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American journal of respiratory cell and molecular biology, ISSN 1044-1549, 11/2015, Volume 53, Issue 5, pp. 585 - 600
Idiopathic pulmonary fibrosis (IPF) is a restrictive lung disease that is associated with high morbidity and mortality. Current medical therapies are not fully... 
lung | matrix metalloproteinase | fibrosis | idiopathic pulmonary fibrosis | interstitial lung disease | ACTIVATED PROTEIN-KINASE | ALVEOLAR EPITHELIAL-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCED LUNG INJURY | GELATINASE-A MMP-2 | COLLAGENASE-3 MMP-13 EXPRESSION | CELL BIOLOGY | RESPIRATORY SYSTEM | GENE-EXPRESSION | TISSUE INHIBITOR | EXTRACELLULAR-MATRIX | GROWTH-FACTOR | PERIPHERAL-BLOOD BIOMARKERS | Matrix Metalloproteinases, Secreted - metabolism | Idiopathic Pulmonary Fibrosis - genetics | Macrophages, Alveolar - pathology | Humans | Matrix Metalloproteinases, Secreted - antagonists & inhibitors | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Molecular Targeted Therapy | Lung - enzymology | Matrix Metalloproteinase Inhibitors - therapeutic use | Disease Models, Animal | Lung - pathology | Signal Transduction | Extracellular Matrix - drug effects | Gene Expression Regulation | Matrix Metalloproteinases, Secreted - classification | Macrophages, Alveolar - enzymology | Randomized Controlled Trials as Topic | Cell Movement - drug effects | Animals | Macrophages, Alveolar - drug effects | Lung - drug effects | Matrix Metalloproteinases, Secreted - genetics | Idiopathic Pulmonary Fibrosis - pathology | Idiopathic Pulmonary Fibrosis - enzymology | Mice | Extracellular Matrix - pathology | Idiopathic Pulmonary Fibrosis - drug therapy | Transcription factors | Pulmonary fibrosis | Pathogenesis | Rodents | Collagen | Mortality | MicroRNAs | DNA methylation | Epigenetics | Gene expression | Adapter proteins | Growth factors
Journal Article
Circulation Research, ISSN 0009-7330, 02/2013, Volume 112, Issue 4, pp. 675 - 688
RATIONALE:Matrix metalloproteinase (MMP)-28 regulates the inflammatory and extracellular matrix responses in cardiac aging, but the roles of MMP-28 after... 
fibroblast | MMP-28 | inflammation | macrophage phenotype | myocardial infarction | LYSYL OXIDASE | CARDIAC & CARDIOVASCULAR SYSTEMS | TGF-BETA | VENTRICULAR-FUNCTION | HEART-FAILURE | CORONARY MICROEMBOLIZATION | REPAIR | MATRIX-METALLOPROTEINASE-9 | PERIPHERAL VASCULAR DISEASE | EPILYSIN MMP-28 | TNF-ALPHA | TARGETED DELETION | HEMATOLOGY | Myocardial Infarction - blood | Cell Adhesion Molecules - genetics | Cicatrix - etiology | Extracellular Matrix Proteins - biosynthesis | Male | Matrix Metalloproteinases, Secreted - deficiency | Pulmonary Edema - etiology | Myocytes, Cardiac - enzymology | Myofibroblasts - metabolism | Ventricular Dysfunction, Left - enzymology | Ventricular Remodeling - genetics | Female | Myocardial Infarction - physiopathology | Transcription, Genetic | Cytokines - genetics | Heart Rupture - enzymology | Receptors, Cytokine - genetics | Matrix Metalloproteinase 9 - blood | Myocardial Infarction - enzymology | Macrophage Activation - physiology | Macrophages - classification | Cell Adhesion Molecules - biosynthesis | Receptors, Cytokine - biosynthesis | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Ventricular Dysfunction, Left - etiology | Inflammation | Ventricular Remodeling - physiology | Macrophages - enzymology | Mice, Knockout | Collagen - metabolism | Myocardial Infarction - complications | Heart Rupture - etiology | Animals | Pulmonary Edema - enzymology | Matrix Metalloproteinases, Secreted - genetics | Matrix Metalloproteinases, Secreted - physiology | Protein-Lysine 6-Oxidase - metabolism | Cicatrix - enzymology | Mice | Cytokines - biosynthesis
Journal Article
Journal Article
Medical Science Monitor, ISSN 1234-1010, 03/2017, Volume 23, pp. 1305 - 1311
Journal Article
Nature Genetics, ISSN 1061-4036, 11/2015, Volume 47, Issue 11, pp. 1260 - 1263
Journal Article
Matrix Biology, ISSN 0945-053X, 2011, Volume 30, Issue 2, pp. 145 - 153
Aggrecan degradation in articular cartilage occurs predominantly through proteolysis and has been attributed to the action of members of the matrix... 
MMP | ADAMTS | Interglobular domain | Chondroitin-sulfate-2 region | Aggrecan catabolism | RHEUMATOID-ARTHRITIS | PROTEOLYTIC CLEAVAGE | CLEAVAGE SITE | ACTIVATION MECHANISMS | LARGE AGGREGATING PROTEOGLYCAN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | HUMAN ARTICULAR-CARTILAGE | MATRIX METALLOPROTEINASES | OSTEOARTHRITIC CARTILAGE | ONCOSTATIN-M | Matrix Metalloproteinases, Secreted - metabolism | Interleukin-1beta - pharmacology | Phenylmercuric Acetate - pharmacology | Matrix Metalloproteinase 12 - genetics | Cartilage - drug effects | Oncostatin M - pharmacology | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 7 - metabolism | Cattle | Isoenzymes - metabolism | Matrix Metalloproteinase 8 - metabolism | Gelatinases - metabolism | Matrix Metalloproteinase 1 - genetics | Recombinant Proteins - metabolism | Endopeptidases - metabolism | Matrix Metalloproteinase 8 - genetics | Peptide Fragments - metabolism | Isoenzymes - genetics | Matrix Metalloproteinase 2 - metabolism | Matrix Metalloproteinase 3 - metabolism | Aggrecans - metabolism | Matrix Metalloproteinase 12 - metabolism | Cartilage - metabolism | Aggrecans - genetics | ADAM Proteins - metabolism | Enzyme Precursors - metabolism | Matrix Metalloproteinase 7 - genetics | Animals | Endopeptidases - genetics | Matrix Metalloproteinases, Secreted - genetics | Phenylmercuric Acetate - analogs & derivatives | ADAM Proteins - genetics | Cartilage - enzymology | Matrix Metalloproteinase 1 - metabolism | Matrix Metalloproteinase 3 - genetics
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 39, pp. 64244 - 64259
The process of tumor invasion requires degradation of extracellular matrix by proteolytic enzymes. Cancer cells form protrusive invadopodia, which produce and... 
Extracellular matrix | LASP1 | Matrix metalloproteinases | C-Fos | AP-1 | MIGRATION | HEPATOCELLULAR-CARCINOMA CELLS | DOMAIN PROTEIN-1 | c-Fos | ACTIN-BINDING | PROLIFERATION | CELL BIOLOGY | INVASION | EPITHELIAL-CELLS | COLORECTAL-CANCER | PROSTATE-CANCER | PODOSOMES | matrix metalloproteinases | extracellular matrix | Matrix Metalloproteinases, Secreted - metabolism | Breast Neoplasms - secretion | Oligonucleotide Array Sequence Analysis | Cytoskeletal Proteins - genetics | Transcription Factor AP-1 - genetics | Humans | Urinary Bladder Neoplasms - enzymology | Gene Expression Regulation, Neoplastic | Databases, Genetic | Podosomes - enzymology | Male | LIM Domain Proteins - metabolism | Prostatic Neoplasms - secretion | Transcription Factor AP-1 - metabolism | Breast Neoplasms - enzymology | Matrix Metalloproteinase 9 - metabolism | Prostatic Neoplasms - genetics | Transfection | Urinary Bladder Neoplasms - genetics | RNA Interference | Urinary Bladder Neoplasms - pathology | Matrix Metalloproteinases, Secreted - secretion | Cytoskeletal Proteins - metabolism | Female | Transcription, Genetic | Binding Sites | Promoter Regions, Genetic | Computational Biology | Gene Expression Profiling - methods | Matrix Metalloproteinase 3 - metabolism | Gene Expression Regulation, Enzymologic | Podosomes - pathology | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Matrix Metalloproteinases, Secreted - genetics | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Prostatic Neoplasms - enzymology | LIM Domain Proteins - genetics | Adaptor Proteins, Signal Transducing - metabolism | Matrix Metalloproteinase 1 - metabolism
Journal Article
Nature Genetics, ISSN 1061-4036, 11/2015, Volume 47, Issue 11, pp. 1363 - 1369
Journal Article
Oncogene, ISSN 0950-9232, 01/2014, Volume 33, Issue 1, pp. 97 - 107
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 01/2014, Volume 95, Issue 1, pp. 9 - 18
Journal Article
Journal Article