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Journal Article
Nature Reviews Neurology, ISSN 1759-4758, 08/2010, Volume 6, Issue 8, pp. 438 - 444
Journal Article
Glia, ISSN 0894-1491, 10/2015, Volume 63, Issue 10, pp. 1714 - 1728
Remyelination is a potent regenerative process in demyelinating diseases, such as multiple sclerosis, the effective therapeutic promotion of which will fill an... 
myelin | oligodendrocyte | remyelination | Oligodendrocyte | Myelin | Remyelination | CONDITIONAL INACTIVATION | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | IGF-I | WHITE-MATTER | MESSENGER-RNA EXPRESSION | SPINAL-CORD | GLIAL-CELLS | NEUROSCIENCES | MULTIPLE-SCLEROSIS | CENTRAL-NERVOUS-SYSTEM | CUPRIZONE-INDUCED DEMYELINATION | Oligodendroglia - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Recovery of Function - drug effects | Demyelinating Diseases - genetics | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Cell Lineage - drug effects | Cell Differentiation - genetics | Oligodendroglia - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase - genetics | Spinal Cord - pathology | Recovery of Function - physiology | Demyelinating Diseases - chemically induced | Demyelinating Diseases - pathology | Cell Lineage - genetics | Disease Models, Animal | Fibroblast Growth Factors - physiology | Animals, Newborn | Cuprizone - toxicity | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Signal Transduction - genetics | 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase - metabolism | Animals | Chelating Agents - toxicity | Signal Transduction - drug effects | Cell Differentiation - drug effects | Signal Transduction - physiology | Mice | Lysophosphatidylcholines - toxicity | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Fibroblast growth factors | Fibroblasts | Growth factors | Rodents
Journal Article
Immunology, ISSN 0019-2805, 02/2016, Volume 147, Issue 2, pp. 212 - 220
Multiple sclerosis ( MS ) is thought to be an autoimmune disorder. It is believed that immunological events in the early stages have great impact on the... 
interleukin ‐22 | myelin basic protein | T cells | multiple sclerosis | interleukin‐17 | Interleukin -22 | CD4 | Multiple sclerosis | Interleukin-17 | Myelin basic protein | TRIALS | IMMUNOLOGY | interleukin-22 | BETA | interleukin-17 | PHASE | THERAPY | GM-CSF | DISABILITY | AFRICAN-AMERICANS | CD4(+) T cells | EXPRESSION | TH17 | Humans | Interleukin-17 - immunology | African Americans | Male | Multiple Sclerosis, Relapsing-Remitting - diagnosis | Interleukins - metabolism | Brain - metabolism | CD4-Positive T-Lymphocytes - immunology | Young Adult | Time Factors | Interleukins - immunology | Multiple Sclerosis, Relapsing-Remitting - ethnology | Adult | Female | Drug Resistance | Multiple Sclerosis, Relapsing-Remitting - metabolism | Disability Evaluation | Multiple Sclerosis, Relapsing-Remitting - immunology | Enzyme-Linked Immunosorbent Assay | Myelin Basic Protein - metabolism | Multiple Sclerosis, Relapsing-Remitting - drug therapy | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Adrenal Cortex Hormones - pharmacology | Myelin Basic Protein - immunology | Brain - drug effects | Interleukin-17 - metabolism | Magnetic Resonance Imaging | Adolescent | Brain - pathology | Hydrocortisone - pharmacology | Brain - immunology | CD4-Positive T-Lymphocytes - drug effects | Brain | Interleukins | Macrophage colony stimulating factor | Hydrocortisone | Myelin proteins | CD4+ T cells | Original
Journal Article
Neurology, ISSN 0028-3878, 02/2011, Volume 76, Issue 6, pp. 534 - 539
Journal Article
Human Brain Mapping, ISSN 1065-9471, 12/2015, Volume 36, Issue 12, pp. 5051 - 5063
Journal Article
Brain, ISSN 0006-8950, 2013, Volume 136, Issue 6, pp. 1799 - 1815
Cortical lesions constitute an important part of multiple sclerosis pathology. Although inflammation appears to play a role in their formation, the mechanisms...