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2004, ISBN 030648238X, [xiv], 218
Book
The New England Journal of Medicine, ISSN 0028-4793, 11/2014, Volume 371, Issue 22, pp. 2061 - 2071
Journal Article
Circulation Research, ISSN 0009-7330, 01/2012, Volume 110, Issue 2, pp. 312 - 324
RATIONALE:Marfan syndrome (MFS) is a systemic connective tissue disorder notable for the development of aortic root aneurysms and the subsequent... 
aneurysm | apoptosis | microRNA | extracellular matrix | Marfan syndrome | FIBROSIS | KAPPA-B ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | AORTIC-ANEURYSM | KINASE | FAILURE | MICRORNA EXPRESSION SIGNATURE | INFLAMMATION | METALLOPROTEINASE SECRETION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | REVEALS | Up-Regulation | Age Factors | Aortic Aneurysm - metabolism | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Aorta - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Marfan Syndrome - therapy | Female | Marfan Syndrome - complications | Microfilament Proteins - metabolism | Aortic Aneurysm - pathology | Microfilament Proteins - genetics | Real-Time Polymerase Chain Reaction | Aortic Aneurysm - prevention & control | Disease Models, Animal | Elastin - metabolism | Fibrillin-1 | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Losartan - pharmacology | Fibrillins | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Marfan Syndrome - genetics | Apoptosis Regulatory Proteins - metabolism | Aorta - pathology | Animals | Elastin - genetics | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Oligonucleotides, Antisense - administration & dosage | Mice | MicroRNAs - genetics | Aortic Aneurysm - genetics | Transforming Growth Factor beta - metabolism | Apoptosis | Genetic Therapy - methods
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2014, Volume 64, Issue 16, pp. 1725 - 1739
Journal Article
Science, ISSN 0036-8075, 4/2006, Volume 312, Issue 5770, pp. 117 - 121
Aortic aneurysm and dissection are manifestations of Marfan syndrome (MFS), a disorder caused by mutations in the gene that encodes fibrillin-1. Selected... 
Airspace | Receptors | Architecture | Dilatation | Root growth | Placebos | Reports | Mice | Aortic aneurysm | Elastic tissue | Marfan syndrome | PATHOGENESIS | GROWTH-FACTOR-BETA | ACTIVATION | FIBRILLIN | MULTIDISCIPLINARY SCIENCES | RATS | RECEPTOR | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | DILATATION | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Propranolol - therapeutic use | Neutralization Tests | Aortic Aneurysm - etiology | Lung Diseases - drug therapy | Marfan Syndrome - drug therapy | Propranolol - administration & dosage | Antibodies - immunology | Transforming Growth Factor beta - antagonists & inhibitors | Female | Marfan Syndrome - complications | Microfilament Proteins - genetics | Adrenergic beta-Antagonists - administration & dosage | Adrenergic beta-Antagonists - therapeutic use | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Aortic Aneurysm - prevention & control | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Lung - pathology | Pulmonary Alveoli - pathology | Signal Transduction | Fibrillins | Pregnancy Complications - drug therapy | Aorta - pathology | Pregnancy | Elastic Tissue - pathology | Animals | Receptor, Angiotensin, Type 1 - metabolism | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Losartan - administration & dosage | Lung Diseases - pathology | Mutation | Transforming Growth Factor beta - metabolism | Complications and side effects | Aortic aneurysms | Genetic aspects | Research | Risk factors
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2015, Volume 35, Issue 4, pp. 911 - 917
Objective-Studies of mice with mild Marfan syndrome (MFS) have correlated the development of thoracic aortic aneurysm (TAA) with improper stimulation of... 
aortic aneurysm | receptor, angiotensin, type 1 | losartan | transforming growth factor β | Marfan syndrome | PATHOGENESIS | TGF-BETA | type 1 | transforming growth factor beta | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | receptor, angiotensin | Aortic Aneurysm, Thoracic - genetics | Phosphorylation | Aortic Aneurysm, Thoracic - prevention & control | Humans | Aortic Rupture - metabolism | Mice, 129 Strain | Angiotensin II Type 1 Receptor Blockers - pharmacology | Marfan Syndrome - drug therapy | Aortic Aneurysm, Thoracic - pathology | Time Factors | Mice, Mutant Strains | Aortic Rupture - prevention & control | Transforming Growth Factor beta - antagonists & inhibitors | Aorta, Thoracic - drug effects | Microfilament Proteins - genetics | Aorta, Thoracic - pathology | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Mice, Inbred C57BL | Losartan - pharmacology | Smad2 Protein - metabolism | Aorta, Thoracic - metabolism | Antibodies, Neutralizing - pharmacology | Aortic Rupture - pathology | Fibrillins | Disease Progression | Marfan Syndrome - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Aortic Aneurysm, Thoracic - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Aortic Rupture - genetics | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Mutation | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2014, Volume 124, Issue 3, pp. 1329 - 1339
Patients with Marfan syndrome (MFS), a multisystem disorder caused by mutations in the gene encoding the extracellular matrix (ECM) protein fibrillin 1, are... 
MEDICINE, RESEARCH & EXPERIMENTAL | LOSARTAN | AORTIC-ANEURYSM | MYOCARDIAL FIBROSIS | MECHANICAL-STRESS | SIGNIFICANT VALVULAR REGURGITATION | MOUSE MODEL | ANGIOTENSIN-II | DILATED CARDIOMYOPATHY | DYSFUNCTION | LEFT-VENTRICULAR FUNCTION | Cardiomyopathy, Dilated - pathology | Humans | Extracellular Matrix - metabolism | Male | Marfan Syndrome - physiopathology | Angiotensin II Type 1 Receptor Blockers - pharmacology | MAP Kinase Signaling System | Myocardium - metabolism | Adult | Marfan Syndrome - complications | Microfilament Proteins - metabolism | Child | Focal Adhesion Kinase 1 - metabolism | Fibrillin-1 | Cross-Sectional Studies | Losartan - pharmacology | Organ Size | Fibrillins | Mice, Transgenic | Myocardium - pathology | Cardiomyopathy, Dilated - metabolism | Mechanotransduction, Cellular | Animals | Myocytes, Cardiac - drug effects | Cardiomyopathy, Dilated - etiology | Cardiomyopathy, Dilated - physiopathology | Receptor, Angiotensin, Type 1 - metabolism | Marfan Syndrome - metabolism | Myocytes, Cardiac - metabolism | Marfan Syndrome - pathology | Mice | Heart cells | Physiological aspects | Cellular signal transduction | Glycoproteins | Research | Properties | Marfan syndrome | Studies | Heart | Microscopy | Pathogenesis | Cardiomyopathy | Rodents | Homeostasis | Cardiomyocytes | Mutation | Bioavailability
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 132, Issue 2, pp. 378 - 386
Journal Article