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International Journal of Cancer, ISSN 0020-7136, 12/2014, Volume 135, Issue 11, pp. 2711 - 2720
Cyclophosphamide–dacarbazine–vincristine regimen is recommended for the treatment of malignant pheochromocytoma and paraganglioma (MPP); however, dacarbazine... 
succinate dehydrogenase B (SDHB) | temozolomide | malignant pheochromocytoma | O‐methylguanine‐DNA methyltransferase (MGMT) | paraganglioma | O-methylguanine-DNA methyltransferase (MGMT) | NEUROENDOCRINE TUMORS | SOLID TUMORS | BONE METASTASES | CYCLOPHOSPHAMIDE | FOLLOW-UP | CHROMOGRANIN-A | DACARBAZINE | ONCOLOGY | NEURON-SPECIFIC ENOLASE | SOMATOSTATIN RECEPTOR SCINTIGRAPHY | Adrenal Gland Neoplasms - drug therapy | Prognosis | Follow-Up Studies | Dacarbazine - therapeutic use | Humans | Middle Aged | Brain Neoplasms - pathology | DNA Repair Enzymes - genetics | Male | Pheochromocytoma - mortality | Promoter Regions, Genetic - genetics | Paraganglioma - secondary | Adrenal Gland Neoplasms - mortality | DNA Methylation | Neoplasm Metastasis | Paraganglioma - mortality | Tumor Suppressor Proteins - genetics | Dacarbazine - analogs & derivatives | Polymerase Chain Reaction | Aged, 80 and over | Adult | Female | Retrospective Studies | Brain Neoplasms - mortality | Pheochromocytoma - secondary | Adrenal Gland Neoplasms - pathology | Paraganglioma - genetics | Brain Neoplasms - genetics | Paraganglioma - drug therapy | Survival Rate | Brain Neoplasms - drug therapy | Mutation - genetics | Antineoplastic Agents, Alkylating - therapeutic use | Pheochromocytoma - drug therapy | DNA Modification Methylases - genetics | Succinate Dehydrogenase - genetics | Pheochromocytoma - genetics | Aged | Biomarkers, Tumor - genetics | Neoplasm Staging | Adrenal Gland Neoplasms - genetics | Hypertension | Immunohistochemistry | Antimitotic agents | Care and treatment | Pheochromocytoma | Gene mutations | Transferases | Genetic aspects | Metastasis | Methylation | Antineoplastic agents | Chemotherapy | Dehydrogenases | Mutation | Drug therapy | Tumors | Cancer | Succinate dehydrogenase | Toxicity | Methylguanine | Paraganglioma | Gene expression | Patients | Vincristine | Metastases | Anticancer properties | O6-methylguanine-DNA methyltransferase | Dacarbazine | Cyclophosphamide | Lymphopenia | DNA methylation | DNA methyltransferase | Temozolomide | Deoxyribonucleic acid--DNA | Neuroendocrine tumors | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2006, Volume 177, Issue 2, pp. 1062 - 1069
Journal Article
Journal Article
Cell, ISSN 0092-8674, 01/2006, Volume 124, Issue 2, pp. 287 - 299
Journal Article
PLOS ONE, ISSN 1932-6203, 11/2015, Volume 10, Issue 11, p. e0143216
Chromosomal translocations are driver mutations of human cancers, particularly leukemias. They define disease subtypes and are used as prognostic markers, for... 
CHROMOSOMAL TRANSLOCATIONS | LEUCINE-ZIPPER | GENE | ACUTE-LYMPHOBLASTIC-LEUKEMIA | E2A | MULTIDISCIPLINARY SCIENCES | CHIMERIC TRANSCRIPTION FACTOR | ANTIAPOPTOTIC ACTIVITY | TRANSACTIVATION DOMAINS | LINEAGE LEUKEMIA | TRANSGENIC MICE | Hepatomegaly - pathology | Translocation, Genetic | Oncogene Proteins, Fusion - metabolism | Basic-Leucine Zipper Transcription Factors - metabolism | Humans | Precursor Cells, B-Lymphoid - metabolism | Antigens, CD19 - genetics | Splenomegaly - pathology | Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Cell Death - genetics | Precursor Cells, B-Lymphoid - pathology | Cell Transformation, Neoplastic - genetics | Integrases - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Disease Models, Animal | Promoter Regions, Genetic | Gene Expression | Splenomegaly - genetics | Mice, Transgenic | Basic-Leucine Zipper Transcription Factors - genetics | Hepatomegaly - genetics | Splenomegaly - metabolism | Cell Transformation, Neoplastic - metabolism | Gene Knock-In Techniques | CD79 Antigens - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Animals | Genetic Engineering | Oncogene Proteins, Fusion - genetics | Antigens, CD19 - metabolism | Hepatomegaly - metabolism | Mice | CD79 Antigens - genetics | Cell Transformation, Neoplastic - pathology | Myxovirus Resistance Proteins - genetics | Integrases - genetics | Myxovirus Resistance Proteins - metabolism | Genetic aspects | B cells | Health aspects | Leukemia | Hematopoietic stem cells | Flow cytometry | Transformation | Transcription factors | Laboratories | Genomics | Genes | Proteins | Precursors | Transgenic animals | Rodents | Bone marrow | Translocation | Immunoglobulins | CD19 antigen | Anemia | Mortality | Minimal residual disease | Myelocytes | Progenitor cells | Hemopoiesis | Pathology | Chromosome translocations | Leukocytosis | Lymphocytes B | Lymphopenia | Cell death | Medical prognosis | Cells (biology) | Stem cells | Comparative studies | Infiltration | Mutation | Apoptosis
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 2, pp. 634 - 642.e5
Background Signaling through the T-cell receptor (TCR) is critical for T-cell development and function. Linker for activation of T cells (LAT) is a... 
Allergy and Immunology | T lymphopenia | immunodeficiency | linker for activation of T cells | Severe combined immunodeficiency | genetic defect | T-cell receptor signaling | TYROSINE PHOSPHORYLATION | linker for activation of T cells | ANTIGEN RECEPTOR COMPLEX | DEATH | IMMUNOLOGY | DEFICIENCY | SIGNAL-TRANSDUCTION | ALLERGY | DISEASE | MICE | CD3-EPSILON GENE | ADAPTER PROTEIN | EXPRESSION | T-Lymphocytes - physiology | Receptors, Antigen, T-Cell - metabolism | Jurkat Cells | Lymphocyte Activation | Membrane Proteins - genetics | Humans | Transgenes - genetics | Genotype | Male | Homozygote | Severe Combined Immunodeficiency - genetics | Pakistan | Pedigree | Adaptor Proteins, Signal Transducing - genetics | Female | Cell Differentiation | Consanguinity | Membrane Proteins - metabolism | Receptors, Antigen, T-Cell - genetics | Adaptor Proteins, Signal Transducing - metabolism | Calcium Signaling - genetics | Apoptosis | Sequence Deletion - genetics | Medical genetics | Bone marrow | Genetic aspects | Transplantation | Children | Research institutes | T cells | Health aspects | Consulting services | Killer cells | Analysis | Genetic research | Genetic polymorphisms | Phosphorylation | Immunoglobulins | Transplants & implants | Genes | Kinases | Defects | Proteins | Studies | Signal transduction | Lymphocytes | Rodents | Mutation | Deoxyribonucleic acid--DNA
Journal Article
Nature Immunology, ISSN 1529-2908, 06/2017, Volume 18, Issue 7, pp. 813 - 823
The transcriptional programs that guide lymphocyte differentiation depend on the precise expression and timing of transcription factors (TFs). The TF BACH2 is... 
TRANSCRIPTION FACTORS | METAANALYSIS | DATABASE | AUTOIMMUNITY | COMMON VARIABLE IMMUNODEFICIENCY | CELL DIFFERENTIATION | CLASSIFICATION | IMMUNOLOGY | IDENTIFICATION | T-CELLS | GENOME-WIDE ASSOCIATION | Recurrence | Colitis - genetics | Fever - complications | Humans | Middle Aged | Colitis - complications | Colitis - pathology | Male | Tomography, X-Ray Computed | Adrenal Cortex Hormones - therapeutic use | Respiratory Tract Infections - genetics | Autoimmune Diseases - genetics | Young Adult | Splenomegaly - complications | Pancytopenia - drug therapy | Adult | Female | Respiratory Tract Infections - complications | Immunologic Deficiency Syndromes - complications | Splenomegaly - genetics | Autoimmune Diseases - complications | Basic-Leucine Zipper Transcription Factors - genetics | Lymphopenia - complications | Syndrome | Haploinsufficiency | Pancytopenia - complications | Pedigree | Fever - genetics | Immunologic Deficiency Syndromes - genetics | Heterozygote | Polymorphism, Single Nucleotide | Mutation | Lymphopenia - genetics | Fever - drug therapy | Pancytopenia - genetics | Respiratory Tract Infections - diagnostic imaging | Lymphocytes | Immunodeficiency | Research | Transcription factors | Maturation | Architecture | Genes | Genomes | Lymphocytes T | Agglomeration | Immunity | Defects | Gene sequencing | Diseases | Enhancers | Lymphocytes B | Intestine | Rodents | Autoimmune diseases | Differentiation
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2011, Volume 108, Issue 19, pp. 7890 - 7895
Transporters at the hepatic canalicular membrane are essential for the formation of bile and the prevention of cholestatic liver disease. One such example is... 
Cholic acids | Bile acids | Hyperbilirubinemia | Hepatocytes | B lymphocytes | Liver | Mice | Genetic mutation | Blood plasma | Bile | Jaundice | Gallbladder | Uterus | Enterohepatic circulation | Bile acid | HOMOZYGOUS DISRUPTION | HOMEOSTASIS | gallbladder | ACID | BILE-SALTS | bile acid | MULTIDISCIPLINARY SCIENCES | jaundice | IDENTIFICATION | DEFICIENCY | uterus | DISEASE | MICE | P-GLYCOPROTEIN GENE | enterohepatic circulation | FAMILIAL INTRAHEPATIC CHOLESTASIS | Genetic Diseases, X-Linked - enzymology | Male | DNA Primers - genetics | Lymphopenia - enzymology | Lymphopenia - pathology | Mitochondrial Proton-Translocating ATPases - genetics | Genes, X-Linked | Mice, Mutant Strains | Base Sequence | Female | Genetic Diseases, X-Linked - genetics | B-Lymphocytes - pathology | Cholic Acid - toxicity | Disease Models, Animal | Cholestasis, Intrahepatic - genetics | Dystocia - genetics | Hyperbilirubinemia, Hereditary - genetics | Cholestasis, Intrahepatic - pathology | Mice, Inbred C57BL | Molecular Chaperones - genetics | Molecular Chaperones - physiology | Cholestasis, Intrahepatic - enzymology | Dystocia - enzymology | Pregnancy | Homozygote | Hyperbilirubinemia, Hereditary - enzymology | Phenotype | Animals | Mitochondrial Proton-Translocating ATPases - physiology | Mutation | Lymphopenia - genetics | Cholic Acid - administration & dosage | Care and treatment | Jaundice, Obstructive | Genetic aspects | Gene mutations | Identification and classification | Cholestasis | Biological Sciences
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2011, Volume 186, Issue 7, pp. 3874 - 3881
Journal Article
NATURE IMMUNOLOGY, ISSN 1529-2908, 01/2018, Volume 19, Issue 1, pp. 29 - 29
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 08/2015, Volume 181, Issue 2, pp. 343 - 356
Summary Exaggerated activation of lymphocytes contributes to the pathogenesis of inflammatory bowel disease (IBD). Medical therapies are linked to the BCL‐2... 
BCL‐2 | apoptosis | IBD | mucosal T cell turnover | ABT‐737 | BIM | Mucosal T cell turnover | ABT-737 | BCL-2 | Apoptosis | SURVIVAL | SYSTEMIC-LUPUS-ERYTHEMATOSUS | CROHNS-DISEASE | AZATHIOPRINE | T-CELL | IMMUNOLOGY | LAMINA PROPRIA | INFLAMMATORY-BOWEL-DISEASE | IN-VIVO | RESISTANCE | MICE | Colitis | Lymphocytes | Analysis | Translational
Journal Article