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Journal Article
Clinical Immunology, ISSN 1521-6616, 2010, Volume 137, Issue 1, pp. 89 - 101
Abstract Activated macrophages have been characterized as M1 and M2 according to their inflammatory response pattern. Here we analyzed the M2 marker expression... 
Allergy and Immunology | M2 phenotype | Th2 cytokines | Pulmonary fibrosis | IL-1RA | CCL22 | Sarcoidosis | JAK/STAT pathway | CCL18 | Alveolar macrophages | CCL17 | INTERLEUKIN-1 RECEPTOR ANTAGONIST | CYTOKINE | IMMUNOLOGY | SYSTEMIC-SCLEROSIS | STAT3 ACTIVATION | INTERSTITIAL LUNG-DISEASES | MANNOSE RECEPTOR | GENE-EXPRESSION | TNF-ALPHA RELEASE | GROWTH-FACTOR | UP-REGULATION | Mannose-Binding Lectins - metabolism | Tumor Necrosis Factor-alpha - metabolism | Gene Expression - drug effects | Humans | Middle Aged | STAT Transcription Factors - metabolism | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Monocytes - metabolism | Monocytes - immunology | Sarcoidosis, Pulmonary - diagnosis | Idiopathic Pulmonary Fibrosis - metabolism | Young Adult | Lectins, C-Type - metabolism | Signal Transduction - immunology | Interleukin-4 - pharmacology | Interleukin 1 Receptor Antagonist Protein - metabolism | Scleroderma, Systemic - complications | Aged, 80 and over | Adult | Female | Bronchoalveolar Lavage Fluid - cytology | Pulmonary Fibrosis - metabolism | Interleukin-8 - metabolism | Macrophages, Alveolar - immunology | Pulmonary Fibrosis - etiology | Sarcoidosis, Pulmonary - complications | Cytokines - metabolism | Macrophage Activation - immunology | Scleroderma, Systemic - metabolism | Pulmonary Fibrosis - immunology | Receptors, Cell Surface - metabolism | Idiopathic Pulmonary Fibrosis - immunology | Monocytes - drug effects | Signal Transduction - drug effects | Macrophages, Alveolar - drug effects | Sarcoidosis, Pulmonary - metabolism | Chemokines, CC - genetics | Aged | Macrophages, Alveolar - metabolism | Macrophage Activation - drug effects | Interleukin-10 - pharmacology | Chemokines, CC - metabolism | Respiratory tract diseases | Systemic scleroderma | Transforming growth factors | Macrophages | Scleroderma (Disease) | Alveoli
Journal Article
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 11/2010, Volume 182, Issue 10, pp. 1292 - 1304
Rationale We identified a 6 year old girl with pulmonary alveolar proteinosis (PAP), impaired granulocyte macrophage colony stimulating factor (GM CSF)... 
Surfactant | Gm-csf receptor | Alveolar macrophage | Whole lung lavage | Genetic disease | alveolar macrophage | INTERLEUKIN-3 | COLONY-STIMULATING FACTOR | GM CSF | DISORDERS | COMMON BETA-SUBUNIT | DEFICIENT MICE | GM-CSF AUTOANTIBODIES | receptor | whole lung lavage | LUNG LAVAGE | GENE | RESPIRATORY SYSTEM | genetic disease | MACROPHAGE DIFFERENTIATION | surfactant | CRITICAL CARE MEDICINE | Humans | Genetic Diseases, Inborn - genetics | Child, Preschool | Infant | Male | Dyspnea - etiology | Receptors, Granulocyte Colony-Stimulating Factor - genetics | Genetic Diseases, Inborn - therapy | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Pulmonary Alveolar Proteinosis - genetics | Receptors, Granulocyte Colony-Stimulating Factor - blood | Female | Genetic Diseases, Inborn - etiology | Child | Genetic Diseases, Inborn - diagnosis | Granulocyte-Macrophage Colony-Stimulating Factor - blood | Lung - pathology | Pulmonary Alveolar Proteinosis - therapy | Genotype | Disease Progression | Pulmonary Alveolar Proteinosis - pathology | Receptors, Granulocyte Colony-Stimulating Factor - physiology | Autoantibodies - physiology | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - physiology | Pedigree | Genetic Markers - genetics | Age of Onset | Mutation | Pulmonary Alveolar Proteinosis - diagnosis | F. Pediatrics and Lung Development | GM-CSF receptor
Journal Article