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British Journal of Haematology, ISSN 0007-1048, 05/2013, Volume 161, Issue 3, pp. 389 - 401
Current therapeutic regimens for acute myeloid leukaemia ( AML ) are still associated with high rates of relapse. Immunotherapy with T ‐cells genetically... 
AML | 123 | cells | CIK | acute myeloid leukaemia | CAR | chimeric antigen receptor | leukaemia stem cell | LSC | cytokine‐induced killer | Leukaemia stem cell (LSC) | Chimeric antigen receptor (CAR) | Acute myeloid leukaemia (AML) | Cytokine-induced killer (CIK) cells | IL3RA (CD123) | acute myeloid leukaemia (AML) | INTERLEUKIN-3 RECEPTOR | chimeric antigen receptor (CAR) | STEM-CELLS | cytokine-induced killer (CIK) cells | ACUTE MYELOGENOUS LEUKEMIA | leukaemia stem cell (LSC) | SUICIDE GENE-THERAPY | ANTIBODY-LIKE IMMUNORECEPTORS | SIGNAL-TRANSDUCTION | ALPHA-CHAIN | HEMATOLOGY | PHASE-I | T-CELLS | ADOPTIVE IMMUNOTHERAPY | Human Umbilical Vein Endothelial Cells | Tumor Stem Cell Assay | Cytotoxicity Tests, Immunologic | Immunotherapy, Adoptive - methods | Endothelial Cells | Transduction, Genetic | Leukemia, Monocytic, Acute - pathology | Leukemia, Myeloid, Acute - pathology | Coculture Techniques | Humans | Cytokines - secretion | Recombinant Fusion Proteins - physiology | Male | Cytokine-Induced Killer Cells - immunology | Monocytes | Leukemia, Myelomonocytic, Acute - pathology | HEK293 Cells | Female | Receptors, Cell Surface - physiology | Cell Line, Tumor - secretion | Hematopoietic Stem Cells | Interleukin-3 Receptor alpha Subunit - antagonists & inhibitors | Antigens | Killer cells | Cytokines | Immunotherapy | Stem cells | Genetically modified organisms | T cells | Health aspects | Index Medicus
Journal Article
Science, ISSN 0036-8075, 11/2009, Volume 326, Issue 5954, pp. 867 - 871
In metazoan organisms, terminal differentiation is generally tightly linked to cell cycle exit, whereas the undifferentiated state of pluripotent stem cells is... 
Monocytes | Cell growth | Stem cells | Cell cycle | Kupffer cells | Cultured cells | Reports | Macrophages | Pluripotent stem cells | Progenitor cells | Cellular differentiation | TRANSFORMATION | STEM-CELLS | DENDRITIC CELLS | MECHANISM | MONOCYTIC DIFFERENTIATION | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | PLURIPOTENCY | CELL-CYCLE EXIT | TERMINAL DIFFERENTIATION | IMMORTALIZATION | Macrophages - transplantation | Up-Regulation | Cell Proliferation | Monocytes - cytology | Proto-Oncogene Proteins c-myc - physiology | Macrophage Colony-Stimulating Factor - pharmacology | Myeloid Progenitor Cells - cytology | MafB Transcription Factor - deficiency | Cell Differentiation | Monocytes - physiology | Macrophages - physiology | MafB Transcription Factor - genetics | Proto-Oncogene Proteins c-maf - genetics | Cells, Cultured | Kruppel-Like Transcription Factors - physiology | Macrophage Colony-Stimulating Factor - metabolism | Macrophages - cytology | MafB Transcription Factor - physiology | Mice, Knockout | Proto-Oncogene Proteins c-maf - physiology | Animals | Proto-Oncogene Proteins c-maf - deficiency | Myeloid Progenitor Cells - physiology | Cell Transformation, Neoplastic | Mice | Proto-Oncogene Proteins c-myc - genetics | Kruppel-Like Transcription Factors - genetics | Phagocytosis | Research | Properties | Cell differentiation | Proteins | Leucocytes | Immune system | Index Medicus | Life Sciences | Immunology
Journal Article
European Journal of Immunology, ISSN 0014-2980, 11/2013, Volume 43, Issue 11, pp. 2930 - 2942
Tumor growth coincides with an accumulation of myeloid‐derived suppressor cells ( MDSC s), which exert immune suppression and which consist of two main... 
Polymorphonuclear MDSC (PMN‐MDSC) | T‐cell activation | Monocytic myeloid‐derived suppressor cell (MO‐MDSC) | CD8 | Nitric oxide | + | T‐cell suppression | Polymorphonuclear MDSC (PMN-MDSC) | Monocytic myeloid-derived suppressor cell (MO-MDSC) | T-cell suppression | T-cell activation | CTL SUPPRESSION | SUBPOPULATIONS | IFN-GAMMA | SELECTIN | IMMUNOLOGY | CD8(+) T-cell activation | NITRIC-OXIDE | DIFFERENTIATION | BEARING MICE | TRANSCRIPTION FACTOR | EXPRESSION | CD8(+) T-cell suppression | ANTIGEN | Up-Regulation | Cell Proliferation | Antigens, CD - biosynthesis | Membrane Glycoproteins - biosynthesis | Lectins, C-Type - biosynthesis | fas Receptor - biosynthesis | L-Selectin - biosynthesis | Receptor, Macrophage Colony-Stimulating Factor - metabolism | STAT5 Transcription Factor - metabolism | Signal Transduction - immunology | Lymphocyte Activation - immunology | STAT1 Transcription Factor - metabolism | Interferon Regulatory Factor-1 | Myeloid Cells - immunology | Antigens, Ly - metabolism | Female | Granzymes - biosynthesis | Interleukin-2 - metabolism | Cell Line | Mice, Inbred C57BL | Neutrophils - immunology | Hyaluronan Receptors - biosynthesis | Mice, Knockout | Cell Adhesion - immunology | Animals | Apoptosis - immunology | Interleukin-2 Receptor alpha Subunit - metabolism | Neoplasms - immunology | Mice | Antigens, Differentiation, T-Lymphocyte - biosynthesis | CD11b Antigen - metabolism | CD8-Positive T-Lymphocytes - immunology | Nitric Oxide - metabolism | Interferon-gamma - biosynthesis | T cells | Tumors | Cytotoxicity | T cell receptors | Immunology | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2017, Volume 12, Issue 5, pp. e0177935 - e0177935
Allogeneic transplantation of blood stem cells from a CCR5-Δ32 homozygous donor to an HIV-infected individual, the "Berlin patient", led to a cure. Since then... 
SYSTEMATIC ANALYSIS | SHRNA | MULTIDISCIPLINARY SCIENCES | SHORT-INTERFERING RNAS | GENE-THERAPY | DICER | STEM-CELL TRANSPLANTATION | MAMMALIAN-CELLS | EXPRESSION | LENTIVIRAL VECTORS | T-CELLS | Cell Line | Leukocytes, Mononuclear - metabolism | RNA, Small Interfering - genetics | HIV-1 - pathogenicity | Down-Regulation | HIV Infections - virology | Humans | Receptors, CCR5 - genetics | HIV-1 - genetics | Leukocytes, Mononuclear - virology | T-Lymphocytes - virology | HIV Infections - pathology | RNA Interference | T-Lymphocytes - metabolism | RNA, Small Interfering - therapeutic use | HEK293 Cells | HIV Infections - therapy | Care and treatment | Physiological aspects | Development and progression | Genetic aspects | Research | HIV infection | Chemokine receptors | Competition | Peptides | Cytotoxicity | Nucleotides | Drug resistance | Inactivation | Design | Acquired immune deficiency syndrome--AIDS | Betaine | Temperature effects | Human immunodeficiency virus--HIV | Biocompatibility | Sensors | Inhibition | Deoxyribonucleic acid--DNA | Construction | CCR5 protein | Complementarity | Base pairs | Denaturation | Vectors | Nucleic acids | Argonaute 2 protein | Gene expression | Antiretroviral therapy | Amplification | Side effects | Inhibitors | Ribonucleic acids | Passengers | Stem cells | Affinity | Mutation | Gene therapy | Logistics | Tropism | Biotechnology | Monocytic leukemia | Accessibility | Transplants & implants | Microbiology | Laboratories | Toxicity | Leukemia | Oligonucleotides | Acute monocytic leukemia | Viruses | Lymphocytes T | Transplantation | Genomes | Infections | Safety engineering | Antiviral activity | Evolution | Nucleotide sequence | Myeloid leukemia | Adhesion | Virology | Reagents | Cell lines | Interferon | Differentiation | Chemokines | Cancer | Index Medicus | Acquired immune deficiency syndrome | AIDS | Human immunodeficiency virus | Deoxyribonucleic acid | HIV | DNA
Journal Article
Cardiovascular Diabetology, ISSN 1475-2840, 06/2018, Volume 17, Issue 1, pp. 78 - 78
Background: High-density lipoproteins (HDLs) can exert anti-atherogenic effects. On top of removing excess cholesterol through reverse cholesterol transport,... 
Endothelial function | Type 2 diabetes mellitus | High-density lipoprotein cholesterol | Endothelial progenitor cells | Monocytic angiogenic cells | APOPTOSIS | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTECTION | ATHEROSCLEROSIS | PROLIFERATION | HDL | OBESITY | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR-DISEASE | COMPLICATIONS | Cell Proliferation | Reactive Oxygen Species - metabolism | Cell Survival | Humans | Middle Aged | Cells, Cultured | Neovascularization, Pathologic | Male | Monocytes - metabolism | Biomarkers - blood | Cell Adhesion | Diabetes Mellitus, Type 2 - diagnosis | Diabetes Mellitus, Type 2 - blood | Phenotype | Endothelial Progenitor Cells - pathology | Monocytes - pathology | Cholesterol, HDL - blood | Female | Aged | Cell Movement | Endothelial Progenitor Cells - metabolism | Cell culture | Diabetic retinopathy | Boyden chamber | Galactosidase | Leukocytes (mononuclear) | Cardiovascular disease | Fluorescent indicators | Density | Epidemiology | Blood | Fibronectin | Angiogenesis | Tubes | Vascular endothelial growth factor | Obesity | Exposure | Regression analysis | Apolipoproteins | Low density lipoprotein | Endothelium | Body mass | Correlation analysis | Stem cells | Viability | Cell migration | Cell proliferation | Adherent cells | Lipoproteins (low density) | Reactive oxygen species | Senescence | Hydrogen peroxide | Lipids | Ethylenediaminetetraacetic acids | Peripheral blood mononuclear cells | Quartiles | Lipoproteins (high density) | Diabetes mellitus (non-insulin dependent) | Hypertension | Diabetes mellitus | Adhesion | Progenitor cells | Cholesterol | Optical density | Monocytes | Lipoproteins | Weight control | Womens health | Cells (biology) | Kidney diseases | Diabetes | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e55481 - e55481
AML1-ETO fusion protein (AE) is generated by t(8;21)(q22;q22) chromosomal translocation, which is one of the most frequently observed structural abnormalities... 
AML1-ETO | PATHWAYS | MONOCYTIC LEUKEMIA | MULTIDISCIPLINARY SCIENCES | G1/S TRANSITION | AML1/ETO | TRANSCRIPTION | KINASE | FUSION PROTEIN | CBP/P300 | ACUTE MYELOID-LEUKEMIA | Oncogene Proteins, Fusion - metabolism | p300-CBP Transcription Factors - antagonists & inhibitors | Apoptosis - drug effects | Humans | Leukemia, Myeloid, Acute - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | p300-CBP Transcription Factors - genetics | Leukemia, Myeloid, Acute - drug therapy | Female | Proto-Oncogene Proteins c-kit - genetics | Hematopoietic Stem Cells - drug effects | Core Binding Factor Alpha 2 Subunit - metabolism | Leukemia, Myeloid, Acute - pathology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Gene Expression Regulation, Leukemic - drug effects | Hematopoietic Stem Cells - metabolism | RUNX1 Translocation Partner 1 Protein | Granulocyte Colony-Stimulating Factor - pharmacology | p300-CBP Transcription Factors - metabolism | Acetylation - drug effects | Animals | Histones - genetics | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Oncogene Proteins, Fusion - genetics | Hematopoietic Stem Cells - cytology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Histones - metabolism | Core Binding Factor Alpha 2 Subunit - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Leukemia, Myeloid, Acute - genetics | Physiological aspects | Genetic aspects | Research | Transferases | Cell proliferation | Transcription | Bcl-2 protein | Leukemia | Event-related potentials | Kinases | Proteins | Cell growth | Allografts | Granulocyte colony-stimulating factor | Peripheral blood | Cell cycle | Colony-stimulating factor | Inhibition | Acetylation | Fusion protein | G1 phase | Translocation | Hematology | Colonies | Myeloid leukemia | Abnormalities | Melanoma | Gene expression | Histone acetyltransferase | c-Kit protein | Patients | AML1 protein | Inhibitors | Cell lines | Stem cells | Leukemogenesis | Leukocytes (granulocytic) | Laboratory animals | Acute myeloid leukemia | Prostate cancer | Histone H3 | Apoptosis | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 12/2013, Volume 191, Issue 12, pp. 6250 - 6260
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e98929 - e98929
Journal Article
Cell Communication and Signaling, ISSN 1478-811X, 2012, Volume 10, Issue 1, pp. 23 - 23
Background: Hepatocyte-like cells (NeoHepatocytes) generated from a peripheral blood monocyte-derived stem cell-like cell (the PCMO) are a promising... 
Extracellular signal-regulated kinase | Hepatocyte-like cells | MEK | Proliferation | EGF | Programmable cells of monocytic origin | PRB | PROTEIN | CELL BIOLOGY |