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inflammation (157) 157
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medicine, research & experimental (107) 107
up-regulation (107) 107
kinases (106) 106
oxidative stress (106) 106
s-transferase m1 (105) 105
cell proliferation - drug effects (104) 104
breast cancer (103) 103
breast-cancer (103) 103
research article (101) 101
immunohistochemistry (96) 96
invasion (96) 96
multidisciplinary sciences (96) 96
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cytokines (78) 78
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1. Full Text FOXM1, a typical proliferation-associated transcription factor
by Wierstra, Inken and Alves, Jürgen
Biological Chemistry, ISSN 1431-6730, 12/2007, Volume 388, Issue 12, pp. 1257 - 1274
FOXM1 is a typical proliferation-associated transcription factor: it stimulates proliferation by promoting S-phase entry as well as M-phase entry and is... 
Cell proliferation | Tumorigenesis | S-phase | Tissue regeneration | Mitosis | Cell cycle | FORKHEAD BOX M1B | cell proliferation | cell cycle | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION PROFILES | FACTOR TRIDENT | mitosis | tissue regeneration | BREAST-CANCER CELLS | M-PHASE PHOSPHOPROTEINS | GENOME-WIDE ANALYSIS | BINDING-SITE | tumorigenesis | HEPATOCELLULAR CARCINOMAS | MOUSE-LIVER REGENERATION | Gene Targeting | Transcriptional Activation - genetics | Animals | Cell Proliferation | Neoplasms - genetics | Humans | Forkhead Transcription Factors - physiology | Mice | Forkhead Box Protein M1 | Forkhead Transcription Factors - genetics | Mice, Knockout | Protein Isoforms - genetics
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2. Full Text Multiple faces of FoxM1 transcription factor
: Lessons from transgenic mouse models
by Kalin, Tanya V and Ustiyan, Vladimir and Kalinichenko, Vladimir V
Cell Cycle, ISSN 1538-4101, 02/2011, Volume 10, Issue 3, pp. 396 - 405
FoxM1 transcription factor (previously called HFH-11B, Trident, FoxM1b, Win, and MPP2) is expressed in actively dividing cells and critical for cell cycle... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Embryonic development | Forkhead transcription factor | Cellular proliferation | Cell cycle | Transgenic mice | FoxM1 | Winged helix DNA binding domain | EMBRYONIC-DEVELOPMENT | cell cycle | winged helix DNA binding domain | embryonic development | DNA-BINDING-DOMAIN | CELL-PROLIFERATION | CELL BIOLOGY | BREAST-CANCER | PULMONARY VASCULATURE | transgenic mice | S-PHASE | HEPATOCYTE ENTRY | TUMOR-SUPPRESSOR | cancer | HEPATOCELLULAR CARCINOMAS | forkhead transcription factor | FORKHEAD BOX M1 | cellular proliferation | Humans | Embryonic Development - genetics | Gene Silencing | DNA Replication | Mice, Transgenic | Forkhead Transcription Factors - physiology | Forkhead Transcription Factors - genetics | Mitosis - genetics | Embryo, Mammalian - metabolism | Cell Lineage | Animals | Neoplasms - genetics | Forkhead Transcription Factors - metabolism | Regeneration - genetics | Cell Cycle - physiology | Mice | Forkhead Box Protein M1 | Extra View
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3. Full Text FOXM1: A key oncofoetal transcription factor in health and disease
by Bella, Laura and Zona, Stefania and Nestal de Moraes, Gabriela and Lam, Eric W.-F
Seminars in Cancer Biology, ISSN 1044-579X, 2014, Volume 29, Issue C, pp. 32 - 39
Abstract Forkhead Box M1 (FOXM1) is a bona fide oncofoetal transcription factor, which orchestrates complex temporal and spatial gene expression throughout... 
Hematology, Oncology and Palliative Medicine | Development | Oncofoetal protein | FOXM1 | Transcription factor | Cancer | PROGENITOR CELLS | STEM-CELLS | INCREASED EXPRESSION | EPITHELIAL-MESENCHYMAL TRANSITION | ONCOLOGY | GASTRIC-CANCER | TARGETING FOXM1 | DNA-REPAIR | UP-REGULATION | PROMOTES | GEFITINIB IRESSA | Antigens, Neoplasm - genetics | Cellular Senescence - genetics | Forkhead Transcription Factors - biosynthesis | Cell Proliferation - genetics | Humans | Transcriptional Activation | Antigens, Neoplasm - biosynthesis | Gene Expression Regulation, Neoplastic | Neoplastic Stem Cells | DNA Repair - genetics | Epithelial-Mesenchymal Transition - genetics | Forkhead Transcription Factors - genetics | Cell Movement - genetics | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Gene Expression Regulation, Developmental | Neovascularization, Pathologic - genetics | Forkhead Box Protein M1 | Embryonic development | Growth | Fetus
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4. Full Text Foxm1 transcription factor is required for lung fibrosis and epithelial‐to‐mesenchymal transition
by Balli, David and Ustiyan, Vladimir and Zhang, Yufang and Wang, I‐Ching and Masino, Alex J and Ren, Xiaomeng and Whitsett, Jeffrey A and Kalinichenko, Vladimir V and Kalin, Tanya V
The EMBO Journal, ISSN 0261-4189, 01/2013, Volume 32, Issue 2, pp. 231 - 244
Alveolar epithelial cells (AECs) participate in the pathogenesis of pulmonary fibrosis, producing pro‐inflammatory mediators and undergoing... 
pulmonary inflammation | radiation‐induced lung fibrosis | EMT | Foxm1 | Snail1 | radiation-induced lung fibrosis | CELLS | TGF-BETA | FACTOR SNAIL | BIOCHEMISTRY & MOLECULAR BIOLOGY | RADIATION PNEUMONITIS | MECHANISMS | FORKHEAD | RESPIRATORY EPITHELIUM | CELL BIOLOGY | MESSENGER-RNA | GENE-EXPRESSION | PULMONARY-FIBROSIS | Epithelial-Mesenchymal Transition - physiology | Humans | Forkhead Transcription Factors - physiology | Pulmonary Fibrosis - genetics | Epithelial-Mesenchymal Transition - genetics | Fibrosis - metabolism | Forkhead Transcription Factors - metabolism | Lung - radiation effects | Lung - metabolism | Pulmonary Fibrosis - etiology | Pneumonia - genetics | Radiation Injuries, Experimental - genetics | Radiation Injuries, Experimental - metabolism | Fibrosis - genetics | Lung - pathology | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation - physiology | Mice, Transgenic | Pulmonary Fibrosis - pathology | Radiation Injuries, Experimental - pathology | Forkhead Transcription Factors - genetics | Animals | Fibrosis - etiology | Mice | Forkhead Box Protein M1 | Pneumonia - metabolism | Lungs | Gene expression | Ribonucleic acid--RNA | Inflammatory diseases
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5. Full Text The transcription factor FOXM1 is a cellular target of the natural product thiostrepton
by Hegde, Nagaratna S and Sanders, Deborah A and Rodriguez, Raphaël and Balasubramanian, Shankar
Nature Chemistry, ISSN 1755-4330, 09/2011, Volume 3, Issue 9, pp. 725 - 731
Transcription factors are proteins that bind specifically to defined DNA sequences to promote gene expression. Targeting transcription factors with small... 
BREAST-CANCER CELLS | BIOLOGY | SMALL ORGANIC-MOLECULES | ESTROGEN | INDUCE APOPTOSIS | MELANOMA-CELLS | PROTEASOME INHIBITORS | EXPRESSION | CHEMISTRY, MULTIDISCIPLINARY | PROGRESSION | DISCOVERY | Thiostrepton - pharmacology | Humans | Thiostrepton - analogs & derivatives | Anti-Bacterial Agents - metabolism | Biological Products - pharmacology | DNA - metabolism | Thiostrepton - metabolism | Biological Products - chemistry | Forkhead Transcription Factors - metabolism | Anti-Bacterial Agents - chemistry | Drug Design | Biological Products - metabolism | Cell Line, Tumor | Protein Binding | Cell Extracts | Anti-Bacterial Agents - pharmacology | Forkhead Transcription Factors - antagonists & inhibitors | Forkhead Box Protein M1
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6. Full Text Nutrient sensor O-GlcNAc transferase regulates breast cancer tumorigenesis through targeting of the oncogenic transcription factor FoxM1
by Caldwell, S.A and Jackson, S.R and Shahriari, K.S and Lynch, T.P and Sethi, G and Walker, S and Vosseller, K and Reginato, M.J
Oncogene, ISSN 0950-9232, 05/2010, Volume 29, Issue 19, pp. 2831 - 2842
Cancer cells upregulate glycolysis, increasing glucose uptake to meet energy needs. A small fraction of a cell's glucose enters the hexosamine biosynthetic... 
Glucose metabolism | Breast cancer | FoxM1 | O-GlcNAc | OGT | p27(Kip1) | CELLS | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | breast cancer | GLYCOSYLATION | BETA | CELL BIOLOGY | ONCOLOGY | INSULIN-RESISTANCE | GLUCOSE | GENETICS & HEREDITY | INHIBITORS | glucose metabolism | NUCLEOCYTOPLASMIC PROTEINS | EXPRESSION | LINKED N-ACETYLGLUCOSAMINE | Humans | N-Acetylglucosaminyltransferases - genetics | Acetylglucosamine - metabolism | Breast Neoplasms - metabolism | Gene Knockdown Techniques | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Breast Neoplasms - enzymology | N-Acetylglucosaminyltransferases - deficiency | Forkhead Transcription Factors - metabolism | Neoplasm Invasiveness - pathology | Base Sequence | Female | Gene Expression Regulation, Neoplastic - drug effects | Matrix Metalloproteinase 2 - metabolism | Enzyme Inhibitors - pharmacology | Oncogene Proteins - metabolism | N-Acetylglucosaminyltransferases - metabolism | Up-Regulation - drug effects | Matrix Metalloproteinase 2 - genetics | Phenotype | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | N-Acetylglucosaminyltransferases - antagonists & inhibitors | Cell Proliferation - drug effects | Mice | Cell Transformation, Neoplastic - drug effects | Cell Cycle - drug effects | Forkhead Box Protein M1 | Post-translational modification | Care and treatment | Control | Transcription factors | Transferases | Physiological aspects | Genetic aspects | Research | Proteins | Glucose | Gene expression | Metabolism | Cell cycle
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7. Full Text Critical role and regulation of transcription factor foxm1 in human gastric cancer angiogenesis and progression
by Li, Qiang and Zhang, Nu and Jia, Zhiliang and Le, Xiangdong and Dai, Bingbing and Wei, Daoyan and Huang, Suyun and Tan, Dongfeng and Xie, Keping
Cancer Research, ISSN 0008-5472, 04/2009, Volume 69, Issue 8, pp. 3501 - 3509
The mammalian forkhead box (Fox) transcription factor FoxM1b is implicated in tumorigenesis. However, the presence of expression and role of Fo-xM1b in gastric... 
GLIOMA-CELLS | SURVIVAL | FACTOR SP1 | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | LIVER-REGENERATION | GENE-EXPRESSION | OVER-EXPRESSION | S-PHASE | FORKHEAD | ENDOTHELIAL GROWTH-FACTOR | Stomach Neoplasms - genetics | Forkhead Transcription Factors - biosynthesis | RNA, Small Interfering - genetics | Up-Regulation | Vascular Endothelial Growth Factor A - biosynthesis | Humans | Transcriptional Activation | Stomach Neoplasms - metabolism | Cell Growth Processes - physiology | Stomach Neoplasms - pathology | Transplantation, Heterologous | Forkhead Transcription Factors - genetics | Stomach Neoplasms - blood supply | Disease Progression | Neoplasm Metastasis | Animals | Transfection | Mice, Nude | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Forkhead Box Protein M1 | FoxM1b | stomach | metastasis | angiogenesis | VEGF
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8. Full Text Targeting forkhead box M1 transcription factor in breast cancer
by O'Regan, Ruth M and Nahta, Rita
Biochemical Pharmacology, ISSN 0006-2952, 08/2018, Volume 154, pp. 407 - 413
Breast cancer continues to be the most commonly diagnosed malignancy and second most common cause of cancer-related deaths among women in the United States.... 
Experimental therapeutics | Breast cancer | Pharmacology | Drug resistance | FoxM1 | DOWNSTREAM TARGET | FACTOR FOXM1 | CELL-PROLIFERATION | OVARIAN-CANCER | PHASE-II TRIAL | MEK INHIBITION | POLO-LIKE KINASE-1 | MUTANT P53 | PHARMACOLOGY & PHARMACY | INHIBITOR BI 2536 | MASTER REGULATOR | Forkhead Box Protein M1 - biosynthesis | Humans | Gene Expression Regulation, Neoplastic | Antineoplastic Agents - administration & dosage | Breast Neoplasms - drug therapy | Breast Neoplasms - metabolism | Drug Delivery Systems | Antineoplastic Agents - metabolism | Breast Neoplasms - genetics | Cell Line, Tumor | Female | Forkhead Box Protein M1 - genetics | Drug Resistance, Neoplasm - physiology | Forkhead Box Protein M1 - antagonists & inhibitors | Drug Resistance, Neoplasm - drug effects | Medical colleges | Chemotherapy | Development and progression | Metastasis | Cancer
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9. Glioblastoma multiforme formation and EMT: Role of FoxM1 transcription factor
by Wang, Zhongyong and Zhang, Sicong and Siu, Timothy L and Huang, Suyun
Current Pharmaceutical Design, ISSN 1381-6128, 01/2015, Volume 21, Issue 10, pp. 1268 - 1271
Glioblastoma multiforme (GBM) is one of the most malignant cancers in human brain. The prognosis of GBM is extremely poor because it is resistant to... 
Wnt/β-catenin signaling | Angiogenesis | Glioblastoma | Tumorigenesis | EMT | FoxM1 | Invasion | TARGET | angiogenesis | DOWN-REGULATION | Wnt/beta-catenin signaling | MESENCHYMAL TRANSITION | BETA-CATENIN | CANCER | MALIGNANT GLIOMA | invasion | PHARMACOLOGY & PHARMACY | tumorigenesis | EXPRESSION | CELL | PROMOTES | PROGRESSION | Epithelial-Mesenchymal Transition - physiology | Humans | Brain Neoplasms - pathology | Carcinogenesis - genetics | Brain Neoplasms - genetics | Forkhead Transcription Factors - physiology | Carcinogenesis - metabolism | Brain Neoplasms - metabolism | Carcinogenesis - pathology | Animals | Glioblastoma - genetics | Glioblastoma - pathology | Glioblastoma - metabolism | Transcription Factors | Forkhead Box Protein M1
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10. Full Text The Forkhead Transcription Factor FOXM1 Controls Cell Cycle-Dependent Gene Expression through an Atypical Chromatin Binding Mechanism
by Xi Chen and Gerd A. Müller and Marianne Quaas and Martin Fischer and Namshik Han and Benjamin Stutchbury and Andrew D. Sharrocks and Kurt Engeland
Molecular and Cellular Biology, ISSN 0270-7306, 01/2013, Volume 33, Issue 2, pp. 227 - 236
OA  Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+... 
CHIP-SEQ | ELEMENTS | PROTEIN | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | COMPLEXES | MITOTIC PROGRESSION | B-MYB | PROMOTER | FACTOR FOXK2 | PROGRAM | CELL BIOLOGY | Chromatin - metabolism | NIH 3T3 Cells | Phosphorylation | Mitosis | Humans | Chromatin Immunoprecipitation | Forkhead Transcription Factors - metabolism | Cell Division | Cloning, Molecular | HEK293 Cells | Protein Interaction Domains and Motifs | DNA-Binding Proteins | Promoter Regions, Genetic | HCT116 Cells | Computational Biology | Gene Expression Regulation | Forkhead Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Animals | Cell Cycle | Cell Line, Tumor | Protein Binding | Mice | Forkhead Box Protein M1 | Genes, cdc
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11. Full Text Foxm1 transcription factor is required for the initiation of lung tumorigenesis by oncogenic Kras(G12D)
by Wang, IC and Ustiyan, V and Zhang, Y and Cai, Y and Kalin, TV and Kalinichenko, VV
ONCOGENE, ISSN 0950-9232, 11/2014, Volume 33, Issue 46, pp. 5391 - 5396
Lung cancer is the leading cause of deaths in cancer patients in the United States. Identification of new molecular targets is clearly needed to improve... 
ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADENOCARCINOMAS | FOXM1 | FORKHEAD | CANCER | SIGNATURE | RESPIRATORY EPITHELIUM | CELL BIOLOGY | K-Ras | transgenic mice | GENE | ONCOLOGY | GENETICS & HEREDITY | TUMOR-SUPPRESSOR | EXPRESSION | non-small-cell lung cancer | Lung Neoplasms - drug therapy | Antibiotics, Antineoplastic - pharmacology | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Carcinogenesis - metabolism | Promoter Regions, Genetic - genetics | Forkhead Transcription Factors - metabolism | Lung - metabolism | Proto-Oncogene Proteins p21(ras) - metabolism | Lung Neoplasms - genetics | Epithelium - drug effects | Epithelium - pathology | Lung - pathology | Epithelium - metabolism | Mice, Inbred C57BL | Carcinogenesis - genetics | Mice, Transgenic | Forkhead Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Carcinogenesis - drug effects | Animals | Signal Transduction - drug effects | Lung - drug effects | Protein Binding | Forkhead Box Protein M1 | Doxorubicin - pharmacology
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12. Full Text MicroRNA-149 Suppresses Colorectal Cancer Cell Migration and Invasion by Directly Targeting Forkhead Box Transcription Factor FOXM1
by Xu, Kai and Liu, Xiaobei and Mao, Xiaobei and Xue, Lijun and Wang, Rui and Chen, Longbang and Chu, Xiaoyuan
Cellular Physiology and Biochemistry, ISSN 1015-8987, 01/2015, Volume 35, Issue 2, pp. 499 - 515
Background/Aims: The aim of this study is to investigate the clinicopathological and prognostic values of miR-149 expression and its roles in colorectal cancer... 
Original Paper | miR-149 | FOXM1 | Migration | Invasion | Colorectal cancer | TUMOR INVASION | PHYSIOLOGY | PROLIFERATION | LIVER METASTASIS | CELL BIOLOGY | POOR-PROGNOSIS | GROWTH | UP-REGULATION | EXPRESSION | PROMOTES | Prognosis | Neoplasm Invasiveness | Colorectal Neoplasms - genetics | HCT116 Cells | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | MicroRNAs - metabolism | Forkhead Transcription Factors - genetics | Survival Analysis | Aged, 80 and over | Cell Line, Tumor | Adult | Female | Aged | MicroRNAs - genetics | Colorectal Neoplasms - pathology | Forkhead Box Protein M1 | Colorectal Neoplasms - metabolism | Cell Movement
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13. Full Text Loss of the forkhead transcription factor FoxM1 causes centrosome amplification and mitotic catastrophe
by Wonsey, Diane R and Follettie, Maximillian T
Cancer Research, ISSN 0008-5472, 06/2005, Volume 65, Issue 12, pp. 5181 - 5189
Expression of the forkhead transcription factor FoxM1 correlates with proliferative status in a variety of normal and transformed cell types. Elevated... 
FACTOR HFH-11B | PROTEIN | ONCOLOGY | DNA-DAMAGE | S-PHASE | HEPATOCYTE ENTRY | FACTOR TRIDENT | CELL-CYCLE PROGRESSION | HEPATOCELLULAR CARCINOMAS | EXPRESSION | AURORA-A | Cell Cycle - genetics | RNA, Small Interfering - genetics | Mitosis | Humans | Transcription Factors - deficiency | Transcription Factors - biosynthesis | Transcription Factors - genetics | Breast Neoplasms - metabolism | Breast Neoplasms - genetics | Gene Amplification | Breast Neoplasms - pathology | Ploidies | Cell Line, Tumor | Forkhead Box Protein M1 | Nucleic Acid Conformation | Spindle Apparatus - physiology | Forkhead Transcription Factors | Centrosome | Gene Expression Regulation, Neoplastic - genetics
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14. Full Text Chk2 Mediates Stabilization of the FoxM1 Transcription Factor To Stimulate Expression of DNA Repair Genes
by Yongjun Tan and Pradip Raychaudhuri and Robert H. Costa
Molecular and Cellular Biology, ISSN 0270-7306, 02/2007, Volume 27, Issue 3, pp. 1007 - 1016
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CELLS | DAMAGE-INDUCED PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | LIVER | CARCINOMAS | S-PHASE | CHECKPOINT | PROGRESSION | P53 | KINASES | CELL BIOLOGY | Transcription, Genetic - drug effects | Fibroblasts - enzymology | Humans | Transcription, Genetic - radiation effects | DNA Repair - radiation effects | Molecular Sequence Data | DNA Repair - genetics | DNA-Binding Proteins - metabolism | Thermodynamics | Ultraviolet Rays | Forkhead Transcription Factors - metabolism | Infrared Rays | Forkhead Transcription Factors - chemistry | DNA Breaks | Phosphorylation - drug effects | Signal Transduction - radiation effects | Forkhead Transcription Factors - deficiency | Protein-Serine-Threonine Kinases - metabolism | Amino Acid Sequence | DNA Repair - drug effects | Gene Expression Regulation - genetics | Tumor Suppressor Protein p53 - metabolism | Etoposide - pharmacology | DNA-Binding Proteins - genetics | Serine - metabolism | Phosphorylation - radiation effects | Animals | BRCA2 Protein - metabolism | Fibroblasts - radiation effects | Signal Transduction - drug effects | Fibroblasts - drug effects | Checkpoint Kinase 2 | Fibroblasts - cytology | Mice | Forkhead Box Protein M1 | X-ray Repair Cross Complementing Protein 1 | BRCA2 Protein - genetics
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