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Annals of Oncology, ISSN 0923-7534, 2008, Volume 19, Issue 11, pp. 1894 - 1902
Journal Article
Journal of Biomedical Nanotechnology, ISSN 1550-7033, 11/2015, Volume 11, Issue 11, pp. 2057 - 2066
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 1999, Volume 65, Issue 6, pp. 1647 - 1655
Journal Article
Atherosclerosis, ISSN 0021-9150, 2007, Volume 198, Issue 1, pp. 166 - 173
Abstract Background From initiation to plaque rupture, immune system components contribute to atherosclerosis. We investigated variation in... 
Cardiovascular | Myocardial infarction | Inflammation | Ischemic stroke | Genetic epidemiology | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | myocardial infarction | SUSCEPTIBILITY | MARKERS | VARIANT | GENOME | genetic epidemiology | POLYMORPHISMS | inflammation | COPY NUMBER | PERIPHERAL VASCULAR DISEASE | LYMPHOTOXIN-ALPHA GENE | CORONARY-HEART-DISEASE | ASSOCIATION | ischemic stroke | Myocardial Infarction - genetics | Interleukin-18 - immunology | Myocardial Infarction - epidemiology | Vasculitis - epidemiology | Intracranial Arteriosclerosis - immunology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Brain Ischemia - genetics | Male | Brain Ischemia - immunology | Interleukin-1beta - genetics | Case-Control Studies | Myocardial Infarction - immunology | Stroke - genetics | Incidence | Vasculitis - immunology | Genetic Variation | Vasculitis - genetics | Tumor Necrosis Factor-alpha - immunology | Adult | Female | Stroke - epidemiology | Genetic Predisposition to Disease - epidemiology | Stroke - immunology | Brain Ischemia - epidemiology | Interleukin-6 - genetics | Lymphotoxin-beta - immunology | Lymphotoxin-beta - genetics | Risk Factors | Intracranial Arteriosclerosis - genetics | Interleukin-1beta - immunology | C-Reactive Protein - genetics | Interleukin-10 - genetics | Intracranial Arteriosclerosis - epidemiology | Coronary Artery Disease - genetics | Interleukin-6 - immunology | Aged | Coronary Artery Disease - epidemiology | Coronary Artery Disease - immunology | Interleukin-10 - immunology | Interleukin-18 - genetics | C-Reactive Protein - immunology | Stroke (Disease) | Genetic research | Ischemia | Tumor necrosis factor | Heart attack | Atherosclerosis
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 24, Issue 2, pp. 315 - 324
Summary Objective The aetiology of OA is not fully understood although several adipokines such as leptin are known mediators of disease progression. Since... 
Rheumatology | Smad1 | Human chondrocytes | Hip osteoarthritis | β-Catenin | Dedifferentiation | Leptin | COLLAGEN | RHEUMATOLOGY | DIFFERENTIATED PHENOTYPE | HYPERTROPHY | IN-VITRO | SYNOVIAL FIBROBLASTS | HUMAN OSTEOARTHRITIC CARTILAGE | SIGNALING PATHWAY | beta-Catenin | ARTICULAR CHONDROCYTES | ORTHOPEDICS | KNEE OSTEOARTHRITIS | EXPRESSION | Receptors, Transforming Growth Factor beta - genetics | Humans | Leptin - metabolism | Middle Aged | Male | Lymphotoxin-alpha - metabolism | RNA, Messenger - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Lymphotoxin-alpha - genetics | Cartilage, Articular - metabolism | Smad5 Protein - metabolism | Smad2 Protein - genetics | Aged, 80 and over | Smad2 Protein - drug effects | Protein-Serine-Threonine Kinases - metabolism | Collagen Type X - drug effects | SOX9 Transcription Factor - metabolism | Matrix Metalloproteinase 13 - genetics | Smad2 Protein - metabolism | Prednisolone - pharmacology | beta Catenin - metabolism | Matrix Metalloproteinase 13 - metabolism | Glycogen Synthase Kinase 3 - genetics | Receptors, Transforming Growth Factor beta - drug effects | Activin Receptors, Type II - genetics | Protein-Serine-Threonine Kinases - drug effects | Activin Receptors, Type II - metabolism | Osteoarthritis, Hip - metabolism | Glycogen Synthase Kinase 3 - drug effects | Activin Receptors, Type II - drug effects | Chondrocytes - drug effects | Collagen Type X - metabolism | beta Catenin - drug effects | Smad1 Protein - genetics | Adult | Female | Smad5 Protein - genetics | Chondrocytes - metabolism | RNA, Messenger - drug effects | Cell Dedifferentiation - drug effects | Cartilage, Articular - cytology | Lymphotoxin-alpha - drug effects | Protein-Serine-Threonine Kinases - genetics | Collagen Type X - genetics | SOX9 Transcription Factor - drug effects | Matrix Metalloproteinase 13 - drug effects | Glycogen Synthase Kinase 3 - metabolism | beta Catenin - genetics | Osteoarthritis, Hip - genetics | Smad5 Protein - drug effects | Core Binding Factor Alpha 1 Subunit - drug effects | Wnt Signaling Pathway - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Wnt Signaling Pathway - genetics | Glucocorticoids - pharmacology | Smad1 Protein - metabolism | Aged | Smad1 Protein - drug effects | Cartilage, Articular - drug effects | In Vitro Techniques | Cell Dedifferentiation - genetics | Core Binding Factor Alpha 1 Subunit - genetics
Journal Article
Chest, ISSN 0012-3692, 09/2003, Volume 124, Issue 3, pp. 1103 - 1115
Genetic epidemiologic studies suggest a strong genetic influence on the outcome from sepsis, and genetics may explain the wide variation in the individual... 
Heat-shock proteins | Sepsis syndrome | Interleukins | Septicemia | Tumor necrosis factor | Polymorphism | DEPENDENT DIABETES-MELLITUS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANTAGONIST | SUSCEPTIBILITY | polymorphism | MAJOR HISTOCOMPATIBILITY COMPLEX | heat-shock proteins | INTERLEUKIN-6 | interleukins | REGION | septicemia | tumor necrosis factor | RESPIRATORY SYSTEM | NECROSIS-FACTOR TNF | DISEASE | ALPHA PROMOTER | sepsis syndrome | ASSOCIATION | Toll-Like Receptor 2 | Toll-Like Receptor 4 | Humans | Tumor Necrosis Factor-alpha - genetics | Antibodies, Monoclonal - therapeutic use | Male | Acute-Phase Proteins - genetics | Systemic Inflammatory Response Syndrome - genetics | Protein C - antagonists & inhibitors | Heat-Shock Proteins - genetics | Lymphotoxin-alpha - genetics | Toll-Like Receptors | Shock, Septic - drug therapy | Adult | Cytokines - genetics | Shock, Septic - genetics | Gene Expression Regulation - physiology | Genotype | Membrane Glycoproteins - genetics | Alleles | Polymorphism, Genetic - genetics | Systemic Inflammatory Response Syndrome - drug therapy | Lymphotoxin-alpha - antagonists & inhibitors | Protein C - genetics | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Receptors, Cell Surface - genetics | Evaluation | Bacterial infections | Physiological aspects | Heat shock proteins | Influence | Septic shock | Genetic aspects | Statistics | Genetic polymorphisms
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e46113
Journal Article
Gut, ISSN 0017-5749, 08/2004, Volume 53, Issue 8, pp. 1082 - 1089
Journal Article
Genes and Immunity, ISSN 1466-4879, 09/2006, Volume 7, Issue 6, pp. 450 - 467
Since the complete sequencing of a human major histocompatibility complex (MHC) haplotype, interest in non-human leucocyte antigen (HLA) genes encoded in the... 
LINKAGE-DISEQUILIBRIUM MAP | NOTCH4 GENE | human leukocyte antigens | immunogenetics | INCLUSION-BODY MYOSITIS | major histocompatibility complex | IMMUNOLOGY | CLOZAPINE-INDUCED AGRANULOCYTOSIS | CENTRAL MHC GENE | SHOCK PROTEIN-70 HSP70-2 | CLASS-III REGION | GENETICS & HEREDITY | genetic predisposition to disease | HLA-DRB GENES |