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Proceedings of the National Academy of Sciences, ISSN 0027-8424, 11/2012, Volume 109, Issue 46, pp. E3186 - E3195
Although macrophages are widely recognized to have a profibrotic role in inflammation, we have used a highly tractable CCl 4 -induced model of reversible... 
Degradation | Kupffer cell | Proliferation | Collagen | Myofibroblast | Kupffer Cell | LUNG FIBROSIS | proliferation | myofibroblast | MULTIDISCIPLINARY SCIENCES | collagen | degradation | HEPATIC-FIBROSIS | RENAL FIBROSIS | IMMUNE-RESPONSES | APOPTOTIC CELLS | IN-VIVO | MONOCYTE SUBSETS | GENE-EXPRESSION | PULMONARY-FIBROSIS | T-CELLS | Liver Cirrhosis - immunology | Antigens, Ly - genetics | Matrix Metalloproteinase 9 - immunology | Matrix Metalloproteinase 12 - genetics | Insulin-Like Growth Factor I - genetics | Monocytes - immunology | MAP Kinase Signaling System - immunology | Liver Cirrhosis - chemically induced | Insulin-Like Growth Factor I - immunology | CD11b Antigen - genetics | MAP Kinase Signaling System - genetics | Matrix Metalloproteinase 9 - genetics | Monocytes - pathology | Liver Cirrhosis - genetics | Macrophages - immunology | CD11b Antigen - immunology | Macrophages - pathology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Matrix Metalloproteinase 12 - immunology | Mice, Transgenic | Carbon Tetrachloride - toxicity | Carbon Tetrachloride Poisoning - pathology | Gene Expression Regulation - drug effects | Animals | MAP Kinase Signaling System - drug effects | Antigens, Ly - immunology | Carbon Tetrachloride Poisoning - immunology | Liver Cirrhosis - pathology | Carbon Tetrachloride Poisoning - genetics | Mice | Proteins | Genotype & phenotype | Signal transduction | Liver diseases | Gene expression | Rodents | Biological Sciences | PNAS Plus
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 05/2016, Volume 68, Issue 5, pp. 1111 - 1123
Journal Article
Hypertension, ISSN 0194-911X, 10/2014, Volume 64, Issue 4, pp. 875 - 882
To test the hypothesis that adoptive transfer of regulatory T cells (Tregs) may dose-dependently inhibit the formation of angiotensin II–induced abdominal... 
Regulatory | T-lymphocytes | Inflammation | Abdominal | Macrophages | Aortic aneurysm | APOPTOSIS | ACTIVATION | TYPE-2 RECEPTOR | aortic aneurysm, abdominal | ATHEROSCLEROSIS | ANTAGONISM | PROTECTS | macrophages | INHIBITION | inflammation | T-lymphocytes, regulatory | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | E-DEFICIENT MICE | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - immunology | Apolipoproteins E - deficiency | Humans | Middle Aged | Matrix Metalloproteinase 9 - immunology | NF-kappa B - immunology | Apoptosis - genetics | Male | NF-kappa B - metabolism | Adoptive Transfer | MAP Kinase Signaling System - immunology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 9 - genetics | Gene Expression - immunology | Inflammation Mediators - metabolism | Angiotensin II | Cytokines - genetics | Myocytes, Smooth Muscle - metabolism | Macrophages - immunology | Matrix Metalloproteinase 2 - immunology | Cytokines - immunology | Inflammation Mediators - immunology | Cytokines - metabolism | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Aortic Aneurysm, Abdominal - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Matrix Metalloproteinase 2 - genetics | Macrophages - metabolism | Animals | Apoptosis - immunology | Apolipoproteins E - genetics | Apolipoproteins E - immunology | Myocytes, Smooth Muscle - immunology | Mice | T-Lymphocytes, Regulatory - transplantation
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2010, Volume 184, Issue 7, pp. 3478 - 3486
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 07/2015, Volume 74, Issue 7, pp. 1417 - 1424
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 148, Issue 3, pp. 565 - 578.e4
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0137979
Background Tumor necrosis factor (TNF)-alpha and matrix metalloproteinases (MMPs) are elevated in pleural fluids of tuberculous pleuritis (TBP) where pleural... 
MATRIX METALLOPROTEINASES | ACTIVATION | FIBRINOLYTIC ENZYMES | EFFUSIONS | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTORS | TISSUE INHIBITORS | MATRIX-METALLOPROTEINASE-9 | PROINFLAMMATORY CYTOKINES | NF-KAPPA-B | GRANULOMA | Cell Line | Humans | Middle Aged | Matrix Metalloproteinase 9 - immunology | Mycobacterium tuberculosis - immunology | Epithelial Cells - pathology | Male | Tuberculosis, Pulmonary - pathology | MAP Kinase Signaling System - immunology | Matrix Metalloproteinase 1 - immunology | Pleura - immunology | Tuberculosis, Pulmonary - immunology | Up-Regulation - immunology | Epithelial Cells - immunology | Pleura - pathology | Tumor Necrosis Factor-alpha - immunology | Aged, 80 and over | Toll-Like Receptor 2 - immunology | Adult | Female | Aged | Phosphorylation | Mesothelium | Matrix metalloproteinases | Pathogenesis | mRNA | Infections | Matrix metalloproteinase | Kinases | Surgery | Pleurisy | Toll-like receptors | Fibroblasts | Tumor necrosis factor-TNF | Measurement methods | Heart diseases | Interstitial collagenase | Heart failure | Cytokines | Effusion | Internal medicine | Review boards | Fluid | Extracellular signal-regulated kinase | Pharmacology | TLR4 protein | Tumor necrosis factor-α | Gene expression | Patients | Gelatinase B | Medicine | Signaling | Tuberculosis | Human performance | TLR2 protein | Fibrosis | Pleural fluid | Neutralization
Journal Article