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Toxicology and Applied Pharmacology, ISSN 0041-008X, 02/2013, Volume 266, Issue 3, pp. 439 - 442
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 09/2012, Volume 263, Issue 2, pp. 203 - 209
Journal Article
Journal Article
Journal of the American Chemical Society, ISSN 0002-7863, 10/2008, Volume 130, Issue 40, p. 13214
Journal Article
Journal of Molecular Structure, ISSN 0022-2860, 05/2018, Volume 1160, pp. 57 - 62
After microsynthesis, structures of mustard gas polysulfide analogues were characterized using electron impact (EI) mass spectrometry. General EI fragmentation... 
Polysulfides | Episulfonium ion | Chemical weapons convention | Mass spectrometry | DFT calculations | Sulfur mustard | CHEMISTRY, PHYSICAL | BACILLUS-ANTHRACIS
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e46149
Chemical warfare agent sulfur mustard (HD) inflicts delayed blistering and incapacitating skin injuries. To identify effective countermeasures against... 
CELLS | EFFICACY | INDUCED TOXICITY | MULTIDISCIPLINARY SCIENCES | SKH-1 HAIRLESS MICE | DNA-DAMAGE | CYTOKINE | CANCER | SULFIDE-INDUCED SKIN | MOUSE SKIN | MOLECULAR-MECHANISMS | Inflammation - pathology | Skin - cytology | Apoptosis - drug effects | Skin - metabolism | Humans | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | Cyclooxygenase 2 - genetics | Matrix Metalloproteinase 9 - genetics | Mustard Gas - analogs & derivatives | Female | Silymarin - pharmacology | Deoxyguanosine - analogs & derivatives | Antidotes - pharmacology | Deoxyguanosine - antagonists & inhibitors | Cells, Cultured | Mice, Hairless | Gene Expression Regulation - drug effects | Mustard Gas - toxicity | Animals | Nitric Oxide Synthase Type II - genetics | Signal Transduction - drug effects | Cyclooxygenase 2 - metabolism | Deoxyguanosine - metabolism | Inflammation - prevention & control | Mice | Chemical Warfare Agents - toxicity | DNA Damage | Oxidative Stress - drug effects | Skin - drug effects | Aldehydes - chemistry | Nitric Oxide Synthase Type II - metabolism | Prevention | Oxidative stress | Dermatology | Formulae, receipts, prescriptions | Physiological aspects | Causes of | Dermatologic agents | Chemical warfare | Inflammation | Research | Sulfide | Transcription factors | 4-Hydroxynonenal | Toxicity | DNA damage | Reversing | Kinases | Skin injuries | Cancer therapies | Optimization | Octanoic acid | Toxicology | Protein adducts | Rodents | Transcription activation | Cell cycle | Fibroblasts | Attenuation | Peroxidase | Bioindicators | Sulfur | Injuries | Pharmaceutical sciences | Deoxyribonucleic acid--DNA | NF-κB protein | Effectiveness | Cytokines | Activator protein 1 | Exposure | Hairless | Nitric-oxide synthase | Gelatinase B | Studies | Deoxyguanosine | Cell death | Mustard gas | Nitric oxide | Silibinin | Biomarkers | Blistering | Skin | Cyclooxygenase-2 | Viability | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Journal Article