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Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2010, Volume 207, Issue 8, pp. 1589 - 1597
Uncontrolled extracellular matrix production by fibroblasts in response to tissue injury contributes to fibrotic diseases, such as idiopathic pulmonary... 
MEDICINE, RESEARCH & EXPERIMENTAL | PHOSPHATASE | MECHANISMS | GROWTH-FACTOR | IMMUNOLOGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | MICRORNA | Oligonucleotides - genetics | Pulmonary Fibrosis - therapy | Idiopathic Pulmonary Fibrosis - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Actins - metabolism | MicroRNAs - metabolism | Pulmonary Fibrosis - genetics | Idiopathic Pulmonary Fibrosis - metabolism | Actins - genetics | Antisense Elements (Genetics) - therapeutic use | Collagen - genetics | Lung - metabolism | Phosphorylation - drug effects | Smad7 Protein - genetics | Extracellular Matrix Proteins - metabolism | Transforming Growth Factor beta1 - pharmacology | Fibroblasts - metabolism | Smad7 Protein - metabolism | Cell Line | Lung - pathology | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Smad2 Protein - metabolism | Mice, Transgenic | Pulmonary Fibrosis - pathology | Transforming Growth Factor beta1 - genetics | Fibroblasts - pathology | Fibronectins - metabolism | Collagen - metabolism | Animals | Fibroblasts - drug effects | Antisense Elements (Genetics) - genetics | Idiopathic Pulmonary Fibrosis - pathology | Pulmonary Fibrosis - chemically induced | Bleomycin - pharmacology | Fibronectins - genetics | Mice | MicroRNAs - genetics | Brief Definitive Report
Journal Article
Circulation Research, ISSN 0009-7330, 06/2010, Volume 106, Issue 11, pp. 1675 - 1680
Fibrosis is one of the largest groups of diseases for which there is no therapy but is believed to occur because of a persistent tissue repair program. During... 
TGF | Endothelin | Scarring | PDGF | Fibrosis | CARDIAC & CARDIOVASCULAR SYSTEMS | scarring | GENE-REGULATION | FIBROTIC PHENOTYPE | MATRIX CONTRACTION | endothelin | GROWTH-FACTOR-BETA | MYOCARDIAL FIBROSIS | IN-VIVO | PERIPHERAL VASCULAR DISEASE | fibrosis | SMOOTH-MUSCLE-CELLS | DERMAL FIBROBLASTS | TGF beta | MICE LACKING SMAD3 | HEMATOLOGY | MYOFIBROBLAST DIFFERENTIATION
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e35144
Interleukin-6 (IL-6) is an important cytokine participating in multiple biologic activities in immune regulation and inflammation. IL-6 has been associated... 
VASCULAR FIBROSIS | HYPERTROPHY | MYOCARDIAL-INFARCTION | INFLAMMATION | BIOLOGY | RECEPTOR | MYOFIBROBLASTS | DYSFUNCTION | INTERLEUKIN-6 | HYPERTENSION | TRANSFORMING GROWTH FACTOR-BETA | Angiotensin II - pharmacology | Interleukin-6 - genetics | Mice, Inbred C57BL | Transforming Growth Factor beta1 - metabolism | Male | Myocardium - pathology | Smad3 Protein - metabolism | Interleukin-6 - deficiency | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Fibroblasts - drug effects | Myocardium - metabolism | Fibrosis | Interleukin-6 - biosynthesis | Collagen - biosynthesis | Mice | Phosphorylation - drug effects | Transforming Growth Factor beta1 - biosynthesis | Fibroblasts - metabolism | Immunohistochemistry | Heart | Oxidative stress | Collagen (type I) | Phosphorylation | Smad protein | Cardiomyopathy | Transforming growth factor | Interleukin | Staining | Smooth muscle | Cardiovascular disease | Smad3 protein | Kinases | Macrophages | Interleukin 6 | Infusion | Immunology | Actin | Rodents | Fibroblasts | Angiotensin II | Growth factors | Heart diseases | Culture | Heart failure | Cardiovascular system | Hypertension | Cytokines | Immunoregulation | Muscles | Cardiomyocytes | Inflammation | Gene expression | Hospitals | Angiotensin | Sepsis | Laboratory animals | Cardiac muscle | Antibodies | Transforming growth factor- beta 1 | Transforming growth factor- beta | cardiomyocytes | Cell activation
Journal Article
Journal Article
Journal Article
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