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Nature, ISSN 0028-0836, 11/2015, Volume 527, Issue 7579, pp. 472 - 476
The role of epithelial-to-mesenchymal transition (EMT) in metastasis is a longstanding source of debate, largely owing to an inability to monitor transient and... 
CANCER-CELLS | STEM-CELLS | THERAPY | MULTIDISCIPLINARY SCIENCES | RESISTANCE | INDUCTION | MODEL | MIR-200 FAMILY | EXPRESSION | BREAST | PLASTICITY | Lung Neoplasms - drug therapy | Apoptosis - drug effects | Antineoplastic Agents, Alkylating - pharmacology | Epithelial-Mesenchymal Transition - drug effects | Lung Neoplasms - pathology | Male | Epithelial-Mesenchymal Transition - genetics | Cyclophosphamide - therapeutic use | Mammary Neoplasms, Experimental - genetics | Cell Tracking | Lung Neoplasms - secondary | Neoplasm Metastasis - drug therapy | Mammary Neoplasms, Experimental - pathology | Female | Disease Models, Animal | Mammary Neoplasms, Experimental - drug therapy | Lung Neoplasms - genetics | Reproducibility of Results | Disease Progression | Antineoplastic Agents, Alkylating - therapeutic use | Cell Lineage | Neoplasm Metastasis - genetics | Drug Resistance, Neoplasm - genetics | Animals | Neoplasm Metastasis - pathology | Cyclophosphamide - pharmacology | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Drug Resistance, Neoplasm - drug effects | Development and progression | Metastasis | Drug resistance | Cell differentiation | Health aspects | Lung cancer | Chemotherapy | Analysis | Stem cells | Cancer | Studies | Genotype & phenotype | Transgenic animals | Rodents | Morphology | Fibroblasts | Breast cancer | Cancer therapies | Tumors | Index Medicus
Journal Article
Science Translational Medicine, ISSN 1946-6234, 03/2011, Volume 3, Issue 73, pp. 73ra21 - 73ra21
Journal Article
Journal Article
Journal of the American Chemical Society, ISSN 0002-7863, 08/2014, Volume 136, Issue 33, pp. 11748 - 11756
All drugs for cancer therapy face several transportation barriers on their tortuous journey to the action sites. To overcome these barriers, an effective drug... 
GOLD NANOPARTICLES | THERAPEUTICS | STABILITY | IN-VIVO | NANOMEDICINE | RESISTANCE | SUPRAMOLECULAR NANOSTRUCTURES | NANOCAPSULES | COPOLYMER MICELLES | CHEMISTRY, MULTIDISCIPLINARY | CHEMOTHERAPY | Prodrugs - administration & dosage | Small Molecule Libraries - pharmacology | Nanoparticles - chemistry | Apoptosis - drug effects | Small Molecule Libraries - administration & dosage | Drug Resistance, Multiple - drug effects | Humans | Prodrugs - chemistry | Chlorambucil - pharmacology | MCF-7 Cells | Mammary Neoplasms, Experimental - pathology | Molecular Structure | Iridium - chemistry | Mammary Neoplasms, Experimental - drug therapy | Chlorambucil - chemistry | Rats | Breast Neoplasms - drug therapy | Surface-Active Agents - pharmacology | Small Molecule Libraries - chemistry | Animals | Surface-Active Agents - administration & dosage | Breast Neoplasms - pathology | Mice, Nude | Organometallic Compounds - chemical synthesis | Organometallic Compounds - chemistry | Cell Proliferation - drug effects | Mice | Nanoparticles - administration & dosage | Surface-Active Agents - chemistry | Cell Cycle - drug effects | Surface-Active Agents - chemical synthesis | Drug Delivery Systems - methods | Prodrugs - pharmacology | Organometallic Compounds - pharmacology | Drug Resistance, Neoplasm - drug effects | Organometallic Compounds - administration & dosage | Drugs | Drug delivery systems | Care and treatment | Prodrugs | Dosage and administration | Research | Methods | Cancer | Vehicles | Index Medicus
Journal Article
Cancer Discovery, ISSN 2159-8274, 06/2011, Volume 1, Issue 1, pp. 54 - 67
Immune-regulated pathways influence multiple aspects of cancer development. In this article we demonstrate that both macrophage abundance and T-cell abundance... 
CELL LUNG-CANCER | MAMMARY-TUMORS | COLONY-STIMULATING FACTOR | ONCOLOGY | ANGIOGENIC SWITCH | MOUSE MODEL | RECEPTOR EXPRESSION | TUMOR-ASSOCIATED MACROPHAGES | SUPPRESSOR-CELLS | MYELOID CELLS | ENDOTHELIAL GROWTH-FACTOR | Mammary Neoplasms, Experimental - immunology | Lung Neoplasms - drug therapy | Receptor, Macrophage Colony-Stimulating Factor - immunology | Epithelial Cells - metabolism | Paclitaxel - pharmacology | Prognosis | Follow-Up Studies | Breast Neoplasms - immunology | Epithelial Cells - drug effects | Humans | Lung Neoplasms - metabolism | Mammary Neoplasms, Experimental - metabolism | Receptor, Macrophage Colony-Stimulating Factor - metabolism | Breast Neoplasms - metabolism | Leukocytes - immunology | Signal Transduction - immunology | Neoplasm Metastasis | Lung Neoplasms - secondary | CD8-Positive T-Lymphocytes - metabolism | Female | Macrophages - immunology | Mammary Neoplasms, Experimental - drug therapy | Survival Rate | Macrophage Colony-Stimulating Factor - metabolism | Breast Neoplasms - drug therapy | Macrophage Colony-Stimulating Factor - immunology | Tumor Microenvironment - immunology | Disease-Free Survival | Lung Neoplasms - immunology | Macrophages - metabolism | Animals | Signal Transduction - drug effects | CD8-Positive T-Lymphocytes - drug effects | Epithelial Cells - immunology | Leukocytes - drug effects | Macrophages - drug effects | Aged | Mice | CD8-Positive T-Lymphocytes - immunology | Leukocytes - metabolism | Cohort Studies | Index Medicus
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 05/2011, Volume 71, Issue 9, pp. 3364 - 3376
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 2014, Volume 10, Issue 7, pp. 558 - 566
PTP1B, a validated therapeutic target for diabetes and obesity, has a critical positive role in HER2 signaling in breast tumorigenesis. Efforts to develop... 
BREAST-CANCER CELLS | OVEREXPRESSION | MAMMARY-TUMORS | METASTASIS | TYROSINE-PHOSPHATASE 1B | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACQUIRED-RESISTANCE | BINDING-SITE | KINASE INHIBITOR | CONTRIBUTES | PROGRESSION | Receptor, ErbB-2 - genetics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Molecular Targeted Therapy | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - enzymology | Protein Binding - drug effects | Mammary Neoplasms, Experimental - pathology | Female | Antineoplastic Agents - pharmacology | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - genetics | Mammary Neoplasms, Experimental - enzymology | Spermine - analogs & derivatives | Mammary Neoplasms, Experimental - drug therapy | Allosteric Regulation - drug effects | Protein Structure, Tertiary | Catalytic Domain | Protein Structure, Secondary | Signal Transduction | Allosteric Site - drug effects | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - antagonists & inhibitors | Spermine - chemistry | Models, Molecular | Antineoplastic Agents - chemistry | Breast Neoplasms - drug therapy | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - metabolism | Animals | Breast Neoplasms - genetics | Spermine - pharmacology | Breast Neoplasms - pathology | Cholestanes - chemistry | Cholestanes - pharmacology | Mice | Kinetics | Breast cancer | Tumorigenesis | Diabetes | Drug therapy | Index Medicus | Cholestanes | Allosteric Regulation | Breast Neoplasms | Life Sciences | Biomolecules | Biochemistry, Molecular Biology | Antineoplastic Agents | Spermine | Protein Tyrosine Phosphatase, Non-Receptor Type 1 | Mammary Neoplasms, Experimental | Allosteric Site | Protein Binding | Receptor, ErbB-2
Journal Article
Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 2516 - 2516
Cancer and stromal cells actively exert physical forces (solid stress) to compress tumour blood vessels, thus reducing vascular perfusion. Tumour interstitial... 
INTERSTITIAL FLUID PRESSURE | BREAST-CANCER | GROWTH-FACTOR-BETA | PANCREATIC STELLATE CELLS | TGF-BETA | AORTIC-ANEURYSM | SOLID STRESS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | GENE-EXPRESSION | II TYPE-2 RECEPTOR | Index Medicus
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 20, Issue 6, pp. 797 - 809
Journal Article