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1. Full Text Human macrophage polarization in vitro : Maturation and activation methods compared
by Vogel, Daphne Y.S and Glim, Judith E and Stavenuiter, Andrea W.D and Breur, Marjolein and Heijnen, Priscilla and Amor, Sandra and Dijkstra, Christine D and Beelen, Robert H.J
Immunobiology, ISSN 0171-2985, 2014, Volume 219, Issue 9, pp. 695 - 703
Abstract Macrophages form a heterogeneous cell population displaying multiple functions, and can be polarized into pro- (M1) or anti-inflammatory (M2)... 
Allergy and Immunology | Advanced Basic Science | Macrophage activation | Multiple sclerosis | Wound healing | Macrophage maturation | M1 M2 macrophages | Fibrosis | M2 MACROPHAGES | CD163 | DENDRITIC CELLS | MARKERS | IMMUNOLOGY | TUMOR-ASSOCIATED MACROPHAGES | REMYELINATION | TYPE-2 MACROPHAGES | EXPRESSION | ALTERNATIVE ACTIVATION | PROGRESSION | Immunologic Techniques | Macrophage Activation - immunology | Humans | Cells, Cultured | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Macrophages - cytology | Cell Differentiation - immunology | Cell Differentiation - drug effects | Macrophage Colony-Stimulating Factor - pharmacology | Macrophages - drug effects | Serum | In Vitro Techniques | Macrophage Activation - drug effects | Macrophages - immunology | Fc receptors | Surgery, Plastic | Bone morphogenetic proteins | Macrophage colony stimulating factor | Comparative analysis | Macrophages | Transforming growth factors | Methods | Index Medicus
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2. Full Text Macrolide derivatives reduce proinflammatory macrophage activation and macrophage‐mediated neurotoxicity
by Zhang, Bei and Kopper, Timothy J and Liu, Xiaodong and Cui, Zheng and Van Lanen, Steven G and Gensel, John C
CNS Neuroscience & Therapeutics, ISSN 1755-5930, 05/2019, Volume 25, Issue 5, pp. 591 - 600
Introduction Azithromycin (AZM) and other macrolide antibiotics are applied as immunomodulatory treatments for CNS disorders. The immunomodulatory and... 
spinal cord injury | brain | microglia | stroke | erythromycin | M2 | CELLS | ANTIBIOTICS | REGENERATION | INJURY | AZITHROMYCIN | NEUROSCIENCES | FORMS | RECOVERY | PHARMACOLOGY & PHARMACY | ISCHEMIC-STROKE | Evaluation | Drug resistance in microorganisms | Protein biosynthesis | Erythromycin | Derivatives (Financial instruments) | Macrophages | Neuroprotection | Cytokines | Immunomodulation | Azithromycin | Macrolide antibiotics | Inflammation | Lipopolysaccharides | Neurotoxicity | Cell activation | Antibiotics | Antibiotic resistance | Bone marrow | Sugar | Enzyme-linked immunosorbent assay
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3. Full Text Orchestration of macrophage polarization
by Mantovani, Alberto and Locati, Massimo
Blood, ISSN 0006-4971, 2009, Volume 114, Issue 15, pp. 3135 - 3136
In this issue of Blood, Ishii and colleagues shed new, fresh light on molecular mechanisms underlying one form of macrophage activation. 
TUMOR-ASSOCIATED MACROPHAGES | M2 MACROPHAGES | ACTIVATION | HEMATOLOGY
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4. Full Text MicroRNA let-7c regulates macrophage polarization
by Banerjee, Sami and Xie, Na and Cui, Huachun and Tan, Zheng and Yang, Shanzhong and Icyuz, Mert and Abraham, Edward and Liu, Gang
Journal of Immunology, ISSN 0022-1767, 06/2013, Volume 190, Issue 12, pp. 6542 - 6549
Macrophages demonstrate a high level of plasticity, with the ability to undergo dynamic transition between M1 and M2 polarized phenotypes. The role of... 
RESPONSES | ACTIVATION | COLONY-STIMULATING FACTOR | RNA | APOPTOTIC CELLS | GENE-EXPRESSION | ALVEOLAR MACROPHAGES | RECEPTOR | PHENOTYPES | IMMUNOLOGY | PULMONARY-FIBROSIS | Cell Polarity | Mice, Inbred C57BL | Macrophages - cytology | MicroRNAs - immunology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Cell Differentiation - immunology | Phenotype | Animals | Flow Cytometry | Transfection | Mice | Mice, Inbred BALB C | Real-Time Polymerase Chain Reaction | Macrophages - immunology | Index Medicus | Abridged Index Medicus | EBP-δ | M2 macrophage | microRNA let-7c | M1 macrophage
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5. Full Text IL-33 promotes ST2-dependent lung fibrosis by the induction of alternatively activated macrophages and innate lymphoid cells in mice
by Li, Dong, PhD and Guabiraba, Rodrigo, PhD and Besnard, Anne-Gaëlle, PhD and Komai-Koma, Mousa, PhD and Jabir, Majid S., MSc and Zhang, Li, BSc and Graham, Gerard J., PhD and Kurowska-Stolarska, Mariola, PhD and Liew, Foo Y., PhD and McSharry, Charles, PhD and Xu, Damo, PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 134, Issue 6, pp. 1422 - 1432.e11
Background The initiation and regulation of pulmonary fibrosis are not well understood. IL-33, an important cytokine for respiratory diseases, is overexpressed... 
Allergy and Immunology | type 2 innate lymphoid cells | alternatively activated macrophages | IL-33 | lung fibrosis | IMMUNITY | RECRUITMENT | NEUTROPHIL | MOUSE | AIRWAY INFLAMMATION | BLEOMYCIN | CYTOKINE | MECHANISMS | IMMUNOLOGY | ALLERGY | IN-VIVO | PULMONARY-FIBROSIS | Interleukin-1 Receptor-Like 1 Protein | Lung - pathology | Receptors, Interleukin - immunology | Interleukin-33 | Mice, Inbred C57BL | Interleukin-13 - immunology | Transforming Growth Factor beta1 - genetics | Receptors, Interleukin - genetics | Mice, Knockout | Interleukins - genetics | Transforming Growth Factor beta1 - immunology | Interleukin-13 - genetics | Animals | Lymphocytes - immunology | Interleukins - immunology | Fibrosis | Lung - immunology | Macrophages, Alveolar - immunology | Macrophages | Transforming growth factors | Studies | Pulmonary fibrosis | Cytokines | Transgenic animals | Lymphocytes | Rodents | Collagen | Extracellular matrix | Software | Index Medicus | Abridged Index Medicus | CT, Computed tomography | flIL-33, Full-length IL-33 | ICOS, Inducible costimulator | M2 macrophage, Alternatively activated macrophage | BAL, Bronchoalveolar lavage | Nos2, Inducible nitric oxide synthase 2 gene | WT, Wild-type | APC, Allophycocyanin | Arg1, Arginase 1 | IPF, Idiopathic pulmonary fibrosis | qPCR, Quantitative PCR | M1 macrophage, Classically activated macrophage | PE, Phycoerythrin | FITC, Fluorescein isothiocyanate | ILC2, Type 2 innate lymphoid cell | mIL-33, Mature IL-33 | BMDM, Bone marrow–derived macrophage | Mechanisms of Allergy and Clinical Immunology
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6. Full Text Titanium surface characteristics, including topography and wettability, alter macrophage activation
by Hotchkiss, Kelly M and Reddy, Gireesh B and Hyzy, Sharon L and Schwartz, Zvi and Boyan, Barbara D and Olivares-Navarrete, Rene
Acta Biomaterialia, ISSN 1742-7061, 02/2016, Volume 31, pp. 425 - 434
Biomaterial surface properties including chemistry, topography, and wettability regulate cell response. Previous studies have shown that increasing surface... 
Wettability | Immune response | M1 activation | Titanium | Surface roughness | Macrophages | M2 activation | MATERIALS SCIENCE, BIOMATERIALS | VIVO | ENGINEERING, BIOMEDICAL | TISSUE-REPAIR | IMPLANT SURFACES | OSSEOINTEGRATION | IN-VITRO | HYDROPHILICITY | BIOMATERIALS | BONE | POLARIZATION | MICROSTRUCTURE | Tumor Necrosis Factor-alpha - metabolism | Osseointegration | Biocompatible Materials - chemistry | Humans | Titanium - chemistry | Inflammation | Macrophages - cytology | Polystyrenes - chemistry | Macrophage Activation | Macrophages - metabolism | Anti-Inflammatory Agents - chemistry | Interleukin-1beta - metabolism | Surface Properties | Hydrophobic and Hydrophilic Interactions | Interleukin-10 - metabolism | Cell Differentiation | Osteoblasts - cytology | Interleukin-6 - metabolism | Osteogenesis | Biological products | Interleukins | Implant dentures | Stem cells | Biomedical engineering | Index Medicus | Surgical implants | Biomedical materials | Surface chemistry | Biocompatibility | Activation | Titanium base alloys
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7. Full Text Iron oxide nanoparticles inhibit tumour growth by inducing pro-inflammatory macrophage polarization in tumour tissues
by Zanganeh, Saeid and Hutter, Gregor and Spitler, Ryan and Lenkov, Olga and Mahmoudi, Morteza and Shaw, Aubie and Pajarinen, Jukka Sakari and Nejadnik, Hossein and Goodman, Stuart and Moseley, Michael and Coussens, Lisa Marie and Daldrup-Link, Heike Elisabeth
Nature Nanotechnology, ISSN 1748-3387, 11/2016, Volume 11, Issue 11, pp. 986 - 994
Until now, the Food and Drug Administration (FDA)-approved iron supplement ferumoxytol and other iron oxide nanoparticles have been used for treating iron... 
CELL LUNG-CANCER | DEFICIENCY ANEMIA | DRUG | OXIDATIVE STRESS | M2 MACROPHAGES | STEM-CELLS | THERAPY | PARTICLES | MATERIALS SCIENCE, MULTIDISCIPLINARY | FERUMOXYTOL | NANOSCIENCE & NANOTECHNOLOGY | EXPRESSION | Inflammation - pathology | Lung Neoplasms - drug therapy | Humans | Metal Nanoparticles - chemistry | Liver Neoplasms - drug therapy | Metal Nanoparticles - therapeutic use | Ferrosoferric Oxide - pharmacology | Ferrosoferric Oxide - chemistry | Antineoplastic Agents - chemistry | Mice, Inbred Strains | Xenograft Model Antitumor Assays - methods | Animals | Lung Neoplasms - secondary | Mammary Neoplasms, Experimental - pathology | Cell Line, Tumor | Female | Macrophages - drug effects | Mice, Inbred NOD | Antineoplastic Agents - pharmacology | Cell Polarity - drug effects | Liver Neoplasms - secondary | Mammary Neoplasms, Experimental - drug therapy | Index Medicus | Drugs | Liver | Iron | Inhibition | Macrophages | Iron oxides | Tumors | Cancer
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8. Full Text Possible involvement of the M2 anti‐inflammatory macrophage phenotype in growth of human gliomas
by Komohara, Y and Ohnishi, K and Kuratsu, J and Takeya, M
The Journal of Pathology, ISSN 0022-3417, 09/2008, Volume 216, Issue 1, pp. 15 - 24
Within tumours, many non‐neoplastic cells such as fibroblasts, endothelial cells, and macrophages assist tumour growth by producing various growth factors and... 
M2 phenotype | CD163 | M‐CSF | glioma | macrophage | CD204 | pro‐inflammatory | microglia | Glioma | M-CSF | Pro-inflammatory | Macrophage | Microglia | MESSENGER-RNA EXPRESSION | PATHOLOGY | HUMAN MONOCYTES | pro-inflammatory | SCAVENGER RECEPTOR CD163 | STIMULATING FACTOR-I | ALTERNATIVELY ACTIVATED MACROPHAGES | ONCOLOGY | ANTICANCER THERAPIES | TUMOR-ASSOCIATED MACROPHAGES | POLYMERASE CHAIN-REACTION | ANTIGEN-PRESENTING CELLS | Microglia - metabolism | Scavenger Receptors, Class A - analysis | Humans | Middle Aged | Receptors, Cell Surface - analysis | Brain Neoplasms - pathology | Male | Brain Neoplasms - metabolism | Glioma - metabolism | Antigens, CD - analysis | Glioma - genetics | Glioma - pathology | Polymerase Chain Reaction | Aged, 80 and over | Microglia - pathology | Adult | Female | Macrophage Colony-Stimulating Factor - genetics | Enzyme-Linked Immunosorbent Assay | Macrophages - pathology | Macrophage Colony-Stimulating Factor - metabolism | Macrophages - metabolism | Phenotype | Antigens, Differentiation, Myelomonocytic - analysis | Aged | Index Medicus
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9. Full Text High salt reduces the activation of IL-4- and IL-13-stimulated macrophages
by Binger, Katrina J and Gebhardt, Matthias and Heinig, Matthias and Rintisch, Carola and Schroeder, Agnes and Neuhofer, Wolfgang and Hilgers, Karl and Manzel, Arndt and Schwartz, Christian and Kleinewietfeld, Markus and Voelkl, Jakob and Schatz, Valentin and Linker, Ralf A and Lang, Florian and Voehringer, David and Wright, Mark D and Hubner, Norbert and Dechend, Ralf and Jantsch, Jonathan and Titze, Jens and Müller, Dominik N
Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4223 - 4238
A high intake of dietary salt (NaCl) has been implicated in the development of hypertension, chronic inflammation, and autoimmune diseases. We have recently... 
MEDICINE, RESEARCH & EXPERIMENTAL | M2 MACROPHAGES | NA+ STORAGE | TISSUE | GENE-EXPRESSION | URINARY SODIUM | TRANSCRIPTION FACTOR | POTASSIUM EXCRETION | ALTERNATIVE ACTIVATION | BLOOD-PRESSURE | T-CELLS | Sodium Chloride, Dietary - pharmacology | Sodium Chloride - pharmacology | Male | Glycolysis - drug effects | Proto-Oncogene Proteins c-akt - genetics | Oxidative Phosphorylation - drug effects | Interleukin-4 - pharmacology | Histone Code - drug effects | Bone Marrow Cells - drug effects | TOR Serine-Threonine Kinases - physiology | Wound Healing - drug effects | Macrophages - immunology | Macrophages - classification | Immunity, Innate - drug effects | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Inflammation | Proto-Oncogene Proteins c-akt - physiology | Mitochondria - drug effects | Random Allocation | Interleukin-13 - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Sodium Chloride, Dietary - toxicity | Chitin - toxicity | Macrophages - drug effects | Mice | Macrophage Activation - drug effects | Salt | Medical research | Immune response | Medicine, Experimental | Research | Macrophages | Properties | Observations | Biological control systems | Health aspects | Hypertension | Wound healing | Sodium | Cytokines | Kinases | Metabolism | Gene expression | Experiments | Acquisitions & mergers | Index Medicus | Abridged Index Medicus
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10. Full Text Alternatively activated alveolar macrophages in pulmonary fibrosis—mediator production and intracellular signal transduction
by Pechkovsky, Dmitri V and Prasse, Antje and Kollert, Florian and Engel, Kathrin M.Y and Dentler, Jan and Luttmann, Werner and Friedrich, Karlheinz and Müller-Quernheim, Joachim and Zissel, Gernot
Clinical Immunology, ISSN 1521-6616, 2010, Volume 137, Issue 1, pp. 89 - 101
Abstract Activated macrophages have been characterized as M1 and M2 according to their inflammatory response pattern. Here we analyzed the M2 marker expression... 
Allergy and Immunology | M2 phenotype | Th2 cytokines | Pulmonary fibrosis | IL-1RA | CCL22 | Sarcoidosis | JAK/STAT pathway | CCL18 | Alveolar macrophages | CCL17 | INTERLEUKIN-1 RECEPTOR ANTAGONIST | CYTOKINE | IMMUNOLOGY | SYSTEMIC-SCLEROSIS | STAT3 ACTIVATION | INTERSTITIAL LUNG-DISEASES | MANNOSE RECEPTOR | GENE-EXPRESSION | TNF-ALPHA RELEASE | GROWTH-FACTOR | UP-REGULATION | Mannose-Binding Lectins - metabolism | Tumor Necrosis Factor-alpha - metabolism | Gene Expression - drug effects | Humans | Middle Aged | STAT Transcription Factors - metabolism | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Monocytes - metabolism | Monocytes - immunology | Sarcoidosis, Pulmonary - diagnosis | Idiopathic Pulmonary Fibrosis - metabolism | Young Adult | Lectins, C-Type - metabolism | Signal Transduction - immunology | Interleukin-4 - pharmacology | Interleukin 1 Receptor Antagonist Protein - metabolism | Scleroderma, Systemic - complications | Aged, 80 and over | Adult | Female | Bronchoalveolar Lavage Fluid - cytology | Pulmonary Fibrosis - metabolism | Interleukin-8 - metabolism | Macrophages, Alveolar - immunology | Pulmonary Fibrosis - etiology | Sarcoidosis, Pulmonary - complications | Cytokines - metabolism | Macrophage Activation - immunology | Scleroderma, Systemic - metabolism | Pulmonary Fibrosis - immunology | Receptors, Cell Surface - metabolism | Idiopathic Pulmonary Fibrosis - immunology | Monocytes - drug effects | Signal Transduction - drug effects | Macrophages, Alveolar - drug effects | Sarcoidosis, Pulmonary - metabolism | Chemokines, CC - genetics | Aged | Macrophages, Alveolar - metabolism | Macrophage Activation - drug effects | Interleukin-10 - pharmacology | Chemokines, CC - metabolism | Respiratory tract diseases | Systemic scleroderma | Transforming growth factors | Macrophages | Scleroderma (Disease) | Index Medicus | Alveoli
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11. Full Text Macrophage polarity in cancer: A review
by Najafi, Masoud and Hashemi Goradel, Nasser and Farhood, Bagher and Salehi, Eniseh and Nashtaei, Maryam Shabani and Khanlarkhani, Neda and Khezri, Zahra and Majidpoor, Jamal and Abouzaripour, Morteza and Habibi, Mohsen and Kashani, Iraj Ragerdi and Mortezaee, Keywan
Journal of Cellular Biochemistry, ISSN 0730-2312, 03/2019, Volume 120, Issue 3, pp. 2756 - 2765
Macrophages are the most abundant cells within the tumor stroma displaying noticeable plasticity, which allows them to perform several functions within the... 
M1 cell | cancer | therapy | M2 cell | polarity | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DELIVERY | CELL BIOLOGY | ZOLEDRONIC ACID | NANOPARTICLES | TUMOR-ASSOCIATED MACROPHAGES | POLARIZATION | Cell proliferation | Reversing | Cytotoxicity | Lymphocytes T | Macrophages | Suppressor cells | Genotype & phenotype | Signal transduction | Angiogenesis | Receptors | Killer cells | Lymphocytes | Fibroblasts | Polarity | Natural killer cells | Inducers | Phenotypes | Cell survival | Immunoregulation | Invasiveness | Stroma | Inflammation | Radiation therapy | Immunosuppression | Immune checkpoint | Major histocompatibility complex | Tumor suppressor genes | Hypoxia | Transduction | Histocompatibility | Cancer | Tumors
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12. Full Text Decidual CD68+HLA‐DR+CD163− M1 macrophages increase in miscarriages with normal fetal chromosome
by Shimada, Shigeki and Ebina, Yasuhiko and Iijima, Norifumi and Deguchi, Masashi and Yamada, Hideto
American Journal of Reproductive Immunology, ISSN 1046-7408, 02/2018, Volume 79, Issue 2, pp. e12791 - n/a
ProblemIs an abnormal increase or decrease of M1/M2 macrophages observed in the deciduae of miscarriages with normal fetal chromosome (MN)? Methods of... 
M2 macrophages | macrophages | fetal chromosomes | miscarriages | M1 macrophages | LOCALIZATION | CELLS | IMMUNOLOGY | PREGNANCY | REPRODUCTIVE BIOLOGY | CSF | ABORTION | LINK | BLOCKADE | UP-REGULATION | EXPRESSION | Pregnancy | CD163 antigen | Flow cytometry | Fetuses | Monoclonal antibodies | Histocompatibility antigen HLA | Miscarriage | Decidua | Macrophages | Chromosomes | Index Medicus
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