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European Journal of Neuroscience, ISSN 0953-816X, 06/2009, Volume 29, Issue 11, pp. 2177 - 2186
Journal Article
JOURNAL OF NEUROSCIENCE, ISSN 0270-6474, 03/2016, Volume 36, Issue 10, pp. 3049 - 3063
Journal Article
Parkinsonism and Related Disorders, ISSN 1353-8020, 2011, Volume 18, Issue 1, pp. S183 - S185
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 11/2008, Volume 107, Issue 4, pp. 1147 - 1157
The blood‐brain barrier (BBB) is important physiologically. Pathologically, BBB disruption has been implicated in a wide spectrum of neurological disorders... 
tight junction proteins | matrix metalloproteinase | astrocytes | microglia | Parkinson’s disease | Parkinson's disease | Matrix metalloproteinase | Astrocytes | Tight junction proteins | Microglia | MICROGLIAL ACTIVATION | PERMEABILITY | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | COFFEE CONSUMPTION | REACTIVE MICROGLIA | SUBSTANTIA-NIGRA | NEUROSCIENCES | 1-METHYL-4-PHENYL-1,2,3,6-TETRAHYDROPYRIDINE MPTP | IN-VIVO | GROWTH-FACTOR | PARKINSONS-DISEASE | Tyrosine 3-Monooxygenase - metabolism | Central Nervous System Stimulants - pharmacology | Gliosis - chemically induced | Male | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Occludin | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 9 - genetics | Caffeine - pharmacology | MPTP Poisoning - pathology | Membrane Proteins - metabolism | Corpus Striatum - pathology | Gliosis - prevention & control | Disease Models, Animal | MPTP Poisoning - drug therapy | Blood-Brain Barrier - drug effects | Gene Expression Regulation - drug effects | Animals | Evans Blue | Zonula Occludens-1 Protein | Analysis of Variance | Corpus Striatum - drug effects | Mice | MPTP Poisoning - chemically induced | Proteins | Tyrosine | Physiological aspects | Analysis | Caffeine | Neurology | Biochemistry | Rodents | Parkinsons disease | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 2013, Volume 3, Issue 1, p. 1837
The role of microtubule (MT) dysfunction in Parkinson's disease is emerging. It is still unknown whether it is a cause or a consequence of neurodegeneration.... 
BETA-TUBULIN | BRAIN TUBULIN | IN-VITRO | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | MITOCHONDRIA | DYNAMICS | NEURONS | AXONAL-TRANSPORT | DYSFUNCTION | COGNITIVE DEFICITS | Neuroprotective Agents - therapeutic use | Neurons - pathology | Tyrosine 3-Monooxygenase - metabolism | Substantia Nigra - pathology | Dopaminergic Neurons - pathology | Male | Axonal Transport - drug effects | Epothilones - therapeutic use | Substantia Nigra - metabolism | Immunoenzyme Techniques | Microtubules - metabolism | Tubulin - metabolism | Protein Processing, Post-Translational - drug effects | Microtubules - drug effects | Dopaminergic Neurons - metabolism | Dopaminergic Neurons - drug effects | MPTP Poisoning - pathology | Neurons - metabolism | Neurons - drug effects | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Mice, Inbred C57BL | Parkinsonian Disorders - chemically induced | Microtubules - pathology | MPTP Poisoning - prevention & control | Blotting, Western | Tubulin Modulators - therapeutic use | Substantia Nigra - drug effects | Animals | Parkinsonian Disorders - pathology | Dopamine Agents - toxicity | Mice | Parkinsonian Disorders - prevention & control | MPTP Poisoning - chemically induced | Nerve Degeneration | Neuroprotection | Basal ganglia | Dopamine | Parkinson's disease | Neurodegenerative diseases | Central nervous system diseases | Mitochondria | Tubulin | Neurodegeneration | Cytoskeleton | MPTP | Axonal transport | Movement disorders | Dopamine receptors
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 03/2009, Volume 29, Issue 5, pp. 954 - 963
Rosiglitazone is a commonly prescribed insulin-sensitizing drug with a selective agonistic activity on the peroxisome proliferator-activated receptor-gamma... 
Inflammation | Tyrosine hydroxylase | Dopamine | Glia | MPP | Motor behaviour | MICROGLIAL ACTIVATION | MPTP MODEL | ALZHEIMERS-DISEASE | glia | SUBSTANTIA-NIGRA | NEUROSCIENCES | tyrosine hydroxylase | inflammation | ALPHA-SYNUCLEIN AGGREGATION | ACTIVATED-RECEPTOR-GAMMA | REACTIVE ASTROCYTES | motor behaviour | NEUROBLASTOMA SH-SY5Y CELLS | NIGROSTRIATAL SYSTEM | dopamine | NIGRA PARS COMPACTA | Tyrosine 3-Monooxygenase - metabolism | Chromatography, High Pressure Liquid - methods | Dynorphins - genetics | Catalepsy | Motor Activity - drug effects | Male | Glial Fibrillary Acidic Protein - metabolism | PPAR gamma - metabolism | RNA, Messenger - metabolism | Smell - drug effects | Globus Pallidus - metabolism | Thiazolidinediones - therapeutic use | Drug Interactions | MPTP Poisoning - pathology | Dynorphins - metabolism | Tandem Mass Spectrometry - methods | Dopamine - metabolism | Disease Models, Animal | Locomotion - drug effects | 3,4-Dihydroxyphenylacetic Acid - metabolism | Mice, Inbred C57BL | Psychomotor Performance - drug effects | MPTP Poisoning - prevention & control | Gene Expression Regulation - drug effects | Globus Pallidus - drug effects | Animals | MPTP Poisoning - metabolism | PPAR gamma - agonists | Mice | CD11b Antigen - metabolism | MPTP Poisoning - chemically induced | Chronic Disease | Tyrosine | Parkinson's disease | RNA | Intermediate filament proteins | Probenecid | Index Medicus
Journal Article
Neurochemistry International, ISSN 0197-0186, 10/2016, Volume 99, pp. 221 - 232
Parkinson’s disease (PD), the most common progressive neurodegenerative movement disorder, results from loss of dopaminergic neurons of substantia nigra pars... 
Dopaminergic neuroprotection | Intracellular calcium levels | SH-SY5Y cell line | Motor functions in mice | Mitochondrial membrane potential | INDUCED CELL-DEATH | MONOAMINE-OXIDASE | MOUSE STRIATUM | SUBSTANTIA-NIGRA NEURONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MPTP-TREATED MICE | COMPLEX-I ACTIVITY | MODEL | NEUROSCIENCES | DOPAMINE NEURONS | STRIATAL LEVEL | DISEASE | Neuroprotective Agents - therapeutic use | Reactive Oxygen Species - metabolism | Humans | Calcium Channel Blockers - therapeutic use | Male | Nimodipine - pharmacology | Neuroprotective Agents - pharmacology | Parkinson Disease, Secondary - metabolism | Parkinson Disease, Secondary - prevention & control | Parkinson Disease, Secondary - chemically induced | Cell Survival - physiology | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Cell Survival - drug effects | Calcium Channels, L-Type - physiology | Mitochondria - metabolism | Mitochondria - drug effects | Calcium Channel Blockers - pharmacology | MPTP Poisoning - prevention & control | Nimodipine - therapeutic use | Reactive Oxygen Species - antagonists & inhibitors | Animals | MPTP Poisoning - metabolism | Cell Line, Tumor | Mice | Mice, Inbred BALB C | MPTP Poisoning - chemically induced | Tyrosine | Medical colleges | Neurosciences | Parkinson's disease | Nimodipine | Neurons | Analysis | Fluorescence | Epidemiology | Resveratrol | Hydroxylases
Journal Article
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 02/2014, Volume 67, pp. 10 - 18
Endoplasmic reticulum (ER) stress has been implicated in Parkinson disease. We previously reported that thioredoxin 1 (Trx-1) suppressed the ER stress caused... 
MPP+/MPTP | Parkinson disease | ER stress | Free radicals | Thioredoxin-1 | MPTP | MPP | APOPTOSIS | ACTIVATION | UNFOLDED-PROTEIN RESPONSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEATH | NEUROTOXICITY | P38 MAP KINASE | IRE1 | ENDOCRINOLOGY & METABOLISM | PC12 CELLS | TRANSCRIPTION FACTOR | Transcription Factor CHOP - genetics | Endoribonucleases - genetics | TNF Receptor-Associated Factor 2 - genetics | MPTP Poisoning - physiopathology | Endoplasmic Reticulum Stress - genetics | PC12 Cells | Heat-Shock Proteins - genetics | MPTP Poisoning - genetics | MAP Kinase Kinase 4 - metabolism | Thioredoxins - genetics | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - analogs & derivatives | Thioredoxins - metabolism | Protein-Serine-Threonine Kinases - metabolism | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Endoplasmic Reticulum Stress - drug effects | Endoribonucleases - metabolism | Signal Transduction | Caspase 12 - genetics | 1-Methyl-4-phenylpyridinium - pharmacology | Heat-Shock Proteins - metabolism | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Rats | TNF Receptor-Associated Factor 2 - metabolism | Mice, Transgenic | Caspase 12 - metabolism | Animals | MPTP Poisoning - metabolism | MAP Kinase Kinase 4 - genetics | Mice | MPTP Poisoning - chemically induced | Transcription Factor CHOP - metabolism | Glucose metabolism | Enzymes | Inositol | Parkinson's disease | Genetic engineering | Stress (Physiology) | Glucose | Thioredoxin | Dextrose
Journal Article