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FEBS Letters, ISSN 0014-5793, 02/2016, Volume 590, Issue 3, pp. 396 - 407
Journal Article
Cell Reports, ISSN 2211-1247, 02/2016, Volume 14, Issue 7, pp. 1774 - 1786
Special AT-rich sequence-binding protein 1 (Satb1) governs genome-wide transcriptional programs. Using a conditional knockout mouse, we find that Satb1 is... 
CHROMATIN | IN-VIVO | GENES | SIGNALS | OVARIAN-CANCER | ANTITUMOR IMMUNITY | DIFFERENTIATION | EXPRESSION | LINEAGE | PROGRESSION | CELL BIOLOGY | Neoplasm Transplantation | RNA, Small Interfering - genetics | Cell Proliferation | Galectin 1 - immunology | Dendritic Cells - immunology | Humans | Matrix Attachment Region Binding Proteins - antagonists & inhibitors | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Dendritic Cells - pathology | Galectin 1 - genetics | Ovarian Neoplasms - genetics | RNA, Small Interfering - immunology | Female | Cell Differentiation | Transcription Factors - immunology | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | Promoter Regions, Genetic | Histones - immunology | Interleukin-6 - genetics | Signal Transduction | Immune Tolerance | Receptor, Notch1 - immunology | Transcription Factors - genetics | Matrix Attachment Region Binding Proteins - immunology | Mice, Knockout | Animals | Histocompatibility Antigens Class II - immunology | Histones - genetics | Cell Transformation, Neoplastic | Immunoglobulin J Recombination Signal Sequence-Binding Protein - immunology | Matrix Attachment Region Binding Proteins - genetics | Interleukin-6 - immunology | Protein Binding | Mice | Histocompatibility Antigens Class II - genetics | Receptor, Notch1 - genetics | Ovarian Neoplasms - immunology
Journal Article
PLoS Genetics, ISSN 1553-7390, 09/2013, Volume 9, Issue 9, p. e1003750
Functional characterization of causal variants present on risk haplotypes identified through genome-wide association studies (GWAS) is a primary objective of... 
RHEUMATOID-ARTHRITIS | GENE | ENZYME A20 | CELL LYMPHOMA | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | FUNCTIONAL VARIANT | RISK | CHROMOSOME CONFORMATION CAPTURE | NF-KAPPA-B | GENOME-WIDE ASSOCIATION | Haplotypes | Promoter Regions, Genetic | Genetic Predisposition to Disease | Signal Transduction | Matrix Attachment Region Binding Proteins - metabolism | Humans | Matrix Attachment Region Binding Proteins - antagonists & inhibitors | Gene Expression Regulation | Nuclear Proteins - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Lupus Erythematosus, Systemic - etiology | DNA-Binding Proteins - genetics | DNA-Binding Proteins - metabolism | Enhancer Elements, Genetic | NF-kappa B - genetics | Lupus Erythematosus, Systemic - genetics | Matrix Attachment Region Binding Proteins - genetics | Alleles | HEK293 Cells | Tumor Necrosis Factor alpha-Induced Protein 3 | Polymorphism, Single Nucleotide | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Lupus Erythematosus, Systemic - pathology | Systemic lupus erythematosus | Promoters (Genetics) | Genetic variation | Physiological aspects | Genetic aspects | Research | Gene expression | Lupus | Enzymes | Disease | Editing | Homeostasis | Genomes | Autoimmune diseases | Kinases | Experiments | Mass spectrometry | Binding sites
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2014, Volume 135, Issue 11, pp. 2537 - 2546
Journal Article
FEBS Letters, ISSN 0014-5793, 05/2015, Volume 589, Issue 12, pp. 1359 - 1368
Liver fibrosis is a worldwide clinical issue. Activation of hepatic stellate cells (HSCs) is the central event during liver fibrosis. We investigated the role... 
Liver fibrosis | Hepatic stellate cell | SATB1 | Ras/Raf-1/MEK/ERK/Ets-1 | collagen type I alpha 1 | hematoxylin–eosin staining | CTGF | collagen type III alpha 1 | α-SMA | multiplicity of infection | standard error of measurement | smooth muscle α-actin | MARs | FBS | matrix attachment regions | epithelial–mesenchymal transition | MOI | H&E | special AT-rich binding protein-1 | EMT | ECM | thioacetamide | connective tissue growth factor | COL1A1 | fetal bovine serum | COL3A1 | TAA | Hepatic stellate cells | HSCs | SEM | extracellular matrix | PROMOTER ACTIVITY | TGF-BETA | PROTEIN | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | CELL BIOLOGY | TISSUE GROWTH-FACTOR | MULTIPLE GENES | BIOPHYSICS | TUMOR-GROWTH | EXPRESSION | BINDING | Cell Proliferation | Homeodomain Proteins - metabolism | Humans | Matrix Attachment Region Binding Proteins - antagonists & inhibitors | Actins - metabolism | Hepatic Stellate Cells - metabolism | Male | Actins - genetics | MAP Kinase Signaling System | Liver Cirrhosis - metabolism | Collagen - biosynthesis | Hepatic Stellate Cells - pathology | Disease Models, Animal | Recombinant Proteins - metabolism | Cell Line | Matrix Attachment Region Binding Proteins - metabolism | Connective Tissue Growth Factor - antagonists & inhibitors | Cells, Cultured | Gene Expression Regulation | Gene Silencing | Recombinant Proteins - chemistry | Random Allocation | Rats, Sprague-Dawley | Homeodomain Proteins - genetics | Animals | Homeodomain Proteins - antagonists & inhibitors | Matrix Attachment Region Binding Proteins - genetics | Liver Cirrhosis - pathology | Connective Tissue Growth Factor - genetics | Connective Tissue Growth Factor - metabolism | Cell Movement | Liver | Liver diseases | Fibrosis
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53527
Disease aggressiveness remains a critical factor to the progression of prostate cancer. Transformation of epithelial cells to mesenchymal lineage, associated... 
EPITHELIAL-MESENCHYMAL TRANSITION | OVEREXPRESSION | CARCINOMA CELLS | NUCLEAR-MATRIX | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | RICH | E-CADHERIN | DIFFERENTIATION | DNA-BINDING PROTEIN | REGION | Prostatic Neoplasms - metabolism | RNA, Small Interfering - genetics | Up-Regulation | Adenocarcinoma - pathology | Humans | Matrix Attachment Region Binding Proteins - antagonists & inhibitors | Gene Expression Regulation, Neoplastic | Male | Epithelial-Mesenchymal Transition - genetics | Adenocarcinoma - metabolism | Prostatic Neoplasms - genetics | Biomarkers, Tumor - metabolism | Adenocarcinoma - genetics | Biomarkers, Tumor - antagonists & inhibitors | Cadherins - genetics | Prostatic Neoplasms - pathology | Signal Transduction | Matrix Attachment Region Binding Proteins - metabolism | Neoplasm Invasiveness | Gene Knockout Techniques | Disease Progression | Xenograft Model Antitumor Assays | Animals | Cell Nucleus - genetics | Biopsy | Mice, Nude | Matrix Attachment Region Binding Proteins - genetics | Cell Line, Tumor | Biomarkers, Tumor - genetics | Mice | Cadherins - deficiency | Cell Movement | Analysis | Stem cells | Development and progression | Nucleotide sequencing | Cell differentiation | Prostate cancer | Papillomavirus infections | Protein binding | DNA sequencing | Transformation | Mesenchyme | Epithelial cells | Metastasis | Tissues | Nuclei | Stomach cancer | E-cadherin | Urology | Cell adhesion & migration | Gene sequencing | Proteins | Human papillomavirus | Transfection | Localization | Deoxyribonucleic acid--DNA | Aggressive behavior | Nucleotide sequence | Invasiveness | Benign | Breast cancer | Gene expression | Medical prognosis | Cell lines | Prostate | Tumors | Cancer | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Blood, ISSN 0006-4971, 09/2010, Volume 116, Issue 9, pp. 1443 - 1453
Journal Article
Stem Cell Research, ISSN 1873-5061, 03/2017, Volume 19, Issue C, pp. 139 - 147
Breast tumors are heterogeneous and carry a small population of progenitor cells that can produce various subtypes of breast cancer. SATB2 (special AT-rich... 
TRANSCRIPTION FACTORS | SELF-RENEWAL | PLURIPOTENCY | HUMAN BREAST-CANCER | BETA-CATENIN | CANCER STEM-CELLS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | CARCINOGENESIS | CADMIUM | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | DIFFERENTIATION | EXPRESSION | Cell Proliferation | Epithelial Cells - metabolism | Humans | Matrix Attachment Region Binding Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-bcl-2 - metabolism | Chromatin Immunoprecipitation | RNA Interference | Epithelial-Mesenchymal Transition | Female | Lentivirus - genetics | Epithelial Cells - cytology | Cell Line | Mammary Glands, Human - cytology | Promoter Regions, Genetic | Matrix Attachment Region Binding Proteins - metabolism | Genetic Vectors - metabolism | Transcription Factors - antagonists & inhibitors | Transcription Factors - genetics | Genetic Vectors - genetics | X-Linked Inhibitor of Apoptosis Protein - genetics | Transcription Factors - metabolism | Matrix Attachment Region Binding Proteins - genetics | X-Linked Inhibitor of Apoptosis Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Cell Movement | RNA, Small Interfering - metabolism | Care and treatment | Stem cells | Breast cancer | Transplantation | Research | Health aspects | Risk factors | Epithelial cells | Oncology, Experimental | Physiological aspects | DNA binding proteins | Cancer
Journal Article