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Journal of Cellular Biochemistry, ISSN 0730-2312, 11/2017, Volume 118, Issue 11, pp. 3756 - 3764
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 04/2006, Volume 47, Issue 7, pp. 1369 - 1378
OBJECTIVES: We examined the effect of C-reactive protein (CRP) on matrix metalloproteinase (MMP) and inhibitor expression in endothelial cells and in patients... 
COMPLEMENT | ACTIVATION | ATHEROGENESIS | CARDIAC & CARDIOVASCULAR SYSTEMS | MMP-1 | ACUTE MYOCARDIAL-INFARCTION | CARDIOVASCULAR-DISEASE | GENE-EXPRESSION | PLAQUES | INDUCED IN-VITRO | LESIONS | Carotid Arteries - metabolism | MAP Kinase Signaling System - physiology | Mammary Arteries - metabolism | Humans | Middle Aged | Male | Matrix Metalloproteinase 1 - biosynthesis | Metalloendopeptidases - metabolism | Gene Expression Profiling | Atherosclerosis - etiology | C-Reactive Protein - metabolism | Microarray Analysis | C-Reactive Protein - pharmacology | Enzyme Induction - drug effects | Adult | Female | Umbilical Veins - cytology | Tissue Inhibitor of Metalloproteinase-1 - blood | Cells, Cultured | Recombinant Proteins - pharmacology | C-Reactive Protein - physiology | Matrix Metalloproteinase 10 | Atherosclerosis - metabolism | Immunohistochemistry - methods | Metalloendopeptidases - biosynthesis | Matrix Metalloproteinase 1 - blood | Staining and Labeling | Metalloendopeptidases - blood | Aorta - cytology | Matrix Metalloproteinase 1 - metabolism | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Heart attacks | Disease | Triglycerides | Gene expression | Cholesterol | Proteins | Body mass index | Lipoproteins | Risk assessment | Atherosclerosis | Blood pressure | Deoxyribonucleic acid--DNA | Veins & arteries
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 09/2014, Volume 74, pp. 99 - 107
Aberrant matrix metalloproteinase-1 (MMP-1) expression contributes to the pathogenesis of many degenerative disease processes that are associated with... 
Oxidative stress | Redox-dependent | Hydrogen peroxide | Free radicals | Epigenetic modification | Steady-state H2O2 concentration | Matrix metalloproteinase-1 | Histone deacetylase-2 | Acetylation | Manganese superoxide dismutase | HISTONE DEACETYLASE 2 | TRANSCRIPTIONAL ACTIVATION | C-FOS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MANGANESE SUPEROXIDE-DISMUTASE | POOR-PROGNOSIS | ENDOCRINOLOGY & METABOLISM | HYDROGEN-PEROXIDE | PROMOTER | NF-KAPPA-B | RNA, Small Interfering - genetics | Superoxide Dismutase - genetics | Oxidation-Reduction | Oxidative Stress | Catalase - genetics | Epigenesis, Genetic | Humans | Transgenes - genetics | JNK Mitogen-Activated Protein Kinases - metabolism | Promoter Regions, Genetic - genetics | Hydrogen Peroxide - metabolism | Catalase - metabolism | Gene Expression Regulation, Enzymologic | Ubiquitination | Cell Line, Tumor | Transcription, Genetic | Histone Deacetylase 2 - metabolism | Histones - metabolism | JNK Mitogen-Activated Protein Kinases - genetics | Proteasome Endopeptidase Complex - metabolism | Matrix Metalloproteinase 1 - genetics | Superoxide Dismutase - metabolism | Antioxidants | Enzymes | Genes | Genetic research | Histones | Superoxide | Gene expression | Cells | acetylation | redox-dependent | steady state H2O2 concentration (SS-[H2O2]) | matrix metalloproteinase-1 (MMP-1) | hydrogen peroxide | epigenetic modification | manganese superoxide dismutase (Sod2) | histone deacetylase-2 | oxidative stress
Journal Article
Immunology, ISSN 0019-2805, 11/2013, Volume 140, Issue 3, pp. 301 - 313
Summary We reported recently that treatment of diabetic apolipoprotein E‐deficient mice with the Toll‐like receptor 4 (TLR4) antagonist Rs‐LPS, a... 
mononuclear cells | Toll‐like receptor | interferon‐γ | inflammation | MD‐2 | MD-2 | Inflammation | Interferon-γ | Mononuclear cells | Toll-like receptor | ACTIVATION | IFN-GAMMA | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | interferon | LPS | GRAM-NEGATIVE BACTERIA | LIPOPOLYSACCHARIDE-RECOGNITION | MMP-1 EXPRESSION | E-DEFICIENT MICE | TRANSCRIPTION FACTOR | TLR4 | RNA, Small Interfering - genetics | Hypoadrenocorticism, Familial | Lymphocyte Antigen 96 - metabolism | Humans | Antibodies, Blocking - pharmacology | Monocytes - immunology | Carbohydrate Metabolism, Inborn Errors - immunology | Lymphocyte Antigen 96 - genetics | Glycerol Kinase - deficiency | U937 Cells | Inflammation Mediators - metabolism | Toll-Like Receptor 4 - antagonists & inhibitors | Matrix Metalloproteinase 1 - genetics | Inflammation Mediators - immunology | Glycerol Kinase - immunology | Atherosclerosis - immunology | Gene Expression Regulation - genetics | Glucose - immunology | Signal Transduction - genetics | Toll-Like Receptor 4 - metabolism | Lymphocyte Antigen 96 - antagonists & inhibitors | Mice, Knockout | Gene Expression Regulation - drug effects | Monocytes - drug effects | Animals | Rhodobacter sphaeroides - immunology | Apolipoproteins E - genetics | Interferon-gamma - immunology | Lipopolysaccharides - pharmacology | Mice | Matrix Metalloproteinase 1 - metabolism | Interferon-gamma - pharmacology | interferon-γ | Original
Journal Article
by Huang, CC and Wang, CH and Wu, PW and He, JR and Chang, PH and Fu, CH and Lee, TJ
SCIENTIFIC REPORTS, ISSN 2045-2322, 10/2019, Volume 9, Issue 1, pp. 1 - 10
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, p. e25103
Journal Article
Journal Article
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 05/2004, Volume 109, Issue 17, pp. 2046 - 2049
Journal Article