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Journal of experimental medicine, ISSN 0022-1007, 2008, Volume 205, Issue 7, pp. 1621 - 1634
Although dendritic cells (DCs) play an important role in mediating protection against influenza virus, the precise role of lung DC subsets, such as CD11b(-)... 
MEDICINE, RESEARCH & EXPERIMENTAL | IIFN-PRODUCING CELLS | INHALED ANTIGEN | IN-VIVO DEPLETION | ANTIGEN PRESENTATION | STEADY-STATE | IMMUNOLOGY | PULMONARY INFECTION | ANTIBODY-RESPONSE | CD8(+) T-CELLS | RESPIRATORY SYNCYTIAL VIRUS | LANGERHANS CELLS | Dendritic Cells - immunology | Lectins, C-Type - immunology | Male | Lectins, C-Type - genetics | CD11c Antigen - immunology | Trachea - virology | Orthomyxoviridae - immunology | Orthomyxoviridae Infections - genetics | CD4-Positive T-Lymphocytes - immunology | Cell Movement - immunology | CD11b Antigen - genetics | Lung - virology | CD11c Antigen - genetics | Female | CD4-Positive T-Lymphocytes - virology | Dendritic Cells - virology | CD11b Antigen - immunology | Plasma Cells - virology | Antigens, Surface - genetics | Intercellular Signaling Peptides and Proteins - genetics | Antigen Presentation - genetics | Mice, Transgenic | Lymph Nodes - virology | Lymph Nodes - immunology | CD8 Antigens - immunology | Trachea - immunology | Heparin-binding EGF-like Growth Factor | Animals | Mannose-Binding Lectins - genetics | Mannose-Binding Lectins - immunology | CD8-Positive T-Lymphocytes - virology | CD8 Antigens - genetics | Dogs | Antibodies, Viral - immunology | Nucleocapsid Proteins - immunology | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | Antigens, Surface - immunology | Antibody Formation - genetics | Intercellular Signaling Peptides and Proteins - immunology | Lung - immunology | Orthomyxoviridae Infections - immunology | Plasma Cells - immunology
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 140, Issue 1, pp. 221 - 230.e3
Background & Aims Anti–tumor necrosis factor (TNF)α antibodies are effective in treating patients with Crohn's disease whereas soluble TNFα receptors have not... 
Gastroenterology and Hepatology | Autoimmunity | IBD | Inflammatory Response | Type 2 Macrophage | MONOCLONAL-ANTIBODY | POLYMORPHISM | ACTIVE CROHNS-DISEASE | INFLAMMATORY-BOWEL-DISEASE | ACCENT-I | DOUBLE-BLIND | PLACEBO-CONTROLLED TRIAL | TNF-ALPHA | INFLIXIMAB | GASTROENTEROLOGY & HEPATOLOGY | T-LYMPHOCYTES | Cell Proliferation | Certolizumab Pegol | Humans | Lectins, C-Type - immunology | Anti-Inflammatory Agents - immunology | Caspase 3 - immunology | Antibodies, Monoclonal, Humanized | Lymphocyte Activation - immunology | Immunoglobulin Fc Fragments - immunology | Etanercept | Infliximab | Immunoglobulin G - immunology | T-Lymphocytes - drug effects | Leukocytes, Mononuclear - immunology | Tumor Necrosis Factor-alpha - immunology | Immunoglobulin G - pharmacology | Immunosuppressive Agents - pharmacology | Antibodies, Monoclonal - immunology | Macrophages - immunology | Cytokines - immunology | Polyethylene Glycols - pharmacology | Leukocytes, Mononuclear - drug effects | Anti-Inflammatory Agents - pharmacology | Cells, Cultured | Cytokines - secretion | Crohn Disease - immunology | Receptors, Cell Surface - immunology | Immunoglobulin Fab Fragments - pharmacology | Mannose-Binding Lectins - immunology | Receptors, Fc - immunology | Immunosuppressive Agents - immunology | Lymphocyte Activation - drug effects | Caspase 3 - analysis | T-Lymphocytes - immunology | Immunoglobulin Fab Fragments - immunology | Adalimumab | Receptors, Tumor Necrosis Factor - immunology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Apoptosis
Journal Article
Nature immunology, ISSN 1529-2908, 2009, Volume 10, Issue 10, pp. 1081 - U58
Journal Article
Blood, ISSN 0006-4971, 07/2017, Volume 130, Issue 2, pp. 167 - 175
Langerhans cell histiocytosis (LCH) and Erdheim-Chester disease (ECD) are rare histiocytic disorders induced by somatic mutation of MAPK pathway genes.... 
MANAGEMENT | DENDRITIC CELLS | NEOPLASMS | DISTINCT | MONOCYTES | SUBSETS | NOTCH | MUTATIONS | MATURATION | HEMATOLOGY | BLOOD | Antigens, CD - immunology | Dendritic Cells - immunology | Histiocytosis, Langerhans-Cell - diagnosis | Histiocytosis, Langerhans-Cell - genetics | Humans | Lectins, C-Type - immunology | Dendritic Cells - pathology | Hematopoietic Stem Cells - pathology | Erdheim-Chester Disease - immunology | Male | Receptors, Notch - genetics | Monocytes - immunology | Lectins, C-Type - genetics | Antigens, CD - genetics | Hematopoietic Stem Cells - immunology | Histiocytosis, Langerhans-Cell - pathology | Bone Marrow Cells - immunology | Monocytes - pathology | Adult | Female | Cell Differentiation | Foam Cells - pathology | Glycoproteins - genetics | Transforming Growth Factor beta - immunology | Antigens, CD1 - immunology | Diagnosis, Differential | Erdheim-Chester Disease - genetics | Gene Expression | Bone Marrow Cells - pathology | Immunophenotyping | Lipopolysaccharide Receptors - immunology | Histiocytosis, Langerhans-Cell - immunology | Erdheim-Chester Disease - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Glycoproteins - immunology | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Mannose-Binding Lectins - genetics | Mannose-Binding Lectins - immunology | Transforming Growth Factor beta - genetics | Proto-Oncogene Proteins B-raf - genetics | Proto-Oncogene Proteins B-raf - immunology | Alleles | Erdheim-Chester Disease - diagnosis | Lipopolysaccharide Receptors - genetics | Foam Cells - immunology | Antigens, CD1 - genetics | Mutation | Receptors, Notch - immunology | Myeloid Neoplasia
Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2012, Volume 189, Issue 8, pp. 3957 - 3969
The lectin pathway of complement is an important component of innate immunity. Its activation has been thought to occur via recognition of pathogens by... 
SYSTEM | INDIVIDUALS | PATTERN-RECOGNITION MOLECULES | COMPLEMENT FACTOR-D | MBL | 3MC SYNDROME | STOICHIOMETRY | MUTATIONS | IMMUNOLOGY | IDENTIFICATION | DEFICIENCY | Craniofacial Abnormalities - immunology | Transcriptional Activation - genetics | Complement Pathway, Mannose-Binding Lectin - immunology | Blepharoptosis - genetics | Eye Abnormalities - enzymology | Mannose-Binding Protein-Associated Serine Proteases - physiology | Eye Abnormalities - immunology | Humans | Heart Defects, Congenital - immunology | Craniosynostoses - enzymology | Blepharoptosis - immunology | Developmental Disabilities - genetics | Transcriptional Activation - immunology | Strabismus - genetics | Abdominal Muscles - abnormalities | Abnormalities, Multiple - immunology | Heart Defects, Congenital - genetics | Developmental Disabilities - enzymology | Blepharoptosis - enzymology | Heart Defects, Congenital - enzymology | Mannose-Binding Protein-Associated Serine Proteases - genetics | Complement Pathway, Alternative - genetics | Abnormalities, Multiple - genetics | Craniofacial Abnormalities - genetics | Strabismus - enzymology | Abdominal Muscles - immunology | Craniosynostoses - genetics | Complement Pathway, Mannose-Binding Lectin - genetics | Cryptorchidism - enzymology | Abdominal Muscles - enzymology | Hip Dislocation, Congenital - genetics | Codon, Nonsense | Eye Abnormalities - genetics | Animals | Craniofacial Abnormalities - enzymology | Craniosynostoses - immunology | Cryptorchidism - immunology | Hip Dislocation, Congenital - immunology | Complement Pathway, Alternative - immunology | Strabismus - immunology | Abnormalities, Multiple - enzymology | Developmental Disabilities - immunology | Hip Dislocation, Congenital - enzymology | Cryptorchidism - genetics
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2013, Volume 288, Issue 17, pp. 11761 - 11770
Nonalcoholic fatty liver disease (NAFLD) affects a large proportion of the American population. The spectrum of disease ranges from bland steatosis without... 
TGR5 | DENDRITIC CELLS | FARNESOID X RECEPTOR | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | KUPFFER CELLS | STEATOHEPATITIS | MICE | SCORING SYSTEM | ALTERNATIVE ACTIVATION | INDUCED INSULIN-RESISTANCE | Liver - pathology | Receptors, G-Protein-Coupled - metabolism | Fatty Liver - pathology | Mannose-Binding Lectins - biosynthesis | Humans | Lectins, C-Type - immunology | Intercellular Signaling Peptides and Proteins - biosynthesis | Male | Monocytes - metabolism | Lectins, C-Type - biosynthesis | Monocytes - immunology | Receptors, G-Protein-Coupled - immunology | Liver - immunology | Receptors, Cytoplasmic and Nuclear - immunology | Monocytes - pathology | Non-alcoholic Fatty Liver Disease | Receptors, Cell Surface - biosynthesis | Cyclic AMP - metabolism | Macrophages - immunology | Fatty Liver - metabolism | Gene Expression Regulation - immunology | Macrophage Activation - immunology | Liver - metabolism | Receptors, Cell Surface - immunology | Macrophages - metabolism | Animals | Mannose-Binding Lectins - immunology | Cyclic AMP - immunology | Fatty Liver - immunology | Interleukin-10 - biosynthesis | Mice, Obese | Mice | Interleukin-10 - immunology | Intercellular Signaling Peptides and Proteins - immunology | Receptors, Cytoplasmic and Nuclear - metabolism | FXR | Monocytes | Immunology | Mouse | Liver | Macrophages | IL-10 | Steatosis
Journal Article
Immunity, ISSN 1074-7613, 09/2014, Volume 41, Issue 3, pp. 402 - 413
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 01/2010, Volume 207, Issue 1, pp. 189 - 206
Recent studies have challenged the view that Langerhans cells (LCs) constitute the exclusive antigen-presenting cells of the skin and suggest that the dermal... 
MEDICINE, RESEARCH & EXPERIMENTAL | RESIDENT DCS | MARGINAL ZONE | IN-VIVO | LYMPH-NODES | STEADY-STATE | BIRBECK GRANULES | MICE | SELF-ANTIGENS | SKIN | IMMUNOLOGY | T-CELLS | Antigens, CD - immunology | Lectins, C-Type - immunology | Lectins, C-Type - genetics | Antigens, CD - genetics | Integrin alpha Chains - immunology | Langerhans Cells - immunology | Female | Dermis - cytology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Antigens, Surface - genetics | Gene Expression Regulation - physiology | Mice, Transgenic | Keratinocytes - cytology | Lymph Nodes - immunology | Lymph Nodes - cytology | Organ Specificity | Dermis - immunology | Keratinocytes - immunology | Gene Knock-In Techniques | Animals | Histocompatibility Antigens Class II - immunology | Mannose-Binding Lectins - genetics | Mannose-Binding Lectins - immunology | Antigens - immunology | Mice | Histocompatibility Antigens Class II - genetics | Antigens, Surface - immunology | Integrin alpha Chains - genetics | Antigen Presentation - physiology | Langerhans Cells - cytology | Antigens - genetics | Antigens | Mannose-Binding Lectins | Langerhans Cells | Lectins, C-Type | Gene Expression Regulation | Keratinocytes | Dermis | Lymph Nodes | Life Sciences | Immunology | Antigen Presentation | Histocompatibility Antigens Class II | Antigens, Surface | Integrin alpha Chains | Antigens, CD
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e87131
The possibility to modulate ex vivo human NK cell differentiation towards specific phenotypes will contribute to a better understanding of NK cell... 
NATURAL-KILLER-CELLS | L-SELECTIN | DISPLAY | MANNOSE RECEPTOR | IMMUNOTHERAPY | CD56(BRIGHT) | MULTIDISCIPLINARY SCIENCES | BIOLOGY | GENE-EXPRESSION | TRANSCRIPTION FACTOR | CYTOTOXICITY | Receptors, CCR6 - immunology | Humans | Neoplasm Proteins - immunology | Antibody-Dependent Cell Cytotoxicity - immunology | Inhibitor of Differentiation Proteins - genetics | Receptors, IgG - metabolism | L-Selectin - immunology | Antigens, CD34 - immunology | Receptors, KIR - metabolism | Neoplasm Proteins - genetics | Fetal Blood - immunology | Immunotherapy, Adoptive - methods | Receptors, CXCR3 - metabolism | Antineoplastic Agents - immunology | Rituximab | Fetal Blood - cytology | Receptors, IgG - immunology | Reverse Transcriptase Polymerase Chain Reaction | Cell Differentiation - immunology | Inhibitor of Differentiation Proteins - immunology | K562 Cells | Receptors, CXCR3 - immunology | Cell Line, Tumor | Interleukin-2 - pharmacology | Killer Cells, Natural - drug effects | Killer Cells, Natural - metabolism | Antibodies, Monoclonal, Murine-Derived - pharmacology | Receptors, KIR - immunology | GATA3 Transcription Factor - genetics | Antigens, CD34 - metabolism | GATA3 Transcription Factor - immunology | L-Selectin - metabolism | Dose-Response Relationship, Drug | High Mobility Group Proteins - immunology | Cell Differentiation - genetics | Hematopoietic Stem Cells - immunology | Flow Cytometry | Interleukin-2 - immunology | Receptors, CCR6 - metabolism | Killer Cells, Natural - immunology | Antineoplastic Agents - pharmacology | Hematopoietic Stem Cells - drug effects | Antibodies, Monoclonal, Murine-Derived - immunology | Cells, Cultured | Hematopoietic Stem Cells - metabolism | Fetal Blood - metabolism | Cell Differentiation - drug effects | Antibody-Dependent Cell Cytotoxicity - drug effects | High Mobility Group Proteins - genetics | Fc receptors | Killer cells | Immunotherapy | Leukemia | Stem cells | Cell differentiation | Health aspects | Cytolytic activity | Lymphocyte receptors | Transcription factors | Toxicity | Differentiation (biology) | Cytotoxicity | Biology | Blood | NKG2 antigen | Receptors | Cord blood | Peripheral blood | Killer cell immunoglobulin-like receptors | Physiology | Natural killer cells | CD34 antigen | Cytokines | Maturation | Gynecology | CD62L protein | Interleukin 12 | T cell receptors | Pharmacology | CXCR3 protein | Gene expression | Obstetrics | Thrombosis | CCR6 protein | CD16 antigen | Major histocompatibility complex | Lymphocytes B | GATA-3 protein | Ligands | Umbilical cord | Chemokines | Cancer
Journal Article
Glia, ISSN 0894-1491, 2005, Volume 51, Issue 4, pp. 297 - 305
Perivascular macrophages (PVM) constitute a subpopulation of resident macrophages in the central nervous system (CNS) that by virtue of their strategic... 
perivascular macrophages | antigen presentation and recognition | DC‐SIGN | multiple sclerosis | mannose receptor | costimulatory molecules | Antigen presentation and recognition | Mannose receptor | Multiple sclerosis | Perivascular macrophages | Costimulatory molecules | DC-SIGN | MULTIPLE-SCLEROSIS LESIONS | HUMAN BRAIN | DENDRITIC CELLS | MICROGLIAL CELLS | NEUROSCIENCES | SCAVENGER RECEPTOR | CENTRAL-NERVOUS-SYSTEM | MENINGEAL MACROPHAGES | RAT-BRAIN | IN-SITU | Mannose-Binding Lectins - metabolism | Antigens, CD - immunology | Microglia - metabolism | Central Nervous System - metabolism | Blood-Brain Barrier - cytology | Humans | Middle Aged | Lectins, C-Type - immunology | Male | Cell Adhesion Molecules - immunology | Central Nervous System - immunology | Lectins, C-Type - metabolism | Lymphocyte Activation - immunology | Encephalitis - physiopathology | Microglia - immunology | Encephalitis - metabolism | Inflammation Mediators - metabolism | Female | Histocompatibility Antigens Class II - metabolism | Microcirculation - immunology | Antigens, Differentiation, Myelomonocytic - immunology | Macrophages - immunology | Cerebral Arteries - cytology | Inflammation Mediators - immunology | Antigen-Presenting Cells - metabolism | Blood-Brain Barrier - immunology | Cerebral Arteries - immunology | Receptors, Cell Surface - metabolism | Encephalitis - immunology | Antigen Presentation - immunology | Antigen-Presenting Cells - immunology | Cell Adhesion Molecules - metabolism | Receptors, Cell Surface - immunology | Microcirculation - cytology | Macrophages - metabolism | Histocompatibility Antigens Class II - immunology | Mannose-Binding Lectins - immunology | T-Lymphocytes - immunology | Aged | Central Nervous System - physiopathology
Journal Article
PLoS Pathogens, ISSN 1553-7366, 01/2010, Volume 6, Issue 1, p. e1000714
Journal Article