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Clinical Immunology, ISSN 1521-6616, 2010, Volume 137, Issue 1, pp. 89 - 101
Abstract Activated macrophages have been characterized as M1 and M2 according to their inflammatory response pattern. Here we analyzed the M2 marker expression... 
Allergy and Immunology | M2 phenotype | Th2 cytokines | Pulmonary fibrosis | IL-1RA | CCL22 | Sarcoidosis | JAK/STAT pathway | CCL18 | Alveolar macrophages | CCL17 | INTERLEUKIN-1 RECEPTOR ANTAGONIST | CYTOKINE | IMMUNOLOGY | SYSTEMIC-SCLEROSIS | STAT3 ACTIVATION | INTERSTITIAL LUNG-DISEASES | MANNOSE RECEPTOR | GENE-EXPRESSION | TNF-ALPHA RELEASE | GROWTH-FACTOR | UP-REGULATION | Mannose-Binding Lectins - metabolism | Tumor Necrosis Factor-alpha - metabolism | Gene Expression - drug effects | Humans | Middle Aged | STAT Transcription Factors - metabolism | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Monocytes - metabolism | Monocytes - immunology | Sarcoidosis, Pulmonary - diagnosis | Idiopathic Pulmonary Fibrosis - metabolism | Young Adult | Lectins, C-Type - metabolism | Signal Transduction - immunology | Interleukin-4 - pharmacology | Interleukin 1 Receptor Antagonist Protein - metabolism | Scleroderma, Systemic - complications | Aged, 80 and over | Adult | Female | Bronchoalveolar Lavage Fluid - cytology | Pulmonary Fibrosis - metabolism | Interleukin-8 - metabolism | Macrophages, Alveolar - immunology | Pulmonary Fibrosis - etiology | Sarcoidosis, Pulmonary - complications | Cytokines - metabolism | Macrophage Activation - immunology | Scleroderma, Systemic - metabolism | Pulmonary Fibrosis - immunology | Receptors, Cell Surface - metabolism | Idiopathic Pulmonary Fibrosis - immunology | Monocytes - drug effects | Signal Transduction - drug effects | Macrophages, Alveolar - drug effects | Sarcoidosis, Pulmonary - metabolism | Chemokines, CC - genetics | Aged | Macrophages, Alveolar - metabolism | Macrophage Activation - drug effects | Interleukin-10 - pharmacology | Chemokines, CC - metabolism | Respiratory tract diseases | Systemic scleroderma | Transforming growth factors | Macrophages | Scleroderma (Disease) | Alveoli
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2013, Volume 49, Issue 4, pp. 503 - 510
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 04/2015, Volume 42, Issue 4, pp. 640 - 653
Acute lung injury (ALI) remains a serious health issue with little improvement in our understanding of the pathophysiology and therapeutic approaches. We... 
BACTERIAL LIPOPOLYSACCHARIDE | STERILE INFLAMMATION | SIGNALING PATHWAY | P2X RECEPTORS | ALPHA PRODUCTION | ENDOTHELIAL-CELLS | MOUSE MODEL | IN-VIVO | ACUTE LUNG INJURY | IMMUNOLOGY | INFLAMMATORY RESPONSE | Interleukin-1alpha - immunology | Macrophages, Alveolar - pathology | Calcium - metabolism | Humans | Capillary Permeability - immunology | Neutrophil Infiltration | Necrosis - pathology | Receptors, Purinergic P2X7 - genetics | Necrosis - immunology | Cadherins - immunology | Adenosine Triphosphate - metabolism | HEK293 Cells | Interleukin-1alpha - genetics | Cadherins - genetics | Receptors, Purinergic P2X7 - immunology | Macrophages, Alveolar - immunology | Neutrophils - pathology | Lung - pathology | Protein Precursors - genetics | Signal Transduction | Neutrophils - drug effects | Gene Expression Regulation | Neutrophils - immunology | Lipopolysaccharide Receptors - immunology | Mice, Transgenic | Protein Precursors - immunology | Acute Lung Injury - pathology | Necrosis - chemically induced | Endothelial Cells - immunology | Animals | Acute Lung Injury - chemically induced | Macrophages, Alveolar - drug effects | Lung - drug effects | Lipopolysaccharides - pharmacology | Lipopolysaccharide Receptors - genetics | Acute Lung Injury - immunology | Mice | Endothelial Cells - pathology | Intubation, Intratracheal | Lung - immunology | Endothelial Cells - drug effects | endothelial cells | alveolar macrophage | Ca2+ influx | MyD88 | IL-1α | neutrophil | P2X7R | lung injury | necrosis | ARDS | ALI | sepsis | lung inflammation
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 07/2018, Volume 59, Issue 1, pp. 45 - 55
IFN-beta is reported to improve survival in patients with acute respiratory distress syndrome (ARDS), possibly by preventing sepsis-induced immunosuppression,... 
Immune suppression | Acute lung injury | Innate immunity | IFN-β | Bacterial pneumonia | IFN-beta | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUTROPHILS | PNEUMONIA | acute lung injury | PUNCTURE | CELL BIOLOGY | innate immunity | CECAL LIGATION | RESPIRATORY SYSTEM | IMMUNOSUPPRESSION | INFLAMMATION | bacterial pneumonia | INFECTION | MICE | immune suppression | T-CELLS | Macrophages, Alveolar - pathology | Respiratory Distress Syndrome, Adult - physiopathology | Humans | Male | Respiratory Distress Syndrome, Adult - mortality | Lung Injury - etiology | Interferon-beta - therapeutic use | Respiratory Distress Syndrome, Adult - blood | Sepsis - drug therapy | Pneumonia - blood | Lung Injury - drug therapy | Disease Models, Animal | Cytokines - metabolism | Immunity, Innate - drug effects | Lung Injury - blood | Mice, Inbred C57BL | Treatment Outcome | Respiratory Distress Syndrome, Adult - drug therapy | Sepsis - physiopathology | Lung Injury - physiopathology | Animals | Interferon-beta - pharmacology | Signal Transduction - drug effects | Macrophages, Alveolar - drug effects | Models, Biological | Survival Analysis | Pneumonia - complications | Sepsis - blood | Pneumonia | β-Interferon | Pathogenesis | Immune clearance | Infections | Macrophages | Recruitment | Rodents | Tumor necrosis factor-TNF | Alveoli | Trachea | Edema | Cell survival | Bacterial infections | Cytokines | Secretion | Mortality | Cecum | Neutrophils | Leukocytes (neutrophilic) | Inflammation | Tumor necrosis factor-α | Immunosuppression | Respiratory distress syndrome | Lungs | Nitric oxide | Interleukin 10 | Sepsis | Phagocytosis | Viral infections
Journal Article
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 06/2016, Volume 23, Issue 6, pp. 1026 - 1037
Journal Article