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PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, pp. e95588 - e95588
The PI3K/Akt and mTOR signaling pathways are important for cell survival and growth, and they are highly activated in cancer cells compared with normal cells.... 
T(14/18) LYMPHOMA-CELLS | BREAST-CANCER CELLS | LUNG-CANCER | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | IN-VIVO | FACTOR-KAPPA-B | ANTITUMOR-ACTIVITY | PHOSPHATIDYLINOSITOL 3-KINASE/MAMMALIAN TARGET | PI3K/MTOR INHIBITOR | Cell Survival - drug effects | Apoptosis - drug effects | Humans | Curcumin - pharmacology | Cell Survival - genetics | Imidazoles - pharmacology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Quinolines - pharmacology | TOR Serine-Threonine Kinases - antagonists & inhibitors | Carcinoma, Renal Cell - metabolism | Flow Cytometry | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | RNA, Small Interfering | DNA Fragmentation - drug effects | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | TOR protein | Post-transcription | Regulations | Bcl-2 protein | p53 Protein | Gene regulation | AKT protein | Kinases | Cancer therapies | Signal transduction | Rhizomes | Pathways | Bioactive compounds | Fibroblasts | Mesangial cells | Curcumin | Colon | Inhibition | Medical research | Chemopreventive agents | Cell survival | Mortality | Gene expression | Biological activity | Mcl-1 protein | 1-Phosphatidylinositol 3-kinase | Signaling | Inhibitors | Cell death | Skin | Combined treatment | Synergistic effect | Clear cell-type renal cell carcinoma | Cancer | Apoptosis | Kidney transplantation | Index Medicus
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 11/2017, Volume 6, Issue 11, p. n/a
Background-Activated cardiac fibroblasts (CFs), preglomerular vascular smooth muscle cells (PGVSMCs), and glomerular mesangial cells (GMCs) proliferate, cause... 
kidney | stromal cell‐derived factor | fibroblasts | sitagliptin | cardiac fibroblasts | C‐X‐C motif chemokine receptor 4 | glomerular mesangial cells | dipeptidyl peptidase 4 | hypertension | Hypertension | Dipeptidyl peptidase 4 | Glomerular mesangial cells | Fibroblasts | C-X-C motif chemokine receptor 4 | Sitagliptin | Stromal cell-derived factor | Kidney | CXCR4 ANTAGONISM | CARDIAC & CARDIOVASCULAR SYSTEMS | DIPEPTIDYL-PEPTIDASE-IV | HEART-FAILURE | stromal cell-derived factor | RECEPTOR | INHIBITION | MYOCARDIAL FIBROSIS | FACTOR-1-ALPHA | KIDNEY FIBROSIS | PHARMACOLOGY | RACK1 | Cell Proliferation | Renal Artery - pathology | Muscle, Smooth, Vascular - metabolism | Microvessels - metabolism | Microvessels - pathology | Rats, Inbred WKY | Male | RNA - genetics | Chemokine CXCL12 - genetics | Myocardium - metabolism | Kidney Glomerulus - metabolism | Receptors, CXCR4 - genetics | Renal Artery - metabolism | Collagen - biosynthesis | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Fibroblasts - metabolism | Rats, Inbred SHR | Cardiomyopathy, Hypertrophic - genetics | Cardiomyopathy, Hypertrophic - metabolism | Cells, Cultured | Gene Expression Regulation | Rats | Myocardium - pathology | Kidney Glomerulus - pathology | Mesangial Cells - metabolism | Fibroblasts - pathology | Blotting, Western | Muscle, Smooth, Vascular - pathology | Animals | Receptors, CXCR4 - biosynthesis | Mesangial Cells - pathology | Chemokine CXCL12 - biosynthesis | Cardiomyopathy, Hypertrophic - pathology | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e46453 - e46453
The loss of glomerular podocytes is a key event in the progression of chronic kidney disease resulting in proteinuria and declining function. Podocytes are... 
PLURIPOTENT STEM-CELLS | SOMATIC-CELLS | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | PARIETAL | PROLIFERATION | GLOMERULAR PODOCYTES | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | EXPRESSION | FIBROBLASTS | BETA | Cell Proliferation | Coculture Techniques | Humans | Cell Membrane Permeability | Fluorescein-5-isothiocyanate - metabolism | Fluorescent Dyes - metabolism | Octamer Transcription Factor-3 - genetics | Induced Pluripotent Stem Cells - metabolism | Tretinoin - pharmacology | Gene Expression | Podocytes - metabolism | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - physiology | Tissue Culture Techniques | Mice, Inbred C57BL | Cells, Cultured | Fluorescein-5-isothiocyanate - analogs & derivatives | Kidney - cytology | Mesangial Cells - metabolism | Animals | Activins - pharmacology | Podocytes - physiology | Cell Differentiation - drug effects | Octamer Transcription Factor-3 - metabolism | Cell Aggregation | Mice | Bone Morphogenetic Protein 7 - pharmacology | Serum Albumin - metabolism | Care and treatment | Chronic kidney failure | Stem cells | Angiotensin | Research | Permeability | Tumor proteins | Health aspects | Risk factors | Cell culture | Nephrology | Transplants & implants | Hemodialysis | Laboratories | Pathogenesis | Oct-4 protein | Fluorescence | Biology | mRNA | Regeneration (physiology) | Proteins | Immunology | Rodents | DNA methylation | Fibroblasts | Physiology | Localization | Angiotensin II | Explants | Contractility | Talin | Gene expression | Regeneration | Morphology | Kidney diseases | Differentiation | Pluripotency | Cytoplasm | Proteinuria | Tumors | Kidney transplantation | Index Medicus
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 08/2017, Volume 92, Issue 2, pp. 504 - 513
Journal Article
Journal Article
Diabetologia, ISSN 0012-186X, 1/2013, Volume 56, Issue 1, pp. 204 - 217
Many of the effects of resveratrol are consistent with the activation of AMP-activated protein kinase (AMPK), silent information regulator T1 (SIRT1) and... 
Oxidative stress | Medicine & Public Health | Human Physiology | Metabolic Diseases | Resveratrol | Internal Medicine | Diabetic nephropathy | Apoptosis | TRANSCRIPTIONAL COACTIVATOR PGC-1 | PROTEIN-KINASE | MITOCHONDRIAL-FUNCTION | FATTY-ACID-METABOLISM | PROLIFERATOR-ACTIVATED RECEPTOR | DIABETIC-NEPHROPATHY | SIRT1 DEACETYLASE | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | ALPHA ERR-ALPHA | NF-KAPPA-B | Sirtuin 1 - metabolism | AMP-Activated Protein Kinases - metabolism | Kidney - pathology | Stilbenes - therapeutic use | Male | Diabetes Mellitus, Type 2 - metabolism | Protective Agents - therapeutic use | Stilbenes - pharmacology | Sirtuin 1 - genetics | Mesangial Cells - drug effects | Kidney - metabolism | Protein Processing, Post-Translational - drug effects | RNA Interference | Mice, Mutant Strains | Protective Agents - pharmacology | Sirtuin 1 - chemistry | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Kidney - physiopathology | Kidney - drug effects | AMP-Activated Protein Kinases - antagonists & inhibitors | Mice, Inbred C57BL | Cells, Cultured | Mesangial Cells - metabolism | Enzyme Activation - drug effects | Sirtuin 1 - antagonists & inhibitors | AMP-Activated Protein Kinases - chemistry | Lipotropic Agents - therapeutic use | Transcription Factors - metabolism | Animals | Signal Transduction - drug effects | Lipid Metabolism - drug effects | Mesangial Cells - pathology | Lipotropic Agents - pharmacology | Mice | Diabetes Mellitus, Type 2 - pathology | Oxidative Stress - drug effects | Transcription Factors - agonists | Diabetes Mellitus, Type 2 - drug therapy | AMP-Activated Protein Kinases - genetics | Index Medicus
Journal Article
American Journal of Pathology, ISSN 0002-9440, 11/2009, Volume 175, Issue 5, pp. 2014 - 2022
Viral RNA can trigger interferon signaling in dendritic cells via the innate recognition receptors melanoma-differentiation-associated gene (MDA)-5 and... 
TOLL-LIKE RECEPTOR-3 | IMMUNE-COMPLEXES | DOUBLE-STRANDED-RNA | DENDRITIC CELLS | LUPUS NEPHRITIS | RECOGNITION | ALPHA/BETA | RIG-I | PATH