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Nature, ISSN 0028-0836, 11/2015, Volume 527, Issue 7579, pp. 472 - 476
The role of epithelial-to-mesenchymal transition (EMT) in metastasis is a longstanding source of debate, largely owing to an inability to monitor transient and... 
CANCER-CELLS | STEM-CELLS | THERAPY | MULTIDISCIPLINARY SCIENCES | RESISTANCE | INDUCTION | MODEL | MIR-200 FAMILY | EXPRESSION | BREAST | PLASTICITY | Lung Neoplasms - drug therapy | Apoptosis - drug effects | Antineoplastic Agents, Alkylating - pharmacology | Epithelial-Mesenchymal Transition - drug effects | Lung Neoplasms - pathology | Male | Epithelial-Mesenchymal Transition - genetics | Cyclophosphamide - therapeutic use | Mammary Neoplasms, Experimental - genetics | Cell Tracking | Lung Neoplasms - secondary | Neoplasm Metastasis - drug therapy | Mammary Neoplasms, Experimental - pathology | Female | Disease Models, Animal | Mammary Neoplasms, Experimental - drug therapy | Lung Neoplasms - genetics | Reproducibility of Results | Disease Progression | Antineoplastic Agents, Alkylating - therapeutic use | Cell Lineage | Neoplasm Metastasis - genetics | Drug Resistance, Neoplasm - genetics | Animals | Neoplasm Metastasis - pathology | Cyclophosphamide - pharmacology | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Drug Resistance, Neoplasm - drug effects | Development and progression | Metastasis | Drug resistance | Cell differentiation | Health aspects | Lung cancer | Chemotherapy | Analysis | Stem cells | Cancer | Studies | Genotype & phenotype | Transgenic animals | Rodents | Morphology | Fibroblasts | Breast cancer | Cancer therapies | Tumors | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2013, Volume 110, Issue 24, pp. 9920 - 9925
The ten-eleven translocation (TET) family of methylcytosine dioxygenases initiates demethylation of DNA and is associated with tumorigenesis in many cancers;... 
Genes | DNA | Cell lines | Breast cancer | Breast neoplasms | Gene expression regulation | Promoter regions | Metastasis | Embryonic stem cells | Tumors | MAMMALIAN DNA | 5-METHYLCYTOSINE | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | MOUSE | TISSUE | GENE-EXPRESSION | 5-HYDROXYMETHYLCYTOSINE | ACTIVE DNA DEMETHYLATION | TET PROTEINS | MLL | Wnt1 Protein - genetics | Prognosis | Oligonucleotide Array Sequence Analysis | Homeodomain Proteins - metabolism | Humans | Immunoblotting | Male | Transplantation, Heterologous | Gene Expression Profiling | Wnt1 Protein - metabolism | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Mixed Function Oxygenases | Neoplasm Metastasis | HMGA2 Protein - metabolism | RNA Interference | HMGA2 Protein - genetics | Mammary Neoplasms, Experimental - pathology | Female | Proto-Oncogene Proteins - metabolism | Mice, Inbred C57BL | Kaplan-Meier Estimate | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Signal Transduction - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Homeodomain Proteins - genetics | Mice, Knockout | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | Genetic aspects | Research | Translocation (Genetics) | Genetic regulation | Genetic engineering | Rodents | Binding sites | Index Medicus | Biological Sciences
Journal Article
Cell, ISSN 0092-8674, 10/2013, Volume 155, Issue 2, pp. 397 - 409
Journal Article
BBA - Molecular Cell Research, ISSN 0167-4889, 07/2014, Volume 1843, Issue 7, pp. 1414 - 1426
The oncofetal H19 gene transcribes a long non-coding RNA(lncRNA) that is essential for tumor growth. Here we found that numerous established inducers of... 
Epithelial to mesenchymal transition | H19 | miR-675 | Positive loop | Slug | E-cadherin | MiR-675 | ACTIVATION | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | EMT | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | NONCODING RNA | CROSS-TALK | GENE | RESISTANCE | Oxygen - pharmacology | Cadherins - metabolism | Drug Resistance, Multiple | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Epithelial-Mesenchymal Transition - genetics | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Oxygen - metabolism | Proto-Oncogene Proteins c-akt - genetics | Breast Neoplasms - metabolism | Cell Hypoxia | Neoplasm Metastasis | Mammary Neoplasms, Experimental - pathology | Female | Cadherins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Snail Family Transcription Factors | Signal Transduction | Mammary Glands, Animal - pathology | RNA, Long Noncoding - genetics | Transcription Factors - genetics | Phosphatidylinositol 3-Kinases - genetics | Transcription Factors - metabolism | Transforming Growth Factor beta - pharmacology | Drug Resistance, Neoplasm - genetics | Feedback, Physiological | Animals | Breast Neoplasms - genetics | Mammary Glands, Animal - metabolism | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | MicroRNAs - genetics | RNA, Long Noncoding - metabolism | Development and progression | Metastasis | RNA | Transforming growth factors | Analysis
Journal Article
Journal Article
Journal Article
Science, ISSN 0036-8075, 1/2012, Volume 335, Issue 6066, pp. 348 - 353
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2013, Volume 110, Issue 27, pp. 11091 - 11096
Using gene-expression data from over 6,000 breast cancer patients, we report herein that high CD73 expression is associated with a poor prognosis in... 
T lymphocytes | Medical research | Up regulation | Anthracyclines | Chemotherapy | Purinergic P1 receptors | Prognosis | Breast cancer | Gene expression | Tumors | Immunotherapy | Ectonucleotidase | Immunogenic cell death | ECTO-5'-NUCLEOTIDASE | HETEROGENEITY | CELLS | THERAPY | MULTIDISCIPLINARY SCIENCES | immunogenic cell death | immunotherapy | ADENOSINE | TUMOR-GROWTH | ectonucleotidase | ESTROGEN-RECEPTOR | CHEMOTHERAPY | 5'-Nucleotidase - antagonists & inhibitors | Mammary Neoplasms, Experimental - immunology | 5'-Nucleotidase - genetics | Doxorubicin - therapeutic use | Breast Neoplasms - immunology | Humans | GPI-Linked Proteins - biosynthesis | Antibodies, Monoclonal - therapeutic use | Antineoplastic Agents - therapeutic use | Mammary Neoplasms, Experimental - genetics | Adaptive Immunity - genetics | Breast Neoplasms - therapy | Female | GPI-Linked Proteins - antagonists & inhibitors | 5'-Nucleotidase - biosynthesis | Drug Resistance, Neoplasm - immunology | Mice, SCID | Mammary Neoplasms, Experimental - therapy | Mice, Knockout | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Cell Line, Tumor | Mice | Mice, Inbred BALB C | Anthracyclines - therapeutic use | GPI-Linked Proteins - genetics | Physiological aspects | Dosage and administration | Diagnosis | Research | Cancer | Rodents | Cells | Index Medicus | Biological Sciences
Journal Article
Cancer Discovery, ISSN 2159-8274, 2013, Volume 3, Issue 2, pp. 224 - 237
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 507, Issue 7493, pp. 508 - 512
Tumour metastasis is the primary cause of mortality in cancer patients and remains the key challenge for cancer therapy(1). New therapeutic approaches to block... 
ACTIVATION | MOUSE NK CELLS | WARFARIN | AXL | IMMUNOTHERAPY | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GROWTH | TYROSINE KINASES | IDENTIFICATION | CELLULAR TARGETS | Mammary Neoplasms, Experimental - immunology | Proto-Oncogene Proteins c-cbl - metabolism | Melanoma, Experimental - drug therapy | Male | Mammary Neoplasms, Experimental - genetics | Warfarin - pharmacology | Warfarin - therapeutic use | Ubiquitination | Melanoma, Experimental - immunology | Neoplasm Metastasis - drug therapy | Neoplasm Metastasis - immunology | Neoplasm Metastasis - prevention & control | c-Mer Tyrosine Kinase | Mammary Neoplasms, Experimental - pathology | Killer Cells, Natural - immunology | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Female | Proto-Oncogene Proteins c-cbl - deficiency | Mammary Neoplasms, Experimental - drug therapy | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins c-cbl - genetics | Proto-Oncogene Proteins - antagonists & inhibitors | Mice, Inbred C57BL | Ubiquitin-Protein Ligases - metabolism | Melanoma, Experimental - pathology | Anticoagulants - therapeutic use | Receptor Protein-Tyrosine Kinases - metabolism | Animals | Melanoma, Experimental - genetics | Adaptor Proteins, Signal Transducing - deficiency | Anticoagulants - pharmacology | Adaptor Proteins, Signal Transducing - genetics | Ubiquitin-Protein Ligases - deficiency | Killer Cells, Natural - drug effects | Mice | Mice, Inbred BALB C | Killer Cells, Natural - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Ubiquitin-Protein Ligases - genetics | Cell receptors | Killer cells | Ligases | Physiological aspects | Development and progression | Genetic aspects | Metastasis | Research | Proteins | Lungs | Rodents | Melanoma | Cytotoxicity | Ligands | Kinases | Immune system | Tumors | Cancer | Index Medicus
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 2014, Volume 10, Issue 7, pp. 558 - 566
PTP1B, a validated therapeutic target for diabetes and obesity, has a critical positive role in HER2 signaling in breast tumorigenesis. Efforts to develop... 
LAPATINIB | OVEREXPRESSION | TYROSINE-PHOSPHATASE 1B | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACQUIRED-RESISTANCE | BINDING-SITE | MECHANISMS | INDUCTION | HUMAN BREAST-CANCER | CONTRIBUTES | TUMORS | Receptor, ErbB-2 - genetics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Molecular Targeted Therapy | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - enzymology | Protein Binding - drug effects | Mammary Neoplasms, Experimental - pathology | Female | Antineoplastic Agents - pharmacology | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - genetics | Mammary Neoplasms, Experimental - enzymology | Spermine - analogs & derivatives | Mammary Neoplasms, Experimental - drug therapy | Allosteric Regulation - drug effects | Protein Structure, Tertiary | Catalytic Domain | Protein Structure, Secondary | Signal Transduction | Allosteric Site - drug effects | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - antagonists & inhibitors | Spermine - chemistry | Models, Molecular | Antineoplastic Agents - chemistry | Breast Neoplasms - drug therapy | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - metabolism | Animals | Breast Neoplasms - genetics | Spermine - pharmacology | Breast Neoplasms - pathology | Cholestanes - chemistry | Cholestanes - pharmacology | Mice | Kinetics | Breast cancer | Tumorigenesis | Diabetes | Drug therapy | Index Medicus | Cholestanes | Allosteric Regulation | Breast Neoplasms | Life Sciences | Biomolecules | Biochemistry, Molecular Biology | Antineoplastic Agents | Spermine | Protein Tyrosine Phosphatase, Non-Receptor Type 1 | Mammary Neoplasms, Experimental | Allosteric Site | Protein Binding | Receptor, ErbB-2
Journal Article