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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 09/2009, Volume 297, Issue 3, pp. 1096 - 1102
Transient receptor potential vanilloid 4 (TRPV4) channels have been implicated as mediators of calcium influx in both endothelial and vascular smooth muscle... 
Calcium-activated potassium channels | Blood pressure | Epoxyeicosatrienoic acid | Vascular smooth muscle | Transient receptor potential vanilloid 4 | Endothelium | endothelium | blood pressure | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | vascular smooth muscle | calcium-activated potassium channels | transient receptor potential vanilloid 4 | CA2+-ACTIVATED K+ CHANNEL | ARACHIDONIC-ACID | BLOOD-PRESSURE | HYPERPOLARIZING FACTOR | epoxyeicosatrienoic acid | ENDOTHELIAL-CELLS | CATION CHANNEL TRPV4 | PERIPHERAL VASCULAR DISEASE | RECEPTOR POTENTIAL VANILLOID-4 | MICE | EPOXYEICOSATRIENOIC ACIDS | SMALL-CONDUCTANCE | Mesenteric Arteries - physiology | Potassium Channels, Calcium-Activated - physiology | Vasodilator Agents - pharmacology | Mesenteric Arteries - drug effects | Hypertension - physiopathology | TRPV Cation Channels - genetics | Vascular Resistance - physiology | Endothelium, Vascular - physiology | Mice, Knockout | TRPV Cation Channels - metabolism | 8,11,14-Eicosatrienoic Acid - analogs & derivatives | Animals | Vascular Resistance - drug effects | Blood Pressure - physiology | Mice | Nitric Oxide Synthase - metabolism | Potassium Channels, Calcium-Activated - antagonists & inhibitors | Vasodilation - drug effects | Vasodilation - physiology | 8,11,14-Eicosatrienoic Acid - pharmacology | Muscle, Smooth, Vascular - physiology
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 03/2005, Volume 288, Issue 3, pp. 1016 - 1021
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2005, Volume 25, Issue 10, pp. 2106 - 2113
OBJECTIVE—Angiotensin (Ang) II-induced vascular damage may be partially mediated by reactive oxygen species generation and inflammation. Homozygous... 
Hypertension | Macrophages | Reactive oxygen species | KAPPA-B ACTIVATION | SPONTANEOUSLY HYPERTENSIVE-RATS | CHRONIC GRANULOMATOUS-DISEASE | CONVERTING-ENZYME-INHIBITION | BLOOD-PRESSURE | macrophages | NAD(P)H OXIDASE | reactive oxygen species | IN-VIVO | PERIPHERAL VASCULAR DISEASE | SUPEROXIDE PRODUCTION | GROWTH-FACTOR | HEMATOLOGY | hypertension | SMOOTH-MUSCLE CELLS | Vasculitis - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress - physiology | Vasculitis - pathology | NADPH Oxidases - metabolism | Mesenteric Arteries - immunology | Mesenteric Arteries - pathology | Aorta - metabolism | Vasculitis - immunology | Mice, Mutant Strains | Inflammation Mediators - metabolism | Macrophage Colony-Stimulating Factor - genetics | Macrophages - immunology | Endothelium, Vascular - immunology | Inflammation Mediators - immunology | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Osteopetrosis - pathology | Vascular Resistance | Macrophages - pathology | Aorta - immunology | Osteopetrosis - metabolism | Macrophage Colony-Stimulating Factor - metabolism | Mesenteric Arteries - metabolism | Mice, Inbred Strains | Osteopetrosis - immunology | Aorta - pathology | Phenotype | Animals | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Mice | Oxidative Stress - drug effects
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0138142
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2018, Volume 38, Issue 9, pp. 1969 - 1985
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 04/2017, Volume 174, Issue 8, pp. 657 - 671
Background and Purpose The cofactor tetrahydrobiopterin (BH4) is a critical regulator of endothelial NOS (eNOS) function, eNOS‐derived NO and ROS signalling in... 
DIABETES-MELLITUS | HYPERPOLARIZING FACTOR | CONCISE GUIDE | NITRIC-OXIDE SYNTHASE | IN-VIVO | PHARMACOLOGY & PHARMACY | INDUCED RELAXATION | HYDROGEN-PEROXIDE | GTP CYCLOHYDROLASE-1 | BLOOD-PRESSURE | VASCULAR SUPEROXIDE-PRODUCTION | Mesenteric Arteries - cytology | Nitric Oxide - biosynthesis | Reactive Oxygen Species - metabolism | Biopterin - deficiency | Endothelial Cells - metabolism | GTP Cyclohydrolase - deficiency | Cells, Cultured | Male | GTP Cyclohydrolase - metabolism | Mesenteric Arteries - metabolism | Biopterin - analogs & derivatives | Biopterin - metabolism | Mice, Knockout | Animals | Mice | Nitric Oxide Synthase Type III - metabolism | Nitric Oxide - metabolism | GTP Cyclohydrolase - genetics | COX-2 inhibitors | Enzymes | Vasodilators | Analysis | Cardiac patients | Physiological aspects | Acetylcholine | Tubulins | High performance liquid chromatography | Arteries | Endothelium | GTP | Hydrogen peroxide | Homeostasis | Biosynthesis | Vasodilation | Western blotting | Prostaglandin endoperoxide synthase | Proteins | Clonal deletion | Tetrahydrobiopterin | Rodents | Phenylephrine | Deletion | Therapeutic applications | Vasoconstriction | Liquid chromatography | High-performance liquid chromatography | Nitric-oxide synthase | Endothelial cells | GTP cyclohydrolase | Nitric oxide | Microvasculature | Cardiovascular diseases | Veins & arteries | Research Papers | Research Paper
Journal Article
Journal of Hypertension, ISSN 0263-6352, 03/2014, Volume 32, Issue 3, pp. 542 - 554
Journal Article
Cardiovascular Research, ISSN 0008-6363, 06/2013, Volume 98, Issue 3, pp. 428 - 436
Journal Article