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Autophagy, ISSN 1554-8627, 09/2011, Volume 7, Issue 9, pp. 1045 - 1051
Multiple stress pathways result in the induction of autophagy and apoptosis. Current methods (e.g., protein gel blot, microscopy) do not offer quantitative... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Autophagy | Multispectral imaging cytometry | Influenza | New methodology | Apoptosis | influenza | CROSS-TALK | new methodology | autophagy | BECLIN 1 | multispectral imaging cytometry | apoptosis | CELL-DEATH | CELL BIOLOGY | Leukocytes, Mononuclear - metabolism | Image Cytometry - methods | Apoptosis - drug effects | Apoptosis - radiation effects | Autophagy - radiation effects | Microtubule-Associated Proteins - metabolism | Humans | Autophagy - drug effects | Lysosomes - radiation effects | Chloroquine - pharmacology | Lysosomes - metabolism | Ultraviolet Rays | Time Factors | Leukocytes, Mononuclear - radiation effects | Signal Transduction - radiation effects | Fibroblasts - metabolism | Lysosomes - drug effects | Leukocytes, Mononuclear - drug effects | Animals | Fibroblasts - radiation effects | Signal Transduction - drug effects | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Leukocytes, Mononuclear - cytology | Fibroblasts - cytology | Mice | Autophagy/radiation effects | Signal Transduction/radiation effects | Image Cytometry/methods | Autophagy/drug effects | Life Sciences | Lysosomes/drug effects | Immunology | Fibroblasts/metabolism | Leukocytes, Mononuclear/radiation effects | Fibroblasts/drug effects | Leukocytes, Mononuclear/metabolism | Apoptosis/radiation effects | Leukocytes, Mononuclear/drug effects | Fibroblasts/cytology | Apoptosis/drug effects | Fibroblasts/radiation effects | Lysosomes/metabolism | Leukocytes, Mononuclear/cytology | Signal Transduction/drug effects | Chloroquine/pharmacology | Embryo, Mammalian/cytology | Lysosomes/radiation effects | Microtubule-Associated Proteins/metabolism
Journal Article
Antimicrobial agents and chemotherapy, ISSN 1098-6596, 2017, Volume 61, Issue 3
As new pathogenic viruses continue to emerge, it is paramount to have intervention strategies that target a common denominator in these pathogens. The fusion... 
Envelope protein | Antiviral | Zika virus | Virus entry | Chikungunya virus | Resistant mutant | LYSOSOMAL FUNCTION | envelope protein | MICROBIOLOGY | SEMLIKI-FOREST-VIRUS | chikungunya virus | CHLOROQUINE | FULL-LENGTH CDNA | IN-VITRO | antiviral | virus entry | PHARMACOLOGY & PHARMACY | INFECTION | resistant mutant | MUTATIONS | CELL ENTRY | ANTIVIRALS | Zika Virus - genetics | Chikungunya virus - genetics | Membrane Glycoproteins - metabolism | Epithelial Cells - drug effects | Humans | Yellow fever virus - growth & development | Semliki forest virus - growth & development | Virus Internalization - drug effects | Endosomes - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Lysosomes - metabolism | West Nile virus - genetics | Hydrogen-Ion Concentration - drug effects | Endosomes - drug effects | Viral Envelope Proteins - metabolism | Cell Membrane - drug effects | Hepatocytes - drug effects | Membrane Fusion - drug effects | Lysosomes - drug effects | Cell Line | Virus Replication - drug effects | Antiviral Agents - pharmacology | Cricetinae | Gene Expression | Viral Envelope Proteins - genetics | Zika Virus - growth & development | West Nile virus - growth & development | Zika Virus - drug effects | West Nile virus - drug effects | Chikungunya virus - growth & development | Membrane Glycoproteins - genetics | Chikungunya virus - drug effects | Drug Resistance, Viral - genetics | Pyrroles - pharmacology | Yellow fever virus - drug effects | Animals | Semliki forest virus - genetics | Neutral Red - metabolism | Epithelial Cells - virology | Semliki forest virus - drug effects | Hepatocytes - virology | Cell Membrane - virology | Mutation | Proto-Oncogene Proteins c-bcl-2 - genetics | Yellow fever virus - genetics
Journal Article
CELL DEATH & DISEASE, ISSN 2041-4889, 01/2018, Volume 9, Issue 2, pp. 96 - 15
Journal Article
Autophagy, ISSN 1554-8627, 09/2012, Volume 8, Issue 9, pp. 1333 - 1341
Photodynamic therapy (PDT) involves photosensitizing agents that, in the presence of oxygen and light, initiate formation of cytotoxic reactive oxygen species... 
apoptosis | lysosomes | autophagy | subcellular localization | photodynamic therapy | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Lysosomes | Photodynamic therapy | Autophagy | Subcellular localization | Apoptosis | CANCER-CELLS | ACTIVATION | AUTOPHAGIC RESPONSES | WST11 | CELL BIOLOGY | CYTOCHROME-C | BCL-2 | PHOTOSENSITIZER NPE6 | REACTIVE OXYGEN | MEMBRANE PERMEABILIZATION | Apoptosis - drug effects | Apoptosis - radiation effects | Microtubule-Associated Proteins - metabolism | Fluorescence | Phagosomes - radiation effects | Protein Transport - drug effects | Recombinant Fusion Proteins - metabolism | Vacuoles - drug effects | Cell Shape - radiation effects | Lysosomes - radiation effects | Photosensitizing Agents - pharmacology | Cell Nucleus - metabolism | Lysosomes - metabolism | Cell Nucleus - radiation effects | Light | Phagosomes - ultrastructure | Microtubule-Associated Proteins - deficiency | Phagosomes - drug effects | Lysosomes - drug effects | Cell Survival - drug effects | Green Fluorescent Proteins - metabolism | Permeability - drug effects | Subcellular Fractions - drug effects | Bacteriochlorophylls - pharmacology | Porphyrins - pharmacology | Cell Nucleus - ultrastructure | Amines - metabolism | Cell Survival - radiation effects | Subcellular Fractions - metabolism | Cell Shape - drug effects | Vacuoles - radiation effects | Autophagy-Related Protein 7 | Animals | Permeability - radiation effects | Protein Transport - radiation effects | Subcellular Fractions - radiation effects | Bacteriochlorophylls - chemistry | Cell Line, Tumor | Vacuoles - metabolism | Mice | Cell Nucleus - drug effects | Basic Research Paper
Journal Article
Nanomedicine: Nanotechnology, Biology, and Medicine, ISSN 1549-9634, 2010, Volume 6, Issue 3, pp. 427 - 441
Abstract Identification of pharmacological and toxicological profiles is of critical importance for the use of nanoparticles as drug carriers in nanomedicine and for the biosafety evaluation... 
Internal Medicine | Lysosomes | Mitochondria | Target treatment | Alzheimer disease | Single-walled carbon nanotubes | Organelles | MEDICINE, RESEARCH & EXPERIMENTAL | BIOCOMPATIBILITY | RATS | NANOSCIENCE & NANOTECHNOLOGY | MAMMALIAN-CELLS | DELIVERY | NANOPARTICLES | REACTIVE OXYGEN | IN-VIVO | HUMAN EPIDERMAL-KERATINOCYTES | CYTOTOXICITY | Memory - drug effects | Organ Specificity - drug effects | Drug Carriers - adverse effects | Intestinal Absorption - drug effects | Drug Carriers - toxicity | Mitochondria - ultrastructure | Autophagy - drug effects | Alzheimer Disease - pathology | Lysosomes - metabolism | Nanotubes, Carbon - toxicity | Brain - ultrastructure | Spectrum Analysis, Raman | Lysosomes - drug effects | Cell Survival - drug effects | Alzheimer Disease - physiopathology | Reproducibility of Results | Subcellular Fractions - drug effects | Acetylcholine - pharmacology | Alzheimer Disease - drug therapy | Nanotubes, Carbon - ultrastructure | Mitochondria - metabolism | Mitochondria - drug effects | Subcellular Fractions - metabolism | Brain - drug effects | Lysosomes - ultrastructure | Acetylcholine - administration & dosage | Animals | Nanomedicine - methods | Brain - pathology | Nanotubes, Carbon - adverse effects | Drug Carriers - pharmacokinetics | Mice | Health Status | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2013, Volume 288, Issue 33, pp. 24247 - 24263
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2018, Volume 293, Issue 10, pp. 3562 - 3587
Multidrug resistance (MDR) is a major obstacle in cancer treatment due to the ability of tumor cells to efflux chemotherapeutics via drug transporters (e.g. P-glycoprotein (Pgp; ABCB1... 
DI-2-PYRIDYLKETONE | ISONICOTINOYL HYDRAZONE CLASS | IRON CHELATORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DRUG-RESISTANCE | MULTIDRUG-RESISTANCE | ANTITUMOR-ACTIVITY | HYPOXIA | CANCER | MODULATION | MEMBRANE PERMEABILIZATION | Neoplasms - metabolism | Apoptosis - drug effects | Drug Resistance, Multiple - drug effects | Humans | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Neoplasm Proteins - antagonists & inhibitors | ATP Binding Cassette Transporter, Subfamily B - agonists | Protein Transport - drug effects | Neoplasm Proteins - metabolism | Antineoplastic Agents - metabolism | Cell Hypoxia | Organelle Biogenesis | Lysosomes - metabolism | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | ATP Binding Cassette Transporter, Subfamily B - genetics | Antineoplastic Agents - pharmacology | Biological Transport - drug effects | Doxorubicin - metabolism | Thiosemicarbazones - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - agonists | Lysosomes - drug effects | Tumor Microenvironment - drug effects | Tetrahydroisoquinolines - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Hydrogen Peroxide - pharmacology | ATP Binding Cassette Transporter, Subfamily B - metabolism | Neoplasms - drug therapy | Neoplasm Proteins - agonists | ATP Binding Cassette Transporter, Subfamily B - antagonists & inhibitors | Models, Biological | Acridines - pharmacology | Cell Line, Tumor | Cell Proliferation - drug effects | Neoplasms - pathology | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | P-glycoprotein | ABCB1 | drug transport | tumor micro-environment | drug delivery | drug resistance | tumor micro-environmental stress | lysosome | Cell Biology
Journal Article
International journal of nanomedicine, ISSN 1178-2013, 2016, Volume 11, pp. 3655 - 3675
.... Although many cancer drugs have been developed to dramatically reduce the size of tumors, most cancers eventually relapse, posing a critical problem to overcome... 
Caspase activity | Salinomycin | Cell viability | Autophagy | Silver nanoparticles | Apoptosis | Ovarian cancer | MOLECULAR-MECHANISM | OXIDATIVE STRESS | autophagy | ovarian cancer | DNA-DAMAGE | caspase activity | apoptosis | NANOSCIENCE & NANOTECHNOLOGY | salinomycin | DEATH | TOXICITY | ANTITUMOR-ACTIVITY | IN-VITRO | cell viability | silver nanoparticles | STEM-CELL | PHARMACOLOGY & PHARMACY | INDUCED CYTOTOXICITY | UP-REGULATION | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Ovarian Neoplasms - pathology | Membrane Potential, Mitochondrial - drug effects | Autophagy - drug effects | Ovarian Neoplasms - genetics | Autophagosomes - drug effects | Lysosomes - metabolism | Biomarkers, Tumor - metabolism | Female | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Pyrans - pharmacology | Real-Time Polymerase Chain Reaction | Metal Nanoparticles - ultrastructure | Lysosomes - drug effects | Cell Survival - drug effects | Silver - pharmacology | Metal Nanoparticles - chemistry | Autophagosomes - metabolism | Autophagosomes - ultrastructure | Cell Shape - drug effects | Lysosomes - ultrastructure | Up-Regulation - drug effects | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | Bacillus - drug effects | Oxidative Stress - drug effects | Nanoparticles | Silver | Care and treatment | Mortality | Drug therapy | Cancer | Gynecology | Cytotoxicity | Metastasis | Cancer therapies | Stem cells | Cell cycle | Tumorigenesis | Tumors
Journal Article
Hepatology (Baltimore, Md.), ISSN 0270-9139, 2014, Volume 59, Issue 4, pp. 1366 - 1380
Journal Article
Autophagy, ISSN 1554-8627, 2015, Volume 11, Issue 9, pp. 1537 - 1560
Autophagy, a lysosomal degradative pathway, is potently stimulated in the myocardium by fasting and is essential for maintaining cardiac function during... 
fasting | autophagy | ischemia-reperfusion | lysosome | myocardial infarction | Ischemia-reperfusion | Myocardial infarction | Fasting | Autophagy | Lysosome | LIFE-SPAN | ACTIVATED PROTEIN-KINASE | CHAPERONE-MEDIATED AUTOPHAGY | MITOCHONDRIAL AUTOPHAGY | DIETARY RESTRICTION | STARVATION-INDUCED AUTOPHAGY | CELL BIOLOGY | TRANSCRIPTION FACTOR TFEB | CARDIOMYOCYTE DEATH | HEART | CALORIE RESTRICTION | Oxygen - pharmacology | Transcription, Genetic - drug effects | Reactive Oxygen Species - metabolism | Male | Autophagy - drug effects | Myocardial Reperfusion Injury - pathology | Ventricular Remodeling | Lysosomes - metabolism | Myocardium - metabolism | Cytoprotection - drug effects | Ultrasonography | Female | Autophagy - genetics | Gene Ontology | Myocardium - ultrastructure | Lysosomes - drug effects | Cell Hypoxia - drug effects | Mice, Inbred C57BL | Feeding Behavior | Myocardium - pathology | Mitochondria - metabolism | Mitochondria - drug effects | Down-Regulation - drug effects | Myocardial Reperfusion Injury - physiopathology | Up-Regulation - drug effects | Myocardial Reperfusion Injury - diagnostic imaging | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Heterozygote | Oxidative Stress - drug effects | Lysosomal-Associated Membrane Protein 2 - metabolism | Myocardial Reperfusion Injury - prevention & control
Journal Article
Journal Article
Blood, ISSN 0006-4971, 12/2016, Volume 128, Issue 25, pp. 2976 - 2987
Chorea-acanthocytosis is one of the hereditary neurodegenerative disorders known as the neuroacanthocytoses. Chorea-acanthocytosis is characterized by... 
MITOCHONDRIAL CLEARANCE | HSP90 | COMPLEX | DOCKING | MOUSE MODEL | RED-CELLS | PROTEIN-PHOSPHORYLATION | HEMATOLOGY | ANION TRANSPORT ACTIVITY | MEMBRANE ASSOCIATION | PEROXIREDOXIN-2 | Neuroacanthocytosis - pathology | Multivesicular Bodies - drug effects | Demography | Molecular Weight | Autophagy-Related Protein 7 - metabolism | Humans | Middle Aged | Cytosol - drug effects | Erythrocytes - ultrastructure | Erythropoiesis - drug effects | Male | Autophagy-Related Protein-1 Homolog - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Bortezomib - pharmacology | Autophagy - drug effects | Proteolysis - drug effects | Lysosomes - metabolism | Anion Exchange Protein 1, Erythrocyte - metabolism | src-Family Kinases - metabolism | Erythrocytes - pathology | Adult | Female | Phosphorylation - drug effects | Lysosomes - drug effects | Heat-Shock Proteins - metabolism | Mitochondria - metabolism | Erythroid Cells - ultrastructure | Mitochondria - drug effects | Lactams, Macrocyclic - pharmacology | Erythrocytes - drug effects | Benzoquinones - pharmacology | Erythroid Cells - drug effects | Cell Differentiation - drug effects | Erythrocytes - metabolism | Cytosol - metabolism | Erythroid Cells - pathology | Multivesicular Bodies - metabolism | Proteasome Endopeptidase Complex - metabolism | Index Medicus | Abridged Index Medicus | Red Cells, Iron, and Erythropoiesis
Journal Article