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Journal of Cellular Biochemistry, ISSN 0730-2312, 12/2017, Volume 118, Issue 12, pp. 4548 - 4557
Journal Article
Cancer Cell, ISSN 1535-6108, 05/2017, Volume 31, Issue 5, pp. 669 - 684.e7
Polyploidy can lead to aneuploidy and tumorigenesis. Here, we report that the Hippo pathway effector Yap promotes the diploid-polyploid conversion and... 
p27 | polyploidy | Hippo | Yap | tumorigenesis | genomic instability | Skp2 | UBIQUITIN-MEDIATED DEGRADATION | ORGAN SIZE | ACTIVATION | DEPENDENT REGULATION | ONCOLOGY | PATHWAY | SIZE-CONTROL | TUMOR SUPPRESSORS | MAMMALIAN-CELLS | CANCER | GENOMIC INTEGRITY | CELL BIOLOGY | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | Phosphoproteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Cytosol - enzymology | Transfection | RNA Interference | Time Factors | Cell Transformation, Neoplastic - genetics | Forkhead Transcription Factors - metabolism | Proteolysis | Carcinoma, Hepatocellular - genetics | Female | Liver Neoplasms - pathology | Acetylation | Protein Stability | Liver Neoplasms - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Protein-Serine-Threonine Kinases - metabolism | Genetic Predisposition to Disease | Liver Neoplasms - genetics | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Carcinoma, Hepatocellular - enzymology | Phosphoproteins - genetics | Forkhead Transcription Factors - genetics | S-Phase Kinase-Associated Proteins - metabolism | Cell Transformation, Neoplastic - metabolism | Hep G2 Cells | Pregnancy | p300-CBP Transcription Factors - metabolism | Phenotype | Animals | Epistasis, Genetic | Ploidies | Adaptor Proteins, Signal Transducing - genetics | Carcinoma, Hepatocellular - pathology | S-Phase Kinase-Associated Proteins - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Cell Transformation, Neoplastic - pathology | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Medical colleges | Molecular biology | Ligases | Liver | Cells | Tumors
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2011, Volume 13, Issue 7, pp. 827 - 837
Compartmentalization of signals generated by receptor tyrosine kinase (RTK) endocytosis has emerged as a major determinant of various cell functions. Here,... 
SIGNAL-TRANSDUCTION | TYROSINE KINASE | CELL-MIGRATION | SPATIAL RESTRICTION | C-MET | MEDIATED ENDOCYTOSIS | ENDOPLASMIC-RETICULUM | EGF RECEPTOR | SOMATIC MUTATIONS | HUMAN CANCER | CELL BIOLOGY | Proto-Oncogene Proteins c-met - metabolism | NIH 3T3 Cells | Dynamins - metabolism | Phosphorylation | Proto-Oncogene Proteins c-cbl - metabolism | Cell Proliferation | Humans | Receptors, Growth Factor - antagonists & inhibitors | Recombinant Fusion Proteins - metabolism | Receptors, Growth Factor - genetics | Transfection | Lung Neoplasms - secondary | RNA Interference | Time Factors | Cell Transformation, Neoplastic - genetics | Female | Soft Tissue Neoplasms - enzymology | Soft Tissue Neoplasms - genetics | Lung Neoplasms - genetics | Lung Neoplasms - enzymology | Signal Transduction | Neoplasm Invasiveness | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Endocytosis - drug effects | Tumor Burden | Cell Transformation, Neoplastic - metabolism | Clathrin - metabolism | Protein Transport | Proto-Oncogene Proteins c-met - genetics | Cell Movement - drug effects | Stress Fibers - metabolism | Animals | Lung Neoplasms - prevention & control | Mice, Nude | Soft Tissue Neoplasms - prevention & control | Soft Tissue Neoplasms - pathology | Endosomes - enzymology | Mice | Protein Kinase Inhibitors - pharmacology | Cell Transformation, Neoplastic - drug effects | Enzyme Activation | Mutation | Cell Transformation, Neoplastic - pathology | GRB2 Adaptor Protein - metabolism | rac1 GTP-Binding Protein - metabolism | Receptors, Growth Factor - metabolism | rac1 GTP-Binding Protein - genetics | Endocytosis | Recycling (Waste, etc.) | Physiological aspects | United Kingdom | Research | France | Carcinogenesis | Proto-oncogenes
Journal Article
Cancer Research, ISSN 0008-5472, 05/2010, Volume 70, Issue 9, pp. 3537 - 3546
K-Ras-induced lung cancer is a very common disease, for which there are currently no effective therapies. Because therapy directly targeting the activity of... 
ACTIVATION | EPITHELIAL-CELLS | ONCOGENIC RAS | ONCOLOGY | PANCREATIC-CANCER | MOUSE MODEL | MEDIATED TRANSFORMATION | IN-VIVO | LINKS INFLAMMATION | TUMOR-GROWTH | CELLULAR-TRANSFORMATION | NF-κB | IKKβ | K-Ras | Lung cancer | p65 | RelA
Journal Article
Journal Article
Journal Article