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Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 7, pp. 678 - 684
Recovery from blood loss requires a greatly enhanced supply of iron to support expanded erythropoiesis. After hemorrhage, suppression of the iron-regulatory... 
OVERLOAD | HOMEOSTASIS | DIFFERENTIATION FACTOR 15 | BETA-THALASSEMIA-INTERMEDIA | ERYTHROPOIESIS | GENETICS & HEREDITY | MOUSE-LIVER | MICE | HEPCIDIN EXPRESSION | ANEMIA | HYPOXIA | Hepcidins - metabolism | beta-Thalassemia - pathology | Liver - pathology | Iron Overload - drug therapy | Molecular Sequence Data | Erythropoiesis - drug effects | Male | Gene Expression Profiling | Iron Overload - pathology | Iron Overload - metabolism | Epoetin Alfa | Erythropoietin - metabolism | Real-Time Polymerase Chain Reaction | Anemia - etiology | Disease Models, Animal | Recombinant Proteins - metabolism | Hormones - pharmacology | Enzyme-Linked Immunosorbent Assay | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Hemorrhage - complications | Hemoglobins - metabolism | Hemorrhage - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Iron - metabolism | Blotting, Western | Erythropoiesis - physiology | Mice, Knockout | Anemia - metabolism | Animals | Mice | beta-Thalassemia - metabolism | Physiological aspects | Genetic research | Genetic aspects | Iron in the body | Hormones | Research | Genetic regulation | Identification and classification | Flow cytometry | Plasma | Phosphorylation | Bone marrow | Homeostasis | Iron | Grants | Metabolism | Gene expression | Experiments | Hemorrhage | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 03/2012, Volume 125, Issue 9, pp. 1134 - 1146
Background-Rheb is a GTP-binding protein that promotes cell survival and mediates the cellular response to energy deprivation (ED). The role of Rheb in the... 
apoptosis | signal transduction | obesity | ischemia | MAMMALIAN TARGET | RAPAMYCIN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | MTOR | SKELETAL-MUSCLE | INFARCTION | IMPACT | PATHWAY | INSULIN-RESISTANCE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Myocardial Ischemia - metabolism | TOR Serine-Threonine Kinases - metabolism | Metabolic Syndrome - metabolism | Ras Homolog Enriched in Brain Protein | Autophagy - physiology | TOR Serine-Threonine Kinases - genetics | Dietary Fats - pharmacology | Metabolic Syndrome - physiopathology | Apoptosis Regulatory Proteins - genetics | Myocardial Ischemia - physiopathology | Neuropeptides - genetics | Energy Metabolism - physiology | Cell Survival - physiology | Beclin-1 | Myocytes, Cardiac - cytology | Cells, Cultured | Rats | Neuropeptides - metabolism | Obesity - physiopathology | Glucose - pharmacology | Monomeric GTP-Binding Proteins - genetics | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Myocardial Ischemia - pathology | Obesity - metabolism | Obesity - pathology | Animals | Myocytes, Cardiac - drug effects | Monomeric GTP-Binding Proteins - metabolism | Myocytes, Cardiac - physiology | Signal Transduction - physiology | Mice | Metabolic Syndrome - pathology | Endoplasmic Reticulum Stress - physiology | Physiological aspects | Autophagy (Cytology) | Obesity | Myocardial ischemia | Research | Metabolic syndrome X | Index Medicus | Abridged Index Medicus | myocardial ischemia | autophagy | Rheb | mTORC1
Journal Article
Journal Article
Cell Metabolism, ISSN 1550-4131, 01/2013, Volume 17, Issue 1, pp. 113 - 124
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2011, Volume 108, Issue 45, pp. 18348 - 18353
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 500, Issue 7462, pp. 283 - 284
[...]the resulting 'empty space' in the proteome - which will be filled with enzymes required for coping with the carbon limitation, such as cAMP-regulated... 
Assimilation | Enzymes | Lactose | Microbiology | Metabolites | E coli | Growth rate | Glucose | Nitrogen | Gene expression | Carbon | Bacteriology
Journal Article
Human molecular genetics, ISSN 0964-6906, 12/2014, Volume 23, Issue 24, pp. 6419 - 6431
Genome-wide association studies have revealed > 60 loci associated with type 2 diabetes ( T2D), but the underlying causal variants and functional mechanisms... 
PROVIDES INSIGHTS | COMMON VARIANTS | RISK LOCI | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUSCEPTIBILITY | GENETICS & HEREDITY | GENE-EXPRESSION | PANCREATIC BETA-CELLS | TYPE-2 DIABETES-MELLITUS | PROINSULIN LEVELS | GENOME-WIDE ASSOCIATION | ADIPOSE-TISSUE | LIM-Homeodomain Proteins - metabolism | Proinsulin - metabolism | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Humans | Diabetes Mellitus, Type 2 - metabolism | Genetic Loci | Transcription Factor 7-Like 2 Protein - metabolism | Maf Transcription Factors, Large - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Maf Transcription Factors, Large - genetics | Islets of Langerhans - metabolism | Trans-Activators - genetics | Transcription, Genetic | Insulin - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Basic Helix-Loop-Helix Transcription Factors - genetics | Transcription Factor 7-Like 2 Protein - genetics | Islets of Langerhans - pathology | Signal Transduction | Gene Expression Regulation | Mice, Transgenic | Proinsulin - genetics | Transcription Factors - genetics | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Insulin - metabolism | LIM-Homeodomain Proteins - genetics | Animals | Alleles | Trans-Activators - metabolism | High-Throughput Nucleotide Sequencing | Mice | Polymorphism, Single Nucleotide | Diabetes Mellitus, Type 2 - pathology | Index Medicus | Biological Sciences | Biochemistry and Molecular Biology | Naturvetenskap | Natural Sciences | Biokemi och molekylärbiologi | Biologiska vetenskaper
Journal Article
Journal Article
Nature Genetics, ISSN 1061-4036, 07/2009, Volume 41, Issue 7, pp. 829 - U89
Journal Article