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American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 06/2015, Volume 308, Issue 9, pp. C699 - C709
Duchenne muscular dystrophy (DMD) is characterized by progressive muscle wasting secondary to repeated muscle damage and inadequate repair. Elevations in... 
Mdx | Mdx/Utr | Calcium | Duchenne muscular dystrophy | Injury | SERCA1 | mdx | MDX MOUSE | CA2+ ENTRY | PHYSIOLOGY | calcium | ECCENTRIC CONTRACTIONS | CONTRACTION-INDUCED INJURY | DEFICIENT MICE | CELL BIOLOGY | CA2+-DEPENDENT PROTEOLYSIS | COUPLING FAILURE | DUCHENNE MUSCULAR-DYSTROPHY | TORQUE PRODUCTION | injury | mdx/Utr(-/-) | CYTOSKELETAL DAMAGE | Up-Regulation | Torque | Utrophin - genetics | Quadriceps Muscle - physiopathology | Necrosis | Utrophin - deficiency | Quadriceps Muscle - enzymology | Muscular Dystrophy, Duchenne - physiopathology | Mice, Inbred mdx | Quadriceps Muscle - pathology | Creatine Kinase, MM Form - blood | Muscle Strength | Calcium Signaling | Disease Models, Animal | Severity of Illness Index | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Muscular Dystrophy, Duchenne - enzymology | Organ Size | Genotype | Mice, Transgenic | Muscular Dystrophy, Duchenne - pathology | Biomarkers - blood | Biomechanical Phenomena | Phenotype | Animals | Muscle Contraction | Muscular Dystrophy, Duchenne - blood | Muscular Dystrophy, Duchenne - genetics | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Hypertrophy | Calcium-transporting ATPases | Muscles | Physiological aspects | Health aspects | Biomarkers | Gene expression | Rodents | Muscular dystrophy | Adenosine triphosphatase | Index Medicus | Utr
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 09/2018, Volume 115, Issue 39, pp. E9182 - E9191
In gene therapy for Duchenne muscular dystrophy there are two potential immunological obstacles. An individual with Duchenne muscular dystrophy has a genetic... 
Gene replacement therapy | Mdx/mTRG2 mice | DNA plasmid | Duchenne muscular dystrophy | Microdystrophin | STEM-CELLS | microdystrophin | TRANSGENIC MDX MICE | MULTIDISCIPLINARY SCIENCES | MUSCULAR-DYSTROPHY | PYRUVATE-KINASE PK | mdx/mTRG2 mice | SOMATIC REVERSION SUPPRESSION | gene replacement therapy | ADENOASSOCIATED VIRAL VECTORS | MYELIN BASIC-PROTEIN | SKELETAL-MUSCLE | MOUSE MODEL | IN-VIVO | DNA - pharmacokinetics | Dependovirus - genetics | Muscle Strength - genetics | Male | Muscle Strength - immunology | DNA - genetics | Plasmids - pharmacology | Animals | Dystrophin - genetics | Genetic Vectors - pharmacology | Muscular Dystrophy, Duchenne - immunology | Mice, Inbred mdx | Plasmids - genetics | Dystrophin - immunology | Mice | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Muscular Dystrophy, Duchenne - therapy | Dystrophin - metabolism | Disease Models, Animal | Genetic Therapy - methods | Care and treatment | Plasmids | Genetic aspects | Gene therapy | Health aspects | Methods | Multiple sclerosis | Antibodies | Viruses | Diabetes mellitus (insulin dependent) | Immunity | Muscular dystrophy | Proteins | Immunology | Technology | Duchenne's muscular dystrophy | Plasmid DNA | Dystrophin | Deoxyribonucleic acid--DNA | Recombinant | Myelin | Diabetes mellitus | Muscles | Gene expression | Insulin | Immunogenicity | Muscle function | Mutation | Dystrophy | Index Medicus | mdx | Biological Sciences | PNAS Plus | mTRG2 mice
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 5/2002, Volume 157, Issue 5, pp. 851 - 864
Three populations of myogenic cells were isolated from normal mouse skeletal muscle based on their adhesion characteristics and proliferation behaviors.... 
Skeletal muscle satellite cells | Stem cells | Multipotent stem cells | Muscles | Cultured cells | Transplantation | Cells | Hematopoietic stem cells | Cell transplantation | Skeletal muscle | Mdx mice | Muscle-derived stem cells (MDSC) | Satellite cells | Dystrophin | MDX MOUSE | dystrophin | IMMUNOSUPPRESSIVE TREATMENT | CELL BIOLOGY | muscle-derived stem cells (MDSC) | cell transplantation | SKELETAL-MUSCLE | IN-VITRO | MYOBLAST TRANSFER THERAPY | mdx mice | HISTOCOMPATIBILITY COMPLEX ANTIGENS | DUCHENNE MUSCULAR-DYSTROPHY | GENE-THERAPY | MYOGENIC PROGENITORS | satellite cells | Stem Cells - immunology | Vascular Endothelial Growth Factor A | Vascular Endothelial Growth Factors | Endothelial Growth Factors - pharmacology | Stem Cells - cytology | Muscle, Skeletal - cytology | Muscular Dystrophy, Animal - pathology | CD4-Positive T-Lymphocytes - immunology | Stem Cell Transplantation | Dystrophin - physiology | Muscle, Skeletal - immunology | Mice, Inbred mdx | Lymphokines - pharmacology | Cell Differentiation - physiology | Cell Separation | Mice, Inbred C57BL | Hematopoietic Stem Cell Transplantation | Nerve Growth Factor - pharmacology | Muscle, Skeletal - physiology | Regeneration - physiology | Cell Division - drug effects | Cell Division - physiology | Animals | Cell Differentiation - drug effects | Biomarkers | Muscle Fibers, Skeletal - cytology | Mice | CD8-Positive T-Lymphocytes - immunology | In Vitro Techniques | Cell research | Striated muscle | Analysis | Identification | Muscular system | Cellular biology | Rodents | Index Medicus | muscle-derived stem cells (MDSC); satellite cells; cell transplantation; dystrophin; mdx mice
Journal Article
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 7/2013, Volume 353, Issue 1, pp. 173 - 187
The renin-angiotensin system (RAS), through angiotensin II and the angiotensin-converting enzyme (ACE), is involved in the genesis and progression of fibrotic... 
Human Genetics | Biomedicine | Mouse ( mdx model of DMD) | Proteomics | Fibrosis | Extracellular matrix | Duchenne muscular dystrophy (DMD) | Molecular Medicine | Angiotensin-converting enzyme | Skeletal muscle | Mouse (mdx model of DMD) | SYSTEM | CELLS | OXIDATIVE STRESS | TGF-BETA | MOUSE | HUMAN MUSCULAR-DYSTROPHY | EXERCISED MDX MICE | FACTOR-I | CELL BIOLOGY | FIBROTIC RESPONSE | ACCUMULATION | Antihypertensive Agents - pharmacology | Transforming Growth Factor beta1 - metabolism | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Connective Tissue Growth Factor - biosynthesis | Renin-Angiotensin System | Mice, Inbred mdx | Muscular Dystrophy, Animal - metabolism | Peptidyl-Dipeptidase A - metabolism | Adenoviridae - genetics | Transforming Growth Factor beta1 - drug effects | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Physical Conditioning, Animal | Transforming Growth Factor beta1 - biosynthesis | Muscle, Skeletal - enzymology | Angiotensin II - metabolism | Mice, Inbred C57BL | Adenoviridae Infections | Animals | Enalapril - pharmacology | Signal Transduction - drug effects | Mice | Muscle Strength - drug effects | Muscle, Skeletal - pathology | Connective Tissue Growth Factor - metabolism | Enzymes | Angiotensin | Muscles | Bone morphogenetic proteins | Transforming growth factors | ACE inhibitors | Angiogenesis | Biomedical research | Muscular system | Muscular dystrophy | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 02/2017, Volume 8, Issue 1, pp. 14454 - 14454
Gene replacement therapies utilizing adeno-associated viral (AAV) vectors hold great promise for treating Duchenne muscular dystrophy (DMD). A related approach... 
CRISPR-CAS9 | SKELETAL | EXPRESSION | VECTORS | MULTIDISCIPLINARY SCIENCES | MDX MICE | Index Medicus
Journal Article
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2015, Volume 21, Issue 7, pp. 786 - 794
Journal Article