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Journal Article
Toxicological Sciences, ISSN 1096-6080, 08/2018, Volume 164, Issue 2, pp. 592 - 602
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 2783 - 14
The importance of the tumor-associated stroma in cancer progression is clear. However, it remains uncertain whether early events in the stroma are capable of... 
BREAST-CANCER | NUCLEAR PTEN | CONTRALATERAL BREAST | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | FOLLOW-UP | DNA-REPAIR | TUMOR MICROENVIRONMENT | MOUSE MODELS | FIBROBLASTS | RADIATION-THERAPY | Epithelial Cells - metabolism | Gamma Rays - adverse effects | Epithelial Cells - drug effects | Humans | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | Mammary Glands, Human - metabolism | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Mammary Neoplasms, Experimental - pathology | Stromal Cells - drug effects | Genomic Instability - drug effects | Female | Antineoplastic Agents - pharmacology | Mammary Glands, Human - radiation effects | Mammary Glands, Human - drug effects | Genomic Instability - radiation effects | PTEN Phosphohydrolase - genetics | Epithelial Cells - radiation effects | PTEN Phosphohydrolase - deficiency | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Signal Transduction | Stromal Cells - metabolism | Radiation Tolerance - genetics | Mammary Neoplasms, Experimental - radiotherapy | Mammary Glands, Animal - drug effects | Mammary Glands, Animal - radiation effects | Breast Neoplasms - radiotherapy | Animals | Breast Neoplasms - genetics | Mammary Glands, Animal - metabolism | Breast Neoplasms - pathology | Cell Transformation, Neoplastic | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Cell Proliferation - radiation effects | Stromal Cells - radiation effects | Hyperplasia | Radiation | Paracrine signalling | Homology | Genomic instability | Epidermal growth factor | Clonal deletion | Deletion | Tumorigenesis | Inhibition | Mammary gland | Tensin | Radiation effects | Stability | Epidermal growth factor receptors | Stroma | Risk reduction | Mammary glands | Breast cancer | Radiation therapy | Epithelium | Patients | Radiation dosage | Breast | Diagnostic systems | PTEN protein | Cancer | Tumors | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 01/2014, Volume 33, Issue 5, pp. 643 - 652
Cancer stem cells (CSCs), a small and elusive population of undifferentiated cancer cells within tumors that drive tumor growth and recurrence, are believed to... 
Cancer stem cell | MCF-10A | reprogramming factors | p16INK4a | oncogenesis | PHOSPHORYLATION | MARKER | BIOCHEMISTRY & MOLECULAR BIOLOGY | IDENTIFICATION | TUMORS | CELL BIOLOGY | BREAST-CANCER | GASTROINTESTINAL CANCER | cancer stem cell | ONCOLOGY | LINE | GENETICS & HEREDITY | PIN1 | EXPRESSION | IMMORTALIZATION | Neoplastic Stem Cells - cytology | Cell Proliferation | Paclitaxel - pharmacology | Epithelial Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Cellular Senescence - drug effects | Cellular Reprogramming | SOXB1 Transcription Factors - metabolism | MCF-7 Cells | Kruppel-Like Transcription Factors - metabolism | Female | Cyclin-Dependent Kinase Inhibitor p16 - pharmacology | Epithelial Cells - cytology | Induced Pluripotent Stem Cells - cytology | CD24 Antigen - biosynthesis | Pyrans - pharmacology | Mammary Glands, Human - drug effects | Mammary Glands, Human - cytology | Spheroids, Cellular - metabolism | Naphthoquinones - pharmacology | Hyaluronan Receptors - biosynthesis | Proto-Oncogene Proteins c-myc - metabolism | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - pathology | Cell Differentiation - drug effects | Mice, Nude | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Mice | Mice, Inbred BALB C | Dactinomycin - pharmacology | Drug Resistance, Neoplasm - drug effects | Epithelial cells | Oncology, Experimental | Stem cells | Cancer cells | Physiological aspects | Mammary glands | Research | Carcinogenesis | Cancer | Studies | Oncology | Metastasis | Cancer therapies | Tumors | Index Medicus
Journal Article
Molecular Cell, ISSN 1097-2765, 08/2018, Volume 71, Issue 4, pp. 606 - 620.e7
Metformin has been reported to possess antitumor activity and maintain high cytotoxic T lymphocyte (CTL) immune surveillance. However, the functions and... 
ERAD | ER accumulation | cancer immunotherapy | PD-L1 | metformin | glycosylation | immune checkpoint blockade | BREAST-CANCER | CELLS | ACTIVATED PROTEIN-KINASE | ADVANCED MELANOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMPK | MECHANISMS | QUALITY-CONTROL | GLYCOSYLATION | EXPRESSION | CHECKPOINT BLOCKADE | CELL BIOLOGY | Endoplasmic Reticulum-Associated Degradation | Mammary Glands, Human - immunology | Phosphorylation | Epithelial Cells - drug effects | Humans | Melanoma, Experimental - drug therapy | Gene Expression Regulation, Neoplastic | Endoplasmic Reticulum - metabolism | T-Lymphocytes, Cytotoxic - drug effects | Melanoma, Experimental - immunology | AMP-Activated Protein Kinases - immunology | Endoplasmic Reticulum - drug effects | Female | Antineoplastic Agents - pharmacology | Epithelial Cells - cytology | Mammary Glands, Human - drug effects | T-Lymphocytes, Cytotoxic - immunology | Mammary Glands, Human - cytology | Endoplasmic Reticulum - genetics | Metformin - pharmacology | Melanoma, Experimental - pathology | CTLA-4 Antigen - genetics | Glycosylation | CTLA-4 Antigen - immunology | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Serine - metabolism | Hypoglycemic Agents - pharmacology | Animals | Melanoma, Experimental - genetics | Epithelial Cells - immunology | Cell Line, Tumor | Mice, Inbred NOD | Mice | T-Lymphocytes, Cytotoxic - cytology | AMP-Activated Protein Kinases - genetics | Metformin | T cells | Proteolysis | Protein kinases | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 10, pp. e26514 - e26514
The transcriptional repressors Snail and Slug are situated at the core of several signaling pathways proposed to mediate epithelial to mesenchymal transition... 
TGF-BETA | PROTEIN | CLAUDIN-LOW | MULTIDISCIPLINARY SCIENCES | REPRESSES E-CADHERIN | GENE-EXPRESSION | SUPERFAMILY | OVARIAN | TGF-BETA-PAR6 POLARITY PATHWAY | EPITHELIAL-MESENCHYMAL TRANSITIONS | FIBROBLASTS | Transcription, Genetic - drug effects | Receptors, Transforming Growth Factor beta - genetics | Humans | Genetic Loci - genetics | Substrate Specificity | Epithelial-Mesenchymal Transition - drug effects | Mammary Glands, Human - metabolism | RNA, Messenger - metabolism | Breast Neoplasms - metabolism | Dioxoles - pharmacology | Transforming Growth Factor beta - antagonists & inhibitors | Female | Benzamides - pharmacology | Snail Family Transcription Factors | Mammary Glands, Human - drug effects | Pyrazoles - pharmacology | RNA, Messenger - genetics | Intercellular Junctions - metabolism | Protein-Serine-Threonine Kinases - genetics | Mammary Glands, Human - pathology | Transcription Factors - genetics | Down-Regulation - drug effects | Transcription Factors - metabolism | Cell Movement - drug effects | Acetylation - drug effects | Phenotype | Pyrroles - pharmacology | Breast Neoplasms - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Cell Differentiation - drug effects | Intercellular Junctions - drug effects | Cell Line, Tumor | Histones - metabolism | Transforming Growth Factor beta - metabolism | Analysis | Genes | Stem cells | Bone morphogenetic proteins | Breast cancer | DNA binding proteins | Metastasis | Transforming growth factors | Health sciences | Transcription factors | Leukocyte migration | Mesenchyme | Laboratories | Epithelial cells | Transforming growth factor-b | Cytology | Kinases | Metastases | Morphogenesis | Proteins | Signal transduction | Pathways | Repressors | Cell cycle | Acetylation | Growth factors | Invasiveness | Environmental health | Gene expression | Ribonucleic acid--RNA | Signaling | DNA microarrays | Collaboration | Medical prognosis | Adenoviruses | Breast | Snail protein | Cell migration | Cancer | Tumors | Index Medicus | RNA | Ribonucleic acid
Journal Article
Breast cancer research : BCR, ISSN 1465-5411, 08/2004, Volume 6, Issue 6, pp. R605 - 615
Introduction Notch signaling has been implicated in the regulation of cell-fate decisions such as self-renewal of adult stem cells and differentiation of... 
Mammary gland development | Mammary stem cells | Notch | Mammary progenitor cells | PROGENITOR CELLS | STEM-CELLS | ACTIVATION | MAMMALIAN NOTCH | mammary stem cells | BREAST-CANCER | mammary gland development | MORPHOGENESIS | IN-VITRO | EPITHELIAL-CELLS | ONCOLOGY | DIFFERENTIATION | mammary progenitor cells | GAMMA-SECRETASE | Epithelial Cells - metabolism | Humans | Stem Cells - cytology | Mammary Glands, Human - metabolism | Proto-Oncogene Proteins - biosynthesis | Receptors, Cell Surface - antagonists & inhibitors | Receptors, Notch | Proto-Oncogene Proteins - agonists | Female | Receptors, Cell Surface - biosynthesis | Receptors, Cell Surface - physiology | Epithelial Cells - cytology | Cell Differentiation - physiology | Mammary Glands, Human - drug effects | Mammary Glands, Human - cytology | Proto-Oncogene Proteins - antagonists & inhibitors | Transcription Factors - physiology | Cells, Cultured | Receptors, Cell Surface - agonists | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Signal Transduction - drug effects | Cell Differentiation - drug effects | Proto-Oncogene Proteins - physiology | Stem Cells - drug effects | Signal Transduction - physiology | Receptor, Notch1 | Transcription Factors - agonists | Receptor, Notch4 | Genetic aspects | Stem cells | Index Medicus
Journal Article
Biomaterials, ISSN 0142-9612, 2010, Volume 31, Issue 14, pp. 3920 - 3929
Journal Article
The Journal of Pathology, ISSN 0022-3417, 12/2017, Volume 243, Issue 4, pp. 496 - 509
Mammography screening has increased the detection of early pre‐invasive breast cancers, termed ductal carcinoma in situ (DCIS), increasing the urgency of... 
breast cancer | cysteine cathepsins | stefin A | myoepithelial cells | cystatin A | 3D culture | PEPTIDE IDENTIFICATION | INHIBITOR STEFIN | HUMAN CATHEPSIN-B | MAMMARY-CANCER | PATHOLOGY | CARCINOMA IN-SITU | MULTISTAGE TUMORIGENESIS | ONCOLOGY | B PROMOTES PROGRESSION | CYSTATIN-C | TUMOR MICROENVIRONMENT | Epithelial Cells - metabolism | Coculture Techniques | Epithelial Cells - drug effects | Humans | Carcinoma, Intraductal, Noninfiltrating - metabolism | Tumor Microenvironment | Carcinoma, Intraductal, Noninfiltrating - genetics | Mammary Glands, Human - metabolism | Breast Neoplasms - metabolism | Transfection | RNA Interference | Tumor Suppressor Proteins - genetics | Biomarkers, Tumor - metabolism | Female | Antineoplastic Agents - pharmacology | Mammary Glands, Human - drug effects | Cystatin A - genetics | Tumor Suppressor Proteins - metabolism | Signal Transduction | Mammary Glands, Animal - pathology | Neoplasm Invasiveness | Cathepsin B - antagonists & inhibitors | Cathepsin B - metabolism | Epithelial Cells - pathology | Mammary Glands, Human - pathology | Mammary Glands, Animal - drug effects | Carcinoma, Intraductal, Noninfiltrating - drug therapy | Cystatin A - metabolism | Breast Neoplasms - drug therapy | Cell Movement - drug effects | Animals | Breast Neoplasms - genetics | Carcinoma, Intraductal, Noninfiltrating - pathology | Mammary Glands, Animal - metabolism | Breast Neoplasms - pathology | Cysteine Proteinase Inhibitors - pharmacology | Cell Line, Tumor | Biomarkers, Tumor - genetics | Mice | Analysis | Breast cancer | Cystatin | Cell culture | Cysteine | Regulators | Ducts | Invasiveness | Breasts | Health risks | Cathepsins | Cultures | Mammography | Pharmacology | Proteinase inhibitors | Protease inhibitors | Cathepsin B | Breast | Tumorigenesis | Three dimensional models | Cancer | Index Medicus
Journal Article