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Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 2007, Volume 320, Issue 1, pp. 1 - 13
Journal Article
Philosophical Transactions: Biological Sciences, ISSN 0962-8436, 12/2012, Volume 367, Issue 1607, pp. 3353 - 3363
Human tissues express cannabinoid CB₁ and CB₂ receptors that can be activated by endogenously released 'endocannabinoids' or exogenously administered compounds... 
Receptors | Cannabinoids | Animal models | Pain | Medical treatment | Agonists | Morphine | Drug design | Endocannabinoids | Cannabinoid receptors | And CB2 | Blood-brain barrier | Cannabinoid receptor agonists | Δ9-tetrahydrocannabinol | Cannabinoid CB1 | Therapeutic applications and strategies | blood-brain barrier | Delta-tetrahydrocannabinol | cannabinoid CB1 and CB2 receptors | CB2 RECEPTOR | LIVER-DISEASES | AMYOTROPHIC-LATERAL-SCLEROSIS | ANXIETY-LIKE BEHAVIOR | MU-OPIOID RECEPTOR | RAT FORMALIN TEST | therapeutic applications and strategies | cannabinoid receptor agonists | NEUROPATHIC PAIN | MOUSE MODEL | BIOLOGY | PENTYLENETETRAZOLE-INDUCED SEIZURE | CENTRAL-NERVOUS-SYSTEM | Endocannabinoids - metabolism | Analgesics - pharmacology | Receptor, Cannabinoid, CB2 - agonists | Cardiovascular Diseases - drug therapy | Humans | Cannabinoid Receptor Agonists - pharmacology | Neurodegenerative Diseases - drug therapy | Pain - metabolism | Receptor, Cannabinoid, CB1 - agonists | Pain - drug therapy | Cannabinoid Receptor Agonists - therapeutic use | Dronabinol - pharmacology | Analgesics - therapeutic use | Cardiovascular Diseases - metabolism | Risk Assessment | Analgesics - administration & dosage | Clinical Trials as Topic | Neurodegenerative Diseases - metabolism | Blood-Brain Barrier - drug effects | Cannabinoid Receptor Agonists - administration & dosage | Drug Discovery | Blood-Brain Barrier - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Dronabinol - therapeutic use | Review
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2013, Volume 288, Issue 32, pp. 22942 - 22960
TGR5 is a G protein-coupled receptor that mediates bile acid (BA) effects on energy balance, inflammation, digestion, and sensation. The mechanisms and... 
GROWTH-FACTOR RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | GPBAR1 TGR5 | DEPENDENT ENDOCYTOSIS | PROTEIN-COUPLED RECEPTORS | RESONANCE ENERGY-TRANSFER | KINASE ACTIVATION | MU-OPIOID RECEPTOR | BETA-ADRENERGIC RECEPTOR | ACTIVATED RECEPTOR-2 | EGF RECEPTOR | Cholagogues and Choleretics - pharmacology | Phenylalanine - analogs & derivatives | Receptors, G-Protein-Coupled - metabolism | Membrane Microdomains - metabolism | Phenylalanine - pharmacology | Humans | ErbB Receptors - genetics | Oleanolic Acid - pharmacology | Arrestins - genetics | Protein Transport - physiology | Protein Transport - drug effects | G-Protein-Coupled Receptor Kinase 2 - genetics | G-Protein-Coupled Receptor Kinase 2 - metabolism | Arrestins - metabolism | beta-Cyclodextrins - pharmacology | Endosomes - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Antineoplastic Agents - pharmacology | Cyclic AMP - genetics | Cyclic AMP - metabolism | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Endosomes - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Endocytosis - drug effects | Enzyme Inhibitors - pharmacology | Thiophenes - pharmacology | Tyrphostins - pharmacology | Endocytosis - physiology | Arrestins - antagonists & inhibitors | G-Protein-Coupled Receptor Kinase 5 - metabolism | Deoxycholic Acid - pharmacology | Mitogen-Activated Protein Kinase 3 - metabolism | beta-Arrestin 2 | beta-Arrestin 1 | beta-Arrestins | Receptors, G-Protein-Coupled - genetics | Quinazolines - pharmacology | G-Protein-Coupled Receptor Kinase 5 - genetics | Membrane Microdomains - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Lipid Raft | Bile Acid | Endocytosis | G Protein-coupled Receptors (GPCR) | Arrestin | Signal Transduction
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2011, Volume 108, Issue 22, pp. 9268 - 9273
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 01/2012, Volume 37, Issue 2, pp. 338 - 349
The capacity of opioids to alleviate inflammatory pain is negatively regulated by the glutamate-binding N-methyl-D-aspartate receptor (NMDAR). Increased... 
mu-opioid receptor | N-methyl-D-aspartate receptor | pain | receptor association | periaqueductal gray | analgesia | PERIAQUEDUCTAL GRAY NEURONS | G-ALPHA-SUBUNITS | PROTEIN-KINASE-C | PSYCHIATRY | D-ASPARTATE RECEPTORS | SYNAPTIC PLASTICITY | NEUROSCIENCES | NEUROPATHIC PAIN | GLUTAMATE RECEPTORS | PHARMACOLOGY & PHARMACY | DEEP BRAIN-STIMULATION | SELECTIVE-MU | ANTINOCICEPTIVE TOLERANCE | Phosphorylation - physiology | Injections, Intraventricular | Periaqueductal Gray - drug effects | Receptors, N-Methyl-D-Aspartate - metabolism | Drug Tolerance - physiology | Male | N-Methylaspartate - pharmacology | G-Protein-Coupled Receptor Kinase 2 - antagonists & inhibitors | Receptor Aggregation - drug effects | Neurons - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Morphine - antagonists & inhibitors | CHO Cells | Cricetinae | Protein Kinase C - physiology | Morphine - pharmacology | Pain - prevention & control | G-Protein-Coupled Receptor Kinase 2 - physiology | Receptors, Opioid, mu - metabolism | Protein Kinase C - antagonists & inhibitors | Mice, Inbred ICR | Periaqueductal Gray - metabolism | Cyclic AMP-Dependent Protein Kinases - physiology | Receptor Aggregation - physiology | Animals | Pain - physiopathology | Mice | Morphine - administration & dosage | Life Sciences | Pharmacology | Pharmaceutical sciences | signal transduction | analgesics | mouse | N-methyl--aspartate receptor | NMDA | Original | glutamate | opioids
Journal Article
Trends in Neurosciences, ISSN 0166-2236, 2012, Volume 36, Issue 3, pp. 195 - 206
The roles of opioid receptors in pain and addiction have been extensively studied, but their function in mood disorders has received less attention.... 
Neurology | delta opioid receptor | depression | kappa opioid receptor | mood | mu opioid receptor | Depression | Mood | Delta opioid receptor | Mu opioid receptor | Kappa opioid receptor | SPRAGUE-DAWLEY RATS | MESSENGER-RNA EXPRESSION | ANTIDEPRESSANT-LIKE ACTIVITIES | CONDITIONED PLACE PREFERENCE | ADULT HIPPOCAMPAL NEUROGENESIS | MORPHINE-INDUCED INCREASES | FORCED SWIM TEST | NEUROSCIENCES | LEARNED HELPLESSNESS MODEL | CENTRAL-NERVOUS-SYSTEM | DORSAL RAPHE NUCLEUS | Mood Disorders - drug therapy | Receptors, Opioid - agonists | Prenatal Exposure Delayed Effects | Opioid Peptides - physiology | Humans | Analgesics, Opioid - pharmacology | Neuronal Plasticity - drug effects | Male | Receptors, Opioid - physiology | Neuronal Plasticity - physiology | Brain-Derived Neurotrophic Factor - physiology | Social Behavior | Receptors, Opioid - deficiency | Female | Antidepressive Agents - pharmacology | Neurogenesis - drug effects | Neurotransmitter Agents - physiology | Affect - drug effects | Nerve Tissue Proteins - physiology | Comorbidity | Affect - physiology | Maternal Behavior - physiology | Maternal Behavior - drug effects | Substance-Related Disorders - physiopathology | Mice, Knockout | Antidepressive Agents - therapeutic use | Pregnancy | Animals | Neurogenesis - physiology | Narcotic Antagonists - pharmacology | Brain Chemistry | Mice | Models, Neurological | Reward | Substance-Related Disorders - drug therapy | Mood Disorders - physiopathology | Mood Disorders - metabolism | Antidepressants | Development and progression | Depression, Mental
Journal Article