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PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, pp. e66796 - e66796
Human protein kinases (HPKs) have profound effects on cellular responses. To better understand the role of HPKs and the signaling networks that influence... 
A VIRUS | SIGNAL-TRANSDUCTION | ANTIVIRAL THERAPY | AVIAN INFLUENZA | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | CELL-CYCLE ARREST | NF-KAPPA-B | RESISTANT INFLUENZA | RESPIRATORY SYNCYTIAL VIRUS | Protein Kinases - metabolism | Up-Regulation | Protein Kinases - genetics | Influenza, Human - pathology | MicroRNAs - antagonists & inhibitors | Virus Replication - genetics | Humans | Influenza A virus - genetics | Influenza, Human - enzymology | Protein Kinases - chemistry | MicroRNAs - metabolism | RNA Interference | NIMA-Related Kinases - antagonists & inhibitors | Base Sequence | Madin Darby Canine Kidney Cells | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Viral Core Proteins - metabolism | Influenza, Human - virology | MicroRNAs - agonists | RNA, Ribosomal, 18S - metabolism | MAP Kinase Kinase Kinase 1 - antagonists & inhibitors | Protein-Serine-Threonine Kinases - metabolism | MAP Kinase Kinase Kinase 1 - metabolism | A549 Cells | Influenza A virus - physiology | RNA, Ribosomal, 18S - genetics | Protein-Serine-Threonine Kinases - genetics | Influenza A virus - isolation & purification | Phenotype | Sequence Alignment | Animals | Influenza, Human - genetics | Dogs | NIMA-Related Kinases - metabolism | NIMA-Related Kinases - genetics | MAP Kinase Kinase Kinase 1 - genetics | RNA-Binding Proteins - metabolism | Influenza viruses | MicroRNA | Health aspects | Protein kinases | Influenza | Genes | Pandemics | Transcription | Genomics | MiRNA | Viruses | Infections | Antagonists | siRNA | Kinases | Gene expression | Ribonucleic acid--RNA | Proteins | Studies | Signal transduction | Signaling | Infectious diseases | Swine flu | MicroRNAs | Cell cycle | Proteomics | Replication | Viral infections | Apoptosis | Index Medicus | RNA | Ribonucleic acid
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 56, Issue 1, pp. 192 - 198
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, pp. e0127885 - e0127885
Recent studies have suggested that 5-aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside (AICAR) increases macrophage phagocytosis through adenosine... 
TGF-BETA | 5-AMINOIMIDAZOLE-4-CARBOXAMIDE RIBOSIDE | SIGNALING PATHWAY | MAP KINASE | MULTIDISCIPLINARY SCIENCES | INHIBITS EFFEROCYTOSIS | ACUTE LUNG INJURY | SAURURUS-CHINENSIS | BACTERIAL PHAGOCYTOSIS | MUSCLE-CELLS | EXPRESSION | AMP-Activated Protein Kinases - metabolism | Apoptosis - drug effects | Apoptosis - genetics | Male | Phagocytosis - genetics | Aminoimidazole Carboxamide - pharmacology | Gene Knockdown Techniques | Ribonucleotides - pharmacology | Phosphorylation - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | MAP Kinase Kinase Kinases - antagonists & inhibitors | Phagocytosis - drug effects | MAP Kinase Kinase Kinases - genetics | AMP-Activated Protein Kinases - antagonists & inhibitors | Macrophages, Peritoneal - enzymology | p38 Mitogen-Activated Protein Kinases - genetics | Imidazoles - pharmacology | MAP Kinase Kinase Kinases - metabolism | Enzyme Activation - drug effects | Animals | Aminoimidazole Carboxamide - analogs & derivatives | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mice | Mice, Inbred BALB C | Pyridines - pharmacology | Enzyme Activation - genetics | AMP-Activated Protein Kinases - genetics | Bone morphogenetic proteins | Inflammation | Adenylic acid | Macrophages | Transforming growth factors | Mitogens | Protein kinases | Apoptosis | Phosphorylation | Transforming growth factor-b | Homeostasis | AKT protein | Activation | Kinases | AMP-activated protein kinase | Medical schools | Proteins | Phagocytes | Cell activation | Pain | Penicillin | Cell cycle | Inhibition | Growth factors | Adenosine | Adenosine monophosphate | AMP | TAK1 protein | Neutrophils | Dipyridamole | MAP kinase | Leukocytes (neutrophilic) | siRNA | 1-Phosphatidylinositol 3-kinase | Medicine | Studies | Inhibitors | Thymocytes | Lungs | Adenosine kinase | Stem cells | Anesthesiology | Laboratory animals | Transporter | Phagocytosis | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2013, Volume 33, Issue 12, pp. 2792 - 2799
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 07/2006, Volume 145, Issue 1, pp. 162 - 172
The Th2 cytokines interleukin (IL)‐4 and IL‐13 and chemokine monocyte chemoattractant protein‐1 (MCP‐1) are significantly involved in bronchial hyperreactivity... 
chemokines/monokines | allergy | epithelial cells | cytokines/interleukins | signalling/signal transduction | Allergy | Cytokines/interleukins | Epithelial cells | Chemokines/monokines | Signalling/signal transduction | ALLERGIC INFLAMMATION | CYTOKINES | CHEMOKINES | KAPPA-B | RELEASE | IMMUNOLOGY | HUMAN EOSINOPHILS | EOTAXIN-2 | ASTHMA PATHOGENESIS | ALVEOLAR MACROPHAGES | AIRWAY HYPERRESPONSIVENESS | Up-Regulation | Epithelial Cells - metabolism | Gene Expression - drug effects | Protein-Tyrosine Kinases - metabolism | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - metabolism | Interleukins - metabolism | MAP Kinase Signaling System | Dose-Response Relationship, Drug | Janus Kinase 2 | Bronchi - metabolism | Chemokine CCL2 - metabolism | Flavonoids - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins - metabolism | Cell Survival - drug effects | Proto-Oncogene Proteins - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Interleukin-4 - metabolism | Imidazoles - pharmacology | Tyrphostins - pharmacology | Interleukin-13 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mitogen-Activated Protein Kinase 3 - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Pyridines - pharmacology | Enzyme Activation | Cell Line, Transformed | Protein-Tyrosine Kinases - antagonists & inhibitors | Proteins | Mutual fund industry | Protein kinases | Index Medicus | signal transduction | monokines | cytokines | Basic Immunology | chemokines | signalling | interleukins
Journal Article
The Plant Cell, ISSN 1040-4651, 12/2012, Volume 24, Issue 12, pp. 4948 - 4960
Spatiotemporal-specific cell proliferation and cell differentiation are critical to the formation of normal tissues, organs, and organisms. The highly... 
Phenotypes | Cell growth | Receptors | Inflorescences | Architecture | RESEARCH ARTICLES | Gene expression regulation | Plants | Pedicels | Genotypes | Plant cells | ACTIVATION | PHYTOALEXIN BIOSYNTHESIS | STOMATAL DEVELOPMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | PLANT DEVELOPMENT | PLANT SCIENCES | CELL BIOLOGY | SALT STRESS | OVULE DEVELOPMENT | THALIANA | TRANSCRIPTION FACTOR | SIGNALING SPECIFICITY | INNATE IMMUNITY | Arabidopsis Proteins - genetics | Mitogen-Activated Protein Kinase Kinases - genetics | Arabidopsis - enzymology | Cell Proliferation | Plants, Genetically Modified - genetics | MAP Kinase Kinase Kinases - genetics | Arabidopsis - cytology | Plants, Genetically Modified - enzymology | Protein-Serine-Threonine Kinases - genetics | Gene Expression Regulation, Plant - genetics | Receptors, Cell Surface - metabolism | MAP Kinase Kinase Kinases - metabolism | Arabidopsis - metabolism | Arabidopsis - genetics | Arabidopsis Proteins - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Plants, Genetically Modified - metabolism | Gene Expression Regulation, Plant - physiology | Mitogen-Activated Protein Kinases - genetics | Plants, Genetically Modified - cytology | Protein-Serine-Threonine Kinases - metabolism | Mitogen-Activated Protein Kinases - metabolism | Receptors, Cell Surface - genetics | Arabidopsis thaliana | Cell proliferation | Physiological aspects | Research | Cell differentiation | Health aspects | Mitogen-activated protein kinases | Index Medicus | s
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2015, Volume 290, Issue 32, pp. 19900 - 19909
Accumulating evidence suggests that activation of mitogen-activated protein kinases (MAPKs) and nuclear factor NF-kappa B exacerbates early brain injury (EBI)... 
SIGNALING PATHWAYS | VASOSPASM | LEARNING-DEFICITS | PHOSPHORYLATION | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | MECHANISMS | NF-KAPPA-B | EXPRESSION | PROTECTS | Neurons - pathology | Phosphorylation | Transcription Factor RelA - antagonists & inhibitors | Apoptosis - drug effects | Injections, Intraventricular | Male | Cerebrovascular Trauma - metabolism | I-kappa B Proteins - metabolism | I-kappa B Proteins - genetics | Transcription Factor RelA - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | MAP Kinase Kinase 4 - metabolism | Neuroprotective Agents - pharmacology | I-kappa B Proteins - antagonists & inhibitors | MAP Kinase Kinase 4 - antagonists & inhibitors | Neurons - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | MAP Kinase Kinase Kinases - antagonists & inhibitors | Neurons - drug effects | Subarachnoid Hemorrhage - drug therapy | Disease Models, Animal | NF-KappaB Inhibitor alpha | Signal Transduction | MAP Kinase Kinase Kinases - genetics | Gene Expression Regulation | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Zearalenone - analogs & derivatives | MAP Kinase Kinase Kinases - metabolism | Subarachnoid Hemorrhage - genetics | Subarachnoid Hemorrhage - pathology | Rats, Sprague-Dawley | Stereotaxic Techniques | Subarachnoid Hemorrhage - metabolism | Animals | Transcription Factor RelA - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cerebrovascular Trauma - pathology | MAP Kinase Kinase 4 - genetics | Cerebrovascular Trauma - prevention & control | Protein Kinase Inhibitors - pharmacology | Cerebrovascular Trauma - genetics | Zearalenone - pharmacology | Index Medicus | TAK1 | neuroprotection | 5Z-7-oxozeaenol | subarachnoid hemorrhage | Neurobiology | mitogen-activated protein kinase (MAPK) | apoptosis | brain | NF-κB (NF-KB)
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 01/2011, Volume 300, Issue 1, pp. E103 - E110
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2014, Volume 124, Issue 8, pp. 3566 - 3578
The MAP kinase kinase kinase TGF beta-activated kinase 1 (TAK1) is activated by TLRs, IL-1, TNF, and TGF beta and in turn activates IKK-NF-kappa beta and JNK,... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | HEPATOCELLULAR-CARCINOMA | ACTIVATED PROTEIN-KINASE | LIPID-METABOLISM | TAK1 | INSULIN-RESISTANCE | IN-VIVO | NF-KAPPA-B | CANCER | HEPATOCYTE-SPECIFIC DELETION | AMP-Activated Protein Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Autophagy - physiology | Hepatocytes - metabolism | Liver Neoplasms, Experimental - prevention & control | Autophagy - drug effects | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | TOR Serine-Threonine Kinases - antagonists & inhibitors | Liver Neoplasms, Experimental - metabolism | Multiprotein Complexes - metabolism | Hepatocytes - cytology | MAP Kinase Kinase Kinases - deficiency | Liver Neoplasms, Experimental - genetics | Fatty Acids - metabolism | Hepatocytes - drug effects | Fatty Liver - genetics | Fatty Liver - metabolism | Oxidation-Reduction | MAP Kinase Kinase Kinases - genetics | Mice, Inbred C57BL | Fatty Liver - prevention & control | Mice, Transgenic | MAP Kinase Kinase Kinases - metabolism | Sirolimus - pharmacology | Mice, Knockout | Animals | Mice | PPAR alpha - metabolism | Prevention | Autophagy (Cytology) | Liver diseases | Physiological aspects | Biological oxidation (Metabolism) | Research | Protein kinases | Studies | Phosphorylation | Rodents | Liver | Lipids | Biosynthesis | Tumorigenesis | Kinases | Laboratory animals | Gene expression | Autophagy | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e100880 - e100880
Tumor cells upregulate many cell signaling pathways, with AKT being one of the key kinases to be activated in a variety of malignancies. GSK2110183 and... 
BREAST-CANCER | APOPTOSIS | ACTIVATING MUTATION | PHOSPHORYLATION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | PLECKSTRIN HOMOLOGY DOMAIN | RICTOR | INDUCTION | EGFR | Ribosomal Protein S6 Kinases - metabolism | Pyrazoles - chemical synthesis | Antineoplastic Agents - chemical synthesis | Humans | Gene Expression Regulation, Neoplastic | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Pancreatic Neoplasms - drug therapy | PTEN Phosphohydrolase - antagonists & inhibitors | Female | Antineoplastic Agents - pharmacology | Drug Evaluation, Preclinical | MAP Kinase Kinase Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | Pyrazoles - pharmacology | PTEN Phosphohydrolase - genetics | Protein Kinase Inhibitors - chemical synthesis | Signal Transduction | Administration, Oral | MAP Kinase Kinase Kinases - genetics | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - enzymology | PTEN Phosphohydrolase - metabolism | Pancreatic Neoplasms - genetics | MAP Kinase Kinase Kinases - metabolism | Mice, SCID | Drug Synergism | Phosphatidylinositol 3-Kinases - genetics | Xenograft Model Antitumor Assays | Animals | Tumor Burden - drug effects | Cell Line, Tumor | Diamines - pharmacology | Mice | Protein Kinase Inhibitors - pharmacology | Blood Glucose - metabolism | Diamines - chemical synthesis | Ribosomal Protein S6 Kinases - antagonists & inhibitors | Ribosomal Protein S6 Kinases - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Cellular signal transduction | Glucose | Phosphotransferases | Dextrose | Tumors | Biotechnology | Phosphorylation | Animal models | Leukemia | Homeostasis | Oncology | AKT protein | Kinases | K-Ras protein | Anticancer properties | Feedback | Polyethylene glycol | Inhibition | Enzymes | Tumor cells | MAP kinase | Breast cancer | Bioavailability | Substrates | 1-Phosphatidylinositol 3-kinase | Studies | Signaling | Inhibitors | Pancreatic cancer | Cell lines | Mutation | Laboratory animals | ATP | PTEN protein | Cancer | Apoptosis | Index Medicus
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 09/2011, Volume 194, Issue 5, pp. 751 - 764
The c-Jun N-terminal kinase (JNK) signaling pathway is essential for neuronal degeneration in multiple contexts but also regulates neuronal homeostasis. It... 
PROGRAMMED CELL-DEATH | SIGNALING PATHWAY | N-TERMINAL KINASE | WALLERIAN DEGENERATION | SYMPATHETIC NEURONS | ACTIVATED PROTEIN-KINASES | C-JUN | MICROTUBULE DYNAMICS | MICE LACKING | NERVE GROWTH-FACTOR | CELL BIOLOGY | RNA, Small Interfering - genetics | Caspase 9 - metabolism | Central Nervous System - metabolism | Apoptosis - drug effects | Homeodomain Proteins - metabolism | Caspase 3 - metabolism | Central Nervous System - pathology | Axons - physiology | Ganglia, Spinal - cytology | Central Nervous System - embryology | Neurons - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Protein Kinase Inhibitors - pharmacokinetics | Embryo, Mammalian - pathology | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Ganglia, Spinal - embryology | MAP Kinase Kinase Kinases - genetics | Mice, Transgenic | MAP Kinase Kinase Kinases - metabolism | Mice, Knockout | Central Nervous System - cytology | Embryo, Mammalian - embryology | Axons - pathology | Ganglia, Spinal - pathology | Nerve Growth Factor - deficiency | Mice | Protein Binding - physiology | Neurons - pathology | Phosphorylation | Spinal Cord - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Protein Transport - physiology | Extracellular Signal-Regulated MAP Kinases - metabolism | MAP Kinase Kinase Kinases - deficiency | Spinal Cord - pathology | Neurons - drug effects | Proto-Oncogene Proteins c-jun - genetics | Cells, Cultured | Axons - metabolism | Nerve Growth Factor - pharmacology | Mice, Inbred Strains | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Spinal Cord - embryology | Animals | Proto-Oncogene Proteins c-jun - metabolism | Adaptor Proteins, Signal Transducing - genetics | Apoptosis - physiology | Adaptor Proteins, Signal Transducing - metabolism | Physiological aspects | Neurons | Apoptosis | Signal transduction | Kinases | Cells | Index Medicus | s
Journal Article