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Journal Article
Nature, ISSN 0028-0836, 09/2015, Volume 525, Issue 7567, pp. 119 - 123
Journal Article
Science, ISSN 0036-8075, 9/2010, Volume 329, Issue 5998, pp. 1492 - 1499
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Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2012, Volume 287, Issue 43, pp. 36593 - 36608
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Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1944 - 12
Cancer recurrence after initial diagnosis and treatment is a major cause of breast cancer (BC) mortality, which results from the metastatic outbreak of dormant... 
INHIBITION | ROLES | MULTIDISCIPLINARY SCIENCES | GROWTH | EXTRACELLULAR-MATRIX | MECHANISMS | MITOPHAGY | OUTGROWTH | CARCINOMA | PROGRESSION | PRINCIPLES | RNA, Small Interfering - genetics | Recurrence | Reactive Oxygen Species - metabolism | Autophagy-Related Protein 7 - metabolism | Humans | Gene Expression Regulation, Neoplastic | Autophagy - drug effects | Reactive Oxygen Species - agonists | Breast Neoplasms - metabolism | Caspases - metabolism | Female | Autophagy - genetics | Mammary Neoplasms, Animal - genetics | Autophagy-Related Protein 7 - antagonists & inhibitors | Cell Survival - drug effects | Adenine - analogs & derivatives | Signal Transduction | Caspases - genetics | Beclin-1 - antagonists & inhibitors | Adenine - pharmacology | Lymphatic Metastasis | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Autophagy-Related Protein 7 - genetics | Mammary Neoplasms, Animal - pathology | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Mammary Neoplasms, Animal - metabolism | Beclin-1 - genetics | Cell Line, Tumor | Hydroxychloroquine - pharmacology | Cell Proliferation - drug effects | Collagen Type I - pharmacology | Mammary Neoplasms, Animal - drug therapy | Mice | Beclin-1 - metabolism | RNA, Small Interfering - metabolism | Cell proliferation | Animal models | Cell survival | Outbreaks | Breast cancer | Pharmacology | Metastasis | Autophagy | Survival | Metastases | Dormancy | Signal transduction | Mitochondria | Inhibition | Damage accumulation | Phagocytosis | Cancer | Tumors | Apoptosis
Journal Article
Nature, ISSN 0028-0836, 08/2016, Volume 537, Issue 7618, pp. 63 - 68
Journal Article
Cell, ISSN 0092-8674, 2007, Volume 129, Issue 4, pp. 723 - 733
Transcriptional activation of the nuclear receptor RAR by retinoic acid (RA) often leads to inhibition of cell growth. However, in some tissues, RA promotes... 
ACUTE-PROMYELOCYTIC-LEUKEMIA | PPAR-BETA | BREAST-CANCER CELLS | TERATOCARCINOMA STEM-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MAMMARY-CARCINOMA | SYMPATHETIC NEURONS | E-FABP | FATTY-ACIDS | TARGET GENE | BINDING PROTEIN-II | CELL BIOLOGY | Active Transport, Cell Nucleus - physiology | Apoptosis - drug effects | Humans | Transcriptional Activation - drug effects | Cell Survival - genetics | Apoptosis - genetics | Mammary Neoplasms, Experimental - physiopathology | Fatty Acid-Binding Proteins - metabolism | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Receptors, Retinoic Acid - genetics | Cell Nucleus - metabolism | Mammary Neoplasms, Animal - physiopathology | Female | Gene Expression Regulation, Neoplastic - physiology | Mammary Neoplasms, Animal - genetics | Tretinoin - pharmacology | Receptors, Retinoic Acid - drug effects | Cell Survival - drug effects | PPAR-beta - metabolism | Keratinocytes | Receptors, Retinoic Acid - metabolism | Cell Transformation, Neoplastic - metabolism | Fatty Acid-Binding Proteins - genetics | Animals | PPAR-beta - drug effects | Active Transport, Cell Nucleus - drug effects | Mammary Neoplasms, Animal - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Transcriptional Activation - physiology | Cell Transformation, Neoplastic - drug effects | Cell Nucleus - drug effects | Growth | Tretinoin
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