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Journal of Biological Chemistry, ISSN 0021-9258, 10/2007, Volume 282, Issue 41, pp. 30014 - 30021
The transcriptional coactivator peroxisome proliferator-activated receptor gamma coactivator 1 alpha (PGC-1 alpha) is a key integrator of neuromuscular... 
OXIDATIVE-PHOSPHORYLATION | MDX MOUSE | SIGNALING PATHWAYS | MITOCHONDRIAL BIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INFLAMMATORY MYOPATHIES | GENE-EXPRESSION | PGC-1 FAMILY | DUCHENNE MUSCULAR-DYSTROPHY | ENERGY-METABOLISM | RECEPTOR-ALPHA
Journal Article
PLOS ONE, ISSN 1932-6203, 06/2018, Volume 13, Issue 6
The lack of dystrophin in Duchenne muscular dystrophy (DMD) compromises the integrity and function of muscle fibers. Skeletal muscles, except the diaphragm, do... 
SKELETAL-MUSCLE | MDX MOUSE | ACTIVATION | MULTIDISCIPLINARY SCIENCES | DEFICIENT | MUSCULAR-DYSTROPHY | SARCOLEMMA | PATHOLOGY | MODEL | EXPRESSION | PATHOPHYSIOLOGY
Journal Article
Cell Death and Disease, ISSN 2041-4889, 11/2012, Volume 3, Issue 11, pp. e418 - e418
A resolutive therapy for Duchene muscular dystrophy, a severe degenerative disease of the skeletal muscle, is still lacking. Because autophagy has been shown... 
Therapy | Autophagy | Duchenne muscular dystrophy | MDX MOUSE | autophagy | therapy | FOXO3 | CELL BIOLOGY | SKELETAL-MUSCLE | PATHWAY | DOWNSTREAM | DEACETYLASE INHIBITORS | IN-VIVO | NITRIC-OXIDE | MICE | DIFFERENTIATION | Index Medicus
Journal Article
Cell, ISSN 0092-8674, 12/2017, Volume 171, Issue 7, pp. 1495 - 1507.e15
Journal Article
PLOS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1
Various therapeutic approaches have been studied for the treatment of Duchenne muscular dystrophy (DMD), but none of these approaches have led to significant... 
MDX MOUSE | STEM-CELLS | UTROPHIN | NORMAL MYOGENIC CELLS | REGENERATION | MULTIDISCIPLINARY SCIENCES | INJECTIONS | DEFICIENT MICE | MYOFIBERS | EXPRESSION | TRANSPLANTATION
Journal Article
by Li, M and Arner, A
PLOS ONE, ISSN 1932-6203, 11/2015, Volume 10, Issue 11
Muscular dystrophies are often caused by genetic alterations in the dystrophin-dystroglycan complex or its extracellular ligands. These structures are... 
SKELETAL-MUSCLE | MDX MOUSE | PROTEIN | EMBRYO | MULTIDISCIPLINARY SCIENCES | CONTRACTION-INDUCED INJURY | MICE | MODEL
Journal Article
Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography, ISSN 0894-7317, 8/2017, Volume 30, Issue 8, pp. 815 - 827.e9
Journal Article
FASEB JOURNAL, ISSN 0892-6638, 07/2019, Volume 33, Issue 7, pp. 8110 - 8124
The C57BL/10ScSn-Dmd(mdx)/J (BL10-mdx) mouse has been the most commonly used model for Duchenne muscular dystrophy (DMD) for decades. Their muscle dysfunction... 
MDX MOUSE | pathology | regeneration | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHENOTYPE | SATELLITE CELLS | muscle function | CELL BIOLOGY | GENE | BIOLOGY | fibrosis | MICE | calcification | Research
Journal Article
SCIENCE TRANSLATIONAL MEDICINE, ISSN 1946-6234, 12/2014, Volume 6, Issue 267
We have previously observed that Wnt signaling activates a fibrogenic program in adult muscle stem cells, called satellite cells, during aging. We genetically... 
MEDICINE, RESEARCH & EXPERIMENTAL | MDX MOUSE | TGF-BETA | WNT/BETA-CATENIN | SIGNALING PATHWAY | COLLAGEN-PRODUCING CELLS | TRANSFORMING GROWTH-FACTOR-BETA-1 | DUCHENNE MUSCULAR-DYSTROPHY | SATELLITE CELL | INJURED SKELETAL-MUSCLE | MOLECULAR-MECHANISMS | CELL BIOLOGY
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 03/2019, Volume 28, Issue 6, pp. 942 - 951
Abstract Duchenne muscular dystrophy is a deadly muscle-wasting disorder caused by loss of dystrophin protein. Studies suggest that metabolic alterations are... 
SKELETAL-MUSCLE | MDX MOUSE | COMPLEX | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | NITRIC-OXIDE | SARCOLEMMA | DUCHENNE MUSCULAR-DYSTROPHY | MICE | EXERCISE | DEFICIENCY | PATHOPHYSIOLOGY
Journal Article
Nature Medicine, ISSN 1078-8956, 2015, Volume 21, Issue 3, pp. 270 - 275
Journal Article
HUMAN MOLECULAR GENETICS, ISSN 0964-6906, 12/2013, Volume 22, Issue 24, pp. 4914 - 4928
Phosphorodiamidate morpholino oligomer (PMO)-mediated exon skipping is among the more promising approaches to the treatment of several neuromuscular disorders... 
MDX MOUSE | TARGETED DISRUPTION | PHOSPHORODIAMIDATE MORPHOLINO OLIGOMERS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | MUSCLE | ANTISENSE OLIGONUCLEOTIDES | PLASMA-MEMBRANE | DUCHENNE DYSTROPHY | CELLULAR UPTAKE | ALPHA-2 CHAIN
Journal Article
JER, ISSN 2288-176X, 03/2019, Volume 15, Issue 2, pp. 213 - 223
Exercise therapy on skeletal muscle of muscular dystrophies has no defined parameters. The effect of low-intensity treadmill training on the oxidative stress... 
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2018, Volume 315, Issue 6, pp. H1544 - H1552
Cardiomyopathy is a significant contributor to morbidity and mortality in Duchenne muscular dystrophy (DMD). Membrane instability, leading to intracellular... 
Heart | Mdx | Calcium | Dystrophin | Duchenne muscular dystrophy | mdx | MDX MOUSE | COMPLEX | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | calcium | MECHANISMS | dystrophin | DILATED CARDIOMYOPATHY | heart | EVOLUTION | PERIPHERAL VASCULAR DISEASE | DUCHENNE MUSCULAR-DYSTROPHY | MICE | DYSFUNCTION
Journal Article