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Nature, ISSN 0028-0836, 04/2017, Volume 544, Issue 7651, pp. 493 - 497
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2014, Volume 192, Issue 6, pp. 2837 - 2845
Inflammation is associated with development of atherosclerosis, and cholesterol crystals (CC) have long been recognized as a hallmark of atherosclerotic... 
ATHEROSCLEROTIC LESIONS | TUMOR-NECROSIS-FACTOR | C5A RECEPTOR | MEMBRANE ATTACK | STERILE INFLAMMATION | EPITHELIAL-CELLS | CARDIOVASCULAR EVENTS | INTERLEUKIN-1-BETA | OXIDATIVE BURST | ANAPHYLATOXINS | IMMUNOLOGY | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology
Journal Article
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 4/2011, Volume 121, Issue 4, pp. 445 - 458
Recent studies demonstrated that primary immune responses can be induced within the brain depending on vessel-associated cells expressing markers of dendritic... 
Pathology | Neurosciences | Medicine & Public Health | Neurodegeneration | Multiple sclerosis (MS) | Innate immunity | Immune privilege | Alzheimer | Microglia | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | DENDRITIC CELLS | LEUKOCYTE EXTRAVASATION | MICROGLIAL CELLS | PATHOLOGY | ENTORHINAL CORTEX LESION | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CD8(+) T-CELLS | CLINICAL NEUROLOGY | MULTIPLE-SCLEROSIS | CERVICAL LYMPH-NODES | CHOROID-PLEXUS | Whole-Body Irradiation | Age Factors | Humans | Nervous System - metabolism | Green Fluorescent Proteins - genetics | Glial Fibrillary Acidic Protein - metabolism | T-Lymphocytes - ultrastructure | CD11c Antigen - metabolism | Lectins, C-Type - metabolism | Neuropil - ultrastructure | Neuroglia - cytology | CD11c Antigen - genetics | T-Lymphocytes - metabolism | Time Factors | Cerebral Cortex - injuries | Macrophage-1 Antigen - metabolism | Imaging, Three-Dimensional | Organ Culture Techniques | Animals, Newborn | Calcium-Binding Proteins - metabolism | Microscopy, Electron, Transmission | Nervous System - cytology | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Mice, Transgenic | Cell Adhesion Molecules - metabolism | Neuropil - metabolism | Animals | Microfilament Proteins | Neuroglia - metabolism | Mice | CD11b Antigen - metabolism | Bone Marrow Cells - metabolism | Antigens | Brain | Nervous system diseases | Multiple sclerosis | Dendritic cells | Bone marrow | Fluorescence | Transplantation | T cells | Alzheimer's disease
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 10/2017, Volume 134, Issue 4, pp. 585 - 604
Treatment of acute ischemic stroke with the thrombolytic tissue plasminogen activator (tPA) can significantly improve neurological outcomes; however,... 
Pathology | Neurosciences | Stroke | Medicine & Public Health | LRP1 | Tissue plasminogen activator | α M β 2 | CD11b/CD18 | Mac-1 | Platelet-derived growth factor-CC | Blood–brain barrier | TISSUE-PLASMINOGEN ACTIVATOR | FOCAL CEREBRAL-ISCHEMIA | PATHOLOGY | IMATINIB TREATMENT | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | alpha(M)beta | SUBSTRATE-SPECIFICITY | Blood-brain barrier | FACTOR-ALPHA RECEPTOR | INTRACEREBRAL HEMORRHAGE | CENTRAL-NERVOUS-SYSTEM | DENSITY-LIPOPROTEIN RECEPTOR | INNATE IMMUNITY | Capillary Permeability - physiology | Cerebral Hemorrhage - metabolism | Leukocytes - pathology | Microglia - metabolism | Brain Ischemia - metabolism | Capillary Permeability - drug effects | Male | Tissue Plasminogen Activator - adverse effects | Stroke - pathology | Fibrinolytic Agents - adverse effects | Lymphokines - metabolism | Microglia - pathology | Female | Macrophage-1 Antigen - metabolism | Disease Models, Animal | Cerebral Hemorrhage - chemically induced | Tumor Suppressor Proteins - metabolism | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Cells, Cultured | Receptors, LDL - metabolism | Mice, Transgenic | Stroke - drug therapy | Arterioles - drug effects | Blood-Brain Barrier - drug effects | Blood-Brain Barrier - metabolism | Arterioles - pathology | Blood-Brain Barrier - pathology | Fibrinolytic Agents - pharmacology | Platelet-Derived Growth Factor - metabolism | Stroke - metabolism | Animals | Arterioles - metabolism | Tissue Plasminogen Activator - pharmacology | Macrophage-1 Antigen - genetics | Brain Ischemia - drug therapy | Brain Ischemia - pathology | CD11b Antigen - metabolism | Cerebral Hemorrhage - pathology | Leukocytes - metabolism | Bone Marrow Cells - metabolism | Stroke (Disease) | Platelet-derived growth factor | Ischemia | Analysis | Bone marrow | Transplantation | Permeability | Integrins | Phosphorylation | CD11b antigen | Arterioles | Membrane permeability | Leukocytes | Macrophages | Hemorrhage | Thrombolysis | t-Plasminogen activator | Microglia | Cerebrovascular system | Cell activation | Cerebral blood flow | Mac1 protein | Rodents | Immunofluorescence | αMβ2 | CD18 | Original Paper | CD11b
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 04/2018, Volume 58, Issue 4, pp. 492 - 499
Severe influenza virus infection can lead to life-threatening pathology through immune-mediated tissue damage. In various experimental models, this damage is... 
Influenza | CD11b | Neutrophils | influenza | VIRUS-INFECTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | LUNG INJURY | ANTIBODY | MECHANISMS | CELL BIOLOGY | EMIGRATION | T-CELL RESPONSES | RESPIRATORY SYSTEM | INFLAMMATION | DISEASE | MICE | neutrophils | Chemotaxis, Leukocyte | CD18 Antigens - immunology | Macrophage-1 Antigen - immunology | Adoptive Transfer | T-Lymphocytes - virology | Viral Load | Orthomyxoviridae Infections - genetics | CD11b Antigen - genetics | Lung - virology | T-Lymphocytes - metabolism | Time Factors | Influenza A virus - pathogenicity | Female | Influenza A virus - immunology | Macrophage-1 Antigen - metabolism | Lung - metabolism | Neutrophils - metabolism | Disease Models, Animal | Lung - pathology | CD11b Antigen - immunology | Signal Transduction | Orthomyxoviridae Infections - metabolism | Mice, Inbred C57BL | Neutrophils - immunology | Mice, Transgenic | Neutrophils - virology | CD18 Antigens - metabolism | Host-Pathogen Interactions | Animals | Macrophage-1 Antigen - genetics | Weight Loss | Neutrophils - transplantation | T-Lymphocytes - immunology | CD11b Antigen - metabolism | Orthomyxoviridae Infections - virology | Lung - immunology | Orthomyxoviridae Infections - immunology | Immunohistochemistry | Pathogens | Immunoglobulins | CD11b antigen | Animal models | Laboratories | Viruses | Infections | Lymphocytes T | Leukocytes (neutrophilic) | Macrophages | Adoptive transfer | Pathology | Hypotheses | Lungs | Lymphocytes | Mac1 protein | Rodents | Bone marrow | CD18 antigen
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Journal Article
Journal Article