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Journal Article
PLoS biology, ISSN 1544-9173, 2014, Volume 12, Issue 9, p. e1001945
Malignant carcinomas that recur following therapy are typically de-differentiated and multidrug resistant (MDR). De-differentiated cancer cells acquire MDR by... 
Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Phosphorylation | eIF-2 Kinase - metabolism | Humans | Gene Expression Regulation, Neoplastic | Neoplasm Recurrence, Local - drug therapy | Gene Expression Profiling | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Neoplasm Recurrence, Local - pathology | Neoplasm Grading | Mammary Neoplasms, Experimental - pathology | Female | NF-E2-Related Factor 2 - genetics | Transcription, Genetic | Antineoplastic Agents - pharmacology | Carcinoma - pathology | Mammary Neoplasms, Experimental - drug therapy | eIF-2 Kinase - genetics | Carcinoma - drug therapy | Neoplasm Recurrence, Local - metabolism | Oxidation-Reduction | Signal Transduction | Mammary Glands, Animal - pathology | Mammary Glands, Animal - drug effects | Mice, SCID | Breast Neoplasms - drug therapy | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Mammary Glands, Animal - metabolism | Breast Neoplasms - pathology | NF-E2-Related Factor 2 - metabolism | Cell Line, Tumor | Carcinoma - genetics | Mice, Inbred NOD | Neoplasm Recurrence, Local - genetics | Carcinoma - metabolism | Cell Dedifferentiation - genetics | Cell research | Oncology, Experimental | Cancer cells | Cellular signal transduction | Research | Drug resistance | Cell differentiation | Health aspects | Cancer | Medical research | Enzymes | Transcription factors | Biomedical research | Breast cancer | Kinases | Gene expression | Experiments | Cancer therapies | Antioxidants | Medical prognosis | Data collection | Tumors
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2015, Volume 125, Issue 2, pp. 621 - 635
The gene encoding F-box protein FBXW7 is frequently mutated in many human cancers. Although most previous studies have focused on the tumor-suppressive... 
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | F-Box-WD Repeat-Containing Protein 7 | Receptors, Notch - metabolism | Stromal Cells - pathology | Humans | Gene Expression Regulation, Neoplastic | Receptors, Notch - genetics | CD2 Antigens - genetics | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Breast Neoplasms - therapy | Neoplasm Metastasis | Mammary Neoplasms, Experimental - pathology | Cell Cycle Proteins - genetics | Female | CD2 Antigens - metabolism | Chemokine CCL2 - metabolism | Retrospective Studies | Neoplasm Proteins - genetics | Macrophages - pathology | Neoplasm Proteins - biosynthesis | Stromal Cells - metabolism | F-Box Proteins - biosynthesis | Chemokine CCL2 - genetics | Mice, Transgenic | Cell Cycle Proteins - biosynthesis | Ubiquitin-Protein Ligases - biosynthesis | Mammary Neoplasms, Experimental - therapy | Macrophages - metabolism | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | Ubiquitin-Protein Ligases - genetics | F-Box Proteins - genetics | Development and progression | Genetic aspects | Metastasis | Carrier proteins | Health aspects | Cellular control mechanisms | Proteins | Statistical analysis | Genes | Rodents | Melanoma | Breast cancer | Tumorigenesis | Mutation | Chemokines | Tumors | Recruitment
Journal Article
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 01/2008, Volume 118, Issue 1, pp. 64 - 78
overexpression of the receptor tyrosine kinase EPH receptor A2 (EphA2) is commonly observed in aggressive breast cancer and correlates with a poor prognosis.... 
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Adenocarcinoma - pathology | Cell Proliferation | Receptor, ErbB-2 - genetics | Humans | Receptor, EphA2 - metabolism | Receptor, ErbB-2 - metabolism | Male | Mammary Glands, Human - metabolism | Mammary Neoplasms, Experimental - metabolism | Cell Movement - genetics | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Breast Neoplasms - therapy | Neoplasm Metastasis | Adenocarcinoma - metabolism | MAP Kinase Signaling System - genetics | Cell Transformation, Neoplastic - genetics | Mammary Neoplasms, Experimental - pathology | Female | Adenocarcinoma - genetics | Antigens, Polyomavirus Transforming - genetics | Antigens, Polyomavirus Transforming - metabolism | Mammary Glands, Animal - pathology | rho GTP-Binding Proteins - genetics | Mammary Glands, Human - pathology | Mice, Transgenic | Cell Transformation, Neoplastic - metabolism | Mammary Neoplasms, Experimental - therapy | Animals | Breast Neoplasms - genetics | Adenocarcinoma - therapy | Mammary Glands, Animal - metabolism | Breast Neoplasms - pathology | rho GTP-Binding Proteins - metabolism | Receptor, EphA2 - genetics | Mice | Cell Transformation, Neoplastic - pathology | Adenocarcinoma | Polymerase chain reaction | Cell receptors | Genetic aspects | Research | Health aspects | Risk factors
Journal Article
Journal Article
Nature communications, ISSN 2041-1723, 10/2013, Volume 4, Issue 1, p. 2516
Cancer and stromal cells actively exert physical forces (solid stress) to compress tumour blood vessels, thus reducing vascular perfusion. Tumour interstitial... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 05/2013, Volume 110, Issue 24, pp. 9920 - 9925
Journal Article
Molecular cancer research, ISSN 1541-7786, 11/2013, Volume 11, Issue 11, pp. 1412 - 1424
Journal Article