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British Journal of Haematology, ISSN 0007-1048, 12/2015, Volume 171, Issue 5, pp. 736 - 751
Journal Article
Immunity, ISSN 1074-7613, 06/2015, Volume 42, Issue 6, pp. 1100 - 1115
Journal Article
Cancer Cell, ISSN 1535-6108, 2009, Volume 16, Issue 4, pp. 295 - 308
Hepatitis B and C viruses (HBV and HCV) cause chronic hepatitis and hepatocellular carcinoma (HCC) by poorly understood mechanisms. We show that cytokines... 
CELLCYCLE | LYMPHOID-TISSUES | ONCOLOGY | SIGNAL PATHWAY | CHRONIC HEPATITIS-C | LIVER | VIRUS CORE PROTEIN | OVAL CELLS | CELL-LINES | NF-KAPPA-B | EXPRESSION | BETA-RECEPTOR | Liver - virology | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Hepatitis B, Chronic - immunology | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Male | Lymphotoxin-alpha - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | RNA, Messenger - metabolism | Case-Control Studies | Lymphotoxin beta Receptor - metabolism | Lymphotoxin-alpha - genetics | Liver - immunology | Lymphocytes - immunology | I-kappa B Kinase - metabolism | Lymphocytes - virology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Carcinoma, Hepatocellular - genetics | Biomarkers, Tumor - metabolism | Carcinoma, Hepatocellular - immunology | Liver Neoplasms - virology | Recombinant Proteins - metabolism | Liver Neoplasms - genetics | Signal Transduction | Lymphotoxin-beta - genetics | Mice, Inbred C57BL | Liver Neoplasms - immunology | Mice, Transgenic | Carcinoma, Hepatocellular - virology | I-kappa B Kinase - genetics | Hepatocytes - immunology | Receptors, Tumor Necrosis Factor, Type I - genetics | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | Receptors, Tumor Necrosis Factor, Type II - genetics | Homeodomain Proteins - genetics | Mice, Knockout | Lymphotoxin-beta - metabolism | Animals | Lymphotoxin beta Receptor - genetics | Chromosome Aberrations | Hepatitis C, Chronic - immunology | Hepatocytes - virology | Ligands | Chemokines - metabolism | Mice | Cell Transformation, Viral | Medical colleges | Liver cancer | Hepatoma | Hepatitis C | Lymphocytes | Health aspects
Journal Article
Atherosclerosis, ISSN 0021-9150, 2007, Volume 198, Issue 1, pp. 166 - 173
Abstract Background From initiation to plaque rupture, immune system components contribute to atherosclerosis. We investigated variation in... 
Cardiovascular | Myocardial infarction | Inflammation | Ischemic stroke | Genetic epidemiology | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | myocardial infarction | SUSCEPTIBILITY | MARKERS | VARIANT | GENOME | genetic epidemiology | POLYMORPHISMS | inflammation | COPY NUMBER | PERIPHERAL VASCULAR DISEASE | LYMPHOTOXIN-ALPHA GENE | CORONARY-HEART-DISEASE | ASSOCIATION | ischemic stroke | Myocardial Infarction - genetics | Interleukin-18 - immunology | Myocardial Infarction - epidemiology | Vasculitis - epidemiology | Intracranial Arteriosclerosis - immunology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Brain Ischemia - genetics | Male | Brain Ischemia - immunology | Interleukin-1beta - genetics | Case-Control Studies | Myocardial Infarction - immunology | Stroke - genetics | Incidence | Vasculitis - immunology | Genetic Variation | Vasculitis - genetics | Tumor Necrosis Factor-alpha - immunology | Adult | Female | Stroke - epidemiology | Genetic Predisposition to Disease - epidemiology | Stroke - immunology | Brain Ischemia - epidemiology | Interleukin-6 - genetics | Lymphotoxin-beta - immunology | Lymphotoxin-beta - genetics | Risk Factors | Intracranial Arteriosclerosis - genetics | Interleukin-1beta - immunology | C-Reactive Protein - genetics | Interleukin-10 - genetics | Intracranial Arteriosclerosis - epidemiology | Coronary Artery Disease - genetics | Interleukin-6 - immunology | Aged | Coronary Artery Disease - epidemiology | Coronary Artery Disease - immunology | Interleukin-10 - immunology | Interleukin-18 - genetics | C-Reactive Protein - immunology | Stroke (Disease) | Genetic research | Ischemia | Tumor necrosis factor | Heart attack | Atherosclerosis
Journal Article
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 6, pp. 533 - 542
Known genetic loci explain only a small proportion of the familial relative risk of colorectal cancer (CRC). We conducted a genome-wide association study of... 
METAANALYSIS | LYMPHOTOXIN-BETA-RECEPTOR | VARIANTS | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | PROLIFERATION | NUCLEOREDOXIN | CELL-GROWTH | MUTATIONS | EXPRESSION | GENOME-WIDE ASSOCIATION | Quantitative trait loci | Genetic susceptibility | Colorectal cancer | Genetic research | Genetic aspects | Research | Identification and classification | Risk factors | Studies | Medical research | Genes
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2012, Volume 71, Issue 6, pp. 981 - 988
Journal Article
Immunological Reviews, ISSN 0105-2896, 11/2011, Volume 244, Issue 1, pp. 169 - 187
The tumor necrosis factor (TNF) receptor superfamily member herpesvirus entry mediator (HVEM) (TNFRSF14) regulates T‐cell immune responses by activating both... 
asthma | cancer | cytokines | autoimmunity | T cells | inflammation | Autoimmunity | Inflammation | Cytokines | Asthma | Cancer | DECOY RECEPTOR-3 | T-LYMPHOCYTE ATTENUATOR | IMMUNOLOGY | VERSUS-HOST-DISEASE | CUTTING EDGE | MHC CLASS-I | B-LYMPHOCYTE | LYMPHOTOXIN-BETA-RECEPTOR | NK-CELL ACTIVATION | SIMPLEX-VIRUS | GLYCOPROTEIN-D | Tumor Necrosis Factor-alpha - metabolism | Antigens, CD - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | Lymphotoxin-alpha - metabolism | Antigens, CD - genetics | Lymphotoxin-alpha - immunology | Tumor Necrosis Factor Ligand Superfamily Member 14 - genetics | Antigens, CD - metabolism | Lymphotoxin beta Receptor - metabolism | Lymphotoxin-alpha - genetics | Signal Transduction - immunology | T-Lymphocytes - metabolism | Gene Expression - immunology | Tumor Necrosis Factor-alpha - immunology | Viral Envelope Proteins - metabolism | Herpesvirus 1, Human - immunology | Receptors, Immunologic - immunology | Receptors, Tumor Necrosis Factor, Member 14 - immunology | Receptors, Tumor Necrosis Factor, Member 14 - genetics | GPI-Linked Proteins - immunology | Tumor Necrosis Factor Ligand Superfamily Member 14 - immunology | Lymphocyte Activation | Immunity, Innate | Protein Binding - immunology | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | GPI-Linked Proteins - metabolism | Herpes Simplex - immunology | Mice, Knockout | Herpes Simplex - metabolism | Receptor Cross-Talk - immunology | Herpes Simplex - virology | Receptors, Tumor Necrosis Factor, Member 14 - metabolism | Animals | Lymphotoxin beta Receptor - genetics | T-Lymphocytes - immunology | Viral Envelope Proteins - immunology | Mice | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | GPI-Linked Proteins - genetics | Lymphotoxin beta Receptor - immunology | Immunoglobulins | Analysis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2016, Volume 113, Issue 26, pp. E3716 - E3724
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 08/2017, Volume 23, Issue 15, pp. 4388 - 4401
Purpose: In head and neck squamous cell carcinoma (HNSCC), the incidence of RAS mutation, which is the major cause of cetuximab resistance, is relatively rare... 
EPITHELIAL-MESENCHYMAL TRANSITION | ACTIVATION | ONCOLOGY | COLORECTAL-CANCER | SQUAMOUS-CELL CARCINOMA | RECEPTOR | ALPHA | CHEMORESISTANCE | NF-KAPPA-B | TNF | EXPRESSION | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | ErbB Receptors - genetics | Epithelial-Mesenchymal Transition - drug effects | Protein Kinase Inhibitors - adverse effects | Epithelial-Mesenchymal Transition - genetics | Cetuximab - adverse effects | Squamous Cell Carcinoma of Head and Neck | Snail Family Transcription Factors - genetics | Protein-Arginine N-Methyltransferases - genetics | Disease Models, Animal | NF-kappa B - antagonists & inhibitors | ErbB Receptors - antagonists & inhibitors | Lymphotoxin-beta - genetics | Head and Neck Neoplasms - drug therapy | Repressor Proteins - genetics | Zebrafish | Head and Neck Neoplasms - pathology | Xenograft Model Antitumor Assays | Protein Kinase Inhibitors - administration & dosage | Drug Resistance, Neoplasm - genetics | Animals | Carcinoma, Squamous Cell - drug therapy | NF-kappa B - genetics | Cell Line, Tumor | Cetuximab - administration & dosage | Head and Neck Neoplasms - genetics | Mice | Mutation | Zebrafish Proteins - genetics | Lymphotoxin | Tyrosine | Drugs | NF-κB protein | Squamous cell carcinoma | Epidermal growth factor receptors | Mesenchyme | Protein arginine methyltransferase | Drug resistance | Inhibitors | Arginine | Experimental design | Tumor necrosis factor | Point mutation | Head and neck cancer | In vivo methods and tests | Synergistic effect | Dimerization | Protein-tyrosine kinase | Cancer
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166589
LIGHT (HVEM-L, TNFSF14, or CD258), an entity homologous to lymphotoxins, with inducible nature and the ability to compete with herpes simplex virus... 
VIRUS ENTRY MEDIATOR | MULTIDISCIPLINARY SCIENCES | CO-STIMULATION | METHODOLOGICAL APPROACH | ALPHA | AMYOTROPHIC-LATERAL-SCLEROSIS | LYMPHOTOXIN/LIGHT | SOLUBLE DECOY RECEPTOR-3 | STROMAL CELLS | VERSUS-HOST-DISEASE | TRANSPLANTATION | Osteocytes - drug effects | Cyclin-Dependent Kinases - metabolism | Humans | Cyclins - genetics | Adipocytes - drug effects | Lymphotoxin beta Receptor - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Mesenchymal Stromal Cells - cytology | Cyclins - metabolism | Microarray Analysis | STAT3 Transcription Factor - genetics | Cell Survival - drug effects | Cyclin-Dependent Kinase 2 - metabolism | Mesenchymal Stromal Cells - drug effects | Signal Transduction | Osteocytes - cytology | Cyclin-Dependent Kinase 2 - genetics | Mesenchymal Stromal Cells - metabolism | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | Lymphotoxin beta Receptor - genetics | CDC2 Protein Kinase | Chondrocytes - cytology | Tumor Necrosis Factor Ligand Superfamily Member 14 - pharmacology | Adipocytes - cytology | Smad3 Protein - metabolism | Platelet-Derived Growth Factor - genetics | Tumor Necrosis Factor Ligand Superfamily Member 14 - genetics | Chondrocytes - drug effects | Smad3 Protein - genetics | Bone Marrow Cells - drug effects | Cyclin-Dependent Kinases - genetics | Chondrocytes - metabolism | STAT3 Transcription Factor - metabolism | Recombinant Proteins - metabolism | Osteocytes - metabolism | Bone Marrow Cells - cytology | Gene Expression Regulation | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Platelet-Derived Growth Factor - metabolism | Transforming Growth Factor beta - genetics | Cell Cycle | Cell Differentiation - drug effects | Adipocytes - metabolism | Cell Proliferation - drug effects | Primary Cell Culture | Transforming Growth Factor beta - metabolism | Bone Marrow Cells - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Platelet-derived growth factor | Dendritic cells | Tumor necrosis factor | Stem cells | Transplantation | B cells | T cells | Transforming growth factors | Enzyme-linked immunosorbent assay | Herpes simplex | Immunoblotting | Adipocytes | Alizarin | Proteins | Signal transduction | Receptors | Cell growth | Light | Bone marrow | CD25 antigen | Biocompatibility | Chondrogenesis | Glycoprotein D | Glycoprotein | Survival | Hospitals | Stromal cells | Herpes viruses | Lymphomas | Viability | Cell proliferation | Biomedical research | Mesenchyme | Leukemia | Multiple myeloma | Staining | Homology | Viruses | Oncology | Lymphocytes T | Smad3 protein | Bone diseases | Cell surface | Cyclin B1 | Biomedical materials | Lymphocytes | Cell cycle | Tumor necrosis factor-TNF | Adipogenesis | Cell survival | Hematology | Research & development--R&D | Stat3 protein | Cyclins | Cyclin-dependent kinase 2 | Medicine | Cell number | Osteogenesis | Research & development | R&D
Journal Article