Catalogue
Articles
RSS feedX
Search Filters
Format
Subjects
Library Location
Language
Publication DateDiabetes, ISSN 0012-1797, 04/2002, Volume 51, Issue 4, pp. 1052 - 1059
Insulin Resistance, Defective Insulin Receptor Substrate 2—Associated Phosphatidylinositol-3′ Kinase Activation, and Impaired Atypical Protein Kinase C (ζ/λ)...
3T3-L1 ADIPOCYTES | DEPENDENT DIABETES-MELLITUS | RAT ADIPOSE-CELLS | TRANSPORT | STIMULATION | SKELETAL-MUSCLE CELLS | GLUT4 TRANSLOCATION | ENDOCRINOLOGY & METABOLISM | 3-KINASE ACTIVITY | GLYCOGEN-SYNTHASE ACTIVITY | NIDDM SUBJECTS | Blood Pressure | Glucose Intolerance - metabolism | Phosphorylation | Humans | Middle Aged | Muscle, Skeletal - metabolism | Reference Values | Phosphatidylinositol 3-Kinases - metabolism | Phosphoproteins - metabolism | Phosphotyrosine - metabolism | Protein Kinase C - metabolism | Muscle, Skeletal - drug effects | Insulin Receptor Substrate Proteins | Adult | Deoxyglucose - pharmacokinetics | Biological Transport - drug effects | Glucose Intolerance - physiopathology | Adipose Tissue - anatomy & histology | Body Mass Index | Insulin - pharmacology | Glucose Tolerance Test | Cells, Cultured | Intracellular Signaling Peptides and Proteins | Blood Glucose | Obesity - physiopathology | Obesity - metabolism | Receptor, Insulin - metabolism | Enzyme Activation | Protein research | Receptors | Analysis | Physiological aspects | Insulin resistance | Glucose | Striated muscle | Insulin
3T3-L1 ADIPOCYTES | DEPENDENT DIABETES-MELLITUS | RAT ADIPOSE-CELLS | TRANSPORT | STIMULATION | SKELETAL-MUSCLE CELLS | GLUT4 TRANSLOCATION | ENDOCRINOLOGY & METABOLISM | 3-KINASE ACTIVITY | GLYCOGEN-SYNTHASE ACTIVITY | NIDDM SUBJECTS | Blood Pressure | Glucose Intolerance - metabolism | Phosphorylation | Humans | Middle Aged | Muscle, Skeletal - metabolism | Reference Values | Phosphatidylinositol 3-Kinases - metabolism | Phosphoproteins - metabolism | Phosphotyrosine - metabolism | Protein Kinase C - metabolism | Muscle, Skeletal - drug effects | Insulin Receptor Substrate Proteins | Adult | Deoxyglucose - pharmacokinetics | Biological Transport - drug effects | Glucose Intolerance - physiopathology | Adipose Tissue - anatomy & histology | Body Mass Index | Insulin - pharmacology | Glucose Tolerance Test | Cells, Cultured | Intracellular Signaling Peptides and Proteins | Blood Glucose | Obesity - physiopathology | Obesity - metabolism | Receptor, Insulin - metabolism | Enzyme Activation | Protein research | Receptors | Analysis | Physiological aspects | Insulin resistance | Glucose | Striated muscle | Insulin
Journal Article
Details 
Digital rights
Loading RightsEndocrinology, ISSN 0013-7227, 05/2015, Volume 156, Issue 5, pp. 1770 - 1781
A shift in quadriceps muscle metabolic profile toward decreased oxidative metabolism and increased glycolysis is a consistent finding in chronic obstructive...
OBSTRUCTIVE PULMONARY-DISEASE | ENERGY REGULATION | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | PATHWAY | GLUCOSE | HEART-FAILURE | ENDOCRINOLOGY & METABOLISM | EXERCISE INTOLERANCE | VASTUS LATERALIS | EXPRESSION | COPD | Tumor Necrosis Factor-alpha - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Vascular Endothelial Growth Factor A - genetics | Glycolysis - drug effects | Case-Control Studies | Glycolysis - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Pulmonary Disease, Chronic Obstructive - metabolism | Severity of Illness Index | Cell Line | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Lactic Acid - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Animals | Glucose - metabolism | Mice | Muscle, Skeletal | Quadriceps Muscle - metabolism | NF-kappa B - drug effects
OBSTRUCTIVE PULMONARY-DISEASE | ENERGY REGULATION | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | PATHWAY | GLUCOSE | HEART-FAILURE | ENDOCRINOLOGY & METABOLISM | EXERCISE INTOLERANCE | VASTUS LATERALIS | EXPRESSION | COPD | Tumor Necrosis Factor-alpha - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Vascular Endothelial Growth Factor A - genetics | Glycolysis - drug effects | Case-Control Studies | Glycolysis - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Pulmonary Disease, Chronic Obstructive - metabolism | Severity of Illness Index | Cell Line | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Lactic Acid - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Animals | Glucose - metabolism | Mice | Muscle, Skeletal | Quadriceps Muscle - metabolism | NF-kappa B - drug effects
Journal Article
Details
Digital rights
Loading RightsCell Stem Cell, ISSN 1934-5909, 10/2012, Volume 11, Issue 4, pp. 541 - 553
In response to muscle injury, satellite cells activate the p38α/β MAPK pathway to exit quiescence, then proliferate, repair skeletal muscle, and self-renew,...
PROGENITOR CELLS | CYTOSINE-ARABINOSIDE | STEM-CELLS | INHIBITION | MECHANISM | POLARITY | EXPANSION | SYNDECAN-3 | DIFFERENTIATION | TEMPLATE DNA STRANDS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Biomarkers - metabolism | Cell Line | Protein Kinase C - genetics | Cell Adhesion Molecules - genetics | Cell Proliferation | Cell Survival | Isoenzymes - genetics | MyoD Protein - genetics | Asymmetric Cell Division | MyoD Protein - metabolism | Cell Adhesion Molecules - metabolism | MAP Kinase Signaling System | Multiprotein Complexes - metabolism | Muscle Development | Animals | Isoenzymes - metabolism | Protein Kinase C - metabolism | Satellite Cells, Skeletal Muscle - physiology | Mice | p38 Mitogen-Activated Protein Kinases - metabolism | Enzyme Activation - genetics | Myogenin - metabolism
PROGENITOR CELLS | CYTOSINE-ARABINOSIDE | STEM-CELLS | INHIBITION | MECHANISM | POLARITY | EXPANSION | SYNDECAN-3 | DIFFERENTIATION | TEMPLATE DNA STRANDS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Biomarkers - metabolism | Cell Line | Protein Kinase C - genetics | Cell Adhesion Molecules - genetics | Cell Proliferation | Cell Survival | Isoenzymes - genetics | MyoD Protein - genetics | Asymmetric Cell Division | MyoD Protein - metabolism | Cell Adhesion Molecules - metabolism | MAP Kinase Signaling System | Multiprotein Complexes - metabolism | Muscle Development | Animals | Isoenzymes - metabolism | Protein Kinase C - metabolism | Satellite Cells, Skeletal Muscle - physiology | Mice | p38 Mitogen-Activated Protein Kinases - metabolism | Enzyme Activation - genetics | Myogenin - metabolism
Journal Article
Details
Digital rights
Loading RightsChemPhysChem, ISSN 1439-4235, 03/2014, Volume 15, Issue 4, pp. 687 - 695
The noncovalent equilibrium activation of a fluorogenic malachite green dye and its cognate fluorogen‐activating protein (FAP) can produce a sparse labeling...
super resolution | imaging agents | localization microscopy | fluorescence | proteins | FLUOROPHORES | SUPERRESOLUTION MICROSCOPY | RESOLUTION | PHYSICS, ATOMIC, MOLECULAR & CHEMICAL | SURFACE-PROTEINS | CHEMISTRY, PHYSICAL | LIVE CELLS | STRUCTURED-ILLUMINATION MICROSCOPY | PROBES | FLUORESCENCE MICROSCOPY | STIMULATED-EMISSION | DIFFRACTION-LIMIT | Fluorescent Dyes - chemistry | Antibodies - genetics | Rosaniline Dyes - chemistry | Microscopy, Fluorescence - methods | Antibodies - chemistry | Physiological aspects | Fluorescence | Muscle proteins | Microscope and microscopy | Cells | Detectors | Localization | Superresolution | Fluorescent Protein | Imaging | Live Cell
super resolution | imaging agents | localization microscopy | fluorescence | proteins | FLUOROPHORES | SUPERRESOLUTION MICROSCOPY | RESOLUTION | PHYSICS, ATOMIC, MOLECULAR & CHEMICAL | SURFACE-PROTEINS | CHEMISTRY, PHYSICAL | LIVE CELLS | STRUCTURED-ILLUMINATION MICROSCOPY | PROBES | FLUORESCENCE MICROSCOPY | STIMULATED-EMISSION | DIFFRACTION-LIMIT | Fluorescent Dyes - chemistry | Antibodies - genetics | Rosaniline Dyes - chemistry | Microscopy, Fluorescence - methods | Antibodies - chemistry | Physiological aspects | Fluorescence | Muscle proteins | Microscope and microscopy | Cells | Detectors | Localization | Superresolution | Fluorescent Protein | Imaging | Live Cell
Journal Article
Details
Digital rights
Loading RightsJournal of Biological Chemistry, ISSN 0021-9258, 03/2001, Volume 276, Issue 11, pp. 7957 - 7962
In cultured vascular smooth muscle cells (VSMC), the vasculotrophic factor, angiotensin II (AngII) activates three major MAPKs via the G(q)-coupled AT(1)...
SIGNAL-TRANSDUCTION | PATHWAYS | GROWTH-FACTOR RECEPTOR | DISINTEGRIN | TYROSINE KINASE | TRANSACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL KINASE | CARDIOVASCULAR-DISEASE | PROTEIN-COUPLED RECEPTORS | NECROSIS-FACTOR-ALPHA | Angiotensin II - pharmacology | Transcriptional Activation | Rats | Muscle, Smooth, Vascular - cytology | Rats, Sprague-Dawley | Animals | Mitogen-Activated Protein Kinases - physiology | JNK Mitogen-Activated Protein Kinases | Metalloendopeptidases - physiology | Enzyme Activation | Receptor, Epidermal Growth Factor - physiology | Muscle, Smooth, Vascular - enzymology | p38 Mitogen-Activated Protein Kinases | Mitogen-Activated Protein Kinases - metabolism | Muscle, Smooth, Vascular - drug effects
SIGNAL-TRANSDUCTION | PATHWAYS | GROWTH-FACTOR RECEPTOR | DISINTEGRIN | TYROSINE KINASE | TRANSACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL KINASE | CARDIOVASCULAR-DISEASE | PROTEIN-COUPLED RECEPTORS | NECROSIS-FACTOR-ALPHA | Angiotensin II - pharmacology | Transcriptional Activation | Rats | Muscle, Smooth, Vascular - cytology | Rats, Sprague-Dawley | Animals | Mitogen-Activated Protein Kinases - physiology | JNK Mitogen-Activated Protein Kinases | Metalloendopeptidases - physiology | Enzyme Activation | Receptor, Epidermal Growth Factor - physiology | Muscle, Smooth, Vascular - enzymology | p38 Mitogen-Activated Protein Kinases | Mitogen-Activated Protein Kinases - metabolism | Muscle, Smooth, Vascular - drug effects
Journal Article
Details
Digital rights
Loading RightsJournal of the American College of Cardiology, ISSN 0735-1097, 01/2007, Volume 49, Issue 2, pp. 185 - 194
Objectives: Our study investigated: 1) the contribution of nuclear factor kappa-B (NF-κB) signaling pathway to the enhanced inflammatory response observed in...
AORTIC ENDOTHELIAL-CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | INTERFERON-GAMMA | STATIN THERAPY | ATHEROSCLEROSIS | ACUTE CORONARY SYNDROMES | BLOOD MONOCYTES | ENHANCED INFLAMMATORY RESPONSE | VASCULAR SMOOTH-MUSCLE | TISSUE FACTOR | ARTERY-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Humans | Middle Aged | Lipopolysaccharides - metabolism | Male | NF-kappa B - metabolism | Reference Values | Monocytes - immunology | C-Reactive Protein - metabolism | Angina, Unstable - immunology | Sensitivity and Specificity | Inflammation Mediators - metabolism | C-Reactive Protein - pharmacology | Adult | Female | Azides - pharmacology | Interleukin-6 - metabolism | Angina, Unstable - metabolism | Enzyme-Linked Immunosorbent Assay | Monocytes - drug effects | Microscopy, Confocal | Lipopolysaccharides - pharmacology | Signal Transduction - physiology | Aged | In Vitro Techniques | Proteins | Enzymes | Heart attacks | E coli | Microscopy | Angina pectoris | Acute coronary syndromes | Drug dosages | Cholesterol | Age
AORTIC ENDOTHELIAL-CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | INTERFERON-GAMMA | STATIN THERAPY | ATHEROSCLEROSIS | ACUTE CORONARY SYNDROMES | BLOOD MONOCYTES | ENHANCED INFLAMMATORY RESPONSE | VASCULAR SMOOTH-MUSCLE | TISSUE FACTOR | ARTERY-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Humans | Middle Aged | Lipopolysaccharides - metabolism | Male | NF-kappa B - metabolism | Reference Values | Monocytes - immunology | C-Reactive Protein - metabolism | Angina, Unstable - immunology | Sensitivity and Specificity | Inflammation Mediators - metabolism | C-Reactive Protein - pharmacology | Adult | Female | Azides - pharmacology | Interleukin-6 - metabolism | Angina, Unstable - metabolism | Enzyme-Linked Immunosorbent Assay | Monocytes - drug effects | Microscopy, Confocal | Lipopolysaccharides - pharmacology | Signal Transduction - physiology | Aged | In Vitro Techniques | Proteins | Enzymes | Heart attacks | E coli | Microscopy | Angina pectoris | Acute coronary syndromes | Drug dosages | Cholesterol | Age
Journal Article
Details
Digital rights
Loading RightsMuscle & Nerve, ISSN 0148-639X, 08/2015, Volume 52, Issue 2, pp. 252 - 259
ABSTRACT Introduction: Our knowledge of the neurophysiology of post‐activation potentiation (PAP) is limited. The purpose of this study was to examine the...
reflex potentiation | post‐activation potentiation | H‐reflex | post‐activation depression | plantarflexors | H-reflex | Post-activation depression | Reflex potentiation | Post-activation potentiation | Plantarflexors | H-Reflex - physiology | Young Adult | Long-Term Synaptic Depression - physiology | Humans | Muscle Contraction - physiology | Adolescent | Long-Term Potentiation - physiology | Adult | Female | Male | Muscle, Skeletal - physiology | Electromyography - methods | Rodents | Reflexes
reflex potentiation | post‐activation potentiation | H‐reflex | post‐activation depression | plantarflexors | H-reflex | Post-activation depression | Reflex potentiation | Post-activation potentiation | Plantarflexors | H-Reflex - physiology | Young Adult | Long-Term Synaptic Depression - physiology | Humans | Muscle Contraction - physiology | Adolescent | Long-Term Potentiation - physiology | Adult | Female | Male | Muscle, Skeletal - physiology | Electromyography - methods | Rodents | Reflexes
Journal Article
Details
Digital rights
Loading RightsJournal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2009, Volume 48, Issue 5, pp. 851 - 858
Abstract The Frank–Starling law of the heart describes the interrelationship between end-diastolic volume and cardiac ejection volume, a regulatory system that...
Cardiovascular | Sarcomere length | Regulation | Frank–Starling Law of The Heart | Length–Tension Relationship | Length-Tension Relationship | Frank-Starling Law of The Heart | CARDIAC & CARDIOVASCULAR SYSTEMS | THIN FILAMENT ACTIVATION | RABBIT SKELETAL-MUSCLE | X-RAY-DIFFRACTION | FORCE REDEVELOPMENT KINETICS | CELL BIOLOGY | FRANK-STARLING RELATIONSHIP | GIANT PROTEIN TITIN | MYOFIBRILLAR ADENOSINE-TRIPHOSPHATASE | REGULATORY UNIT INTERACTIONS | MAGNESIUM BINDING-SITES | SKINNED CARDIAC-MUSCLE | Actin Cytoskeleton - metabolism | Animals | Models, Biological | Sarcomeres - metabolism | Heart - physiology | Humans | Myocardial Contraction - physiology | Troponin I - metabolism | Proteins
Cardiovascular | Sarcomere length | Regulation | Frank–Starling Law of The Heart | Length–Tension Relationship | Length-Tension Relationship | Frank-Starling Law of The Heart | CARDIAC & CARDIOVASCULAR SYSTEMS | THIN FILAMENT ACTIVATION | RABBIT SKELETAL-MUSCLE | X-RAY-DIFFRACTION | FORCE REDEVELOPMENT KINETICS | CELL BIOLOGY | FRANK-STARLING RELATIONSHIP | GIANT PROTEIN TITIN | MYOFIBRILLAR ADENOSINE-TRIPHOSPHATASE | REGULATORY UNIT INTERACTIONS | MAGNESIUM BINDING-SITES | SKINNED CARDIAC-MUSCLE | Actin Cytoskeleton - metabolism | Animals | Models, Biological | Sarcomeres - metabolism | Heart - physiology | Humans | Myocardial Contraction - physiology | Troponin I - metabolism | Proteins
Journal Article
Details
Digital rights
Loading RightsMolecular Nutrition & Food Research, ISSN 1613-4125, 10/2014, Volume 58, Issue 10, pp. 1931 - 1940
Scope Dietary polyphenols are suggested to play a role in the prevention of Alzheimer's disease, of which accumulation of aggregated beta amyloid (Aβ) is a key...
Amyloid β peptide | Caenorhabditis elegans | Alzheimer's disease | Autophagy | Proteasome | Unfolded protein response | ALZHEIMERS-DISEASE | MEDITERRANEAN DIET | FOOD SCIENCE & TECHNOLOGY | NEURODEGENERATION | MODEL | IMPAIRMENT | GENE-EXPRESSION | Amyloid beta peptide | PRECURSOR PROTEIN | STRESS | AGGREGATION | Humans | Caenorhabditis elegans Proteins - metabolism | Lysosomes - enzymology | Lysosomes - metabolism | RNA Interference | Alzheimer Disease - prevention & control | Amyloid beta-Peptides - genetics | Protein Aggregation, Pathological - prevention & control | Proteolysis | Amyloid beta-Peptides - metabolism | Quercetin - administration & dosage | Muscles - metabolism | Peptide Fragments - genetics | Disease Models, Animal | Nerve Tissue Proteins - antagonists & inhibitors | Paralysis - chemically induced | Peptide Fragments - metabolism | Protein Aggregation, Pathological - enzymology | Animals, Genetically Modified | Quercetin - therapeutic use | Unfolded Protein Response | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Antioxidants - therapeutic use | Animals | Caenorhabditis elegans Proteins - antagonists & inhibitors | Muscles - enzymology | Peptide Fragments - antagonists & inhibitors | Paralysis - prevention & control | Alzheimer Disease - metabolism | Antioxidants - administration & dosage | Proteasome Endopeptidase Complex - metabolism | Caenorhabditis elegans Proteins - genetics | Dietary Supplements | Protein Aggregation, Pathological - metabolism | Proteins | Analysis | Polyphenols | Genetic engineering | Mitochondrial DNA | Paralysis
Amyloid β peptide | Caenorhabditis elegans | Alzheimer's disease | Autophagy | Proteasome | Unfolded protein response | ALZHEIMERS-DISEASE | MEDITERRANEAN DIET | FOOD SCIENCE & TECHNOLOGY | NEURODEGENERATION | MODEL | IMPAIRMENT | GENE-EXPRESSION | Amyloid beta peptide | PRECURSOR PROTEIN | STRESS | AGGREGATION | Humans | Caenorhabditis elegans Proteins - metabolism | Lysosomes - enzymology | Lysosomes - metabolism | RNA Interference | Alzheimer Disease - prevention & control | Amyloid beta-Peptides - genetics | Protein Aggregation, Pathological - prevention & control | Proteolysis | Amyloid beta-Peptides - metabolism | Quercetin - administration & dosage | Muscles - metabolism | Peptide Fragments - genetics | Disease Models, Animal | Nerve Tissue Proteins - antagonists & inhibitors | Paralysis - chemically induced | Peptide Fragments - metabolism | Protein Aggregation, Pathological - enzymology | Animals, Genetically Modified | Quercetin - therapeutic use | Unfolded Protein Response | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Antioxidants - therapeutic use | Animals | Caenorhabditis elegans Proteins - antagonists & inhibitors | Muscles - enzymology | Peptide Fragments - antagonists & inhibitors | Paralysis - prevention & control | Alzheimer Disease - metabolism | Antioxidants - administration & dosage | Proteasome Endopeptidase Complex - metabolism | Caenorhabditis elegans Proteins - genetics | Dietary Supplements | Protein Aggregation, Pathological - metabolism | Proteins | Analysis | Polyphenols | Genetic engineering | Mitochondrial DNA | Paralysis
Journal Article
Details
Digital rights
Loading RightsAmerican Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2011, Volume 183, Issue 3, pp. 299 - 309
Rationale: Severe asthma (SA) remains poorly understood. Mast cells (MC) are implicated in asthma pathogenesis, but it remains unknown how their phenotype,...
Carboxypeptidase A | Prostaglandin D2 | Chymase | NASAL-MUCOSA | chymase | CLUSTER-ANALYSIS | MICE LACKING | INTESTINAL EPITHELIUM | RESPIRATORY SYSTEM | prostaglandin D2 | carboxypeptidase A | GRANULE CHYMASE | IN-VIVO | TRICHINELLA-SPIRALIS | AIRWAY SMOOTH-MUSCLE | INTEGRIN ALPHA(V)BETA | BRONCHOALVEOLAR ALLERGEN CHALLENGE | CRITICAL CARE MEDICINE | Acute Disease | Respiratory Mucosa - cytology | Cell Count | Humans | Middle Aged | Biomarkers - analysis | Prostaglandin D2 - analysis | Logistic Models | Male | Mast Cells - physiology | Reverse Transcriptase Polymerase Chain Reaction | Prostaglandin D2 - physiology | Young Adult | Phenotype | Asthma - immunology | Adult | Female | Signal Transduction - physiology | Asthma - etiology | Bronchoalveolar Lavage Fluid - chemistry | A. Asthma and Allergy
Carboxypeptidase A | Prostaglandin D2 | Chymase | NASAL-MUCOSA | chymase | CLUSTER-ANALYSIS | MICE LACKING | INTESTINAL EPITHELIUM | RESPIRATORY SYSTEM | prostaglandin D2 | carboxypeptidase A | GRANULE CHYMASE | IN-VIVO | TRICHINELLA-SPIRALIS | AIRWAY SMOOTH-MUSCLE | INTEGRIN ALPHA(V)BETA | BRONCHOALVEOLAR ALLERGEN CHALLENGE | CRITICAL CARE MEDICINE | Acute Disease | Respiratory Mucosa - cytology | Cell Count | Humans | Middle Aged | Biomarkers - analysis | Prostaglandin D2 - analysis | Logistic Models | Male | Mast Cells - physiology | Reverse Transcriptase Polymerase Chain Reaction | Prostaglandin D2 - physiology | Young Adult | Phenotype | Asthma - immunology | Adult | Female | Signal Transduction - physiology | Asthma - etiology | Bronchoalveolar Lavage Fluid - chemistry | A. Asthma and Allergy
Journal Article
Details
Digital rights
Loading RightsToxicology Letters, ISSN 0378-4274, 05/2015, Volume 235, Issue 1, pp. 1 - 7
Hypertension is one of the most frequent complications of solid organ transplantation, and cyclosporin A (CsA) plays a predominant role in the pathophysiology...
ETB receptor | NF-κB | Cyclosporin A | VSMC | MAPK | receptor | ET | RAT | TRANSCRIPTION | ORGAN-CULTURE | MESENTERIC-ARTERY | NF-kappa B | EPITHELIAL-CELLS | CONTRACTION | TOXICOLOGY | ENDOTHELIN-1 | INHIBITORS | HYPERTENSION | EXPRESSION | Up-Regulation | Phosphorylation | Rats, Wistar | Male | NF-kappa B - metabolism | Endothelin B Receptor Antagonists - pharmacology | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Time Factors | p38 Mitogen-Activated Protein Kinases - metabolism | Active Transport, Cell Nucleus | Organ Culture Techniques | Muscle, Smooth, Vascular - drug effects | NF-kappa B - antagonists & inhibitors | Receptor, Endothelin B - drug effects | Immunosuppressive Agents - toxicity | Vasoconstriction - drug effects | Mesenteric Artery, Superior - drug effects | Mesenteric Artery, Superior - enzymology | Cyclosporine - toxicity | Receptor, Endothelin B - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction - drug effects | Protein Kinase Inhibitors - pharmacology | Receptor, Endothelin B - metabolism | Enzyme Activation | Muscle, Smooth, Vascular - enzymology | Mitogen-Activated Protein Kinase 1 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Index Medicus | Proteins | Hypertension | Receptors | Muscles | Activation | Kinases | Gene expression
ETB receptor | NF-κB | Cyclosporin A | VSMC | MAPK | receptor | ET | RAT | TRANSCRIPTION | ORGAN-CULTURE | MESENTERIC-ARTERY | NF-kappa B | EPITHELIAL-CELLS | CONTRACTION | TOXICOLOGY | ENDOTHELIN-1 | INHIBITORS | HYPERTENSION | EXPRESSION | Up-Regulation | Phosphorylation | Rats, Wistar | Male | NF-kappa B - metabolism | Endothelin B Receptor Antagonists - pharmacology | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Time Factors | p38 Mitogen-Activated Protein Kinases - metabolism | Active Transport, Cell Nucleus | Organ Culture Techniques | Muscle, Smooth, Vascular - drug effects | NF-kappa B - antagonists & inhibitors | Receptor, Endothelin B - drug effects | Immunosuppressive Agents - toxicity | Vasoconstriction - drug effects | Mesenteric Artery, Superior - drug effects | Mesenteric Artery, Superior - enzymology | Cyclosporine - toxicity | Receptor, Endothelin B - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction - drug effects | Protein Kinase Inhibitors - pharmacology | Receptor, Endothelin B - metabolism | Enzyme Activation | Muscle, Smooth, Vascular - enzymology | Mitogen-Activated Protein Kinase 1 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Index Medicus | Proteins | Hypertension | Receptors | Muscles | Activation | Kinases | Gene expression
Journal Article
Details
Digital rights
Loading RightsCirculation, ISSN 0009-7322, 10/2003, Volume 108, Issue 16, pp. 1917 - 1923
C-REACTIVE PROTEIN | SOLUBLE CD40 LIGAND | CARDIAC & CARDIOVASCULAR SYSTEMS | HUMAN ATHEROSCLEROTIC PLAQUES | OX-LDL RECEPTOR-1 | LOW-DENSITY-LIPOPROTEIN | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | ACUTE CORONARY SYNDROMES | NF-KAPPA-B | VASCULAR SMOOTH-MUSCLE | TISSUE FACTOR EXPRESSION | Inflammation - complications | Cardiovascular Diseases - complications | Animals | Endothelium, Vascular - metabolism | Humans | Biomarkers - analysis | Endothelium, Vascular - physiopathology | Endothelium, Vascular - pathology | Inflammation - diagnosis | Disease Progression | Cardiovascular Diseases - diagnosis | Inflammation - physiopathology
Journal Article
Details
Digital rights
Loading Rights