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The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 12/2017, Volume 140, Issue 6, pp. 1632 - 1642
Eosinophils are a subset of granulocytes that can be involved in the pathogenesis of different diseases, including allergy. Their effector functions are... 
necrosis | eosinophilic disease | reactive oxygen species | non-apoptotic cell death | RIP kinase | Autophagy | mixed lineage kinase-like | eosinophil cytolysis | necroptosis | ACTIVATION | PROGRAMMED NECROSIS | DOMAIN-LIKE | NECROTIC DEATH | EXTRACELLULAR DNA TRAPS | RELEASE | IMMUNOLOGY | CELL-DEATH | ALLERGY | IN-VIVO | PIECEMEAL DEGRANULATION | RIP3 | Protein Kinases - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Signal Transduction | Humans | Cells, Cultured | Hypersensitivity - immunology | Molecular Targeted Therapy | Cell Adhesion | Eosinophils - immunology | Immunoglobulins, Intravenous - metabolism | Complement C3b - metabolism | Eosinophilic Esophagitis - immunology | Cytotoxicity, Immunologic | Skin - immunology | Skin diseases | Cell death | Mitogens | Protein kinases | Fluorescence microscopy | TOR protein | Immunohistochemistry | Cytolysis | Cell culture | Reactive oxygen species | Phosphorylation | Intravenous administration | Pathogenesis | Fluorescence | Cytotoxicity | Activation | Kinases | Experiments | Proteins | Signal transduction | Cell activation | Gangrene | Immunoglobulins | Oxygen | Blood & organ donations | Leukocytes (eosinophilic) | Hypersensitivity | Esophagitis | MAP kinase | Rapamycin | Pharmacology | Electron microscopy | Adhesion | Asthma | Signaling | Microscopy | Granular materials | Protein kinase | Morphology | Degranulation | Granules | Skin | Leukocytes (granulocytic) | Phagocytosis | Apoptosis | Eosinophils | Index Medicus | Abridged Index Medicus
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 07/2001, Volume 21, Issue 14, pp. 4713 - 4724
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
PROGRAMMED CELL-DEATH | CYTOCHROME-C | BINDING PROTEINS RAC1 | INHIBITS APOPTOSIS | SIGNALING PATHWAYS | MIXED-LINEAGE KINASE-2 | PHOSPHATIDYLINOSITOL 3-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | SYMPATHETIC NEURONS | ZIPPER-BEARING KINASE | NERVE GROWTH-FACTOR | CELL BIOLOGY | Humans | Leucine Zippers | Protein-Serine-Threonine Kinases | Neurons - cytology | cdc42 GTP-Binding Protein - metabolism | PC12 Cells | MAP Kinase Kinase 7 | Mitogen-Activated Protein Kinase Kinases - metabolism | Caspases - metabolism | MAP Kinase Kinase 4 | Ultraviolet Rays | JNK Mitogen-Activated Protein Kinases | Neurons - metabolism | Proto-Oncogene Proteins - metabolism | Gene Expression | Nerve Growth Factor - metabolism | Signal Transduction | MAP Kinase Kinase Kinases - genetics | Sympathetic Nervous System - cytology | Rats | MAP Kinase Kinase Kinases - metabolism | Caspase Inhibitors | Cytochrome c Group - metabolism | Proto-Oncogene Proteins c-akt | Myristic Acid - metabolism | Animals | MAP Kinase Kinase Kinases - physiology | MAP Kinase Kinase Kinase 5 | Enzyme Activation | rac1 GTP-Binding Protein - metabolism | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | MLK protein | c-Jun N-terminal kinase | Rac1 protein | DLK protein | c-Jun amino-terminal kinase | cytochrome c | Index Medicus | Cell Growth and Development
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2014, Volume 34, Issue 7, pp. 1429 - 1436
OBJECTIVE—Mitogen-activated protein kinase pathways play an important role in neointima formation secondary to vascular injury, in part by promoting... 
neointima | mitogen-activated protein kinase kinase kinase | MLK3 | PROTEIN-KINASE | DOWN-REGULATION | PROLIFERATION | HYPERPLASIA | APOLIPOPROTEIN-E | IN-VIVO | NUCLEOTIDE EXCHANGE FACTOR | PERIPHERAL VASCULAR DISEASE | GENE-TRANSFER | HEMATOLOGY | SIGNAL-REGULATED KINASE | Cell Proliferation | Hyperplasia | Myocytes, Smooth Muscle - pathology | JNK Mitogen-Activated Protein Kinases - metabolism | Neointima | MAP Kinase Kinase Kinases - deficiency | Time Factors | rho-Associated Kinases - metabolism | Carotid Arteries - enzymology | Carotid Artery Injuries - genetics | Rho Guanine Nucleotide Exchange Factors - metabolism | Myocytes, Smooth Muscle - drug effects | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction | MAP Kinase Kinase Kinases - genetics | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Mice, Knockout | Muscle, Smooth, Vascular - pathology | Animals | Carotid Artery Injuries - enzymology | rho GTP-Binding Proteins - metabolism | Carotid Arteries - pathology | Mice | Protein Kinase Inhibitors - pharmacology | Muscle, Smooth, Vascular - enzymology | Index Medicus | cJun NH2-terminal kinase | p63RhoGEF | Rho-kinase | vascular smooth muscle cell | mixed-lineage kinase | RhoA
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 10/2010, Volume 191, Issue 1, pp. 211 - 223
Regenerative responses to axonal injury involve changes in gene expression; however, little is known about how such changes can be induced from a distant site... 
Axons | Regeneration | Larvae | Neuroscience | Neurons | Drosophila | Nerves | Physical trauma | Gene expression regulation | Physiological regulation | CYTOPLASMIC DYNEIN | DROSOPHILA NEUROMUSCULAR-JUNCTION | SYNAPTIC GROWTH | NERVOUS-SYSTEM | MIXED LINEAGE KINASE | VESICULAR GLUTAMATE TRANSPORTER | ZIPPER-BEARING KINASE | JNK PATHWAY | MICROTUBULE DYNAMICS | N-TERMINAL KINASES | CELL BIOLOGY | Drosophila melanogaster - physiology | Proto-Oncogene Proteins c-jun - physiology | Phosphoprotein Phosphatases - metabolism | Neurons - cytology | Axons - physiology | Drosophila Proteins - metabolism | Drosophila Proteins - physiology | Cell Nucleus - metabolism | Neurons - physiology | Neurons - metabolism | Nerve Tissue Proteins - physiology | Signal Transduction | MAP Kinase Kinase Kinases - genetics | Drosophila melanogaster - cytology | Gene Expression Regulation | MAP Kinase Kinase Kinases - metabolism | Nerve Regeneration | Cell Nucleus - ultrastructure | Nerve Tissue Proteins - genetics | Protein Transport | Nerve Tissue Proteins - metabolism | Animals | Proto-Oncogene Proteins c-jun - metabolism | Drosophila melanogaster - enzymology | MAP Kinase Kinase Kinases - physiology | Drosophila Proteins - genetics | Mutation | Proteins | Research | Properties | Phosphotransferases | Biochemistry | Insects | Kinases | Index Medicus
Journal Article
The EMBO Journal, ISSN 0261-4189, 06/2004, Volume 23, Issue 11, pp. 2226 - 2234
Mitogen‐activated protein kinases (MAPKs) are integral to the mechanisms by which cells respond to physiological stimuli and to a wide variety of environmental... 
MAP kinase | MAPK phosphatase | C. elegans | heavy metal stress | Heavy metal stress | DORSAL CLOSURE | PROTEIN-KINASE PHOSPHATASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEPENDENT APOPTOSIS | TNF SUPERFAMILY | TRANSDUCTION PATHWAY | DROSOPHILA | CELL BIOLOGY | MIXED-LINEAGE KINASE | MOVEMENT | MKP-7 | EIGER | MAP Kinase Kinase 7 - genetics | Dual Specificity Phosphatase 1 | Caenorhabditis elegans Proteins - chemistry | Humans | MAP Kinase Kinase 7 - chemistry | Caenorhabditis elegans Proteins - metabolism | Cercopithecus aethiops | Molecular Sequence Data | JNK Mitogen-Activated Protein Kinases - metabolism | Protein Tyrosine Phosphatases - metabolism | Recombinant Fusion Proteins - metabolism | MAP Kinase Kinase 7 - metabolism | MAP Kinase Kinase Kinases - chemistry | Protein Tyrosine Phosphatases - chemistry | Protein Tyrosine Phosphatases - genetics | Gene Deletion | p38 Mitogen-Activated Protein Kinases - metabolism | Protein Phosphatase 1 | Protein Structure, Tertiary | Amino Acid Sequence | Cell Line | Catalytic Domain | Caenorhabditis elegans - metabolism | Signal Transduction | Caenorhabditis elegans - genetics | MAP Kinase Kinase Kinases - genetics | Metals, Heavy - pharmacology | MAP Kinase Kinase Kinases - metabolism | Cysteine - chemistry | Recombinant Fusion Proteins - chemistry | Caenorhabditis elegans - embryology | Amino Acid Motifs | Sequence Homology, Amino Acid | Metamorphosis, Biological | Animals | Caenorhabditis elegans - drug effects | Larva | Embryo, Nonmammalian | Dual-Specificity Phosphatases | COS Cells | Caenorhabditis elegans Proteins - genetics | Caenorhabditis elegans - enzymology | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 08/2010, Volume 29, Issue 31, pp. 4399 - 4411
The malignant phenotype in breast cancer is driven by aberrant signal transduction pathways. Mixed-lineage kinase-3 (MLK3) is a mammalian mitogen-activated... 
MLK3 | invasion | JNK | Bim | migration | breast cancer | AP-1 | SIGNALING PATHWAYS | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL KINASE | GENE-EXPRESSION PROFILES | DEPENDENT PATHWAY | CELL BIOLOGY | MIXED-LINEAGE KINASE | SRC-HOMOLOGY-3 DOMAIN | ONCOLOGY | BASEMENT-MEMBRANE CULTURES | GENETICS & HEREDITY | P38 MAPK PATHWAYS | C-JUN | Humans | Mammary Glands, Human - metabolism | Cell Movement - genetics | Breast Neoplasms - metabolism | Transfection | Cell Transformation, Neoplastic - genetics | Female | Antineoplastic Agents - pharmacology | Cell Culture Techniques | Gene Expression Regulation, Neoplastic - drug effects | Drug Evaluation, Preclinical | MAP Kinase Kinase Kinases - antagonists & inhibitors | Sirolimus - analogs & derivatives | MAP Kinase Kinase Kinases - genetics | Neoplasm Invasiveness | RNA, Small Interfering - pharmacology | Cells, Cultured | Mammary Glands, Human - pathology | MAP Kinase Kinase Kinases - metabolism | Cell Transformation, Neoplastic - metabolism | Sirolimus - pharmacology | Phenotype | Breast Neoplasms - genetics | Breast Neoplasms - pathology | MAP Kinase Kinase Kinases - physiology | Physiological aspects | Development and progression | Genetic aspects | Breast cancer | Metastasis | Research | Protein kinases | Risk factors | Genotype & phenotype | Signal transduction | Gene expression | Kinases | Cell adhesion & migration | Index Medicus
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 06/2017, Volume 198, Issue 11, pp. 4435 - 4447
Journal Article
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