Cells, Cultured | Receptors, Prostaglandin E - drug effects | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Animals | Diabetic Retinopathy - enzymology | Prostaglandin-Endoperoxide Synthases - metabolism | Aged | Isoenzymes - antagonists & inhibitors | Receptors, Prostaglandin E - metabolism | Index Medicus | Abridged Index Medicus | Enzyme Inhibitors | Astrocytes | Antigens, CD36 | Neurons and Cognition | Endothelial Growth Factors | Dinoprostone | Intercellular Signaling Peptides | Life Sciences | Cell Division | Diabetic Retinopathy
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2009, Volume 296, Issue 2, pp. 442 - 452
Platelet-derived growth factor (PDGF)-BB is a well-known smooth muscle (SM) cell (SMC) phenotypic modulator that signals by binding to PDGF αα-, αβ-, and... 
Shear stress | Disturbed blood flow | Smooth muscle α-actin | Smooth muscle myosin heavy chain | ALPHA-ACTIN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | smooth muscle alpha-actin | disturbed blood flow | MATRIX METALLOPROTEINASES | shear stress | smooth muscle myosin heavy chain | SHEAR-STRESS | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | CAROTID BIFURCATION | WALL SHEAR | EXPRESSION IN-VIVO | GROWTH FACTOR-BB | MARKER GENES | Up-Regulation | Phosphorylation | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Humans | Protein Multimerization | Actins - metabolism | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Stress, Mechanical | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Myosin Heavy Chains - metabolism | Platelet-Derived Growth Factor - genetics | Lymphokines - genetics | RNA Interference | Time Factors | Kruppel-Like Transcription Factors - metabolism | ets-Domain Protein Elk-1 - metabolism | Muscle Proteins - metabolism | Lymphokines - metabolism | Microfilament Proteins - metabolism | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Genes, Reporter | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Receptor, Platelet-Derived Growth Factor beta - metabolism | Calcium-Binding Proteins - metabolism | Recombinant Proteins - metabolism | Promoter Regions, Genetic | Atherosclerosis - physiopathology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Rats | Regional Blood Flow | Atherosclerosis - metabolism | Mice, Knockout | Phenotype | Platelet-Derived Growth Factor - metabolism | Animals | Apolipoproteins E - genetics | Mice | Protein Kinase Inhibitors - pharmacology | RNA, Small Interfering - metabolism | Proteins | Genotype & phenotype | Membranes | Arteriosclerosis | Genes | Ribonucleic acid--RNA | Cells | Index Medicus | smooth muscle α-actin
Journal Article
Cancer Research, ISSN 0008-5472, 12/2000, Volume 60, Issue 24, pp. 7075 - 7083
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 10/2017, Volume 18, Issue 10, p. 2097
We previously reported that, in multiple sclerosis (MS) patients with a progressive form of the disease, spinal cord periplaques extend distance away from... 
Neuroinflammation | Multiple sclerosis | Astrocytes | Myelin | Bioinformatics | myelin | ANDROGEN RECEPTOR | TGF-BETA | multiple sclerosis | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | CHEMISTRY, MULTIDISCIPLINARY | APPEARING WHITE-MATTER | MESSENGER-RNA | neuroinflammation | bioinformatics | LYSYL HYDROXYLASE | GROWTH | GENE-EXPRESSION | N-MYC | CENTRAL-NERVOUS-SYSTEM | astrocytes | Interleukin-12 Subunit p35 - metabolism | Oligodendroglia - metabolism | Spinal Cord - metabolism | Connexin 43 - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Cell Cycle Proteins - metabolism | Computational Biology | Interleukin-12 - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Glial Fibrillary Acidic Protein - metabolism | Myelin Sheath - metabolism | Interleukin-17 - metabolism | Inflammation - metabolism | Platelet-Derived Growth Factor - metabolism | Interleukin-33 - metabolism | Lymphokines - metabolism | Astrocytes - metabolism | Multiple Sclerosis - metabolism | Spinal cord | Smad protein | Genes | Interleukin | Transforming growth factor-a | Myc protein | Connexin 43 | Sclerosis | Demyelination | Smad2 protein | Oligodendrocytes | Interleukin 1 | Growth factors | Elongation | Translation | Transforming growth factor-b1 | Cytokines | Glial fibrillary acidic protein | Gap junctions | Data processing | Inflammation | Gene expression | Substantia alba | Patients | Gliosis | Translation elongation | Index Medicus | Life Sciences
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 10/2017, Volume 134, Issue 4, pp. 585 - 604
Treatment of acute ischemic stroke with the thrombolytic tissue plasminogen activator (tPA) can significantly improve neurological outcomes; however,... 
Pathology | Neurosciences | Stroke | Medicine & Public Health | LRP1 | Tissue plasminogen activator | α M β 2 | CD11b/CD18 | Mac-1 | Platelet-derived growth factor-CC | Blood–brain barrier | TISSUE-PLASMINOGEN ACTIVATOR | FOCAL CEREBRAL-ISCHEMIA | PATHOLOGY | IMATINIB TREATMENT | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | alpha(M)beta | SUBSTRATE-SPECIFICITY | Blood-brain barrier | FACTOR-ALPHA RECEPTOR | INTRACEREBRAL HEMORRHAGE | CENTRAL-NERVOUS-SYSTEM | DENSITY-LIPOPROTEIN RECEPTOR | INNATE IMMUNITY | Capillary Permeability - physiology | Cerebral Hemorrhage - metabolism | Leukocytes - pathology | Microglia - metabolism | Brain Ischemia - metabolism | Capillary Permeability - drug effects | Male | Tissue Plasminogen Activator - adverse effects | Stroke - pathology | Fibrinolytic Agents - adverse effects | Lymphokines - metabolism | Microglia - pathology | Female | Macrophage-1 Antigen - metabolism | Disease Models, Animal | Cerebral Hemorrhage - chemically induced | Tumor Suppressor Proteins - metabolism | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Cells, Cultured | Receptors, LDL - metabolism | Mice, Transgenic | Stroke - drug therapy | Arterioles - drug effects | Blood-Brain Barrier - drug effects | Blood-Brain Barrier - metabolism | Arterioles - pathology | Blood-Brain Barrier - pathology | Fibrinolytic Agents - pharmacology | Platelet-Derived Growth Factor - metabolism | Stroke - metabolism | Animals | Arterioles - metabolism | Tissue Plasminogen Activator - pharmacology | Macrophage-1 Antigen - genetics | Brain Ischemia - drug therapy | Brain Ischemia - pathology | CD11b Antigen - metabolism | Cerebral Hemorrhage - pathology | Leukocytes - metabolism | Bone Marrow Cells - metabolism | Stroke (Disease) | Platelet-derived growth factor | Ischemia | Analysis | Bone marrow | Transplantation | Permeability | Integrins | Phosphorylation | CD11b antigen | Arterioles | Membrane permeability | Leukocytes | Macrophages | Hemorrhage | Thrombolysis | t-Plasminogen activator | Microglia | Cerebrovascular system | Cell activation | Cerebral blood flow | Mac1 protein | Rodents | Immunofluorescence | Index Medicus | αMβ2 | CD18 | Original Paper | CD11b
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2016, Volume 478, Issue 2, pp. 845 - 851
Platelet-derived growth factor D (PDGF-D) signaling plays significant roles during the development and progression of human malignancies via interacting with... 
TSCC | PDGF-D/PDGFRβ | p38/AKT/ERK/EMT signal pathway | HEAD | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICROENVIRONMENTAL REGULATION | EMT | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | OVEREXPRESSION | BIOPHYSICS | DISSEMINATION | PDGF-D/PDGFR beta | NECK | BIOLOGICAL INSIGHTS | RNA, Small Interfering - genetics | Phosphorylation | Cadherins - metabolism | Vimentin - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | Epithelial-Mesenchymal Transition - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Antigens, CD - genetics | Platelet-Derived Growth Factor - genetics | Proto-Oncogene Proteins c-akt - genetics | Antigens, CD - metabolism | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | Lymphokines - genetics | Snail Family Transcription Factors - genetics | Vimentin - genetics | Lymphokines - antagonists & inhibitors | Lymphokines - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Cadherins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Tongue Neoplasms - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Snail Family Transcription Factors - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Neoplasm Invasiveness | Tongue Neoplasms - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Tongue Neoplasms - pathology | Disease Progression | Platelet-Derived Growth Factor - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Line, Tumor | Platelet-Derived Growth Factor - antagonists & inhibitors | Cell Movement | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 11/2002, Volume 13, Issue 11, pp. 4029 - 4044
Interactions between cancer cells and their microenvironment are critical for the development and progression of solid tumors. This study is the first to... 
Immunohistochemistry | Receptor, Epidermal Growth Factor - genetics | Up-Regulation | Endothelial Growth Factors - metabolism | Receptor, ErbB-3 - metabolism | Adenocarcinoma - pathology | Receptor, ErbB-2 - genetics | Vascular Endothelial Growth Factors | Humans | Receptor, ErbB-2 - chemistry | Sp1 Transcription Factor - metabolism | Receptor, ErbB-3 - chemistry | Adenocarcinoma - metabolism | Lymphokines - metabolism | Transcription, Genetic | Dimerization | Enzyme Inhibitors - metabolism | Neoplasms - blood supply | Receptor, ErbB-3 - genetics | Receptor, Epidermal Growth Factor - chemistry | Mice, Nude | Recombinant Fusion Proteins - genetics | Mice | Mitogen-Activated Protein Kinase 1 - metabolism | Neoplasms - metabolism | Neoplasm Transplantation | Vascular Endothelial Growth Factor A | Neovascularization, Pathologic | Receptor, ErbB-2 - metabolism | Transplantation, Heterologous | Recombinant Fusion Proteins - metabolism | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | Lymphokines - genetics | Genes, Reporter | Cell Line | Nitriles - metabolism | Promoter Regions, Genetic | Intercellular Signaling Peptides and Proteins - genetics | Kruppel-Like Transcription Factors | Transcription Factors - metabolism | Animals | Butadienes - metabolism | Endothelial Growth Factors - genetics | Neuregulin-1 - metabolism | Signal Transduction - physiology | Neoplasms - pathology | Receptor, ErbB-4 | Mitogen-Activated Protein Kinase 3 | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 02/2001, Volume 93, Issue 4, pp. 309 - 314
Journal Article
Experimental Cell Research, ISSN 0014-4827, 03/2017, Volume 352, Issue 1, pp. 146 - 156
Autophagy constitutes a defense mechanism to overcome aging and apoptosis in osteoarthritic cartilage. Several cytokines and transcription factors are linked... 
Platelet rich plasma | Anti-inflammatory cytokines | Osteoarthritis | Autophagy | Apoptosis | PATHWAYS | CELLS | CHONDROCYTES | MECHANISMS | FOXO TRANSCRIPTION FACTORS | PROTECTS | CELL BIOLOGY | PATHOGENESIS | ONCOLOGY | ARTICULAR-CARTILAGE | GENE-EXPRESSION | DEGRADATION | Chondrocytes - cytology | Cell Proliferation | Humans | Middle Aged | Male | Anti-Inflammatory Agents - metabolism | Platelet-Rich Plasma - metabolism | Case-Control Studies | Young Adult | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Interleukin-13 - genetics | Cartilage, Articular - metabolism | Interleukin-10 - metabolism | Adult | Female | Interleukin-4 - genetics | Osteoarthritis - pathology | Real-Time Polymerase Chain Reaction | Chondrocytes - metabolism | Cartilage, Articular - cytology | Osteoarthritis - prevention & control | Interleukin-4 - metabolism | Matrix Metalloproteinase 13 - genetics | RNA, Messenger - genetics | Cells, Cultured | Osteoarthritis - metabolism | Matrix Metalloproteinase 3 - metabolism | Interleukin-13 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Matrix Metalloproteinase 13 - metabolism | Tissue Inhibitor of Metalloproteinase-1 - genetics | Transforming Growth Factor beta - genetics | Interleukin-10 - genetics | Matrix Metalloproteinase 3 - genetics | Transforming Growth Factor beta - metabolism | Physiological aspects | Diagnosis | Anti-inflammatory drugs | Proteases | Analysis | Bone morphogenetic proteins | Index Medicus | TRANSCRIPTION FACTORS | APOPTOSIS | COLLAGEN | ECOLOGICAL CONCENTRATION | MICROTUBULES | SAFETY | TRANSCRIPTION | CYSTEINE | 60 APPLIED LIFE SCIENCES | FIBROBLASTS | CARTILAGE | INSULIN | LYMPHOMAS | NECROSIS | POLYMERASE CHAIN REACTION | THERAPY | CHAIN REACTIONS | LYMPHOKINES | POLYMERASES | CULTURE MEDIA | INFLAMMATION | YEASTS | ENZYME IMMUNOASSAY | PLANT GROWTH
Journal Article