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Nature, ISSN 0028-0836, 07/2017, Volume 547, Issue 7662, pp. 227 - 231
The regenerative capacity of the adult mammalian heart is limited, because of the reduced ability of cardiomyocytes to progress through mitosis(1). Endogenous... 
PRESSURE-OVERLOAD | GENE | ADULT HEART REGENERATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MOUSE MODEL | MUSCLE | DUCHENNE MUSCULAR-DYSTROPHY | MICE | Protein-Serine-Threonine Kinases - deficiency | Phosphorylation | Cell Proliferation | Glycoproteins - metabolism | Dystroglycans - metabolism | Male | Phosphoproteins - metabolism | Heart Failure - prevention & control | Multiprotein Complexes - metabolism | Dystrophin - deficiency | Mice, Inbred mdx | Cardiomyopathies | Glycoproteins - deficiency | Multiprotein Complexes - deficiency | Dystrophin - metabolism | Protein-Serine-Threonine Kinases - metabolism | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | Organ Size | Heart Failure - genetics | Pressure | Multiprotein Complexes - chemistry | Animals | Dystrophin - genetics | Myocytes, Cardiac - metabolism | Protein Binding | Mice | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Adaptor Proteins, Signal Transducing - metabolism | Heart | Genes | Homeostasis | Genomes | Kinases | Muscular dystrophy | Cell growth | Actin | Duchenne's muscular dystrophy | Cell cycle | Extracellular matrix | Heart diseases | Dystrophin | Deoxyribonucleic acid--DNA | Heart failure | Dystroglycan | Glycoprotein | Cardiomyocytes | Studies | Yes-associated protein | Regeneration | Point mutation | Cytoskeleton | Dystrophy | Mutation
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 07/2013, Volume 62, Issue 2, pp. 89 - 95
Journal Article
Circulation, ISSN 0009-7322, 06/2010, Volume 121, Issue 24, pp. 2661 - 2671
Background-Excessive proliferation and impaired apoptosis of pulmonary artery (PA) smooth muscle cells (PASMCs) contribute to vascular obstruction in patients... 
genetic | mitochondrial diseases | epigenesis | hypoxia-inducible factor-1 | gene silencing | pulmonary | oxidation-reduction | subunit | hypertension | superoxide dismutase 2 | MULTIPLE-MYELOMA | APOPTOSIS | METHYLATION | CARDIAC & CARDIOVASCULAR SYSTEMS | hypertension, pulmonary | epigenesis, genetic | HYPOXIA | CANCER | OVEREXPRESSION | GENE | hypoxia-inducible factor-1, alpha-subunit | PATHWAY | DNA METHYLTRANSFERASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | SOD2 EXPRESSION | Superoxide Dismutase - genetics | Cell Proliferation | Mitochondria, Muscle - enzymology | Humans | Myocytes, Smooth Muscle - pathology | Rats | Hypertension, Pulmonary - metabolism | Rats, Sprague-Dawley | Hydrogen Peroxide - metabolism | Epigenesis, Genetic - physiology | Phenotype | Animals | Biomimetics | Epigenesis, Genetic - genetics | Rats, Mutant Strains | Adenoviridae - genetics | Hypertension, Pulmonary - drug therapy | Hypertension, Pulmonary - pathology | Myocytes, Smooth Muscle - metabolism | Superoxide Dismutase - metabolism | Apoptosis | Disease Models, Animal | Cell proliferation | Physiological aspects | Development and progression | Superoxide dismutase | Research | Vascular smooth muscle | Pulmonary hypertension | Epigenetic gene methylation | DNA methyltransferase | Pulmonary arterial hypertension | Hypoxia-inducible factor-1α (HIF-1α) | Voltage-gated potassium channels (Kv1.5)
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 01/2010, Volume 121, Issue 1, pp. 98 - 109
Background-Pulmonary arterial hypertension is a progressive proliferative vasculopathy of the small pulmonary arteries that is characterized by a primary... 
Hypoxia | Nitric oxide | Pulmonary artery smooth muscle cells | Xanthine oxidoreductase | Monocrotaline sodium | hypoxia | nitric oxide | xanthine oxidoreductase | ISCHEMIA/REPERFUSION INJURY | CARDIAC & CARDIOVASCULAR SYSTEMS | ORGANIC NITRATES | monocrotaline sodium | ACTIVATED-PROTEIN-KINASE | CELL-PROLIFERATION | EXERCISE CAPACITY | pulmonary artery smooth muscle cells | SYNTHASE | L-ARGININE | IN-VIVO | PERIPHERAL VASCULAR DISEASE | ISCHEMIA-REPERFUSION | HEMATOLOGY | EXPRESSION | Xanthine Dehydrogenase - metabolism | Hypoxia - drug therapy | Xanthine Dehydrogenase - antagonists & inhibitors | Male | Hypertension, Pulmonary - chemically induced | Hypoxia - metabolism | Sodium Nitrite - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Hypertension, Pulmonary - drug therapy | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Disease Models, Animal | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Administration, Inhalation | Rats | Hypertension, Pulmonary - metabolism | Monocrotaline - toxicity | Rats, Sprague-Dawley | Cell Division - drug effects | Mice, Knockout | Pulmonary Artery - cytology | Animals | Mice | Nitric Oxide - metabolism | Sodium Nitrite - pharmacokinetics | Chronic Disease | Cell proliferation | Control | Nitrites | Physiological aspects | Smooth muscle | Research | Pulmonary hypertension
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2015, Volume 17, Issue 5, pp. 627 - 638
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 9, pp. 1 - 13
Background: It has long been thought that mammalian cardiomyocytes are terminally-differentiated and unable to proliferate. However, myocytes in more primitive... 
PROGENITORS | ABNORMAL QT PROLONGATION | MULTIPOTENT | FIBROBLAST | MULTIDISCIPLINARY SCIENCES | MOUSE | CARDIAC STEM-CELLS | ZEBRAFISH HEART REGENERATION | EXPRESSION | ADULT | ATRIAL-FIBRILLATION | Cell Proliferation | GATA4 Transcription Factor - metabolism | Myocytes, Cardiac - cytology | Homeodomain Proteins - metabolism | Cells, Cultured | Rats | Rats, Inbred WKY | GATA4 Transcription Factor - genetics | Male | Mice, Transgenic | Transcription Factors - genetics | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Homeobox Protein Nkx-2.5 | Animals | Myocytes, Cardiac - metabolism | Cell Differentiation | Mice | Fluorescence | Genetic engineering | Stem cells | Heart cells | Heart | Cell proliferation | Cell culture | Cardiac arrhythmia | Transcription factors | Physicians | Centrifugation | Myocytes | Kinases | Genotype & phenotype | Cell fate | Transgenic animals | Rodents | Cell cycle | Plasticity | Fibroblasts | Extracellular matrix | Cardiology | Heart diseases | Nkx2.5 protein | Explants | Antigens | Cardiac muscle | Transgenic mice | MiRNA | Zebrafish | Cardiomyocytes | Mammals | Tamoxifen | Gene expression | c-Kit protein | Endothelial cells | Medicine | Studies | Polymerase chain reaction | Microscopy | Striations | Green fluorescent protein | Plastic properties | Diabetes | Gene mapping | Pluripotency | Plastic foam
Journal Article
Circulation Research, ISSN 0009-7330, 06/2013, Volume 112, Issue 12, pp. 1557 - 1566
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2015, Volume 65, Issue 9, pp. 892 - 900
Journal Article