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Science, ISSN 0036-8075, 6/2001, Volume 292, Issue 5522, pp. 1728 - 1731
Journal Article
Science, ISSN 0036-8075, 7/2008, Volume 321, Issue 5887, pp. 408 - 411
When intracellular pathogens invade mammalian hosts, naive CD8(+) T cells differentiate into cytotoxic killers, which lyse infected target cells and secrete... 
T lymphocytes | Spleen | Pathology | Lymphocytic choriomeningitis virus | Cytokines | Neutrophils | Viruses | Reports | Infections | Mice | Genotypes | LINEAGE COMMITMENT | EFFECTOR | CD4 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DISTINCT | DIRECTS | DIFFERENTIATION | TRANSCRIPTION FACTOR | IL-21 | Lymphocytic choriomeningitis virus - immunology | T-Box Domain Proteins - physiology | Arenaviridae Infections - virology | Interferon-gamma - metabolism | Lymphocyte Depletion | CD4-Positive T-Lymphocytes - immunology | Lymphocytic choriomeningitis virus - isolation & purification | Wasting Syndrome - immunology | CD8-Positive T-Lymphocytes - metabolism | Wasting Syndrome - virology | Cell Differentiation | Lymphocytic choriomeningitis virus - physiology | Arenaviridae Infections - immunology | T-Box Domain Proteins - deficiency | Mice, Inbred C57BL | Arenaviridae Infections - pathology | T-Box Domain Proteins - genetics | Mice, Knockout | Antigens, Viral - immunology | Interleukin-17 - metabolism | Animals | Virus Replication | CD8-Positive T-Lymphocytes - immunology | Cytotoxicity, Immunologic | Wasting Syndrome - pathology | Virus diseases | Killer cells | CD8 lymphocytes | Influence | Genetic aspects | Cell-mediated cytotoxicity | DNA binding proteins | Research | Properties | Cell differentiation | Immunology | Biomedical research | Cellular biology | Rodents | Tc17 | autoimmunity | IL-17 | IL-23 | Th17 | IL-22 | RORγt
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 06/2012, Volume 166, Issue 4, pp. 1474 - 1489
BACKGROUND AND PURPOSE The endocannabinoid system may regulate glial cell functions and their responses to pathological stimuli, specifically, Alzheimer's... 
Neuroinflammation | beta‐amyloid | astrocytes | TRPV1 | PPAR | Endocannabinoids | beta-amyloid | ENDOCANNABINOID ANANDAMIDE | GLIOMA-CELLS | ACTIVATION | RESPONSES | IN-VITRO | DISEASE | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | CANNABINOIDS | BRAIN | Neurons - cytology | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | PPAR gamma - metabolism | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | TRPV Cation Channels - metabolism | Astrocytes - immunology | Cyclooxygenase 2 - genetics | TRPV Cation Channels - antagonists & inhibitors | Amyloid beta-Peptides - metabolism | Neurons - metabolism | Neurons - drug effects | PPAR gamma - genetics | Amidohydrolases - metabolism | Animals, Newborn | Astrocytes - cytology | Astrocytes - drug effects | Nerve Tissue Proteins - antagonists & inhibitors | Peptide Fragments - metabolism | Cytokines - metabolism | Amidohydrolases - genetics | Mice, Inbred C57BL | Nerve Tissue Proteins - agonists | Cells, Cultured | Enzyme Inhibitors - pharmacology | Neurons - immunology | Amidohydrolases - antagonists & inhibitors | PPAR alpha - genetics | Nerve Tissue Proteins - genetics | TRPV Cation Channels - genetics | Mice, Knockout | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Nitric Oxide Synthase Type II - genetics | Cyclooxygenase 2 - metabolism | PPAR gamma - agonists | PPAR alpha - agonists | Mice | PPAR alpha - metabolism | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Astrocytes - metabolism | Nitric Oxide Synthase Type II - metabolism | Research Papers
Journal Article
Journal Article
The Journal of Pathology, ISSN 0022-3417, 08/2015, Volume 236, Issue 4, pp. 445 - 456
Anaplastic large cell lymphoma (ALCL) is a rare, aggressive, non‐Hodgkin's lymphoma that is characterized by CD30 expression and disease onset in young... 
IL‐22 | IL‐21 | IL‐10 | ALK kinase | ALCL | cytokines | miR‐155 | miR-155 | IL-22 | IL-10 | IL-21 | NPM-ALK | TYROSINE KINASE | PATHOLOGY | IDENTIFICATION | MICRORNA-155 | PATHOGENESIS | ONCOLOGY | GENE-EXPRESSION | HODGKIN LYMPHOMA | LEUKEMIA | PROMOTER | TRANSGENIC MICE | Translocation, Genetic | Humans | Caspase 3 - metabolism | Gene Expression Regulation, Neoplastic | Suppressor of Cytokine Signaling 1 Protein | MicroRNAs - metabolism | Case-Control Studies | Chromosomes, Human, Pair 2 | DNA Methylation | Transfection | Anaplastic Lymphoma Kinase | Lymphoma, Large-Cell, Anaplastic - metabolism | STAT3 Transcription Factor - metabolism | Lymphoma, Large-Cell, Anaplastic - genetics | Promoter Regions, Genetic | Argonaute Proteins - metabolism | Lymphoma, Large-Cell, Anaplastic - therapy | Receptor Protein-Tyrosine Kinases - deficiency | Argonaute Proteins - genetics | Signal Transduction | CCAAT-Enhancer-Binding Protein-beta - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Mice, SCID | CCAAT-Enhancer-Binding Protein-beta - metabolism | Chromosomes, Human, Pair 5 | Xenograft Model Antitumor Assays | Animals | Receptor Protein-Tyrosine Kinases - genetics | Cell Line, Tumor | Mice, Inbred NOD | Lymphoma, Large-Cell, Anaplastic - pathology | MicroRNAs - genetics | Suppressor of Cytokine Signaling Proteins - metabolism | Genetic Therapy - methods | Original Papers
Journal Article
Journal Article
Nature, ISSN 0028-0836, 06/2012, Volume 486, Issue 7402, pp. 256 - 260
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 2013, Volume 15, Issue 12, pp. 1464 - 1472
Journal Article